ライフサイエンスコーパス: lipase

1 e aminotransferase and one grade 4 increased lipase).

2 nditions using immobilised Candida antartica lipase.

3 tic esterification by the Celite-immobilized lipase.

4 s the most powerful (IC50: 76mug/mL) against lipase.

5 ecause of physical losses of the immobilised lipase.

6 rrhage, necrosis, the release of amylase and lipase.

7 t more rapidly degraded in the presence of a lipase.

8 ocalized presynaptically with diacylglycerol lipase.

9 e on the substrate specificity of pancreatic lipase.

10 gs suggested that AtABHD11 is a lyso(phospho)lipase.

11 treated with an inhibitor of lysosomal acid lipase.

12 naling and mice lacking adipose triglyceride lipase.

13 iting the expression of adipose triglyceride lipase.

14 y ePL utilizing Lipozyme(R): sn-1,3 specific lipase.

15 oes not depend on lipid transfer proteins or lipases.

16 p, respectively, in the absence of the other lipases.

17 id droplets, mediated by inhibition of other lipases.

18 membrane trafficking, while others resemble lipases.

19 gests methods for developing these selective lipases.

20 at Yor022c is a Ddl1 (DDHD domain-containing lipase 1) that hydrolyzes CL, phosphatidylethanolamine,

21 ade 3 or worse adverse events were increased lipase (137 [17%] of 784 patients in the orteronel plus

22 ent-related adverse events were increases in lipase (15%), alanine aminotransferase (12%), and aspart

23 tients in the ponatinib group were increased lipase (22 [14%] of 154 vs three [2%] of 152 with imatin

24 ive Hnf4 signaling leads to up-regulation of lipase 3 and enzymes for mitochondrial beta-oxidation.

25 fatty acid selectivity of Candida antarctica lipase A (CAL-A) was applied to produce DHA concentrate

26 d the relevant target of one compound as the lipase ABHD6.

27 the protein content of adipose triglyceride lipase, acetyl-CoA carboxylase 2 and AMP-activated prote

28 and ABHD5 ligands, demonstrating that ABHD5 lipase activation could be dissociated from its other fu

29 t of a novel functional surface required for lipase activation.

30 samples at various time points and measured lipase activities and stabilities.

31 roxidation, as well as alpha-glucosidase and lipase activities were demonstrated, therefore supportin

32 ation of inhibition of alpha-glucosidase and lipase activities.

33 ain or beta5-loop had decreased triglyceride lipase activity similar to that of PNLIPRP2.

34 Three metagenomic enzymes exhibited lipase activity, and seven proteins showed polyester dep

35 iated with reverse cholesterol transport and lipase activity.

36 stantial LD loss via activation of cytosolic lipases adipose triglyceride lipase (ATGL) and hormone-s

37 degrade LD via ATGL (adipocyte triglyceride lipase) after FA loading.

38 AT contains a motif characteristic of serine lipases "AHSLG" and the catalytic triad consisting of se

39 he main 2-AG producing enzyme diacylglycerol lipase alpha (DAGL-alpha).

40 its primary synthetic enzyme, diacylglycerol lipase alpha (DGLalpha), from dopamine D1 receptor-expre

41 the 2-AG-synthesizing enzyme diacylglycerol lipase alpha.

42 ve enzymes were determined by evaluating the lipase, alpha-amylase and alpha-glucosidase activities.

43 id (eCB) biosynthetic enzymes diacylglycerol lipase-alpha (DAGLalpha) and -beta (DAGLbeta) to neurons

44 Increased pancreatic lipase amounts and activity were noted in the extensive

45 Activation of lipoprotein lipase, an enzyme that is inhibited by angiopoietin-like

46 Lipase, an enzyme with industrial significance for catal

47 with increased expression of lysosomal acid lipase, an M2 macrophage marker.

48 Serum lipase and amylase were lower in the DR group than in th

49 ionally related to APOA5, namely lipoprotein lipase and apolipoprotein C-III.

50 ng signals, as well as alkaline phosphatase, lipase and chitinase activities.

