ライフサイエンスコーパス: lip

1 hat measured the skin (r = -0.48, P < .001), lips (r = -0.21, P = .001), and lip rhytids (r = -0.32,

2 in's lymphoma (HR, 3.0; 95% CI, 1.7 to 5.3), lip cancer (HR, 3.4; 95% CI, 2.0 to 6.0), and nonepithel

3 t other metals in a convenience sample of 32 lip products used by young Asian women in Oakland, Calif

4 occurring prior to folding of the activation lip and of loop L16.

5 g and modification of some phonemes although lip closure was still possible allowing food intake.

6 We analyzed lip products by inductively coupled plasma optical emiss

7 , P < .001), lips (r = -0.21, P = .001), and lip rhytids (r = -0.32, P < .001) correlated with the re

8 recorded M1 neurons in the anterior bank and lip of the central sulcus as monkeys performed more natu

9 croelectrode arrays in the anterior bank and lip of the central sulcus in each of two monkeys.

10 icant positive correlation between blood and lip tissue DNA methylation, indicating blood as a suitab

11 Cancers of the breast, uterine cervix, and lip or oral cavity are three of the most common malignan

12 tial coherence between left motor cortex and lip movements and this partial coherence directly predic

13 zed patients with dry eyes and dry mouth and lip biopsies from the Sjogren's International Collaborat

14 regulated by PeMYBs in the sepals/petals and lip.

15 stive mouth movements (tongue protrusion and lip smacking) for the purposes of social communication:

16 fects checklists for problems after skin and lip treatment.

17 related with the reporting of more skin- and lip-related adverse effects.

18 others unrelated (eg, melanoma, thyroid and lip cancers).

19 Performance levels and lipping intensity (an appetitive behavior) both showed t

20 ion included nose, chin, part of cheeks, and lips.

21 ion that was most pronounced on the face and lips and was associated with nail pigmentation, blue pig

22 f nonpitting edema and involves the face and lips.

23 about 17 neurons that supplied the anterior lip region and parts of the central body.

24 Both shell shape and apertural lip thickness contributed to shell strength suggesting t

25 tats showed greater plasticity for apertural lip thickness and apertural area in the presence of crab

26 threat), fearful (fear grin), and appeasing (lip smack)--were presented to the subjects in a blocked

27 orehead, glabella, lateral periorbital area, lips, and marionette lines), with scores ranging from 0

28 involved multiple body parts (residual arm, lips, and feet).

29 de-by-side computer-generated monkey avatars lip-smacking at natural versus sped-up or slowed-down rh

30 sociated increases in affiliative behaviors--lip smacking, visual attention to a caregiver, and time

31 consistently oriented toward the blastopore lip in dorsal and lateral cells, but oriented parallel t

32 e apical surfaces of cells at the blastopore lip, and it functions during blastopore formation and cl

33 At the blastopore lip, Vangl2 is required for the apical accumulation of t

34 -water distribution coefficient (K(lipw) = C(lip)/C(w)) as 450 L/kg lipid, which is independent of do

35 ounty-level mortality rates from 29 cancers: lip and oral cavity; nasopharynx; other pharynx; esophag

36 acial allograft, including mandible, cheeks, lips, and chin, in November 2009.

37 nsistent with the hypothesis that chimpanzee lip-smacks function to coordinate and prolong social gro

38 Cleft lip and/or palate (CL/P) are common structural birth def

39 Cleft lip and/or palate (CL/P), the most frequent human cranio

40 Cleft lip is one of the most common human birth defects, yet o

41 Cleft lip with or without cleft palate (CL/P) is one of the mo

42 Cleft lip with or without cleft palate is the most common cong

43 Cleft lip with or without palate (CLP) and isolated cleft pala

44 Cleft lip, which results from impaired facial process growth a

45 15 cleft lip and palate organisations totalled 26% of both revenu

46 ospadias (0.6%), hydrocephalus (0.6%), cleft lip or palate (0%), and obstetric fistula (0%).

47 observed wider de novo deletions among cleft lip and palate (CLP) cases than seen among cleft palate

48 than seen among cleft palate (CP) and cleft lip (CL) cases.

49 and isolated cleft lip only (CLO) and cleft lip and palate (CLP).

50 p = 0.001), and with SNP rs3923086 and cleft lip and palate in Asian populations (p = 0.004).

