ライフサイエンスコーパス: lidocaine

1 FVEP was unaltered by lidocaine.

2 gent current acts as a natural antagonist of lidocaine.

3 ine release in IM-treated rats receiving RVM lidocaine.

4 se 3 but not interleukin-6 was attenuated by lidocaine.

5 osensitivity of Na(V)1.5 was approximated by lidocaine.

6 treatment with galanin, galanin antibody, or lidocaine.

7 following formalin injection with or without lidocaine.

8 l described stabilization of inactivation by lidocaine.

9 mutations to sensitize the sodium channel to lidocaine.

10 nts who received amiodarone as compared with lidocaine.

11 l blocker, but not by the Na channel blocker lidocaine.

12 rved after immediate pre-choice infusions of lidocaine.

13 from use-dependent block for bupivacaine and lidocaine.

14 the superficial layers we topically applied lidocaine.

15 0 to 6.3; P=0.16); and for amiodarone versus lidocaine, 0.7 percentage points (95% CI, -3.2 to 4.7; P

16 08F in DIII-S4) identified in a rare case of lidocaine (1 mg/kg)-induced Brugada syndrome.

17 amined the effect of sectioning or injecting lidocaine (1-2 microL of 0.5%) into the right vestibular

18 ditions, in both the presence and absence of lidocaine (10 microM).

19 0.07 microM) than ranolazine (16 microM) and lidocaine (17 microM).

20 In seven dogs, lidocaine (2%, 0.4 cc), a neuronal blocker, was injected

21 cleus by microinjecting the local anesthetic lidocaine (2%; 0.1 or 0.3 microl) bilaterally 2 min befo

22 tions of ranolazine (1 to 10 micromol/L) and lidocaine (2.1 to 21 micromol/L) were evaluated in canin

23 ollowing formalin injection, with or without lidocaine; (2) application of lidocaine in the left scia

24 d adenosine, lidocaine, and Mg/adenosine and lidocaine (5.7 mL O2/min/kg [95% CI, 4.7-6.8] to 4.9 mL

25 Addition of lidocaine 90 mumol/L depressed conduction velocity, incr

26 were randomly assigned to amiodarone (974), lidocaine (993), or placebo (1059); of those, 24.4%, 23.

27 Cysteine-labelling studies have shown that lidocaine, a local anaesthetic (LA) that elicits depolar

28 We used lidocaine, a local anesthetic and antiarrhythmic drug, t

29 This isoflurane effect was attenuated by lidocaine, a local anesthetic with anti-inflammatory pro

30 Release profiles of lidocaine, a model hydrophobic drug, were measured by UV

31 y rapid pacing in cocultures; treatment with lidocaine, a Na(+) channel blocker, significantly decrea

32 Lidocaine abbreviated both atrial and ventricular APD90.

33 We conclude that carbamazepine and lidocaine access the sodium channel in similar ways and

34 l-volume 7.5% NaCl adenocaine (adenosine and lidocaine, adenocaine) and Mg on hypotensive resuscitati

35 0.39, 95% confidence interval 0.21-0.71) and lidocaine (adjusted hazard ratio 0.72, 95% confidence in

36 Results demonstrated that lidocaine administration did not prevent the hypersensit

37 cts of neonatal injury could be prevented by lidocaine administration during the initial healing peri

38 ugs and provide a mechanism for benefit from lidocaine administration in clinical acquired long QT sy

39 or determinants of the voltage dependence of lidocaine affinity.

40 of 7.5% NaCl with adenocaine (adenosine and lidocaine, AL) and AL with Mg (ALM) on fluid requirement

41 ections of glucocorticoids plus lidocaine or lidocaine alone.

42 nefit as compared with epidural injection of lidocaine alone.

43 n the glucocorticoid-lidocaine group and the lidocaine-alone group, -1.0 points; 95% confidence inter

44 A dose of 10 micromol/L lidocaine also caused a more negative shift of steady-st

45 d adenosine, lidocaine, and Mg/adenosine and lidocaine also had significantly lower blood lactate (7.

