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1 morphism data, for genomewide association or linkage disequilibrium mapping.
2 apping, in terms of both linkage mapping and linkage disequilibrium mapping.
3 1.1 region with further localization through linkage disequilibrium mapping.
5 l polymorphisms in this gene-dense region by linkage disequilibrium mapping across 6 adjacent genes.
6 utations were genotyped, and a joint linkage/linkage disequilibrium mapping analysis was used to demo
7 ss the genome has important implications for linkage disequilibrium mapping and association studies,
8 Gene mapping efforts are now focussing on linkage disequilibrium mapping and extension of the inte
9 ement of the location of BLM has relied upon linkage-disequilibrium mapping and somatic intragenic re
10 -based gene-mapping methods-likelihood-based linkage-disequilibrium mapping and the transmission/dise
11 ing techniques, such as comparative mapping, linkage disequilibrium mapping, and deletion mapping, an
13 ologies is crucial in haplotype analyses and linkage-disequilibrium mapping for complex diseases.
19 these features severely limit the ability of linkage disequilibrium mapping in Drosophila to resolve
20 ocus that accounts for this relative risk by linkage-disequilibrium mapping in an admixed population
22 systems, association mapping (also known as linkage disequilibrium mapping) is increasingly being ad
26 sion models are proposed for high-resolution linkage disequilibrium mapping of quantitative trait loc
27 ected-only DNA-pooling strategy to carry out linkage disequilibrium mapping of the ED4 gene to 11q23.
29 e we build on recently developed methods for linkage-disequilibrium mapping of quantitative traits to
31 oaches, such as quantitative trait locus and linkage disequilibrium mapping, start with a phenotype o
32 ethodologies will allow thorough genome wide linkage disequilibrium mapping studies in large cohorts
33 A. thaliana and may need to be considered in linkage disequilibrium mapping studies of genetically di
35 ually using logistic regression, followed by linkage disequilibrium mapping to identify the causal lo
36 er requires high-resolution recombination or linkage disequilibrium mapping to nominate putative cand
39 were designated I/I, I/III, and III/III and linkage disequilibrium mapping was used to test the prim
43 is not practical and allelic association or linkage disequilibrium mapping will have to be employed
45 nkage and case-control association analyses, linkage disequilibrium mapping with haplotypes was used.
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