51 ry such as ophthalmology (88, 28%) and cleft lip and palate surgery (70, 22%) were also frequently pe

52 of limb deficiency, cleft palate, and cleft lip cases were, respectively, 1.8 (95% CI: 1.1, 3.1), 1.

53 persistence of the epithelial seam and cleft lip.

54 owth, epithelial seam persistence, and cleft lip.

55 ctrodactyly, ectodermal dysplasia, and cleft lip/palate (EEC) syndrome is caused by single point muta

56 ctrodactyly, ectodermal dysplasia, and cleft lip/palate (EEC) syndrome patients with p63 mutations as

57 associated with cognitive defects and cleft lip/palate, its role in mammalian development and physio

58 y of Fallot, coarctation of the aorta, cleft lip, cleft palate, anorectal atresia/stenosis, and limb

59 longed treatment trajectories, such as cleft lip and palate.

60 sed risk of anencephaly, spina bifida, cleft lip with or without cleft palate (CLP), or cleft palate

61 -agonists were found for spina bifida, cleft lip, anal atresia, severe congenital heart defects in ge

62 aniofacial defects including bilateral cleft lip and palate and tongue agenesis, following the loss o

63 The proband presented with bilateral cleft lip and palate, malformed auricles, and bilateral ectrod

64 directed at ophthalmology, followed by cleft lip and palate surgery.

65 n saliva, and patients with the common cleft lip and palate have a higher prevalence of dental caries

66 ns or liver consumption would decrease cleft lip with or without cleft palate (CL +/- P) risk and tha

67 ing regulatory protein (Esrp1) develop cleft lip and palate.

68 candidate genes for genetic disorders (cleft lip and palate, certain forms of cancer) or solute uptak

69 ing characteristic facial dysmorphism, cleft lip and/or palate, craniosynostosis, learning disability

70 the ectodermal dysplasia-ectrodactyly-cleft lip/palate (EEC) syndrome, comprising SHFM.

71 he cellular mechanism demonstrated for cleft lip pathogenesis, we found that either SHH ligand additi

72 cilium, and these patients often have cleft lip and palate.

73 ed testes, hypospadias, hydrocephalus, cleft lip or palate, and club foot.

74 ed testes, hypospadias, hydrocephalus, cleft lip/palate, and clubfoot) was determined by physical exa

75 birth defects, including hypospadias, cleft lip with or without cleft palate, or hydrocephalus.

76 ental anomalies were more prevalent in cleft lip with cleft palate than other cleft types.

77 disruption of this sequence results in cleft lip.

78 esult is orofacial clefting, including cleft lip and cleft palate (CL/P).

79 use of abnormal development (including cleft lip and/or palate, craniosynostosis and facial dysostose

80 Three common diseases, isolated cleft lip and cleft palate (CLP), hypothyroidism and thyroid c

81 Among individuals with isolated cleft lip and cleft palate, increased risks of intellectual di

82 three affected siblings with isolated cleft lip and palate, we discovered that they share a novel mi

83 , KIAA1598-VAX1, and MAFB and isolated cleft lip only (CLO) and cleft lip and palate (CLP).

84 ung adults who were born with isolated cleft lip only did not differ significantly from unaffected in

85 mortality among persons with isolated cleft lip only.

86 lateral choanal atresia, hearing loss, cleft lip and/or palate, and other craniofacial dysmorphisms.

87 lly treatable conditions (breast mass, cleft lip/palate, club foot, hernia or hydrocele [adult and pa

88 nout, expansion of the facial midline, cleft lip, extensive exencephaly, and microphthalmia or anopht

89 Nonsyndromic cleft lip and/or palate (NSCL/P) is a prevalent birth defect o

90 known candidate genes for nonsyndromic cleft lip and/or palate through genome-wide linkage analysis.

91 e genetic contribution to nonsyndromic cleft lip and/or palate through the analysis of family pedigre

92 icated in the etiology of nonsyndromic cleft lip with or without cleft palate (NSCL/P) in populations

93 Nonsyndromic cleft lip with or without cleft palate (nsCL/P) is among the m

94 genetic risk variants for nonsyndromic cleft lip with or without cleft palate (NSCL/P).