46 We found that lidocaine also has little or no effect when applied intr

47 Lidocaine also preferentially suppressed these parameter

48 Lidocaine, an agent with rapid dissociation kinetics, ha

49 However, ionized ranolazine and charged lidocaine analog (QX-314) failed to block mechanosensiti

50 or by the internal dialysis of low doses of lidocaine analog QX-314.

51 ality of anesthesia than does combination 2% lidocaine and 0.5% bupivacaine in patients undergoing vi

52 diac arrest may have different benefits from lidocaine and amiodarone than previously demonstrated.

53 d potential (FVEP) also were recorded before lidocaine and at relevant times afterwards.

54 A homology model for how lidocaine and benzocaine bind in the closed and open/ina

55 esidue permits clear separation of action of lidocaine and benzocaine into 2 components that can be r

56 properties of diverse LA compounds, such as lidocaine and bupivacaine.

57 lidocaine, bupivacaine, or a combination of lidocaine and bupivacaine.

58 -1 and TASK-2 channels that are inhibited by lidocaine and external acidification.

59 By applying lidocaine and measuring the gating currents, we demonstr

60 e IV bolus of 4 mL/kg 7.5% NaCl + adenosine, lidocaine and Mg (n = 8) or 4 mL/kg 7.5% NaCl (n = 8) at

61 tated by an electrostatic attraction between lidocaine and pi electrons of the aromatic rings of thes

62 d forearm by an anesthetic cream (containing lidocaine and prilocaine) on one of the 2 d, and a place

63 (iii) the analysis of the local anesthetics, lidocaine and prilocaine, in a gel and a cream.

64 The pure Na(+) channel blocker lidocaine and the antianginal ranolazine were additional

65 l as to other pharmacological agents such as lidocaine and ZD7288.

66 for those who received radiocolloid plus 1% lidocaine, and 1.6 (1.3) for those who received radiocol

67 jection (N = 6), 0.70 degrees C (N = 6) with lidocaine, and 1.77 degrees C (N = 6) after sham microin

68 In all, 16 (4.1%) amiodarone, 11 (3.1%) lidocaine, and 6 (1.9%) placebo-treated patients survive

69 care, 97% for sodium bicarbonate, 90% for 1% lidocaine, and 90% for sodium bicarbonate plus 1% lidoca

70 tetrakis(4-chlorophenyl)borate, metergoline, lidocaine, and bromhexine.

71 njections of the cationic anesthetic QX-314, lidocaine, and capsaicin, delays the maturation of both

72 M carbamazepine, suggesting that lacosamide, lidocaine, and carbamazepine all bind to the same site.

73 Low-dose vaginal estrogen, lidocaine, and dehydroepiandrosterone may also be consid

74 ted by membrane repolarization, inhibited by lidocaine, and enhanced by a peptide-mimetic of the beta

75 ation, treatment with 7.5% NaCl + adenosine, lidocaine, and Mg generated significantly higher mean ar

76 translate small-volume 7.5% NaCl adenosine, lidocaine, and Mg hypotensive fluid resuscitation from t

77 n decreased in pigs that received adenosine, lidocaine, and Mg/adenosine and lidocaine (5.7 mL O2/min

78 Pigs that received adenosine, lidocaine, and Mg/adenosine and lidocaine also had signi

79 After 180 minutes, pigs in the adenosine, lidocaine, and Mg/adenosine and lidocaine group had thre

80 Higher cardiac index in the adenosine, lidocaine, and Mg/adenosine and lidocaine group was due

81 sely related to heart rate in the adenosine, lidocaine, and Mg/adenosine and lidocaine group.

82 me resuscitation with 7.5% NaCl + adenosine, lidocaine, and Mg/adenosine and lidocaine provided super

83 nd trial, we compared parenteral amiodarone, lidocaine, and saline placebo, along with standard care,

84 sis of verapamil, citalopram, amitriptyline, lidocaine, and sunitinib in dried blood spots.

85 uroanatomical tracing, localized infusion of lidocaine, and surgical transections.