51 ut neither significantly altered lipoprotein lipase and cholesteryl ester protein mass or measures of

52 , stronger inhibitory activity on pancreatic lipase and comparable and lower activity on alpha-glucos

53 mulated phosphorylation of hormone-sensitive lipase and consequent lipolysis, as do knockouts of dopa

54 induce phosphorylation of hormone-sensitive lipase and consequently activate lipolysis, which then e

55 TGL) serves as a major triacylglycerol (TAG) lipase and controls the bulk of intracellular lipid turn

56 fied that the mutant strain lost most of its lipase and esterase activities and displayed reduced vir

57 me, including alpha-glucosidase, amylase and lipase and exhibited antioxidant activity by different m

58 hthalate-to-adipate ratio by Rhizopus oryzae lipase and Fusarium solani cutinase.

59 ilarly, the addition of cellulase, protease, lipase and glucose-oxidase did not modify this decreasin

60 avascular lipolysis by adipocyte lipoprotein lipase and hepatic uptake of HDL by scavenger receptor B

61 Elevated expressions of adipose triglyceride lipase and hormone sensitive lipase in adipose tissue fu

62 tivity against alpha-glucosidase, pancreatic lipase and hyaluronidase were determined.

63 including alpha-amylase, alpha-glucosidase, lipase and hydroxyl methyl glutaryl CoA reductase.

64 levated phosphorylation of hormone-sensitive lipase and increased lipolysis.

65 pelling evidence revolves around lipoprotein lipase and its endogenous facilitator (APOA5 [apolipopro

66 exclusively in actinomycetes, exhibits both lipase and protease activities, is secreted into macroph

67 upport the involvement of an uncharacterized lipase and/or of multiple hydrolases.

68 dichloromethane and diethyl ether containing lipases and a subsequent concentration with Vigreux colu

69 her perilipins extend beyond protection from lipases and include the preservation of droplet integrit

70 (beta-AR)/cAMP pathway to activate cytosolic lipases and induce their recruitment to the LD surface.

71 expression of bone sialoprotein, lipoprotein lipase, and fatty acid binding protein 4 are the preferr

72 zymes (hormone-sensitive lipase, lipoprotein lipase, and fatty acid binding protein 4) versus sham.

73 howed lower levels of both hormone-sensitive lipase, and its phosphorylated form.

74 ata, did not allow us to pinpoint one PGE2-G lipase, and rather support the involvement of an unchara

75 three different classes of enzymes (amylase, lipase, and sulfatase), relying on two distinct mechanis

76 Therefore, the A. sediminis lipase appears to be a good candidate enzyme for ester s

77 It is the first time that lipases are added to transform the fat into free fatty a

78 The two lipases are structurally similar, yet they have differen

79 Lipase AS catalyzes the selective hydrolysis of the rear

80 The breakdown of LDs requires lipase associating with core retromer and binding to per

81 1-3h at 30 degrees C, the skin treated with lipase at 0.30 units/g dry skin for 3h had the highest l

82 wo fungal esterases (FsC and Rhizopus oryzae lipase) at different temperatures.

83 g lipolysis as indicated by elevation of the lipase ATGL, the lipolysis marker glycerol and release o

84 ives fat loss via the adipocyte triglyceride lipase ATGL-1.

85 e key lipolytic enzymes adipose triglyceride lipase (ATGL) and hormone sensitive lipase (HSL).

86 on of cytosolic lipases adipose triglyceride lipase (ATGL) and hormone-sensitive lipase (HSL).

87 Adipose triglyceride lipase (ATGL) and its coactivator comparative gene ident

88 ed as the "lipolysome." Adipose triglyceride lipase (Atgl) catalyzes the initiating step of TG hydrol

89 others have shown that adipose triglyceride lipase (ATGL) increases the activity of the nuclear rece

90 ation demonstrates that adipose triglyceride lipase (Atgl) is one of the enzymes involved in the JGM-

91 ither protein levels of adipose triglyceride lipase (ATGL) nor phosphorylations of hormone-sensitive

92 In liver, adipose triglyceride lipase (ATGL) serves as a major triacylglycerol (TAG) li

93 T4) was unaffected, and adipose triglyceride lipase (ATGL) was suppressed.

94 patic levels of SRA and adipose triglyceride lipase (ATGL), a major hepatic triacylglycerol (TAG) hyd

95 teasomal degradation of adipose triglyceride lipase (ATGL), a rate limiting enzyme of TAG hydrolysis.