95 Nonsyndromic cleft lip with/without cleft palate (nsCL/P) and nonsyndromic

96 Non-syndromic (NS) cleft lip with or without cleft palate (CL/P) is a common diso

97 WS), the most common syndromic form of cleft lip and palate.

98 erygium syndrome, 2 syndromic forms of cleft lip and palate.

99 Median CERs of cleft lip or palate repair ($47.74 per DALY), general surgery

100 isorder; however, our understanding of cleft lip pathogenesis remains incomplete.

101 es to the etiology and pathogenesis of cleft lip through antagonistic interactions with other gene re

102 (TGFA) polymorphisms with the risk of cleft lip with or without cleft palate (CL/P) or cleft palate

103 understanding the genetic etiology of cleft lip with or without cleft palate, relatively little is k

104 in the upper beak or the equivalent of cleft lip.

105 th syndromic and nonsyndromic forms of cleft lip/palate (CLP).

106 senting 14 specialties (ophthalmology, cleft lip/palate, multidisciplinary teams, orthopaedics, cardi

107 t surgical specialties (ophthalmology, cleft lip/palate, multidisciplinary teams, orthopaedics, cardi

108 steroids were confirmed (cleft palate, cleft lip, anal atresia, and hypospadias).

109 cts, middle-ear defects, cleft palate, cleft lip, limb defects, limb-reduction defects, polydactyly,

110 Non-syndromic cleft lip and/or palate (NSCLP) is a common congenital malform

111 ts (OFCs), which include non-syndromic cleft lip with or without cleft palate (CL/P), are among the m

112 fied in several GWAS for non-syndromic cleft lip with or without cleft palate (NS CL/P).

113 s as candidate genes for non-syndromic cleft lip with or without cleft palate (NSCL/P).

114 Non-syndromic cleft lip with palate (NSCLP) is the most serious sub-phenotyp

115 The cleft lip area, bilateral nostril areas, and the nostril heigh

116 een the facial processes that leads to cleft lip and cleft palate.

117 showed that mutations in IRF6 lead to cleft lip and palate and mandibular abnormalities.

118 20 patients with complete unilateral cleft lip and palate were prospectively recruited.

119 Wnt9b mutations are associated with cleft lip and cleft palate in mice; however, the cause of thes

120 al outcome assessment in patients with cleft lip and for the rehabilitation of patients with facial n

121 ling pathway have been associated with cleft lip and palate in humans and mice, the mechanisms involv

122 mprove nasal symmetry in patients with cleft lip and palate.

123 Patients with cleft lip and/or palate (CLP), who undergo numerous medical in

124 rge multi-ethnic human population with cleft lip identified clusters of single-nucleotide polymorphis

125 e authors identified associations with cleft lip with cleft palate (aOR = 1.23) and anorectal atresia

126 dentified in both mice and humans with cleft lip.

127 is commonly found in individuals with cleft lip/palate (CL/P), we used four large cohorts to evaluat

128 Patients with cleft lip/palate (CLP) have been reported, in some studies, to

129 o investigate if genes associated with cleft lip/palate were also associated with oral cancer, we gen

130 eral types of cancer and recently with cleft lip/palate.

131 ith isolated cleft palate (ie, without cleft lip) had increased mortality (hazard ratio, 3.4; 95% CI,

132 nockout embryos showed fully penetrant cleft lips and short snouts.

133 The cleft is defined as type I ("closed lips") if there are fused clefts in cerebral mantle.

134 me model is proposed as reference to compare lip movement during speech in similar population groups.

135 , seen in a mirror, spoke with corresponding lip movements, and for half of the participants this was

136 irtual mirror, that spoke with corresponding lip movements.

137 mined whether the three electrically coupled lip motoneurons C15/16/17 are controlled by the buccal C

138 A deaf lip-reading interpreter and a hearing American sign lang

139 hus the dynamic behavior of epithelia during lip/palate development.

140 d move with a speech-like 5 Hz rhythm during lip-smacking, but not during chewing.

141 link between WNT9B and FGF signaling during lip and upper jaw development.

142 le for sonic hedgehog (SHH) signaling during lip fusion.

143 s that are regulated by Shh signaling during lip morphogenesis.

144 ics of the macaque monkey vocal tract during lip-smacking (a rhythmic facial expression) versus chewi

145 mild systemic reaction to peanut experienced lip swelling, stomach cramping, and objective tiredness.

146 uctures in Aplysia californica: the external lips and the internal radula.

