ライフサイエンスコーパス: lipid

1 nsport a glycosylated diphosphate isoprenoid lipid.

2 date adjuvanted with alum and monophosphoryl lipid A (MPL), blockade Ab titers peaked early, with no

3 xed with liposomes containing monophosphoryl lipid A as an adjuvant.

4 ucture analysis and to produce a mimetic Kdo-lipid A domain AlmG substrate to that synthesized by V.

5 nt of a minimal keto-deoxyoctulosonate (Kdo)-lipid A domain in E. coli was necessary to facilitate ch

6 pid A substitution is seen in the absence of lipid A glycinylation.

7 Here we report a second lipid A modification mechanism that only functions in th

8 no-residue phosphoethanolamine (pEtN) to the lipid A of V. cholerae El Tor that is not functional in

9 Similarly, efficient pEtN lipid A substitution is seen in the absence of lipid A g

10 Chronic alcohol feeding causes lipid accumulation and apoptosis in the liver.

11 n of bta-miR-23a by its inhibitors increased lipid accumulation and expression of C/EBPalpha, PPARgam

12 AnkB variants that fail to restore normal lipid accumulation and GLUT4 localization in adipocytes

13 but markedly attenuated the obesity-induced lipid accumulation in the kidney and renal dysfunction,

14 tary fat overconsumption leads to myocardial lipid accumulation through mechanisms that are incomplet

15 e in mice alleviated fasting-induced hepatic lipid accumulation.

16 During inflammation, proteins and lipids act in a concerted fashion, calling for combined

17 Protein or lipid addition had no influence on the formation and bio

18 ant products, including a series of phenolic lipids (alkylresorcinols) found only at appreciable conc

19 inability to switch between the oxidation of lipid and carbohydrate appears to be an important featur

20 Dysregulated lipid and carbohydrate metabolism resulting from insulin

21 in the maintenance of proper apical membrane lipid and cell wall composition is further supported by

22 on between CETP and HMGCR scores, changes in lipid and lipoprotein levels, and the risk of cardiovasc

23 istance against oxidative modification, main lipid and protein composition, and size distribution).

24 utophosphorylation, H917A abolished both the lipid and protein kinase activities of PI3Kalpha.

25 , depends strongly on the presence of acidic lipids and is enhanced by cholesterol (Chol).

26 zed metabolic tissue that takes up and burns lipids and is linked to systemic metabolic homeostasis,

27 ay change the molecular organization of skin lipids and proteins, which may in turn alter the protect

28 nguish between molecular components, such as lipids and proteins.

29 association data from a large-scale GWAS of lipids and show that these improvements are largely sust

30 ent, but also with zonal changes of specific lipids and their associated metabolic pathways.

31 red the temporal and spatial compositions of lipids and transcriptomes for two oil yielding organs me

32 available, reduced organic compounds such as lipids and waxes from decomposition.

33 These results demonstrating polar-lipid, and specifically phospholipid, inhibition of prio

34 lts, high blood pressure (BP), adverse serum lipids, and smoking associate with cognitive deficits.

35 The nature of endogenous or environmental lipid antigens encountered by iNKT cells is not well def

36 llowed by direct biochemical analysis of the lipid antigens trapped at the TCR-CD1d interface.

37 We developed a method to directly capture lipid antigens within CD1d-lipid-TCR complexes, while ex

38 meostasis through effects on CD1d-associated lipid antigens.

39 The present work explored the lipid antioxidant capacity of citrus pectin addition to

40 d basis set consisting of macromolecules and lipids apart from metabolites of interest, was used for

41 alyses demonstrated abnormal stratum corneum lipid architecture in AD and IV HEEs, independent of FLG

42 dolipids or lipopentapeptide (L5P), yet both lipids are present in other M. avium subspecies.

43 ity and glucose tolerance; as a class, these lipids are referred to as 'lipokines'.

44 f how it functions and its interactions with lipids are unknown or limited due to inability of obtain

45 The effects of amino acid replacements on lipid association of the C-terminal peptide fully recapi

46 reactions from the cytosol, analogous to the lipid-based membrane of eukaryotic organelles.