86 flammatory agents, gabapentinoids, tramadol, lidocaine, and/or the N-methyl-d-aspartate class of glut

87 tly increased BP more than the group without lidocaine; and (4) there was delayed significant BP incr

88 the PVN were inhibited with intraparenchymal lidocaine, AngII-induced sodium ingestion was exaggerate

89 The local anesthetic lidocaine appears to be able to access its binding site

90 ilaterally; (3) formalin injection following lidocaine application significantly increased BP more th

91 and rapid recovery, whereas mepivacaine and lidocaine are suitable for longer procedures.

92 partment intubation, the use of atropine and lidocaine as premedications, the choice of sedative agen

93 times after blockade of axon transport with lidocaine at either the retrobulbar level (2 muL, 40 mug

94 esuscitation and 0.9% NaCl +/- adenosine and lidocaine at infusion of shed blood.

95 materials formed via a chemical reaction of lidocaine base with a series of acids characterized by a

96 ed by open-channel blockers, suggesting that lidocaine binding is favored by inactivation but prevent

97 shows that the residue N395 is critical for lidocaine binding to Nav1.7 and suggests that the respon

98 Previous studies have shown that lidocaine binding to the pore domain causes a decrease i

99 3C-DIII + R2C-DIV) the IC50 for rested-state lidocaine block decreased from 194 +/- 15 microM in cont

100 extensive (due to ultra-slow recovery), but lidocaine block of Na+ channels was not, P-segment cyste

101 he absence of an intact inactivation gate on lidocaine block was determined for R3C-DIII + ICM, R2C-D

102 These mutations eliminated use-dependent lidocaine block with no effect on tonic/rested state blo

103 olarized conformation increases affinity for lidocaine block, a cysteine residue was substituted for

104 We studied lidocaine blockade of I(Na) generated by wild-type and V

105 ationic lidocaine derivative QX-314 (N-ethyl-lidocaine) blocks effectively when applied internally wi

106 Following CSAR inhibition by epicardial lidocaine, blood pressure, HR, LVSP, dp/dt, LVEDP and ES

107 Lidocaine, bupivacaine and acidic pH depolarized circula

108 ndard deviation) of sensory blockade for the lidocaine, bupivacaine, and combination groups were 2.14

109 tandard deviation) of motor blockade for the lidocaine, bupivacaine, and combination groups were 3.04

110 d on the peribulbar injection they received: lidocaine, bupivacaine, or a combination of lidocaine an

111 Lidocaine but not methylphenidate also induced significa

112 als, comparable in magnitude to infusions of lidocaine, but with a slower onset and longer duration.

113 ous clinically used drugs including Demerol, lidocaine, capecitabine, and CPT-11 are hydrolyzed by th

114 , with potencies 10- to 100-fold higher than lidocaine (compound 1).

115 omfortable intercourse after applying liquid lidocaine compresses to the vulvar vestibule before pene

116 1 fashion to receive the standard topical 4% lidocaine cream and injection of [(99m)Tc]Tc-sulfur coll

117 In constant 100 mum lidocaine, current-clamped Purkinje cells continued to f

118 CSAR inhibition by epicardial lidocaine decreased cardiac contractility to a greater e

119 ocol was further applied to oxidize the drug Lidocaine, demonstrating the potential utility of the de

120 Similarly, the beta4 peptide reduced lidocaine-dependent suppression of spiking in CA3 neuron

121 In contrast, the cationic lidocaine derivative QX-314 (N-ethyl-lidocaine) blocks e

122 introducing the charged, membrane-impermeant lidocaine derivative QX-314 through the pore of the noxi

123 n neuronal entry of the membrane-impermeable lidocaine derivative QX-314, leading to TLR5-dependent b

124 ux across skin-in combination with QX-314, a lidocaine derivative that has relative difficulty penetr

125 l interaction between the surfactant and the lidocaine derivative, not a systemic effect.

126 factants, and also was achieved with another lidocaine derivative, QX-222.

