ライフサイエンスコーパス: light adaptation

1 easurements of rhodopsins to investigate dim-light adaptation.

2 e speeding of incremental dim flashes during light adaptation.

3 ect interaction with PIs and is required for light adaptation.

4 synaptic inputs, and are critical to retinal light adaptation.

5 s in photoreceptor synaptic transmission and light adaptation.

6 pted retina were consistent with its role in light adaptation.

7 on constant of transduction was unaltered by light adaptation.

8 )-mediated modulation of transduction during light adaptation.

9 inergic system and in turn influence retinal light adaptation.

10 esting the importance of the modification in light adaptation.

11 desensitizes rods by a process equivalent to light adaptation.

12 round organization of the visual system with light adaptation.

13 y connecting output and input, and regulates light adaptation.

14 g windmill pattern, similar to the effect of light adaptation.

15 is involved in numerous functions related to light adaptation.

16 ular correlate of classical conditioning and light adaptation.

17 the principal extruder of Ca(2+) ions during light adaptation.

18 nsights into the nature of rod photoreceptor light adaptation.

19 ion of photoreceptor guanylyl cyclase during light adaptation.

20 activation does not appear to play a role in light adaptation.

21 e of a newly described form of photoreceptor light adaptation.

22 ination, it does not participate directly in light adaptation.

23 otoreceptors to tune gain, inactivation, and light adaptation.

24 becomes significantly more protonated during light adaptation.

25 n horizontal cell electrical synapses during light adaptation.

26 he changed titration behavior of Y185F after light adaptation.

27 ng image contrast, color discrimination, and light adaptation.

28 bed cp26 gene that we found had no effect on light adaptation.

29 tion in the photoreceptors in either dark or light adaptation.

30 n's first and most fundamental mechanism for light adaptation.

31 so used by the visual system as a signal for light adaptation.

32 annot be regarded as a general mechanism for light adaptation.

33 storage compartment, resulting in long term light adaptation.

34 s, respectively) that depended on background light adaptation.

35 und illumination and severely impaired their light adaptation.

36 l properties of cones in darkness and during light adaptation.

37 ation of saturated responses shut off during light adaptation.

38 s when arrestin1 translocated in response to light adaptation.

39 rs, an increase of cytoplasmic Ca(2+) mimics light adaptation.

40 insights into the classic problem of retinal light adaptation.

41 e detection limit to higher frequencies upon light adaptation.

42 endent protein kinase II (CaMKII) to mediate light adaptation.

43 amically regulated by a circadian rhythm and light adaptation.

44 ols photoresponse sensitivity, kinetics, and light adaptation.

45 cone bipolar cells was unaltered by dark or light adaptation.

46 ds, thus making an important contribution to light adaptation.

47 sed to monitor cone adaptation after intense light adaptation.

48 RetGC activation in the conditions mimicking light adaptation.

49 thereby may serve as a powerful mechanism of light adaptation.

50 which was shifted to higher frequencies upon light adaptation.

51 cells were indistinguishable after dark and light adaptation.

52 cycle accumulates 11-cis-retinyl esters upon light adaptation.

53 naptic terminals, regardless of the state of light adaptation.

54 mposed flashes grew in amplitude, indicating light adaptation.

55 .1-10 microM Ca(2+)) and accurately mimicked light adaptation.

56 d changes in photoresponse properties during light adaptation.

57 ly through dopamine receptor pathways during light adaptation.

58 , coupling is regulated during the course of light adaptation.

59 t response and modulating sensitivity during light-adaptation.

60 0.97-2.72 log cd-s/m(2)) after 15 minutes of light adaptation (150 cd/m(2)).

61 ed (1.2-2.7 log cd-s/m2) after 15 minutes of light adaptation (150 cd/m2).

62 Different conditions of light adaptation allow for preparation of reaction cente

63 hought to be a major locus for mechanisms of light adaptation and contrast enhancement in the retina.

64 ic protein kinase C (eye-PKC) is involved in light adaptation and deactivation.

65 esponses were greatly suppressed during both light adaptation and early stages of dark adaptation.