147 and P > .16 for individual symptoms of face, lip, tongue, or eyelid swelling).

148 Most patients had swelling of face/lips (78.3%) and tongue (78.3%).

149 hic frequencies of lesions were as followed: lips and oral cavity (n = 46 [93%]) and pharynx and vest

150 xtract three-dimensional coordinate data for lip shape during four spoken words decomposed into seven

151 jectable with low viscosity may be ideal for lip enhancement wherein softness is required, whereas a

152 study was to develop an outcome measure for lip shape during speech using statistical shape analysis

153 both kiss initiators and kiss recipients for lip kissing, and took into consideration differences due

154 AC and MEL; RCM and dermoscopy were used for lips analysis.

155 nd sometimes difficult to differentiate from lip melanoma (MEL).

156 sks that depend implicitly on achieving good lip-synch.

157 was seen with vesicles and bullae around her lips, trunk, axillae, arms, and thighs.

158 Metal content in lip products has been an issue of concern.

159 ripheral nervous system (PNS), especially in lip and foot regions of the anterior integument.

160 Irf6 cKO), bypassing the role of the gene in lip and palate morphogenesis and thus ensuring survival

161 We identified small yet consistent shifts in lip representation contralateral to the missing hand tow

162 ured the McGurk effect (in which incongruent lip-voice pairs evoke illusory phonemes), and also ident

163 tutive shell shape development and inducible lip thickening have evolved jointly to form an effective

164 e interactions (mutual gaze and intermittent lip-smacking) with human caregivers display increased so

165 and a clinical trial of a minimally invasive lip treatment in the United Kingdom and France (clinical

166 Stimulation of the nose, upper lip, lower lip, and chin caused a somatotopic lateral-to-medial, ip

167 a (BM), HP, and transition zone of the lower lip (TZ) after six-day culture.

168 Our results indicate that the lower lip of the active site mouth is preorganized for efficie

169 with Van der Woude express pits on the lower lip that continuously or intermittently drain saliva, an

170 phyrin, in the microcirculation of the lower lip.

171 , often but not always associated with lower lip pits.

172 ween the nipple-areola complex and the lower lips, moves as a rigid body with the cycling motion of t

173 movement, then one hypothesis is that monkey lip-smacking versus chewing should also exhibit these di

174 ch communicative gesture in macaque monkeys, lip-smacking, has motor parallels with speech in its rhy

175 For example, stimulation of the motor lip representation influences specifically discriminatio

176 und that TMS-induced disruption of the motor lip representation modulated specifically the early audi

177 Multiple lip revision operations are often required throughout ch

178 reductase (NarGHI) was examined using a narG-lip (lipase) reporter gene assay in vivo.

179 rded from 10 patients with clinically normal lips (mean [SD] age, 34.5 [6.1] years), 16 patients with

180 In normal lips, the draped pattern-a previously described MAC RCM

181 confocal microscopy (RCM) features of normal lips of different phototypes and to identify features th

182 Group 1 included patients with normal lips and different phototypes, and Group 2 consisted of

183 were concentrated around the midface, nose, lips, and eyes.

184 Objective assessments of lip movement can be beneficial in many disciplines inclu

185 macules (MAC) are the most frequent cause of lip pigmentation and sometimes difficult to differentiat

186 can assist in the differential diagnosis of lip MEL and MAC.

187 on influences specifically discrimination of lip-articulated speech sounds.

188 spectrometry and used previous estimates of lip product usage rates to determine daily oral intakes.

189 perceptual tuning to the natural rhythms of lip-smacking is unknown.

190 or prospective studies examining the role of lip-reading in medical settings.

191 ls of taste buds including the epithelium of lips and olfactory epithelium, and ii) mechanosensory ce

192 ssociated angioedema (defined as swelling of lips, tongue, pharynx, or face during ACE inhibitor use

193 apses in the microglomeruli of the olfactory lip and visual collar.

194 In type II ("open lips") the clefts are separated and filled with cerebros

195 on the ear (RR, 4.67; 95% CI, 1.28-17.12) or lip (RR, 4.55; 95% CI, 1.41-14.69), invasion beyond subc

196 4.63), ear (RR, 2.33; 95% CI, 1.67-3.23), or lip (RR, 2.28; 95% CI, 1.54-3.37).