96 tabolism by attenuating adipose triglyceride lipase (ATGL), but repression of oncogene-induced transf

97 principal TAG hydrolase adipose triglyceride lipase (ATGL), in the regulation of cardiac lipolysis.

98 catabolism mediated by adipose triglyceride lipase (ATGL), the key enzyme for intracellular lipolysi

99 e enzymatic activity of adipose triglyceride lipase (ATGL), which catalyzes the hydrolysis of TGs to

100 conserved regulator of adipose triglyceride lipase (ATGL)-mediated lipolysis that plays important ro

101 ficking itineraries regulated by cytoplasmic lipases, autophagy, and mitochondrial fusion dynamics, e

102 catalyzed by immobilized Candida antarctica lipase B in acetonitrile.

103 atch process catalyzed by Candida antarctica lipase B was optimised, leading to the production of 440

104 Lipases B from Candida antarctica (CALB), Rhizomucor mie

105 l usually requires the use of stereospecific lipases before instrumental identification.

106 I-58 may stimulate an epidermal triglyceride lipase beyond ATGL required for the adequate provision o

107 vation of insulin receptor (InR) and adipose lipase brummer (bmm).

108 ructure by inactivating bile salt-stimulated lipase (BSSL) and partially denaturing some of its prote

109 (BCAL0788), which acts as an outer membrane lipase by removing the primary beta-hydroxymyristate (3-

110 One route is based on a lipase catalyzed cleavage of the protecting group.

111 xylochemistry, followed by hydrogenation and lipase-catalyzed kinetic resolution afforded the target

112 phiphilic lipid alkylsuccinylated tyrosol by lipase-catalyzed succinylation of tyrosol with alkylsucc

113 andem repeat (VNTR) domain of carboxyl ester lipase (CEL) presents an opportunity to study the pathog

114 e production of R-9-PAHSA and carboxyl ester lipase (CEL), a PAHSA degradative enzyme, selectively hy

115 verse events across all doses were increased lipase concentration (12 [9%] of 137), dyspnoea (eight [

116 placebo phase of deferred treatment (raised lipase concentration) were deemed related to study drug.

117 utropenia (four [5%] patients) and increased lipase concentrations (four [5%]).

118 with iso-propanol and hexane the immobilised lipase could be reused in 14 consecutive batches.

119 A commercial preparation of Candida rugosa lipases (CRL) was tested for the production of capsinoid

120 abinoid receptors (CB1Rs) and diacylglycerol lipase (DAGL) in the VTA.

121 nephospholipase D (NAPE-PLD), diacylglycerol lipase (DAGL), or phospholipase C (PLC), and their metab

122 inhibit 2-AG biosynthesis by diacylglycerol lipase (DAGL).

123 Diacylglycerol lipases (DAGLalpha and DAGLbeta) convert diacylglycerol

124 and CNS-active inhibitors for diacylglycerol lipases (DAGLs), enzymes responsible for the biosynthesi

125 e been approved for treatment of lipoprotein lipase deficiency in Europe.

126 Lysosomal acid lipase deficiency is a rare, autosomal recessive conditi

127 y in children and adults with lysosomal acid lipase deficiency, an underappreciated cause of cirrhosi

128 y's disease, Pompe's disease, lysosomal acid lipase deficiency, and five types of mucopolysaccharidos

129 Lysosomal acid lipase deficiency-which can be diagnosed using dry blood

130 uct Glybera for the treatment of lipoprotein lipase deficiency.

131 s in children and adults with lysosomal acid lipase deficiency.

132 ng that SMc01003 also acts as diacylglycerol lipase (DglA) in its native background.

133 Surprisingly, lack of two lipases did not affect expression, localization, and sta

134 1 [11%]), colitis (nine [9%]), and increased lipase (eight [8%]).