66 by eye-protein kinase C (PKC) that promotes light adaptation and fast deactivation, most likely via

67 ession of CaMKII in transgenic flies affects light adaptation and increases prolonged depolarizing af

68 de novo, may play an essential role in rapid light adaptation and photoprotection.

69 kinase (RK) is a conserved component of the light adaptation and recovery pathways shared among rod

70 essential in photoreceptor cells for normal light adaptation and recovery to the dark state.

71 +)]i following light exposure controls their light adaptation and response termination.

72 inaC(P209) heterozygotes displayed abnormal light adaptation and slow deactivation.

73 channel phosphorylation may be important for light adaptation and the regulation of phototransduction

74 hototransduction, reduces sensitivity during light adaptation, and suppresses bleached rhodopsin acti

75 of its population dynamics, growth rate, and light adaptation, and the size and macromolecular and el

76 behavioral and physiological descriptions of light adaptation are reviewed, and recent attempts to mo

77 ceptor outer segments were shortened by 36%, light adaptation as measured by transducin translocation

78 ate that elimination of M1 ipRGCs attenuates light adaptation, as evidenced by an impaired electroret

79 This discrepancy is alleviated by light adaptation: as the mean intensity increases, the r

80 receptive field structure changes less with light adaptation at higher input-to-output cell ratios,

81 ately 1 s or less for the near-completion of light adaptation at this background strength.

82 ion, implicating a nonlinear process--namely light adaptation--at the level of single cone photorecep

83 d by 1-1.5 log units, after which additional light adaptation brought about an uncoupling of cells.

84 +) influx via these channels is required for light adaptation, but although several molecular targets

85 nce image-forming visual function, including light adaptation, but the mechanisms involved are unclea

86 suction electrodes, in the dark, and during light adaptation by backgrounds or by bleaching visual p

87 te that bipolar cell sodium channels mediate light adaptation by controlling retinal signaling gain.

88 pport a dopaminergic role in post-receptoral light adaptation by decreasing HC receptive field diamet

89 Part of this light adaptation by ipRGCs appeared to be triggered by a

90 on of axon collateral-bearing ipRGCs impairs light adaptation by limiting dopamine-dependent facilita

91 ract decreases in retinal sensitivity during light adaptation by preventing the loss of visual inform

92 During light adaptation by steady backgrounds the maximal respo

93 that both DA and NO could be involved in the light-adaptation changes induced by either pattern of in

94 and intensity of light exposures, mediating light adaptation, circadian entrainment, pupillary refle

95 ed in two male subjects under four different light-adaptation conditions for 20 days.

96 nent of the electroretinogram and revealed a light adaptation defect in nrc photoreceptors.

97 Analysis of the b-wave demonstrated a light adaptation defect in nrc that causes saturation at

98 The four conditions of light adaptation did not significantly affect MPOD.

99 tween left and right eyes is attributable to light-adaptation differences between the eyes.

100 ptor cell deactivation, desensitization, and light adaptation, failed to suppress rdgB degeneration u

101 e but, in addition, have now quantified this light adaptation for the M1 ipRGC subtype.

102 totransduction processes, has elucidated how light adaptation happens dynamically through stochastic

103 he understanding of vertebrate photoreceptor light adaptation has come from the discovery that as man

104 However, whether NCKX1 contributes to light adaptation has not been directly tested and the me

105 s a crucial role in our understanding of low-light adaptation in A. vinosum.

106 Here, we report a mechanism of light adaptation in Drosophila, which is regulated by ph

107 including clathrin-mediated endocytosis and light adaptation in Drosophila.

108 s modulate dark-adapted responses as well as light adaptation in mammalian cones.

109 The mechanisms by which Ca2+ regulates light adaptation in microvillar photoreceptors remain po

110 from the holoenzyme, and may be relevant to light adaptation in photoreceptor cells.

111 We find that the low-light adaptation in R. palustris leads to a reduced elem

112 been recognized as one of the mechanisms for light adaptation in rods.

113 pinules in relation to steady and flickering light adaptation in the carp retina.

114 ascade, which leads to a state equivalent to light adaptation in the dark-adapted rod.

115 ions, a feature which might be important for light adaptation in the retina.

116 ory factor, possibly L-DOPA, which regulates light adaptation in the retinal circuitry.

