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1 mation of its proliferative units (crypts of Lieberkuhn).
2 liferative units of the intestine (crypts of Lieberkuhn).
3 tive compartment of the intestine (crypts of Lieberkuhn).
4 s that reside near the base of the crypts of Lieberkuhn.
5 ells positioned at the base of the crypts of Lieberkuhn.
6 l daughters located at the base of crypts of Lieberkuhn.
7 tine is highest at the base of the crypts of Lieberkuhn.
8 nd closer to the stem cell zone in crypts of Lieberkuhn.
9 ed of well-delineated units called crypts of Lieberkuhn.
10 on events that is initiated in the crypts of Lieberkuhn.
11 l epithelial cell proliferation in crypts of Lieberkuhn.
12 driven by epithelial stem cells in crypts of Lieberkuhn.
13 only in the actively proliferating crypts of Lieberkuhn but not in villi.
14 al granulocytes at the base of the crypts of Lieberkuhn in the small intestine of many mammalian spec
15 ase granules into the lumen of the crypts of Lieberkuhn in the small intestine where their component
16 y cells located at the base of the crypts of Lieberkuhn in the small intestine.
17 pattern is restricted to epithelial cells of Lieberkuhn's crypts of the intestine, the spermatocytes
18 ls at the base of small intestinal crypts of Lieberkuhn secrete host defense peptides and proteins, i
19 lls are located at the base of the crypts of Lieberkuhn, tiny invaginations that line the mucosal sur
20 t epithelial stem cells within the crypts of Lieberkuhn to initiate the lethal GI syndrome.
21 tory epithelium and the intestinal crypts of Lieberkuhn, to inform response and recovery of such tiss
22 m cell clonogens (SCCs) within the crypts of Lieberkuhn, which are a subset of cells necessary for po
23 ion augments cell proliferation in crypts of Lieberkuhn, without a compensatory change in basal apopt

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