135 rade 3 treatment-related adverse events were lipase elevation in four (15%) patients, increased alani

136 A stable covalent binding technique for lipase enzyme immobilization on an optical fiber is repo

137 ic acid, lauric acid and hexanoic acid using lipase enzyme was studied and their emulsion functionali

138 ysis of the Xf secretome revealed a putative lipase/esterase (LesA) that was abundantly secreted in b

139 The lipase/esterase also elicited a hypersensitive response

140 This lipase exhibited high efficiency for omega-3 (n-3), and

141 Increased acid lipase expression and accumulation of free fatty acids a

142 by driving endothelial CD36 and lipoprotein lipase expression and facilitate FA transport across hea

143 Pancreatic lipase expression increased lipolysis in 3T3-L1 cells.

144 , pleural effusion (six [8%]), and increased lipase (five [7%]).

145 that beta-AR stimulation mobilizes cytosolic lipases for LD breakdown in hepatocytes, and perturbatio

146 Lipases form lysolipids that emulsify other lipids.

147 f 78 patients; the most common were elevated lipase (four [5%]), elevated amylase (three [4%]), and f

148 rmine the potential of immobilised digestive lipase from Chinook salmon (Oncorhynchus tshawytscha) to

149 Lipase from liver of seabass (Lates calcarifer), with a

150 Thus, lipase from liver of seabass could be used to remove fat

151 as pseudoalcaligenes (PpCutA) and a putative lipase from Pseudomonas pelagia (PpelaLip) were identifi

152 glycine lipoaminoacid was carried out with a lipase from Pseudomonas stutzeri and a protease from Bac

153 Lipozyme RM IM lipase from Rhizomucor miehei was used as biocatalyst.

154 When seabass skin was treated with crude lipase from seabass liver at various levels (0.15 and 0.

155 tion was conducted employing the immobilized lipase from Thermomyces lanuginosus (Lipozyme TL IM), th

156 igated further by studies on the lipoprotein lipase gene.

157 ta/pancreatitis-associated protein, amylase, lipase, glucose, and creatinine levels were quantified a

158 RNA level of hexokinase-2, hormone sensitive lipase, glutathione peroxidase-1, and myosin heavy chain

159 These hydrolases comprise an array of lipases, glycosidases, and proteases and thus, they have

160 Purified lipase had Michaelis-Menten constant (Km) and catalytic

161 1 mutation, deficient in the predominant TAG lipase, had little effect on total FA content but increa

162 racteristics of milk, the immobilised salmon lipase has potential applications in developing dairy pr

163 osphorylation of cytosolic hormone-sensitive lipase (HSL) and perilipin 1 (Plin1) in the lipid drople

164 fective phosphorylation of hormone-sensitive lipase (HSL) at S565, with higher phosphorylation at pro

165 depended on activation of hormone-sensitive lipase (HSL) in cultured adipocytes and in the epididyma

166 imulated glucose uptake in hormone-sensitive lipase (HSL) knockout (KO) mice after treadmill exercise

167 lyceride lipase (ATGL) and hormone-sensitive lipase (HSL).

168 lyceride lipase (ATGL) and hormone sensitive lipase (HSL).

169 fatigue (four [2%]), and asthenia, elevated lipase, hypophosphataemia, and pneumonitis in two (1%) p

170 tion of alpha-amylase, alpha-glucosidase and lipase (IC50: 0.38mg/mL, 0.87mug/mL and 15mug/mL, respec

171 calable methodology to create Candida rugosa lipase-immobilized magnetic nanoparticles (L-MNPs) by th

172 se triglyceride lipase and hormone sensitive lipase in adipose tissue further corroborated our findin

173 glycerol 1) is synthesized by diacylglycerol lipase in pyramidal neurons; 2) travels retrogradely to

174 ng alveoli, albumin in liver parenchyma, and lipase in the stomach lining.

175 suggested that pOPCs reduced the activity of lipase in vitro and triglyceride storage capacity in viv

176 f cross talk between lipophagy and cytosolic lipases in lipid mobilization.