117 changes in the outer retina during dark and light adaptation in unaffected and Best disease subjects

118 Light adaptation in vertebrate photoreceptors is commonl

119 location, for their ability to contribute to light adaptation in zebrafish cones.

120 these cells exhibit all of the hallmarks of light adaptation, including response range compression,

121 A key component in dark/light adaptation is phosducin, a phosphorylatable protei

122 The results indicate that light adaptation is primarily mediated downstream of PLC

123 Light adaptation is thought to be orchestrated by a Ca2+

124 ove rod threshold, after which, with further light adaptation, it stabilized at levels close to those

125 When also studied under light adaptation, most visually responsive SCN neurones

126 a prominent Ca(2+)-independent component of light adaptation not typically seen in rods and cones or

127 First, light adaptation of bacteriorhodopsin (bR) at pHs near t

128 For cells desensitized by bleaching, light adaptation of both components of the dark-adapted

129 Recent studies have revealed that light adaptation of both vertebrate and invertebrate pho

130 g imaging of RGC responses without excessive light adaptation of cones.

131 proposed that phosducin plays a role in the light adaptation of G protein-mediated visual signaling.

132 on are believed to play an important role in light adaptation of photoreceptor cells.

133 Dark and light adaptation of retinal neurons allow our vision to

134 The effect of dantrolene on light adaptation of the photoreceptor was assessed by me

135 Light adaptation of the pigment leads to a phototransfor

136 order to study the relative contribution to light adaptation of the various actions of Ca2+ in rod p

137 We examined the effect of light adaptation on the gap junctional coupling of alpha

138 The effect of light adaptation on these stimulus classes was also asse

139 review how these factors jointly orchestrate light adaptation over a large dynamic range.

140 eptor kinase 1 (GRK1), is a component of the light adaptation pathway expressed in rods and cones.

141 st, ERG flicker analysis after the 15-minute light adaptation period demonstrated no loss in amplitud

142 regulation of the visual response affecting light adaptation, possibly by catalyzing phosphorylation

143 stent with a role for Pd in Ca(2+)-dependent light adaptation processes in photoreceptor cells and al

144 argue for a novel mechanism of photoreceptor light adaptation produced by modulation of GAP-dependent

145 In WT mice, light adaptation reduced outer retinal manganese uptake

146 Light-adaptation reduced glycolysis by 20%.

147 retinal ganglion cells in mouse retina under light adaptation remains unknown.

148 a 'Crawford transformation' derived from the light adaptation results.

149 Light adaptation serves to stabilize AMPARs in a noncycl

150 In addition, our light adaptation studies support the notion than an addi

151 k of NCKX1 did not compromise rod background light adaptation, suggesting additional Ca(2+)-extruding

152 sphorylated and decreased more slowly during light adaptation (t((1/2)) approximately 9 min) to less

153 These data appear to exclude models for light adaptation that postulate high levels of phosphory

154 minutes and continuing through 15 minutes of light adaptation, the cone b-wave amplitudes of WT and A

155 ritical for amplification, inactivation, and light adaptation, the fractional contribution of Ca(2+)

156 nce that they are dynamically gated for dark/light adaptation, the full impact that rod-cone GJs can

157 This performance requires powerful light adaptation, the neural implementation of which has

158 Since cytoplasmic Ca2+ regulates light adaptation, the sensitivities of these processes t

159 During light adaptation, the sensitivity of CNG channels to cGM

160 apidly (t((1/2)) approximately 2 min) during light adaptation to less than 20% phosphorylated.

161 to irradiance responses dissipates following light adaptation to the extent that these receptors make

162 We found that light adaptation using mesopic or photopic background li

163 Light adaptation was normal at low backgrounds but becam

164 83/96/182 cluster on retinal maintenance and light adaptation, we generated a sponge transgenic mouse

165 ssential for cone photoreceptor survival and light adaptation, whereas either Arr1 or Arr4 is necessa

166 sponses were separated from rod responses by light adaptation, whereas rod sensitivity was assessed b

167 .51 mum) 10 to 20 minutes after the start of light adaptation, while Best disease subjects exhibited

168 s in the tiger salamander were used to study light adaptation with positive and negative contrast sti