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1 uted structures, most of which belong to the limbic system.
2 tified abnormal activation in the prefrontal-limbic system.
3 eceptor before delivery to the neocortex and limbic system.
4 d on emotional and situational cues from the limbic system.
5 the balance of excitation-inhibition in the limbic system.
6 y as inhibitory components of the prefrontal-limbic system.
7 changes in the tissue levels of M-Enk in the limbic system.
8 MRI studies had signal abnormalities in the limbic system.
9 t of schizophrenia is on the hippocampus and limbic system.
10 n specific nuclei of the basal forebrain and limbic system.
11 ted within the temporal lobe regions and the limbic system.
12 discrete regions of the ram hypothalamus and limbic system.
13 s expressed in several discrete areas in the limbic system.
14 h emotional behavior, such as the cortex and limbic system.
15 tly expressed in neurons in the thalamus and limbic system.
16 ions of the preoptic area, hypothalamus, and limbic system.
17 oth cortical and subcortical portions of the limbic system.
18 d in cortical and subcortical regions of the limbic system.
19 rved in choroid plexus and components of the limbic system.
20 bnormal volumes are densely connected to the limbic system.
21 on the mechanisms of ketamine on a disrupted limbic system.
22 aminergic and glutamatergic functions in the limbic system.
23 ed fear, and neurochemical parameters in the limbic system.
24 inter-hemispheric integration and within the limbic system.
25 animals, highlighting the importance of the limbic system.
26 TLE is associated with reorganisation of the limbic system.
27 ce implicating other networks, including the limbic system.
28 rk information to spatial signals within the limbic system.
29 arget neurons in subcortical stations of the limbic system.
30 ities in frontal lobe, basal ganglia and the limbic system.
31 eding centers and mood regulators within the limbic system.
32 l, as well as of the hypothalamus and of the limbic system.
33 sly unknown progenitor pool dedicated to the limbic system.
34 s with a theoretical model of the prefrontal-limbic system.
35 es, since they are two key structures of the limbic system.
36 ceive input from different structures of the limbic system.
37 -myoclonus, and encephalitides affecting the limbic system.
38 ula (LHb) which is often associated with the limbic system.
39 izophrenia relates to overstimulation of the limbic system.
40 relin may act at a range of sites within the limbic system.
41 ge glutamatergic neurons innervating the rat limbic system.
42 of the core circuitry of imitation with the limbic system.
43 tween the temporal lobes, frontal lobes, and limbic system.
46 role in regulating neuronal functions of the limbic system, although the cellular targets and molecul
48 ificant signal increases within the affected limbic system and additionally in areas connected to the
49 n 5-HT(2A) receptor mRNA expression occur in limbic system and associated areas following chronic D-A
51 eptide cholecystokinin (CCK) is expressed in limbic system and hypothalamic nuclei that form a circui
52 ndogenous opioids that activate mu-OR in the limbic system and hypothalamus providing a "neurochemica
57 atest differences occurred in regions of the limbic system and primary sensory and posterior parietal
58 subpallial areas strongly implicated in the limbic system and show robust intrahypothalamic connecti
60 hest levels found in specific regions of the limbic system and the brainstem where NPVF-producing neu
61 in extensive neuronal damage throughout the limbic system and the loss of selectively vulnerable neu
63 via tractography of the main bundles in the limbic system and using Tract-Based Spatial Statistics,
66 idely distributed in many regions of cortex, limbic system, and basal ganglia, appear to play a key i
67 ave been described in the prefrontal cortex, limbic system, and cerebellum in individuals with schizo
68 interconnecting the basal ganglia, thalamus, limbic system, and higher cortical centre are implicated
70 found increased Fos in the cerebral cortex, limbic system, and parts of the arousal and autonomic sy
71 filtrates, encephalitis, particularly of the limbic system, and spinal cord white matter inflammation
73 ajor excitatory pathways linking the cortex, limbic system, and thalamus, regions that have been impl
75 hological process in schizophrenia is on the limbic system, and there have been several postmortem re
76 were located in subcortical areas and in the limbic system, and to a lesser degree in the frontal, te
78 omeostatic function, areas in the cortex and limbic system are important for processing information r
80 on cells, grid cells, and place cells in the limbic system, are thought to underlie navigation by pat
81 These results and demonstrations that other limbic system areas make time-limited contributions to l
83 xtensive transneuronal labeling was found in limbic system areas: (1) extended amygdaloid complex, (2
84 tress and neuropathology produced in the rat limbic system as a result of seizure activity elicited b
85 changes were localized mainly to within the limbic system, as well as between the limbic and nocicep
95 have long considered it part of an extended limbic system based on its location posterior to the orb
96 of cognitive and reward functions in cortico-limbic systems (bottom-up) and then on pathways by which
98 renaline and GABA levels in key areas of the limbic system, but not with functional alterations in th
99 l plasticity and seizures, is induced in the limbic system by chronic ethanol consumption, temporally
100 m and additionally in areas connected to the limbic system by neural pathways, including the lingual
101 interface between the visual pathway and the limbic system, by which increasingly refined and specifi
103 volving the occipital lobes, temporal lobes, limbic system, cerebellum, and frontoparietal cortices,
104 volving the occipital lobes, temporal lobes, limbic system, cerebellum, and frontoparietal cortices,
107 The patient, R.M., had extensive bilateral limbic system damage, affecting orbitofrontal cortex, te
108 elieved to be impaired by basal ganglia, not limbic system damage, but there is uncertainty whether l
111 ration throughout the association cortex and limbic system, deposition of amyloid-beta peptide (Abeta
112 ructures, LAMP may play an important role in limbic system development and function, as suggested by
115 estigated functional connectivity within the limbic system during face identification; a primary comp
117 (in layers 3 and 5), several members of the limbic system (e.g., hippocampus, central amygdala, and
118 es and loss of synapses in the neocortex and limbic system each correlate strongly with cognitive imp
119 al network measures in structures within the limbic system, especially the left hippocampus, the left
121 ent increase in neuronal excitability in the limbic system has not been unequivocally determined.
