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1  2 diaminopyimidines, 1 aminocoumarin, and 1 lincosamide).
2  resistance to tetracycline, macrolides, and lincosamides.
3 nd tlrD) conferring resistance to macrolide, lincosamide and streptogramin B type (MLS) antibiotics w
4 istance to beta-lactams, cephalosporins, and lincosamides and were multiresistant.
5 ylase that mediates resistance to macrolide, lincosamide, and streptogramin antibiotics.
6 sents one of the target sites for macrolide, lincosamide, and streptogramin B antibiotics.
7 ycin A against the inducibly MLS (macrolide, lincosamide, and streptogramin B)-resistant organisms.
8 (macrolides, aminoglycosides, tetracyclines, lincosamides, and chloramphenicol), DNA synthesis inhibi
9 luding tetracyclines, macrolides, ketolides, lincosamides, and fluoroquinolones.
10          Multidrug resistance to macrolides, lincosamides, and tetracycline was the most frequent ant
11 le mechanisms of resistance to macrolide and lincosamide antibiotics are present in S. pyogenes strai
12 and infants, and that the development of new lincosamide antibiotics for malaria should be reconsider
13 mutant was hypersusceptible to macrolide and lincosamide antibiotics, even in the presence of the erm
14 rythromycin, the ketolide telithromycin, the lincosamide clindamycin, and a phenicol, chloramphenicol
15 including erythromycin and azithromycin) and lincosamide (including clindamycin) antibiotics are reco
16 crolides, but not to 16-membered macrolides, lincosamides or analogues of streptogramin B.
17 ncy, peptide deformylase inhibitors, and new lincosamide, oxazolidinone, lipopeptide and cephalospori
18                                Macrolide and lincosamide resistance in S. pyogenes is mediated by sev
19 ain isolated in 1980 and the first macrolide/lincosamide-resistant strain isolated in 1984.
20 reus conferred hypersensitivity to macrolide-lincosamide-streptogramin B (MLS(B)) antibiotics on stra
21           Strains contained either macrolide-lincosamide-streptogramin B (MLSB) resistance genes enco
22  or ribosomal target modification (macrolide-lincosamide-streptogramin B [MLSB] resistance; usually e
23 mycin and other antibiotics of the macrolide-lincosamide-streptogramin B group (MLS) is methylation o
24 ed by the possibility of inducible macrolide-lincosamide-streptogramin B resistance (MLSBi).
25 with the constitutive or inducible macrolide-lincosamide-streptogramin B resistance phenotype (cMLS(B
26 LI and ERY resistant (constitutive macrolide-lincosamide-streptogramin B resistance) demonstrated eit
27 azole, and rifampin, but inducible macrolide-lincosamide-streptogramin resistance in a subset of CA-M
28  and M. ulcerans, M. tuberculosis (macrolide-lincosamide-streptogramin resistance protein, MLSRP), an
29 sferases confers resistance to the macrolide-lincosamide-streptogramin type B (MLS) antibiotics throu
30           Tetracycline, multidrug, macrolide-lincosamide-streptogramin, bacitracin, vancomycin, beta-
31 y a gene conferring resistance to macrolides-lincosamides-streptogramin B, showing that differential
32  the effect of fluoroquinolone and macrolide/lincosamide usage on resistance of methicillin-resistant

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