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1  proliferation, and intrastromal invasion by limbal epithelial cells.
2 nces in glycogen content between corneal and limbal epithelial cells.
3 accounted for an average of 40% of the total limbal epithelial cells.
4 superior in vitro proliferative potential of limbal epithelial cells, and the transplanted limbal cel
5 iated corneal epithelial cells, and SSEA4(-) limbal epithelial cells contain a higher proportion of l
6 cently, ex vivo cultivation and expansion of limbal epithelial cells has been performed utilizing AM
7 , as well as from immortalized human corneal-limbal epithelial cell (HCLE) cultures.
8                                        Human limbal epithelial cells (HLE) and corneal stromal stem c
9 /beta-catenin pathway influences the fate of limbal epithelial cells, likely to be progenitor cells,
10                An immortalized human corneal-limbal epithelial cell line (HCLE) expressing the same M
11                An immortalized human corneal limbal epithelial cell line was treated in the presence
12                                          The limbal epithelial cells of FIH-1 null mice had an increa
13 mmunostaining with mAb HE1/11F, however, the limbal epithelial cells stained weakly.
14                      Primary cultures of pig limbal epithelial cells stained with Hoechst 33342 were
15      In the control, most expansion of human limbal epithelial cells started from the limbus from day
16 ained from a healthy area of the limbus, the limbal epithelial cells were cultured on a denuded human
17 thelial cells and immortalized human corneal limbal epithelial cells were cultured on the SF and denu
18 greatest number of Ki67-positive corneal and limbal epithelial cells were present at days 13 to 19, a
19                             Primary cultured limbal epithelial cells were treated with either 10 micr
20                        We have observed that limbal epithelial cells, when removed from their niche a

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