1 egions, including the frontal, temporal, and limbic cortices.

2 t significantly less Fos labeling than Wt in limbic cortices and subcortical structures, including ke

3  in the somatosensory cortex compared to the limbic cortices, and limbic cortical degeneration is not

4  are occupied by heteromodal, paralimbic and limbic cortices, collectively known as transmodal areas.

5 ons as it spread over time from temporal and limbic cortices into frontal and occipital brain regions

6 rast, injections in orbitofrontal and medial limbic cortices labeled neurons in the anterior and tube

7 nduced hyperexcitability of the amygdala and limbic cortices produced exaggerated conditioned fear-po

8 d disruption of the glutamatergic input from limbic cortices, resembling the action of the atypical a

9 rug injection included exacerbated damage in limbic cortices, retrosplenial cortical damage, and redu

10 n in the cortex, extending from the simplest limbic cortices to eulaminate areas with elaborate lamin

11  visual, auditory, somatosensory, motor, and limbic cortices via retrograde pathway tracers injected

12         Projection neurons targeting orbital limbic cortices were more prevalent in the lateral part

13 hypothalamus, whereas those targeting medial limbic cortices were more prevalent in the medial hypoth

14 more strongly linked to parietal, motor, and limbic cortices, whereas lateral extrastriate areas were

15                         Visual, auditory and limbic cortices, which are known to myelinate early, sho

16 l, posterior parietal, superior temporal and limbic cortices with the cerebellum.