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1 We assessed tumorigenicity using limiting dilution analysis.
2 herwise underestimated with the conventional limiting dilution analysis.
3 e of techniques exploiting the principles of limiting dilution analysis.
4 incorporation of [3H]thymidine into DNA and limiting dilution analysis.
5 xic (CTLf) T cells were enumerated by use of limiting dilution analysis.
6 because few cells score as LCMV specific in limiting dilution analysis.
7 ), and cytotoxic T cell precursors (pCTL) by limiting dilution analysis.
8 s of the clade B of HIV-1 were determined by limiting dilution analysis.
9 lopeptide reactive T cells was determined by limiting dilution analysis.
10 -lymphocyte precursor (CTLp) frequency using limiting dilution analysis.
11 the HIC and MLR populations, as assessed by limiting dilution analysis.
12 cine endothelial and lymphoblastoid cells by limiting dilution analysis and analyzed the responding T
13 re PR1-specific CTLs than were identified by limiting dilution analysis and correlated better with ly
14 esponses to all 3 viruses were quantified by limiting dilution analysis and staining with HLA-A*0201
15 L reactive to SIV proteins was determined by limiting-dilution analysis and demonstrated that the res
16 ells in peripheral blood were quantitated by limiting dilution analysis, and their effect on EBV load
17 antigen-specific memory B cells by means of limiting-dilution analysis, and we compared memory B-cel
18 as determined by in vitro proliferation and limiting dilution analysis (approximately equals 1:190,0
19 protein L6, were equivalent as determined by limiting dilution analysis (approximately equals 1:5,000
20 te with similarly high cloning efficiency in limiting dilution analysis as CD28(+) CD45RO(hi) cells o
23 ontrols; because of low cell numbers, formal limiting dilution analysis could only be performed in 10
28 equencies of 1 in 10(5)-10(6) as measured by limiting dilution analysis, estimates of the T cell freq
29 36 mo after transplantation were studied in limiting dilution analysis for allopeptide reactivity.
30 chromium release assay for CTL activity and limiting dilution analysis for CTL precursor frequency,
38 than, CTL precursor frequencies derived from limiting dilution analysis, indicating that this novel p
39 L responses against different virus strains, limiting dilution analysis is particularly appropriate t
40 panded in the presence of IL-2 and tested in limiting dilution analysis (LDA) for reactivity to synth
45 cellular interferon-gamma (IFN-gamma) and by limiting-dilution analysis (LDA) of cytotoxic T-cell pre
46 ecific CD8+ T cells has been accomplished by limiting dilution analysis of cytotoxic precursor cells.
54 M+, IgG+, and RF+ B cells were determined by limiting-dilution analysis of purified peripheral blood
55 nd multiply spliced viral RNAs combined with limiting dilution analysis provided measurements of tran
58 requencies of helper and cytotoxic cells, by limiting dilution analysis, serially measured in vitro c
63 l HCMV-specific CTL response, we have used a limiting-dilution analysis system to quantify HCMV-speci
65 e used monocyte-derived dendritic cells in a limiting dilution analysis to generate Mtb-specific CD8(
66 ral tissues have not been evaluated, we used limiting dilution analysis to measure the concentration
67 d distinguish patients at risk, we performed limiting dilution analysis to quantify anti-EBV cytotoxi
68 of keratinocytes into subcutis combined with limiting dilution analysis, to assess the long-term repo
69 equency of BCR-ABL(+) cells as determined by limiting dilution analysis was consistently higher in 38
77 ated cell sorting, cell transplantation, and limiting dilution analysis, we were able to identify the
79 ong term immunologic memory as determined by limiting dilution analysis, with memory T cells displayi
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