197 Clefts of the palate and/or lip are among the most common human craniofacial malform

198 than repression and formation of the palate, lip, and characteristic folds of the closed Antirrhinum

199 ecurrent herpes labialis receiving permanent lip makeup.

200 g high-frequency vocalizations at the phonic lips, a constriction in the nasal passages just beneath

201 Groomers were also more likely to produce lip-smacks during face-to-face grooming where the visual

202 It is time we routinely provide lip-reading interpreters as well as recognize the need f

203 Although rare, lip abscesses related to isotretinoin therapy present wi

204 An RCM lip algorithm was developed that provided 100% sensitivi

205 nderstanding of the mechanisms that regulate lip morphogenesis is limited.

206 canonical variate discriminated the resting lip shape from articulation of the utterances and accoun

207 to seven visemes (which included the resting lip shape).

208 nt of Pax6 in the development of all rhombic lip (RL) lineages.

209 o the medial ganglionic eminence and rhombic lip, resembling the gnathostome brain.

210 , indicating that VZ/SVZ-derived and rhombic lip-derived progenitor cells show differential requireme

211 ricting vz progenitors from becoming rhombic lip progenitors.

212 the dorsal-most region of the caudal rhombic lip, gives rise to the cochlear and precerebellar nuclei

213 ar nuclei that arise from the caudal rhombic lip.

214 defining molecule of the cerebellar rhombic lip and Pax6 is downstream in the Math1 pathway.

215 reduces the length of the cerebellar rhombic lip.

216 We also examine five rhombic lip cell markers (Wls, Math1, Pax6, Lmx1a, and Tbr2) to

217 odelling of mutations in mouse lower rhombic lip progenitors that generate WNT-subgroup tumours ident

218 non-neural roof plate and the neural rhombic lip.

219 Wls-positive cells in the Math1-null rhombic lip indicates that Wls expression is independent of Math

220 interactions during the presence of Rhombic lip versus the presence of distinct internal granule lay

221 signals is required for induction of rhombic lip-derived cerebellar neurons.

222 contains two germinative zones, the rhombic lip (RL) and the ventricular zone (VZ), which generate d

223 Expression of atonal1 in the rhombic lip adjacent at the roof plate boundary is acutely depen

224 on of cerebellar cell types from the rhombic lip and its upregulation inhibits their production.

225 ved from neuronal progenitors of the rhombic lip and that cerebellar ectopia were derived from granul

226 erebellar nuclei that arise from the rhombic lip and that issue mossy fibers.

227 e of spatial compartmentation in the rhombic lip and the interplay between Wls, Math1, and Pax6 in th

228 cularly distinct compartments in the rhombic lip during cerebellar development.

229 The rhombic lip gives rise to neuronal populations that contribute t

230 These findings suggest that the rhombic lip is dynamically patterned by the expression of Wls, M

231 between Wls, Math1, and Pax6 in the rhombic lip provides novel views of early cerebellar development

232 orsal interneurons that comprise the rhombic lip relay sensory information and coordinate motor outpu

233 he cells in the interior face of the rhombic lip throughout normal mouse cerebellar development.

234 ives rise to isthmic nuclei, and the rhombic lip, which generates deep cerebellar nuclei and granule

235 ule precursor cells derived from the rhombic lip.

236 ocalized to the exterior face of the rhombic lip.

237 However, the cerebellum's upper rhombic lip and the optic tectum are abnormal in clo.

238 ain specification and generate upper rhombic lip derivatives on exposure to bone morphogenetic protei

239 ones, the ventricular zone and upper rhombic lip, which give rise to distinct cell types in the matur

240 ioning of fissures, whereas in upper rhombic lip-derived cells the genes are more important in regula

241 precerebellar nuclei originate from rhombic lips, the first analysis of neuronal migrations from the

242 ve knock-out of P/Q-type channels in rhombic-lip-derived neurons including the PF and MF pathways (qu

243 e that form a deep socket rimmed by a robust lip of bone.

244 een oscillatory brain activity and speaker's lip movements and demonstrated significant entrainment i

245 activity in visual cortex and the speaker's lips.

