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1 effects on postprandial vascular function or lipemia.
2 on of GLP-1 and GLP-2 decreased postprandial lipemia.
3 activity resulted in decreased postprandial lipemia.
4 ic acid (16:0) in the sn-2 position decrease lipemia.
5 of saturated fats may influence postprandial lipemia.
6 exposure, resulting in elevated postprandial lipemia.
7 s a primary role in determining postprandial lipemia.
8 en milk protein and milk fat on postprandial lipemia.
9 ent and an attenuated impact on postprandial lipemia.
10 ized SSOB resulted in 53% lower postprandial lipemia, 23% higher hepatic lipase activity, and a 25% l
13 cturing dietary fat to modulate postprandial lipemia and lipid beta-oxidation in humans with differen
15 understanding the links between postprandial lipemia and the accumulation of lipid within vessels, a
16 t information on the control of postprandial lipemia and the biological effects of chylomicron remnan
17 ucoregulatory function, greater postprandial lipemia, and greater lipid oxidation rates than do their
19 ipoprotein particles; increased postprandial lipemia; and abnormal apolipoprotein A1 and B metabolism
20 dominal obesity and exaggerated postprandial lipemia are independent risk factors for cardiovascular
22 d release (bioaccessibility) on postprandial lipemia by comparing lipid encapsulated by cell walls wi
23 erance (characterized by higher postprandial lipemia, cholesterol enrichment of triglyceride-rich lip
24 ts on postprandial fibrinolytic activity and lipemia, factor VII coagulant (FVII:c) activity, and act
25 se Western dietary habits cause postprandial lipemia for a major part of a day and, moreover, increas
30 impact of the cheese matrix on postprandial lipemia in humans has not yet been evaluated.In healthy
31 sma lipids and lipoproteins and postprandial lipemia in premenopausal African American and white wome
34 experimental data suggest that postprandial lipemia is an independent risk factor for atherosclerosi
36 d in glucomannan or glucomannan-spirulina on lipemia, liver glutathione status, antioxidant enzymes a
38 of TM6SF2 in the regulation of postprandial lipemia, potentially through a similar function for TM6S
40 A compared with DHA only during postprandial lipemia relative to control high-oleic acid meals; the s
41 in may explain the reduction in postprandial lipemia seen in clinical trials of this agent and may pr
42 not associated with fasting or postprandial lipemia test triacylglycerol or free fatty acids (P > or
43 ubgroup (n = 36) also underwent postprandial lipemia tests with lipid oxidation rate measurements.
44 ke had more dramatic effects on postprandial lipemia than did MSF after fat intake, possibly because
45 agents also improve fasting and postprandial lipemia, the latter more significantly than the former.
47 oviral therapy predict enhanced postprandial lipemia, which is an emerging cardiovascular disease ris
48 , we tested the hypothesis that postprandial lipemia with its characteristic elevation of triglycerid
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