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1 metrically according to clinically diagnosed level of consciousness.
2 hnic group, social class, stroke subtype and level of consciousness.
3 ncephalopathy and four of whom had depressed level of consciousness.
4 n cohort, even in one patient with depressed level of consciousness.
5 ous system, which was a reversible depressed level of consciousness.
6 sing the SAS to subjectively determine their level of consciousness.
7 Blink and tail-clamp responses also assessed level of consciousness.
8 used a perturbational approach to assess the levels of consciousness.
9 singly, this phenomenon appears to transcend levels of consciousness.
10 of emotion relate differentially to types or levels of consciousness.
11 rain functional organization that transcends levels of consciousness.
12 mus and basal forebrain underlies decreasing levels of consciousness.
13 h dissection (0.47, 0.22-0.99; p=0.047), low level of consciousness (0.45, 0.31-0.64; p<0.0001), and
16 lowing neurologic abnormalities: an abnormal level of consciousness, an inability to answer two conse
17 tive days of ICU care, against constructs of level of consciousness and delirium, and correlated with
18 diarrhea, anorexia, weight loss, and altered levels of consciousness and pathologically by the presen
19 common emergency conditions (such as altered level of consciousness) and automated paging for "panic
20 mptoms, seizures, memory deficits, decreased level of consciousness, and central hypoventilation asso
21 o 15, with lower scores indicating a reduced level of consciousness] and at least one reactive pupil)
22 exmedetomidine produced differing effects on level of consciousness as assessed by response to tail c
25 edelic phenomenology constitutes an elevated level of consciousness - as measured by neural signal di
26 tress/low SpO2 at 28% (213/748), and altered level of consciousness at 23% (170/748) compared with re
27 = no reaction to pain) to measure patients' level of consciousness at enrollment and 12 additional t
28 cerebellum significantly correlated with the level of consciousness at the time of PET (r = 0.58, P <
31 al CMRglc were plotted against posttraumatic level of consciousness, CMRglc values for the thalamus,
32 neurodeficit score (NDS) which consisted of level of consciousness, cranial nerve, motor-sensory fun
33 was compared with severity of injury and the level of consciousness evaluated using GCSini and the Gl
34 and decreased for those triggered by reduced level of consciousness (from 20% to 17%; difference [95%
35 ; 95% CI: 0.25-0.62), and those with reduced levels of consciousness, GCS <9, (HR: 0.44; CI: 0.33-0.5
37 y be the pathological correlate of depressed level of consciousness in acute disseminated encephalomy
39 ical wake-up test" is needed to evaluate the level of consciousness in patients with severe traumatic
40 ship between sleep electrophysiology and the level of consciousness in severely brain-damaged patient
41 iable nor valid for routinely monitoring the level of consciousness in the critically ill patient.
42 e of human consciousness by manipulating the level of consciousness in volunteers with anesthetic age
43 proven helpful in assessing objectively the level of consciousness irrespective of sensory processin
46 encephalopathy syndrome, seizures, depressed level of consciousness, methotrexate-related stroke-like
48 nesias, autonomic dysfunction, and decreased level of consciousness often requiring ventilatory suppo
50 1; 95% CI, 1.023-1.099; P = .001), decreased level of consciousness (OR, 5.397; 95% CI, 2.660-10.948;
51 t with encephalopathy (P < 0.001), depressed level of consciousness (P < 0.001), headache (P < 0.001)
54 RASS showed significant differences between levels of consciousness (P<.001 for all) and correctly i
56 ntinence, agitation, combativeness, a labile level of consciousness, respiratory depression, and deat
57 linical variables including use of a 6-point level of consciousness scale (1 = awake, 6 = no reaction
59 er controlling for these factors, increasing level of consciousness score at 3 hrs after enrollment a
61 olved issues include psychology's neglect of levels of consciousness that are distinct from access or
63 Patients were prospectively evaluated for level of consciousness using the RASS, SAS, and GCS ever
64 icity (seizures, motor neuropathy, depressed level of consciousness) was attributed to radiation trea
65 of the default mode network (DMN) reflects "level of consciousness," we observed functional uncoupli
66 History of seizures, age, seizure type, and level of consciousness were determined at status epilept
67 to 15, with lower scores indicating a lower level of consciousness) were randomly assigned to intrav
68 ceptors induce hallucinations and reduce the level of consciousness, while the nicotinic receptor is
69 icient hemodynamic conditions, and decreased level of consciousness, who received mild induced hypoth
71 hese findings suggest that variations in the level of consciousness within the same physiological sta
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