47 te acute malnutrition (MAM) are treated with lipid-based nutrient supplement (LNS) or corn-soy blend

48 andomised controlled trial of small-quantity lipid-based nutrient supplements (SQ-LNS) fortified with

49 embly strategies leading to the emergence of lipid bicontinuous single crystals with unprecedented sw

50 t-simulating an entire mammal red blood cell lipid bilayer and cytoskeleton as modeled by multiple mi

51 require membrane protein reconstitution in a lipid bilayer at high concentrations.

52 ed composition, to quantify the influence of lipid bilayer composition on protein-glycolipid binding

53 verse structure and regulated deformation of lipid bilayer membranes are among a cell's most fascinat

54 and amplification of chemical signals across lipid bilayer membranes is of profound significance in m

55 It also indicates that the lipid bilayer modulates channel gating, although it is n

56 release of the protein precursor within the lipid bilayer of the inner membrane, followed by cleavag

57 using approaches that include the supported lipid bilayer platform as well as DNA tension sensor tec

58 howed that the peptide was on the surface of lipid bilayer regardless of the charged lipid ratio.

59 Nanodiscs that hold a lipid bilayer surrounded by a boundary of scaffold prote

60 re matches the polar/apolar interface of the lipid bilayer very well.

61 specifically targeting phospholipids in the lipid bilayer via the production of singlet oxygen ((1)O

62 sed it to study vesicle fusion to a tethered lipid bilayer.

63 m and is connected to its neighbor through a lipid bilayer.

64 n and, uniquely, physical transformations of lipid bilayers can be monitored on a length scale of mic

65 vidual bacteriorhodopsin molecules in native lipid bilayers.

66 ngle RyR1 channels reconstituted into planar lipid bilayers.

67 lic particles smaller than 6 nm can embed in lipid bilayers.

68 Lipid binding by purified MpPR-1 occurs with micromolar

69 rstanding the interaction between SWCNTs and lipid biocorona.

70 mutants defective in AM-specific paralogs of lipid biosynthesis genes (KASI and GPAT6).

71 important gene contributing hybrid vigor in lipid biosynthesis in oil palm.

72 derived sugar provides carbon skeletons for lipid biosynthesis.

73 ome-wide transcriptome analysis reveals that lipid biosynthetic enzymes are among the downstream targ

74 e importance of reactivation of AR-regulated lipid biosynthetic pathways in driving CRPC progression,

75 may be therapeutically enhanced by blocking lipid biosynthetic pathways.

76 BP, serum lipids, body mass index, and smoking were assessed in al

77 Lipid burden was assessed as the lipid core burden index

78 s critical in elucidating the roles of these lipids, but these studies were performed with racemic mi

79 determined by the amount of water, proteins, lipids, carbohydrates and nucleic acids present in a cel

80 s homeostatic control through consumption of lipids, carbohydrates, and amino acids, as well as gover

81 e hypothesis that the mitochondrion-specific lipid cardiolipin functions as a first contact site for

82 rinsic adjuvant activity of the accompanying lipid cargo could be a general essential feature of the

83 ug delivery system comprising nanostructured lipid carrier (NLCs) within liposomes (Lip-NLCs).

84 providing insight into the mechanism of host lipid catabolism by an M. tuberculosis enzyme, augmentin

85 The lipids cholesteryl hemisuccinate, linoleamide/oleamide,

86 al prion protein (PrP(C)) in lipid rafts and lipid cofactors generating infectious prions in in vitro

87 n and trough viscosity values of the amylose-lipid complexed starches were significantly lower than t

88 Injection of Cas9-guide RNA-lipid complexes targeting the Tmc1(Bth) allele into the

89 For the cooked starch/lipid complexes, more profound effect was evident (22.2-

90 nocarriers consists of either a polymer or a lipid component along with other excipients to stabilize

91 asize that spatial determination of the host lipid components of the immune response is crucial to id

92 The LAURDAN spectrum is sensitive to the lipid composition and dipolar relaxation arising from wa

93 ng from water penetration, but disentangling lipid composition from membrane fluidity can be obtained

94 A regime of lipid composition is found where all properties are favo

95 levated sphingolipid turnover and an altered lipid composition of both MAM and mitochondrial membrane

96 tolerogenic, induce different changes in the lipid composition of cultured CD11c(+) cells, and highli

97 with significant alterations in steady-state lipid composition of producer cells and HIV particles.