122 h include the basal ganglia and parts of the limbic system, have key roles in learning, motor control
124 The head-direction (HD) cells found in the limbic system in freely mov ing rats represent the insta
125 he medial prefrontal cortex and the extended limbic system in methadone maintenance patients with a h
126 dies in almost all of the key regions of the limbic system in the forebrain, including the midline an
128 noradrenergic, serotonergic, olfactory, and limbic systems in the modified behavioral response to qu
129 nt nuclei associated with motor, sensory, or limbic systems; in the caudate nucleus, Thy-1 expression
130 lar, cingulate and somatomotor cortices, the limbic system including amygdaloid nuclei, hippocampus a
131 HD cells have been identified throughout the limbic system including the anterodorsal thalamus (ADN)
132 ithin the neocortex and in structures of the limbic system, including the amygdala, hippocampus, and
133 rior cingulate cortex (ACC), a region of the limbic system involved in processing the emotional compo
137 gdaloid complex (AC), a key component of the limbic system, is a brain region critical for the detect
138 LAMP is expressed by neurons composing the limbic system, is highly conserved between rodents and h
139 ressant efficacy have largely focused on the limbic system, leaving it unclear whether this signaling
140 r results suggest that alpha-activity in the limbic system may be a signature of symptom severity in
141 that abnormal neural connections within the limbic system may contribute to the social impairments o
142 findings extend previous reports of altered limbic system microstructural disorganization as a trait
143 enia tecta and other regions of the anterior limbic system of male weanling rats 3 days after four do
145 d in cortical and subcortical regions of the limbic system of the adult and developing rat central ne
149 Abnormal accumulation of alpha-syn in the limbic system, particularly in the hippocampus, correlat
150 s in glucose utilization concentrated in the limbic system, particularly those areas mediating motiva
151 tem damage, but there is uncertainty whether limbic system pathology does in fact leave the SRT unimp
153 recent imaging studies that suggest that the limbic system plays an important role in the mediation o
154 field potential (LFP) activity in the human limbic system reflect disease state and symptom severity
155 eptide Y Y1R differentially expressed in the limbic system regulates anxiety and stress responses via
157 eexperiencing criterion had fewer lesions in limbic system structures (eg, the cingulum) on the right
158 nd selective activation of somatosensory and limbic system structures in the brain and brainstem foll
159 d disorders because they connect subcortical limbic system structures such as the amygdala with the p
161 ely correlated with fMRI signals measured in limbic system structures, including the anterior hypotha
162 long-lasting alterations in the function of limbic system structures, including the nucleus accumben
163 describe how structures supplying NE to the limbic system, such as the nucleus tractus solitarius (N
164 to abnormal structures in the temporal lobe limbic system suggests that p3 may be a contributing fac
165 lls) or head direction (HD cells) in the rat limbic system suggests that these cell types are importa
166 : Although neuropathologic studies show that limbic system tau disease occurs ubiquitously in middle-
167 olve molecules and processes in the immature limbic system that are activated by stressful challenges
168 PY is abundantly expressed in regions of the limbic system that are implicated in arousal and in the
169 ntified a population of cells throughout the limbic system that discharge as a function of the animal
170 g a sensitized BOLD response in the extended limbic system that may subserve the process of dependenc
171 specific subcortical forebrain nuclei of the limbic system that project to the thalamus and to cortic
172 the patterns of binding in the olfactory and limbic systems, that PP receptors might be involved in p
173 , revealed a high level of expression in the limbic system-the hippocampal formation, the amygdala, t
174 in neocortical association areas and in the limbic system throughout life, where the protein might p
176 with the ventromedial prefrontal cortex and limbic system, together with dorsal and fronto-striatal
177 limbic compromise, it is unknown whether the limbic system undergoes dynamic regional perfusion netwo
179 ed a conserved pattern of expression in the "limbic system." We suggest that FnTm2 may be sensitive t
180 mphetamine can produce notable damage to the limbic system when seizures occur and to the basal gangl
181 likely increased activation of the extended limbic system, which was dysfunctional, and therefore le
182 mber of excitatory synaptic loops within the limbic system, which, in addition to being important in
183 both, Ncdh and Pcdh19, in structures of the limbic system with overlapping expression patterns parti
184 yrophilic neurons in discrete regions of the limbic system; within 24 hr of seizures, a significant p
185 reduced the accumulation of alpha-syn in the limbic system without any significant deleterious effect
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