177 regulatory network of yeast triacylglycerol lipases in more detail, we also examined properties of T

178 in SAP, we evaluated the role of pancreatic lipases in SAP-associated visceral fat necrosis, the inf

179 licability of one class of biocatalysts viz."lipases" in synthetic transformations, the resolution of

180 No cytotoxicity and a moderate lipase inhibition activity were highlighted.

181 ne-infused rats with an adipose triglyceride lipase inhibitor blocked corticosterone-induced increase

182 G since it was blocked by the diacylglycerol lipase inhibitor DO34.

183 Pretreatment with the monoacylglycerol (MAG) lipase inhibitor JZL-184 also reduced affective disturba

184 was not preserved with the monoacylglycerol lipase inhibitor JZL184.

185 TAG species as compared with a more general lipase inhibitor orlistat.

186 gonist WIN 55,212-2, or the monoacylglycerol lipase inhibitor URB602.

187 se inhibitor) and JZL184 (a monoacylglycerol lipase inhibitor).

188 B1R antagonist, AM251, or the diacylglycerol lipase inhibitor, DO34.

189 ortical neurons, suggesting that promiscuous lipase inhibitors have the potential to cause metabolic

190 ty acid amide hydrolase and monoacylglycerol lipase inhibitors in paclitaxel-treated mice.

191 Japan including sympathomimetics, pancreatic lipase inhibitors, GABAA receptor activators, a serotoni

192 xidant activity whereas a negative impact on lipase inhibitory activity was observed (p<.0001).

193 Lysosomal acid lipase is an essential lipid-metabolizing enzyme that br

194 zymes are required, only one enzyme, namely, lipase is required in our sensor.

195 An apoplastic GDSL-lipase known as CUTIN SYNTHASE1 (CUS1) is required for c

196 Of note, lysosomal acid lipase (LAL) deficiency facilitates melanoma growth and

197 Lysosomal acid lipase (LAL) is essential for the clearance of endocytos

198 Lysosomal acid lipase (LAL), a key enzyme in the metabolic pathway of n

199 ffect of lalistat, a specific lysosomal acid lipase (LAL/Lipa) inhibitor on LD degradation in HSCs du

200 [5%]), abdominal pain (13; 4%), and elevated lipase level (15; 5%), and in the nilotinib group were a

201 otinib group were anaemia (18; 6%), elevated lipase level (15; 5%), elevated alanine aminotransferase

202 reaction (four [2%] patients) and increased lipase level (three [2%]).

203 PEP), defined by new upper-abdominal pain, a lipase level more than 3-fold the upper limit of normal,

204 ge, 30-100 U/L [0.50-1.66 mukat/L]), a serum lipase level of 391 U/L (6.52 mukat/L) (normal range, 13

205 utations in the gene encoding lysosomal acid lipase (LIPA) that result in reduced or absent activity

206 ntenance of AT; deficiency in lysosomal acid lipase (Lipa), the enzyme required for lysosome lipid ca

207 s: albumin (ALB) in liver carcinoma, gastric lipase (LIPF) in stomach carcinoma, and thyroglobulin (T

208 Ser mutation (rs77960347) in the endothelial lipase (LIPG) gene, occurring with an allele frequency o

209 sion of lipolytic enzymes (hormone-sensitive lipase, lipoprotein lipase, and fatty acid binding prote

210 Angptl4 inhibits extracellular lipoprotein lipase (LPL) activity and stimulates the lipolysis of tr

211 proteins to remnant particles by lipoprotein lipase (LPL) and their uptake by the liver.

212 I (apoC-II) is the co-factor for lipoprotein lipase (LPL) at the surface of triacylglycerol-rich lipo

213 membrane protein that transports lipoprotein lipase (LPL) from the subendothelial space to the lumina

214 tigated whether lipid uptake via lipoprotein lipase (LPL) in astrocytes is required to centrally regu

215 Lipoprotein lipase (LPL) undergoes spontaneous inactivation via glob

216 oactivator 1beta (PGC1beta), and lipoprotein lipase (LPL) were among the up-regulated genes identifie

217 ary endothelial cells that binds lipoprotein lipase (LPL) within the interstitial space and shuttles

218 wered plasma TGs in mice lacking lipoprotein lipase (LPL), hepatic heparan sulfate proteoglycan (HSPG

219 m of action, and relation to the lipoprotein lipase (LPL), however, remain elusive.