98 Membrane lipid composition varies greatly within submembrane comp

99 tive abundances of these IPLs and their core lipid compositions differ systematically between the phy

100 cisely controlling exposure to vesicles with lipid compositions that mimic both bacterial and mammali

101 te in the presence of membranes; conversely, lipid conformation and packing can adapt to the presence

102 The lipid-conjugated prodrug of cidofovir, brincidofovir, ha

103 myloid formation highlight the diverse roles lipid constituents may play in the prion conversion proc

104 ebrafish in order to explore how the dietary lipid content may influence the gut microbiome.

105 Lipid burden was assessed as the lipid core burden index (LCBI), and large LRP were defin

106 of hepatic mTOR in Am mice increased hepatic lipid deposition and HIRI.

107 D11d deficiency led to a marked reduction in lipid deposition in aortas and isolated macrophages.

108 orted, no obvious fingerprint degradation or lipid diffusion is observed with either glass or stainle

109 ABSTRACT: Because the lipid droplet (LD)-associated perilipin (PLIN) proteins

110 doplasmic reticulum membrane morphology, and lipid droplet formation, but not on growth at elevated t

111 y disrupted lipolysis without affecting ATGL lipid droplet translocation or ABHD5 interactions with p

112 Cytoplasmic lipid droplets (LDs) of neutral lipids (triacylglycerols

113 ers in close proximity to the apical side of lipid droplets (LDs).

114 umulation of neutral lipids in intracellular lipid droplets has been associated with the formation an

115 ed with a decreased accumulation of TAGs and lipid droplets.

116 ctural proteins (e.g. collagen, elastin) and lipids (e.g. foam cells, extracellular lipids) in the fi

117 yocellular lipid (IMCL) and extramyocellular lipid (EMCL) content in obesity, we utilized a new four-

118 n erythrocytes 14 d after the test meals.The lipid emulsion given either before or with the meal sign

119 NAFLD is associated not only with lipid enrichment, but also with zonal changes of specifi

120 nt mice lacked a functional corneocyte-bound lipid envelope leading to a severe skin barrier defect a

121 heir high density, the headgroups of anionic lipids experience electrostatic repulsion that, being ex

122 This cooperative lipid extraction mechanism for membrane perforation repr

123 The other motif peptides do not induce lipid flip-flop, suggesting an alternate mechanism.

124 , while excluding CD1d bound to nonantigenic lipids, followed by direct biochemical analysis of the l

125 constitute the roe mass and microencapsulate lipid fraction, so that small oil droplets are entrapped

126 results are reported per SD increase of each lipid fraction.

127 Fasting lipid fractions (triglycerides [TGs], high-density lipop

128 zed by substantial reductions in all 3 major lipid fractions, is caused by mutations that inactivate

129 Total lipids from an M. avium subsp. paratuberculosis (Map) ov

130 al factories have been engineered to produce lipids from carbohydrate feedstocks for production of bi

131 In summary, C. neoformans harvests lipids from macrophages, and the C. neoformans-macrophag

132 Above and below the lipid gel-liquid crystalline temperature, partitioning i

133 er of different substrates (muscle proteins, lipids, glucose, DNA (satellite cells)) can be monitored

134 Recently, lipids have been identified that are released from tissu

135 Lipids have fundamental roles in the structure, energeti

136 en bonds between the 5-HT hydroxyl group and lipid headgroups and allows 5-HT to intercept reactive o

137 ied differential actions of ILP7 and ILP8 in lipid homeostasis and ovarian development.

138 2A or C2C failed to rescue two defects in PM lipid homeostasis observed in E-Syts KO cells, delayed d

139 Circadian clocks play an important role in lipid homeostasis, with impact on various metabolic dise

140 egulate the hepatic acute-phase response and lipid homeostasis.