127 The interaction of symmetrical lidocaine dimers with voltage-gated Na+ channels (VGSCs)

128 was accomplished with bilateral infusions of lidocaine directly into PRh immediately before the sampl

129 pplication of the Na-super++ channel blocker lidocaine disrupted learning when it was applied over a

130 mutagenesis data, that the local anaesthetic lidocaine docks eccentrically below the selectivity filt

131 versible inactivation of the IP nucleus with lidocaine during conditioning evoked a complete disappea

132 to ensure the dissipation of the actions of lidocaine during the choice phase.

133 ons of volatile anesthetics and subcutaneous lidocaine efficacy have been attributed in part to melan

134 erior dorsal hippocampus by microinfusion of lidocaine eliminated the avoidance of the familiar winne

135 ion potential clamp experiments suggest that lidocaine eliminates virtually all inward current elicit

136 -dependent inhibition and speeds recovery of lidocaine-engaged channels.

137 in a 1:2 ratio to receive (1) an intra-oral lidocaine-epinephrine injection with buffered saline nas

138 cyclic aromatic hydrocarbons, angiotensin I, lidocaine, ferrocene, diesel, and rosemary oils were use

139 ertion or intercourse, all patients received lidocaine for 2 months in an open-label trial.

140 etermine the appropriate use of atropine and lidocaine for rapid sequence intubation.

141 PSI cartridge was applied to the analysis of lidocaine from a paper tip using different solvent mixtu

142 ent solvent mixtures, and to the analysis of lidocaine from a serum sample.

143 Discomfort was significantly lower in the lidocaine gel group (P = .01).

144 Premedication with 4% lidocaine gel significantly reduced discomfort during sc

145 ion with acetaminophen, ibuprofen, and/or 4% lidocaine gel.

146 3.4 (5.1) for the sodium bicarbonate plus 1% lidocaine group (p<0.0001).

147 aine, and 90% for sodium bicarbonate plus 1% lidocaine group (p=0.56).

148 bupivacaine group compared with 23.3% in the lidocaine group and 30% in the combination group (P = 0.

149 treatment effect between the glucocorticoid-lidocaine group and the lidocaine-alone group, -1.0 poin

150 e adenosine, lidocaine, and Mg/adenosine and lidocaine group had three-fold higher urinary output (2.

151 e adenosine, lidocaine, and Mg/adenosine and lidocaine group was due to a two-fold increase in stroke

152 oup, 115.83+/-34.49 minutes, and that of the lidocaine group, 94.17+/-49.86 minutes (P < 0.001).

153 dium bicarbonate group, 4.6 (4.5) for the 1% lidocaine group, and 3.4 (5.1) for the sodium bicarbonat

154 e adenosine, lidocaine, and Mg/adenosine and lidocaine group.

155 VEDP and ESPVR decreased in CHF rats whereas lidocaine had little effect in sham rats, indicating tha

156 The anti-arrhythmic drug lidocaine has been shown to have a lower affinity for bl

157 A lidocaine HCl (Xylocaine) injection on the midline at th

158 quid doses of diphenhydramine hydrochloride, lidocaine hydrochloride, diazepam, and fluoxetine hydroc

159 The decreased preload by lidocaine in CHF rats may be due to a reduction in perip

160 ay implicate a possible therapeutic role for lidocaine in decreasing the inappropriate activation, po

161 lates inflammatory processes, the effects of lidocaine in sepsis are still unknown.

162 Ranolazine was more effective than lidocaine in terminating persistent AF and in preventing

163 ith or without lidocaine; (2) application of lidocaine in the left sciatic nerve alone significantly

164 Intrathecal lidocaine in the lithotomy position carries a significan

165 ancreatitis, rats received microinjection of lidocaine in the NG or in the RVM to determine the impor

166 were infused with the sodium channel blocker lidocaine in vehicle or vehicle only during sleep.

167 ork activity in the chick embryo by infusing lidocaine in vivo triggers synaptic scaling in spinal mo

168 Furthermore, lidocaine inactivation (0.4 mul, 4%) studies demonstrate

169 We report the first case of lidocaine-induced Brugada electrocardiogram phenotype.

170 sumatriptan or alphaCGRP(8-37) abolished RVM lidocaine-induced CPP in IM-treated rats.

