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1 lastid biogenesis inhibitors norflurazon and lincomycin.
2 s affected by treatment with norflurazon and lincomycin.
5 aliana plants under long-term treatment with lincomycin, a chloroplast protein synthesis inhibitor.
7 plastid signals, the effects of norflurazon, lincomycin and 3-(3,4-dichlorophenyl)-1,1-dimethylurea (
8 under a variety of treatments (Norflurazon, lincomycin and a far-red light pre-treatment) leading to
11 ring resistance to kanamycin, spectinomycin, lincomycin, and gentamycin/sisomycin, respectively) were
12 ndent transcripts is specifically induced by lincomycin, and the splicing of ndhB transcripts is sign
13 dochii to tiamulin, valnemulin, doxycycline, lincomycin, and tylosin by broth microdilution and that
15 results provide significant insight into the lincomycin biosynthetic pathway, because the activated o
16 e showed that treatment with norflurazon and lincomycin, but not DCMU, decreased the accumulation of
17 ycin), macrolides-lincosamids (erythromycin, lincomycin), coumarin derivatives (coumermycin A(1), nov
18 ein synthesis was blocked by the addition of lincomycin, D1 degradation was again slower in S264P tha
20 ml chloramphenicol, gentamycin, paromomycin, lincomycin, hygromycin, and tetracycline, as well as cyc
21 B. thetaiotaomicron, the homolog of linA, a lincomycin resistance gene from Staphylococcus aureus.
22 red to confer sensitivity to norflurazon and lincomycin, suggesting post-transcriptional regulation.
23 use antibiotics (trimethoprim, tylosin, and lincomycin) to algal and cyanobacterial species in Europ
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