141 is pathway may link outer membrane fusion to lipids homeostasis.

142 second messenger typically derived from the lipid-hydrolyzing activity of PLDalpha1, is a molecular

143 of G-protein signaling (RGS1) protein and a lipid-hydrolyzing enzyme, phospholipase Dalpha1 (PLDalph

144 py-resistant cell state are dependent on the lipid hydroperoxidase GPX4 for survival.

145 As lipid identification remains a bottleneck of modern unta

146 s ranking algorithm were comparable to other lipid identification software annotations, MS-DIAL and G

147 The lipid identifications with the greatest summed intensity

148 exhibit similar changes in intramyocellular lipid (IMCL) and extramyocellular lipid (EMCL) content i

149 e primary mechanism of excessive myocellular lipid import.

150 s shown the full potential of mapping intact lipids in biological systems with better than 10 mum lat

151 nflammation mediators including proteins and lipids in human fibroblasts upon inflammatory stimulatio

152 The accumulation of neutral lipids in intracellular lipid droplets has been associat

153 we study and quantify the molecular order of lipids in myelin at subdiffraction scales, using label-f

154 ane-spanning furrow that provides a path for lipids in scramblases has changed to form an enclosed aq

155 fferentiated into adipocytes and accumulated lipids in the cytoplasm when cultured with butyric acid,

156 in to PG is approximately 1:1 if only the PG lipids in the outer leaflets of membranes are accessible

157 the equally important functions of cellular lipids in virus replication have been gaining full atten

158 ) and lipids (e.g. foam cells, extracellular lipids) in the first 200 mum of the intima provide impor

159 by astrocytes and functions in transporting lipids including cholesterol to support neuronal homeost

160 a provide a wealth of information on protein-lipid interactions for a key protein in photosynthesis.

161 NCE Prion conversion is likely influenced by lipid interactions, given the location of normal prion p

162 ilic groups play important roles in compound-lipid interactions.

163 EIEIO involves irradiating singly charged lipid ions with electrons having kinetic energies of 5-1

164 Although H936A inactivated the lipid kinase activity without affecting autophosphorylat

165 PI3KC3-C1 consists of the lipid kinase catalytic subunit VPS34, the VPS15 scaffold

166 The lipid kinase phosphatidylinositol 4-kinase IIIbeta (PI4K

167 ly (lactic-co-glycolic acid) (PLGA) core and lipid layer containing docetaxel and clinically used inh

168 Diets containing three different lipid levels (high [HFD], medium [MFD], and low [LFD]) w

169 Despite enhanced weight gain and plasma lipid levels compared with Apoe(-/-) controls, EphA2(-/-

170 o, tofacitinib was associated with increased lipid levels.

171 Lipid ligands of G2A were found in increased concentrati

172 %) of patients who reported currently taking lipid-lowering medication, full implementation of the US

173 examining potential benefits of combination lipid-lowering therapy in individuals with CKD are neede

174 cytes, resulting in villous degeneration and lipid malabsorption.

175 sitol 4,5-bisphosphate (PI(4,5)P2), the main lipid marker of the plasma membrane.

176 Nonlaboratory-based risk factors and lipids measured in adolescence independently predicted p

177 pproach that additionally incorporates adult lipid measures.

178 on days 3 and 7 thereafter and subjected to lipid mediator profiling.

179 n of the downstream specialized proresolving lipid mediators (SPMs) 14-hydroxydocosahexaenoic acid, 1

180 into the as yet unrevealed action of PFOS on lipid mediators in affecting testicular functions.

181 Analyses of lipid mediators revealed increased arachidonic acid and

182 Resolvins, a class of lipid mediators, counteract this effect, representing an

183 ptide:lipid ratio, and the properties of the lipid membrane.

184 positions and orientations in the different lipid membranes (DOPC for the liquid disordered phase an

185 sorbent dilution series with solid supported lipid membranes (SSLMs).

