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1 tor, is disrupted specifically in the bovine limb bud mesenchyme.
2 st cells in the first branchial arch and the limb bud mesenchyme.
3 hat restricts ZPA formation to the posterior limb bud mesenchyme.
4 ed to inhibit Shh expression in the anterior limb bud mesenchyme.
5 opment, we deleted POR specifically in mouse limb bud mesenchyme.
6 tured chondrocytic cells and differentiating limb-bud mesenchyme.
7 mination in both bone marrow progenitors and limb-bud mesenchyme.
8 n the paraxial mesoderm and migrate into the limb-bud mesenchyme.
9 is expressed and transcriptionally active in limb bud mesenchyme and in mesenchymal condensations.
10 e associated with decreased proliferation of limb bud mesenchyme and small cartilaginous condensation
11 me-derived cell types: (1) the dermatome and limb bud mesenchyme and, later, the subdermal mesenchyme
13 t severely impairs Tcfap2a expression in the limb bud mesenchyme but generates only a modest reductio
14 terior and posterior polarity of the nascent limb bud mesenchyme by impacting Gli3 and Tbx3 expressio
17 8 function in the forelimb AER, we show that limb bud mesenchyme fails to survive in the absence of b
18 arly pronounced in facial ectomesenchyme and limb bud mesenchyme in association with Lim genes, Lhx-6
19 ent, and its conditional deletion from early limb bud mesenchyme in mice severely affects both initia
20 ing the Cre/loxP system in mice, we rendered limb bud mesenchyme insensitive to BMP signals through t
21 y)-cre line led to Fgfr1 inactivation in all limb bud mesenchyme (LBM) cells during limb initiation.
22 hen a bead soaked in FGF is implanted in the limb bud mesenchyme to maintain outgrowth after extirpat
23 , we conditionally inactivated Hif-1alpha in limb bud mesenchyme using a Prx1 promoter-driven Cre tra
25 is dynamically expressed in the early distal limb bud mesenchyme, with expression becoming downregula
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