171 In rats having learned the water-maze task, lidocaine-induced inactivation of the ReRh right before

172 ions were not correlated with the respective lidocaine-induced slow recovery after short depolarizati

173 ignificantly correlated with the kinetics of lidocaine-induced slow recovery.

174 oscillations were reduced after intraseptal lidocaine infusions in rats.

175 depolarization and antagonized use-dependent lidocaine inhibition of fast-inactivated sodium channels

176 Acute pelvic neurectomy or lidocaine inhibition of lumbosacral dorsal roots facilit

177 l anaesthetic binding site, had no effect on lidocaine inhibition of Nav1.7 current.

178 This study also investigated lidocaine inhibition of the Nav1.7-N395K channel.

179 We therefore recorded lidocaine inhibition of voltage-clamped, tetrodotoxin-se

180 Our observation of reduced lidocaine inhibition on Nav1.7-N395K shows that the resi

181 th a site-3 toxin, anemone toxin II, reduced lidocaine inhibition; this effect was largely occluded b

182 Here, we show that lidocaine inhibits TRPV1 channels expressed in Xenopus l

183 Standard anesthesia consisted of 5 mL of 1% lidocaine injected locally, and conscious sedation consi

184 local anaesthesia (0.5% L-bupivacaine and 1% lidocaine injected subcutaneously) or regional (brachial

185 Lidocaine injection caused rapid (retrobulbar ~10 minute

186 Lidocaine injection inhibited hemorrhagic hypotension an

187 tive responses, mediated in part by TTX- and lidocaine-insensitive channels, were observed in most ce

188 Microinjection of lidocaine into either the NG or the RVM produced a time-

189 These effects are blocked by infusions of lidocaine into the nucleus tractus solitarii (NTS) and b

190 Microinjection of lidocaine into the rostral ventromedial medulla (RVM), o

191 P = 0.017] and the absence of intraoperative lidocaine intravenous perfusion (odds ratio: 0.182, 95%

192 Lidocaine is an antiarrhythmic agent commonly used to tr

193 Systemic lidocaine is anti-inflammatory but is effective only in

194 Lidocaine is generally considered much less cardiotoxic

195 input in the initial healing period and (b) lidocaine is ineffective at preventing long-term spinal

196 ation who survive 3 hrs, amiodarone, but not lidocaine, is associated with an increased risk of death

197 re disrupted by local anesthetic blocks with lidocaine (LID).

198 was produced by cutaneous microinjection of lidocaine (LID).

199 Infusion of lidocaine (LIDO) or vehicle had no effect on the neuroen

200 s are targeted by local anaesthetics such as lidocaine (lignocaine), some patients do not respond to

201 entricular depolarizations and suppressed by lidocaine (lignocaine).

202 ized potentials, raising the question of how lidocaine-like compounds affect neurons with resurgent N

203 Lidocaine microinjection attenuated the hemorrhagic hypo

204 Although the local anesthetic lidocaine modulates inflammatory processes, the effects

205 dimers, in which the tertiary amines of the lidocaine moieties are linked by an alkylene chain (two

206 Unilateral microinjections of lidocaine, muscimol, or glutamate antagonists into the p

207 anglion cell were blocked with intracellular lidocaine N-ethyl bromide (QX-314), the signal-to-noise

208 Patients received lidocaine (n = 664, 59.0%), amiodarone (n = 50, 4.4%), b

209 zed to receive either a placebo (n = 7) or a lidocaine (n = 7) bolus (1.5 mg/kg), followed by continu

210 taining either sodium bicarbonate (n=35), 1% lidocaine (n=35), or sodium bicarbonate and 1% lidocaine

211 docaine (n=35), or sodium bicarbonate and 1% lidocaine (n=35).

212 ed, 194 met criteria for inclusion (n=79 for lidocaine, n=74 for amiodarone, n=41 for combination).

213 , epidural injection of glucocorticoids plus lidocaine offered minimal or no short-term benefit as co

214 utation attenuates the inhibitory effects of lidocaine on both resting and inactivated Nav1.7.