186 o investigate peptide and protein binding to lipid membranes, as it allows for very low amounts of sa

187 ate topology and dimensions to interact with lipid membranes.

188 changes that impact hepatic inflammatory and lipid metabolic pathways, providing new insight into the

189 r, imparting sex differences in hepatic drug/lipid metabolism and disease risk.

190 tions are observed in vesicular trafficking, lipid metabolism and in the endoplasmic reticulum that c

191 lism.HDAC3 is a critical mediator of hepatic lipid metabolism and its loss leads to fatty liver.

192 tion factor EB (TFEB), a master regulator of lipid metabolism and lysosomal biogenesis and function.

193 Among these, lipid metabolism and mitochondrial dysfunction proteins

194 y unidentified link between O-GlcNAcylation, lipid metabolism and the regulation of SREBP-1 in cancer

195 s exposure induced disturbance of energy and lipid metabolism as well as oxidative stress.

196 nity and further suggest that alterations in lipid metabolism may affect iNKT cell homeostasis throug

197 urrent changes in the expression of genes in lipid metabolism pathways.

198 hondrial function, autophagy, ER stress, and lipid metabolism were measured in pancreatic tissue, aci

199 nscript and metabolite subnetworks linked to lipid metabolism, inflammation and glycerophospholipid m

200 in transducing nutritional state to regulate lipid metabolism.

201 elated to nucleotide-dependent processes and lipid metabolism.

202 This study was designed to evaluate serum lipid metabolite changes that are associated with the pr

203 Lipid metabolite oscillations were strongly attenuated u

204 hat orally ingested UFP promoted atherogenic lipid metabolites in both the intestine and plasma via a

205 Lipid metabolites may partially explain the inverse asso

206 sition, accompanied by increased atherogenic lipid metabolites.

207 In addition, the 10-lipid mixture had direct bactericidal effects against St

208 tochastic neighbor embedding of peaks in the lipid molecular mass range (m/z 700-850) distinguishes s

209 iously observed effect of temperature on the lipids' molecular dynamics and inducing an ordering effe

210 Partitioning of lipid molecules in biomimetic membranes is a model syste

211 Lipid nanodiscs are playing increasingly important roles

212 Using lipid nanoparticle formulations of these enhanced sgRNAs

213 MS configuration where ozonolysis of ionized lipids occurred rapidly (10 ms) without prior mass-selec

214 s confined to these lipids, while plastidial lipids of prokaryotic type were characterized by the ove

215 (MTP), a protein involved in the transfer of lipids onto CD1d, regulates liver iNKT cell homeostasis

216 ing, the M2AH induces membrane curvature and lipid ordering, constricting and destabilizing the membr

217 increased the redox potential, promoted the lipid oxidation and elevating the hue color of the morta

218 ining compounds and acetaldehyde, as well as lipid oxidation derived odorants to the overall odor of

219 oinflammatory cytokines, and end products of lipid oxidation had a synergistic effect on TLR2 activat

220 OxyHb promoted lipid oxidation in washed muscle more effectively compar

221 than in Controls, suggesting an increase in lipid oxidation.

222 The intrinsic amphipathic lipid packaging sensor (ALPS) motif within HOPS Vps41, a

223 on the relative proportions of four dominant lipids, PC(32:0), PC(34:1), PC(36:1), and PC(38:5).

224 link between peptide pore formation and both lipid-peptide charge and topological interactions.

225 We explore the complexity of the lipid-peptide interactions governing membrane-disruptive

226 solute scale of inhibition properties of the lipid peroxidation can be devised.

227 egarding the mechanisms of inhibition of the lipid peroxidation in micelles, in view of bibliographic

228 We used it to probe the importance of lipid peroxidation in progression of NASH beyond simple

229 light and is photoprotective, as it reduces lipid peroxidation levels.

230 pha does not abolish their responsiveness to lipid perturbation.