220 temic pretreatment with the monoacylglycerol lipase (MAGL) inhibitor MJN110 (which selectively elevat

221 Monoacylglycerol lipase (MAGL) inhibitors are considered potential therap

222 Monoacylglycerol lipase (MAGL) represents a primary degradation enzyme of

223 cid amide hydrolase (FAAH), monoacylglycerol lipase (MAGL), N-acylethanolamine acid amidase (NAAA), o

224 nt actions of inhibitors of monoacylglycerol lipase (MAGL), the major degradative enzyme of the endoc

225 AG is degraded primarily by monoacylglycerol lipase (MAGL), which is expressed in neurons and astrocy

226 Rhizomucor miehei lipase mediated-hydrolysis of sardine oil was conducted

227 erived fatty acids, generated by lipoprotein lipase-mediated hydrolysis of triglycerides, rather than

228 ecreted protein and inhibitor of lipoprotein lipase-mediated plasma triglyceride clearance.

229 6 (intracellular WWL70) and monoacylglycerol lipase MGL (JZL184) or by blocking GABAergic inhibition

230 The serine hydrolase monoacylglycerol lipase (MGL) functions as the main metabolizing enzyme o

231 a potent beta-lactam-based monoacylglycerol lipase (MGL) inhibitor characterized by an irreversible

232 Monoglyceride lipase (MGL) is required for efficient hydrolysis of the

233 The function of monoacylglycerol lipase (MGL), a key actor in the hydrolytic deactivation

234 nal orthologue of mammalian monoacylglycerol lipase (MGL), contributes >90% of cellular FAEE hydrolas

235 armacological inhibition of monoacylglycerol lipase (MGL)-a lipid hydrolase that degrades 2-AG in pre

236 Monoglyceride lipase (MGLL) expression was analyzed by quantitative RT

237 does not contain any of the acyl binding or lipase motifs that are present in other studied acyltran

238 and pancreatitis, hypocalcemia, and elevated lipase (n = 1; all in same patient) for sunitinib and th

239 atment-related adverse events were increased lipase (none vs 5 [8%] vs none) and diarrhoea (none vs 3

240 with poly(butylene succinate) (PBS) and the lipase of Rhizopus oryzae (RoL), we detected complete hy

241 l4p, and Tgl5p are the major triacylglycerol lipases of the yeast Saccharomyces cerevisiae Recently w

242 rgy storage by orchestrating the activity of lipases on the surface of lipid droplets.

243 ajor 2-AG synthesizing enzyme diacylglycerol lipase or blockade of CB1 receptors abolished the facili

244 e in SUGAR-DEPENDENT1 (SDP1) triacylglycerol lipase or PEROXISOMAL ABC TRANSPORTER 1 (PXA1), here we

245 in receptor binding or provide the protease, lipase, or esterase activity required for entry of the v

246 The stability is confirmed by lipase p-nitrophenyl palmitate (PNP) assay.

247 Pancreatic lipase (PL) plays a central role in fat metabolism and i

248 Furthermore, adipose triglyceride lipase, plasma membrane-associated fatty acid binding pr

249 an Arabidopsis thylakoid membrane-associated lipase, PLASTID LIPASE1 (PLIP1).

250 Pancreatic triglyceride lipase (PNLIP) is essential for dietary fat digestion in

251 ty acid amide hydrolase and monoacylglycerol lipase produce reliable antinociceptive effects, and off

252 polysis in mice lacking adipose triglyceride lipase provokes severe cardiac steatosis and heart dysfu

253 D family members and the structural basis of lipase regulation.

254 en and adults, whereas a homolog, pancreatic lipase-related protein 2 (PNLIPRP2), is critical in newb

255 Cytoplasmic lipases removed FAs from LDs, enabling their transfer in

256 ce of xylanase, alpha-amylase, cellulase and lipase resulted in bread with greater quantities of DON-

257 intestinal transit time, presence of gastric lipase, sample/digestive fluids ratio, concentration and

258 part of a three-gene cluster also encoding a lipase (SBP) and a Nod-like receptor, both of which disp

259 The immobilised lipase showed the highest specificity towards short-chai

260 erse events were increased concentrations of lipase (six [8%]) and amylase (two [3%]).