246 nt a case of a 15-year-old boy with a severe lip abscess requiring incision and drainage and hospital

247 scales that measure appearance of the skin, lips, and facial rhytids (ie, overall, forehead, glabell

248 During continuous speech, lip movements provide visual temporal signals that facil

249 Most of the tested lip products contained high concentrations of titanium a

250 We found that lip-smacking, a distinct multimodal oral gesture produce

251 More precisely, the results show that lips on the Target speaker matched to a secondary (Mask)

252 ed syllables were least likely to engage the lip sensorimotor area and they were least impaired by TM

253 the coherent auditory speech signal from the lip-brain coherence.

254 ptical fibre probe is used to illuminate the lip and acquire fluorescence emission spectra in approxi

255 y cilia of the CNC-derived mesenchyme in the lip and palate region in mice and is a strong candidate

256 medication played in the development of the lip abscess.

257 ing PDGF-C have been linked to clefts of the lip and/or palate in humans, and ablation of PDGF-C in 1

258 Surgical repair of the lip is the only treatment and is usually performed durin

259 om the vermillion and/or mucosal part of the lip were recorded from 10 patients with clinically norma

260 rea and for squamocellular carcinomas of the lip with no detectable cervical lymphoadenopathy.

261 onsive to the red spots in the callus of the lip, and PeMYB12 participated in the full pigmentation i

262 For tumors of the lip, oral cavity and pharynx, digestive organs, respirat

263 Clefting of the lip, with or without palatal involvement (CLP), is assoc

264 full pigmentation in the central lobe of the lip.

265 EL of the vermillion and mucosal part of the lip.

266 for whom lymphocytic foci per 4 mm(2) on the lip biopsies was either 0 (F = 0), <1 (F <1) or > 3 (F >

267 lateral cells, but oriented parallel to the lip in ventral regions.

268 t of multiple articulators (for example, the lips, jaw, tongue and larynx) over rapid time scales.

269 experienced an unequivocal angioedema of the lips as a positive reaction in SBPCPT.

270 and usually present with enlargement of the lips, tongue, nose, hands, and feet and are diagnosed by

271 sented with chronic verrucous papules on the lips, tongue, and buccal mucosa refractory to multiple e

272 We found that the lips, tongue, and hyoid move with a speech-like 5 Hz rhy

273 rasping a water bottle and raising it to the lips), while undergoing fMRI.

274 t analysis of neuronal migrations from these lips was performed with markers of neuroblasts.

275 of the first branchial arch are integral to lip and primary palate development.

276 late several morphogenetic events leading to lip fusion.

277 cally the early auditory-cortex responses to lip-articulated speech sounds when they were attended.

278 The motor cortex in the brain tracks lip movements to help with speech perception.

279 s that include a prominent maxilla and upper lip that readily reveal the upper gingiva, widely spaced

280 s cNCC mesenchyme proliferation during upper lip morphogenesis, and that disruption of this sequence

281 cell (cNCC) mesenchyme is required for upper lip closure.

282 ty of cells contributing to the future upper lip.

283 Stimulation of the nose, upper lip, lower lip, and chin caused a somatotopic lateral-to

284 tinoic acid is a critical regulator of upper lip and primary palate development in Xenopus laevis.

285 f IQR, P-value 0.0371), and wrinkle on upper lip (7.7% more wrinkle on upper lip per increase of IQR,

286 kle on upper lip (7.7% more wrinkle on upper lip per increase of IQR, P-value 0.0218).

287 understand how retinoic acid regulates upper lip and palate morphogenesis we searched for genes downr

288 eyes, a narrow nasal bridge, a tented upper lip, a high palate, an open mouth, tightly adherent skin

289 r understand the complex nature of the upper lip and primary palate development which will lead to im

290 The upper lip and primary palate form an essential separation betw

291 and differentiation necessary for the upper lip and primary palate morphogenesis.

292 also results in a median cleft in the upper lip and primary palate.

293 Rgamma result in a median cleft in the upper lip and primary palate.

294 ate mapping revealed contribute to the upper lip and primary palate.

295 The upper lip region has the most significant change in HDX behavi

296 erm of the medial nasal processes, the upper lip remained intact in mutant mice.

297 poral narrowing, broad nasal tip, thin upper lip, posteriorly rotated or low-set ears, and microretro

298 re-biased patch (MCP) located on the ventral lip of the posterior superior temporal sulcus (STS) in a

299 erium simum), a large African herbivore with lips specialized for grazing in open savannahs, relates

300 the ventilated patient who can mouth words, lip-reading interpretation offers an opportunity for com