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1  with GABAB encephalitis have presented with limbic encephalitis.
2 s, and a mixed movement disorder rather than limbic encephalitis.
3 he cognitive impairment associated with this limbic encephalitis.
4 age-gated potassium channel complex antibody limbic encephalitis.
5 dentify the real autoantigen associated with limbic encephalitis.
6  be the target of antibodies associated with limbic encephalitis.
7 patients (P < 0.0001), who predominantly had limbic encephalitis.
8                       Only 26% had classical limbic encephalitis.
9 y been reported in three cases of reversible limbic encephalitis.
10 romyotonia (NMT), Morvan syndrome (MoS), and limbic encephalitis.
11 ented as classic paraneoplastic syndromes (5 limbic encephalitis, 1 paraneoplastic encephalomyelitis,
12 e neurological presentations (5 brainstem or limbic encephalitis, 3 cerebellar ataxia, 2 Lambert-Eato
13  reactivation is well established as causing limbic encephalitis after haematopoietic stem cell trans
14  seen with symptoms suggestive of autoimmune limbic encephalitis, although they can be paucisymptomat
15  whereas LGI1 autoantibodies are involved in limbic encephalitis, an acquired epileptic disorder asso
16 ients with neuromyotonia, Morvan's syndrome, limbic encephalitis and a few cases of adult-onset epile
17                             One patient with limbic encephalitis and antibodies against LGI1 also had
18 We analysed sera and CSF of 57 patients with limbic encephalitis and antibodies attributed to voltage
19 cerebrospinal fluid of patients with ataxia, limbic encephalitis and certain forms of epilepsy.
20 with various clinical presentations, such as limbic encephalitis and complex and diffuse encephalopat
21 od B-cell populations from two patients with limbic encephalitis and faciobrachial dystonic seizures
22 vant, but not all patients had a 'classical' limbic encephalitis and some did not receive immunothera
23 re found in patients with different forms of limbic encephalitis, and in a few patients with epilepsy
24 h VGKC antibodies described in patients with limbic encephalitis, and the subsequent seminal paper de
25 re considered definitely autoimmune, 10 with limbic encephalitis (antibody specificity: 5 LGI1, 1 con
26                                     The term limbic encephalitis associated with antibodies against v
27 ated potassium channels should be changed to limbic encephalitis associated with LGI1 antibodies, and
28 ibodies, 79 had the presumptive diagnosis of limbic encephalitis, dementia, cognitive dysfunction, or
29 otein-2 (CASPR2), are found in patients with limbic encephalitis, faciobrachial dystonic seizures, Mo
30 otassium channel complex antibody-associated limbic encephalitis has recently been recognized as a fo
31 ness in 13 patients with VGKC antibodies and limbic encephalitis in 2004.
32 a single institution, who developed subacute limbic encephalitis initially considered of uncertain ae
33                                              Limbic encephalitis is typically a paraneoplastic syndro
34                                              Limbic encephalitis (LE) associated with antibodies to t
35                                              Limbic encephalitis (LE) is often associated with lung,
36 actor-related receptor) in PCD and mGluR5 in limbic encephalitis (LE).
37 ons without peripheral involvement is called limbic encephalitis (LE).
38 -responsive clinical presentations including limbic encephalitis, Morvan's syndrome and acquired neur
39 onopathy (n = 7), K(+)-channel antibody with limbic encephalitis (n = 1) or neuromyotonia (n = 1), an
40                                              Limbic encephalitis, one of the most common manifestatio
41 ge: 64.0 +/- 2.55 years, median 4 years post-limbic encephalitis onset; n = 18 controls].
42 and two as stiff person syndrome; five had a limbic encephalitis or epileptic encephalopathy, two had
43                            Syndromes such as limbic encephalitis or opsoclonus-myoclonus should alway
44                               Paraneoplastic limbic encephalitis (PLE) is a rare disorder characteriz
45      LGI1 is the autoantigen associated with limbic encephalitis previously attributed to voltage-gat
46 es showed that antibodies from patients with limbic encephalitis previously attributed to voltage-gat
47 mplex encephalitis, Korsakoff's syndrome and limbic encephalitis should be considered.
48                                              Limbic encephalitis that is associated with anti-voltage
49 ated 1, are associated with a common form of limbic encephalitis that presents with cognitive impairm
50  description of focal cortical dysplasia and limbic encephalitis, the pathology of status epilepticus
51 same period, only one paraneoplastic case of limbic encephalitis was identified between the two main
52 ed to have paraneoplastic or immune-mediated limbic encephalitis were clinically assessed.
53 -treated LGI1 VGKC-complex antibody-mediated limbic encephalitis were investigated using in vivo ultr
54                                  Symptoms of limbic encephalitis were observed in all patients, inclu
55 ium channel (VGKC) complex antibody-mediated limbic encephalitis with generalized hippocampal atrophy
56                                              Limbic encephalitis with leucine-rich, glioma-inactivate
57  with high potassium channel antibodies have limbic encephalitis without tumours.

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