215 l was conducted to investigate the effect of lidocaine on leukocyte recruitment in septic patients.

216 ficantly reduced use-dependent inhibition of lidocaine on Nav1.7 current.

217 ring capacity, indicating a direct effect of lidocaine on signal transduction in neutrophils.

218 We tested the effect of the LA lidocaine on the time course of recovery from short and

219 les to anchor spiral waves and the effect of lidocaine on their attachment.

220 duced by a local VTA microinfusion of either lidocaine or (+/-)2-amino,5-phosphopentanoic acid (AP-5)

221 atly reduced by pretreatment with 300 microM lidocaine or 300 microM carbamazepine, suggesting that l

222 nistration of cocaine, intra-VTA infusion of lidocaine or AP-5 significantly diminished this effect.

223 In the first set, lidocaine or artificial cerebrospinal fluid (ACSF) was m

224 o effect on low-affinity tonic inhibition by lidocaine or its permanently charged derivative QX-314 a

225 epidural injections of glucocorticoids plus lidocaine or lidocaine alone.

226 porary cardiac pacing than did recipients of lidocaine or placebo.

227 Subjects (injured or uninjured) received lidocaine or saline on 1 of 4 administration schedules (

228 inactivation using reagents that influenced (lidocaine) or spared (GABA receptor agonists) fibers of

229 omized, double-blind, to receive amiodarone, lidocaine, or placebo by paramedics.

230 erved as sham controls and were infused with lidocaine outside of V1 (into the CSF).

231 ildren who received amiodarone also received lidocaine (p < .001).

232 ge of 8 mins later in the code compared with lidocaine (p<.001).

233 eceived radiocolloid plus bicarbonate and 1% lidocaine (p<0.0001).

234 Furthermore, we also found that epicardial lidocaine paradoxically decreased left ventricular end-d

235 Furthermore, we found that epicardial lidocaine paradoxically decreased LV end-diastolic volum

236 tion for use and proposal as second line for lidocaine patches, capsaicin high-concentration patches,

237 and botulinum toxin A, and undetermined for lidocaine patches.

238 derate or high for all treatments apart from lidocaine patches; tolerability and safety, and values a

239 Inactivation of the vHipp with lidocaine prevented the sustained, but not acute, antide

240 ition of the rmPFC by the local injection of lidocaine produced an increase in the amplitude of evoke

241 RVM lidocaine produced CPP, increased NAc c-Fos, and dopamin

242 atic fields in the pore model predicted that lidocaine produced the largest positive electrostatic ba

243 As expected, we found that bupivacaine, like lidocaine, protected cysteine residues from MTSEA modifi

244 + adenosine, lidocaine, and Mg/adenosine and lidocaine provided superior cardiovascular, acid-base, m

245 Treatment with lidocaine reduced net fluid secretion in the small intes

246 Concomitantly, lidocaine reduces maximum gating charge (Qmax) by 40% re

247 at concentrations (40-60 mumol/L) for which lidocaine remains specific for inactivated Na(+) channel

248 Users of lidocaine reported less pain during intercourse in the b

249 Overall, neither amiodarone nor lidocaine resulted in a significantly higher rate of sur

250 ylprednisolone) and three local anesthetics (lidocaine, ropivacaine, and bupivacaine) were added to s

251 f domain IVS6, the putative binding site for lidocaine's alkylamino end, with positively and negative

252 n electrostatic barrier to ion permeation by lidocaine's charge.

253 on of the CA3 region of the hippocampus with lidocaine selectively eliminated source memory, suggesti

254 the hippocampus with bilateral infusions of lidocaine selectively eliminated the ability of rats to

255 These data suggest that lidocaine selectively inhibits chemokine-induced arrest

256 re-treatment with lidocaine, suggesting that lidocaine-sensitive K+ channels were responsible for pH-

257 In addition, lidocaine sensitivity was dramatically reduced, which pr

258 Lidocaine shortened APD equally during acute and chronic

259 INa-L block with lidocaine shortened QTc and APD more at 6.5 hours than a

260 ipheral vascular resistance since epicardial lidocaine significantly lowered peripheral (renal) sympa

261 -BLA infusions of a memory-impairing dose of lidocaine significantly reduced Arc protein levels in th

262 ateral injections of buffered and unbuffered lidocaine solutions were given before surgery to patient

263 r a bupivacaine lozenge (L-group, n = 51) or lidocaine spray (S-group, n = 42).

