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1 opic expression of AER-specific genes in the limb ectoderm.
2 A (Bmpr) that disrupted BMP signaling in the limb ectoderm.
4 -7a and Lmx-1, which are expressed in dorsal limb ectoderm and mesoderm, respectively, are thought to
5 proliferation versus differentiation in the limb ectoderm and polarizing activity regions, respectiv
6 s restricted primarily to the distal ventral limb ectoderm and the apical ectodermal ridge, and overl
7 factor, is absent in the mutant presumptive limb ectoderm, and the expression of Fgf10, a mesenchyma
8 al restriction of Wnt and FGF signals to the limb ectoderm, and to a specialized region of it, the ap
9 cal ectodermal ridge (AER), which forms from limb ectoderm at the distal tip of the embryonic limb bu
10 also show that the induction of Fgf-8 in the limb ectoderm by FGF-10 is mediated by the induction of
11 report here that inactivating Fgf8 in early limb ectoderm causes a substantial reduction in limb-bud
15 pression of an early broad ventral domain of limb ectoderm into a narrow rim at the tip and further s
17 ich is expressed ubiquitously throughout the limb ectoderm, is essential for normal limb development
18 iption factor expressed in embryonic ventral limb ectoderm, is essential for ventral limb patterning.
20 ectopic expression of Wnt-7a in the ventral limb ectoderm of En-1 mutants results in the formation o
21 EGF is present in ventral but not dorsal limb ectoderm, suggesting a role for EGF in specificatio
22 ectoderm, nor in presumptive non-AER-forming limb ectoderm, suggesting possible roles in initial limb
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