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1 de of 3 or greater or cystic periventricular leukomalacia.
2 ch as multiple sclerosis and periventricular leukomalacia.
3 crotizing enterocolitis, and periventricular leukomalacia.
4 brain injury in the form of periventricular leukomalacia.
5 (P=0.028) than those without periventricular leukomalacia.
6 ial hemorrhage and/or cystic periventricular leukomalacia.
7 e intracranial hemorrhage or periventricular leukomalacia.
8 traventricular hemorrhage or periventricular leukomalacia.
9 raventricular hemorrhage and periventricular leukomalacia.
10 e period of highest risk for periventricular leukomalacia.
11 lockade in a rodent model of periventricular leukomalacia.
12 e simultaneous occurrence of periventricular leukomalacia.
13 raventricular hemorrhage and periventricular leukomalacia (12.4 percent vs. 23.5 percent; relative ri
14 raventricular hemorrhage and periventricular leukomalacia (33.3 percent and 38.2 percent, respectivel
15 23.9 percent, P=0.03) and of periventricular leukomalacia alone (5.2 percent vs. 9.0 percent, P=0.048
16 injury is thought to mediate periventricular leukomalacia, an age-dependent white matter lesion seen
21 s a therapeutic strategy for periventricular leukomalacia and that the mechanism of protective effica
25 racranial hemorrhage, cystic periventricular leukomalacia, and/or severe retinopathy of prematurity.
26 raventricular hemorrhage, or periventricular leukomalacia; and death or necrotizing enterocolitis.
27 n animal models, features of periventricular leukomalacia can be induced by hypoxia and activation of
28 ay, intracranial hemorrhage, periventricular leukomalacia, chronic lung disease, patent ductus arteri
30 ural brain abnormalities and periventricular leukomalacia, fetal and postnatal cerebral blood flow is
31 ricular hemorrhage >grade 2, periventricular leukomalacia >grade 1, or necrotizing enterocolitis).
32 BAS; however, neonates with periventricular leukomalacia had lower preoperative oxygenation (P=0.026
33 respect to the mechanisms of periventricular leukomalacia in infants and of persistent demyelination
34 traventricular hemorrhage or periventricular leukomalacia in premature infants are associated with ab
36 ential for disorders such as periventricular leukomalacia in the preterm and neonatal seizures in the
40 on or remyelination, such as periventricular leukomalacia leading to cerebral palsy, stroke, and seco
41 o noted in children with the periventricular leukomalacia of cerebral palsy, another major source of
43 of intracranial hemorrhage, periventricular leukomalacia, or ventriculomegaly (17.5 percent vs. 23.9
44 traventricular hemorrhage or periventricular leukomalacia (PIVH/PVL), retinopathy of prematurity (ROP
45 al or physical impairment in periventricular leukomalacia (pre- or perinatal white matter injury lead
46 intraventricular hemorrhage, periventricular leukomalacia, proven sepsis, necrotizing enterocolitis,
49 tive MRI examinations showed periventricular leukomalacia (PVL) in 4 patients (16%) and infarct in 2
51 ite matter, which results in periventricular leukomalacia (PVL), a condition associated with abnormal
52 alsy in premature infants is periventricular leukomalacia (PVL), a disorder of the immature cerebral
53 ught to consist primarily of periventricular leukomalacia (PVL), a distinctive form of cerebral white
54 alsy in premature infants is periventricular leukomalacia (PVL), a lesion of the immature cerebral wh
55 f Huntington's disease (HD), periventricular leukomalacia (PVL), and kidney dysfunction; Fer-1 inhibi
56 hite matter injury, that is, periventricular leukomalacia (PVL), the dominant form of brain injury in
57 o white matter injury termed periventricular leukomalacia (PVL), the leading cause of subsequent neur
58 ding with the peak period of periventricular leukomalacia (PVL), the major disorder underlying cerebr
59 rtant in the pathogenesis of periventricular leukomalacia (PVL), the major pathological substrate of
60 age of immaturity, including periventricular leukomalacia (PVL), was the most common finding (42.5%),
61 e brain pathologies, notably periventricular leukomalacia (PVL), which is distinguished by bilateral
65 o the cerebral white matter [periventricular leukomalacia (PVL)] is a common and leading cause of bra
66 cular cerebral white matter [periventricular leukomalacia (PVL)] results in cerebral palsy and is the
67 ntricular hemorrhage, cystic periventricular leukomalacia, severe bronchopulmonary dysplasia, retinop
68 rade of 3 or greater, cystic periventricular leukomalacia, surgical necrotizing enterocolitis, or sta
69 In preterm infants (n = 44), periventricular leukomalacia was associated with larger vCD (1084 vs. 82
71 premature birth and typical periventricular leukomalacia, who had a dramatic improvement in motor fu
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