261 ansesterification catalyzed by the versatile lipase/sterol esterase from the ascomycete fungus O. pic

262 %] in the reverse sequence group), increased lipase (ten [15%] vs 12 [17%]), and diarrhoea (eight [12

263 The lipases Tgl3 and Tgl4 are required for efficient cell-cy

264 tions to the two other yeast triacylglycerol lipases, Tgl4p and Tgl5p.

265 ures, this trend was more pronounced for the lipase than the cutinase.

266 Mil1 is a predicted lipase that binds Apm2 but not Apm1 and contributes to i

267 lated from oil-contaminated soil contained a lipase that was stable at varying pH and in various solv

268 ysis revealed that the drug inhibits several lipases that are not targeted by PF04457845, a highly se

269 Because the lipases that catabolize LDs in hepatocytes reside on the

270 rovides the necessary glycolytic enzymes and lipases that process lipid and glycolipid antigens, as w

271 ents related to nivolumab included increased lipase (three [1%] of 268 patients), increased alanine a

272 atment-related adverse events were increased lipase (three [8%] and no patients), pneumonitis (two [5

273 H), a plant homolog of yeast Triacylglycerol lipase (TLG2/SYP4) interacting protein Tgl2-Vesicle Prot

274 mance of the reactor, and the ability of the lipase to alter free fatty acid composition and sensory

275 Lipozyme 435 was the most efficient lipase to catalyze the transesterification reaction.

276 ally abundant and sustainable Candida rugose lipase to ordered-assemble into nanoparticles with high

277 HELLP targets the SBP lipase to the membrane, suggesting a synergy between HEL

278 up, their CE core is hydrolyzed by liposomal lipases to generate free cholesterol (FC).

279 hen shifted from the enzymatic activation of lipases to substrate accessibility, mediated by perilipi

280 o report an as-yet-undescribed mechanism for lipase transport in eukaryotic cells, with SDP1 being fi

281 e functional effects of lipids isolated from lipase-treated wheat dough.

282 e been investigated using Candida antarctica lipase type B as biocatalyst.

283 nd diacylglycerols (DATEM, used as control), lipase use did not impact dough extensibility.

284 The lipase was immobilised on hydrophobic resin (Toyopearl(R

285 The lipase was stable when immobilised and there was no sign

286 Thermomyces lanuginosus (TL 100L) lipase was used to partially remove DPA-n6, further conc

287 nzymes (alpha-amylase, alpha-glucosidase and lipase) was evaluated.

288 Using immobilized lipases we successfully acylated A. tequilana fructans w

289 ANGPTL4 inhibits lipoprotein lipase; we therefore searched for mutations in LPL and i

290 vities of T. terrestris and chickpea against lipase were 15.3 +/- 2.03 and 9.74 +/- 1.09 mug/ml, resp

291 L) nor phosphorylations of hormone-sensitive lipase were altered.

292 lein- and arginine-induced serum amylase and lipase were significantly higher in panc-PTP1B KO mice c

293 ble inhibitor of the enzyme monoacylglycerol lipase, which accounts for 85% of the 2-AG degradation i

294 ble inhibitor of the enzyme monoacylglycerol lipase, which accounts for 85% of the 2-arachidonoylglyc

295 nd 4) is metabolized by either monoglyceride lipase, which is located in the inhibitory axon terminal

296 ospholipase A-Igamma3 (At1g51440), a plastid lipase with a high substrate preference for MGDG, and is

297 s study demonstrates that ABH is an esterase/lipase with catalytic Ser-His-Asp triad.

298 des an explanation for absence of reports of lipases with omega-3 fatty acid hydrolyzing ability and

299 However, no lipases with preference for omega-3 fatty acids selectiv

300 The yeast lipase YLLIP2 is stable and has high levels of activity