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1 helial EBV in the pathogenesis of oral hairy leukoplakia.
2 ufficient for the pathogenesis of oral hairy leukoplakia.
3 e tongue epithelium in lesions of oral hairy leukoplakia.
4 BV-associated diseases other than oral hairy leukoplakia.
5 skin pigmentation, nail dystrophy, and oral leukoplakia.
7 ositive subjects with and without oral hairy leukoplakia, a replicative EBV-associated epithelial dis
8 virus (EBV) replication characterizes hairy leukoplakia, an oral epithelial lesion typically occurri
10 tients with either oral candidiasis or hairy leukoplakia and a low CD4:CD8 cell ratio should be caref
15 and its synthetic derivatives, can eradicate leukoplakia and suppress the formation of squamous cell
19 C), pseudomembranous candidiasis (PC), hairy leukoplakia (HL), and warts was computed over follow-up
20 mbrane protein (LMP)-1 is expressed in hairy leukoplakia (HL), but data on LMP-1 sequence variation o
21 deficiency virus (HIV)-associated oral hairy leukoplakia (HLP) and Epstein-Barr virus (EBV) replicati
22 udy, EBV strains were identified in 25 hairy leukoplakia (HLP) biopsies and six matched peripheral bl
23 otein expression in vivo in lesions of hairy leukoplakia (HLP) in which there is abundant EBV replica
27 nails, abnormal skin pigmentation, and oral leukoplakia; Hoyeraal-Hreidarsson syndrome (HH), a clini
30 deficiency virus (HIV)-candidiasis and hairy leukoplakia-in 152 HIV-infected blood transfusion recipi
31 h as nasopharyngeal carcinoma and oral hairy leukoplakia, indicating that the virus can infect epithe
32 f HHV-8 DNA in both the EBV-associated hairy leukoplakia lesions and in the EBV-associated AIDS-relat
33 lytically infected with EBV (from oral hairy leukoplakia lesions) express much more FAS than uninfect
34 EBV was detected by Southern blot in hairy leukoplakia lesions, in a subset of AIDS-related lymphom
35 HIV-positive persons but not in pseudohairy leukoplakia lesions, oral aphthous ulcers, or oral KS le
36 multisystem disorder, characterized by oral leukoplakia, nail dystrophy, and abnormal skin pigmentat
37 sopharyngeal carcinoma (NPC), and oral hairy leukoplakia (OHL) lesions that have lytic infection, fre
39 eal involvement (OR, 2.7 [95% CI, 1.8-4.0]), leukoplakia (OR, 2.6 [95% CI, 1.7-3.9]), papilliform sur
40 e due to lytic infection (such as oral hairy leukoplakia) or latent infection (such as nasopharyngeal
41 has shown potential in the treatment of oral leukoplakia, oral lichen planus, and head and neck cance
42 al premalignant lesions, we examined 84 oral leukoplakia samples from 37 patients who had been enroll
43 diverse pathologies ranging from oral hairy leukoplakia to nasopharyngeal carcinoma, from infectious
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