264 etic compared with standard treatment with a lidocaine spray before UGE.

265 A bupivacaine lozenge compared with a lidocaine spray proved to be a superior option as topica

266 e affinity of the cardiac sodium channel for lidocaine such that the drug assumes Class IC characteri

267 cidic pH was abolished by pre-treatment with lidocaine, suggesting that lidocaine-sensitive K+ channe

268 d radiocolloid injection and pH-adjusted and lidocaine-supplemented formulations for patients undergo

269 or survival were greater after amiodarone or lidocaine than placebo, without increased risk of advers

270 nerve; however, in contrast to the effect of lidocaine, the application of QX-314 and capsaicin toget

271 etting, particularly by using lower doses of lidocaine to reduce the problem of transient neurologic

272 with early breast cancer, the addition of 1% lidocaine to the radioisotope solution can improve patie

273 advanced cardiac life support algorithm for lidocaine toxicity as well as other lipid soluble drug i

274 sion therapy after prolonged and intractable lidocaine toxicity.

275 herapy for intractable cardiac arrest due to lidocaine toxicity.

276 sed to normokalaemic (n = 25, 14 hearts), or lidocaine-treated hypokalaemic (n = 8, five hearts) or n

277 arts), hypokalaemic (n = 8, five hearts), or lidocaine-treated normokalaemic (n = 8, five hearts) or

278 Lidocaine treatment abrogated chemokine-induced neutroph

279 dividuals with hereditary erythromelalgia to lidocaine treatment may be determined, at least in part,

280 After open-label lidocaine use, 37 (90%) of 41 reported comfortable penet

281 acebo were 2.3% (-0.3, 4.8), P=0.08, and for lidocaine versus placebo 1.2% (-1.1, 3.6), P=0.30.

282 nce interval [CI], -0.4 to 7.0; P=0.08); for lidocaine versus placebo, 2.6 percentage points (95% CI,

283 Local lidocaine was applied to the left sciatic nerve to block

284 control channel states during drug exposure, lidocaine was applied with rapid-solution exchange techn

285 Inhibition of Na currents by lidocaine was diminished by either the beta4 peptide or

286 The IC50 for lidocaine was estimated at 500 microM for inactivated wi

287 sions, rats were significantly impaired when lidocaine was infused before the sample phase, regardles

288 seline, use-dependent inhibition of I(Na) by lidocaine was more pronounced in V232I+L1308F versus wil

289 .96, 95% confidence interval 1.21-3.16), but lidocaine was not at 30 days (adjusted hazard ratio 1.19

290 of NsVBa, whereas the local anesthetic drug lidocaine was shown to antagonize NsVBa without affectin

291 The effects of lidocaine were not blocked by tetraethylammonium chlorid

292 nnels also bind exogenous compounds, such as lidocaine, which functionally stabilize inactivation.

293 ) but no local anesthetic properties, and of lidocaine, which has similar local anesthetic effects as

294 VF/pVT who received amiodarone also received lidocaine, while 67% of children who received amiodarone

295 e percent of patients preferred the buffered lidocaine with a scaled pain reduction of 0.9 (P = 0.000

296 Determine if raising the pH of 2% lidocaine with epinephrine 1:100 000 to a physiologic le

297 Buffering 2% lidocaine with epinephrine 1:100 000 with sodium bicarbo

298 ous injections of buffered and unbuffered 2% lidocaine with epinephrine 1:100 000.

299 B]) anaesthesia (0.5% L-bupivacaine and 1.5% lidocaine with epinephrine).

300 nesthesia, and defining the 'safest' dose of lidocaine with epinephrine.