231 calization uncover a novel mechanism whereby lipid phosphatase activity in the nucleus can regulate m

232 From this correlation, we hypothesize that lipids play a so far unrecognized role in organ developm

233 Lipids play central roles in metabolism and cell signali

234 We developed core shell lipid-polymer hybrid nanoparticles (CSLPHNPs) with poly

235 he impacts of CerC16 (up to 20 mol %) on the lipid polymorphism of 1-palmitoyl-2-oleoyl-sn-glycero-3-

236 Lipid profile levels were statistically significant on c

237 sment of insulin resistance, serum ferritin, lipid profile, and liver function tests improved irrespe

238 lysis of variance was used for comparison of lipid profile, whereas Kruskal-Wallis test was used for

239 Lipid profiles of fatm and ram2 suggested that FatM incr

240 starch with non-starch components, including lipids, protein, dietary fibre, phenolics, and minerals,

241 ian chia seeds showed high concentrations of lipids, proteins, total dietary fiber, minerals and vita

242 ophage interaction is modulated by exogenous lipids, providing a new tool for studying nonlytic exocy

243 location of normal prion protein (PrP(C)) in lipid rafts and lipid cofactors generating infectious pr

244 s we show that clustering of gangliosides in lipid rafts is important.

245 llactose-containing gangliosides enriched in lipid rafts to inhibit raft-dependent PI3K signaling.

246 To explain why klotho preferentially targets lipid rafts we show that clustering of gangliosides in l

247 The association of lipid rafts with HCV-induced autophagosomes was confirme

248 e dissolution of ordered microdomains (i.e., lipid rafts).

249 as well as on their lateral accumulation in lipid rafts.

250 ding on the peptide's structure, the peptide:lipid ratio, and the properties of the lipid membrane.

251 e of lipid bilayer regardless of the charged lipid ratio.

252 Thus, we have identified sulfatide as a self-lipid recognized by human iNKT cells and propose that su

253 y have recently been shown to be involved in lipid remodeling and scavenging during replication.

254 rotein precipitation and dichloromethane for lipid removal, was developed to detect and quantify as m

255 signaling inactivation causes exhaustion of lipid reserves in somatic tissues.

256 orbic acid and accumulation of cytosolic and lipid ROS.

257 polar couplings (RDCs) for natural abundance lipid samples to obtain segmental SCH order parameters.

258 erican Heart Association (ACC/AHA) recommend lipid screening in all adults older than 20 years to ide

259 Hybrid lipids show no enrichment at the boundary, but decrease

260 anionic lipids with defined head groups and lipid side chains.

261 hanism was shown to be interference with the lipid signaling pathway, leading to reduced expression o

262 ty is central to PITP-mediated regulation of lipid signaling.

263 species (ROS) can damage DNA, proteins, and lipids, so cells have antioxidant systems that regulate

264 Six lipid species (all belonging to the triacylglycerol clas

265 e allowed fast separation of both individual lipid species and categories.

266 es in these molecules; and 3) 1-y changes in lipid species and subsequent CVD.With the use of a case-

267 VD).We evaluated the associations between 1) lipid species and the risk of CVD (myocardial infarction

268 e lipidomics field, more structurally unique lipid species are detected and annotated.

269 use of a case-cohort design, we profiled 202 lipid species at baseline and after 1 y of intervention

270 anched-chain amino acids, select unsaturated lipid species, and trimethylamine-N-oxide), thus in effe

271 bution with LDs may be linked to the diverse lipid storage in muscle between trained and sedentary in

272 ated fat ingestion rapidly increases hepatic lipid storage, energy metabolism, and insulin resistance

273 second harvest of the Very Large Database of Lipids study to assess for the first time the impact of

274 entified a biochemical toolbox that includes lipid substrates for enzymatic assays, potent inhibitors

275 s free fatty acids, metabolites, and complex lipids such as ceramides, glycerophosphoglycerols, cardi

276 nt hydrophobic surfaces as well as insoluble lipid surfactants, including phospholipids, and proteins

277 ized via glycerol production and release and lipid synthesis (particularly FFA, triglycerides, and ch

278 etal muscle and the role that ACSL6 plays in lipid synthesis in both rodent and human skeletal muscle

279 INO mutation suppressed pah1Delta effects on lipid synthesis, nuclear/endoplasmic reticulum membrane

280 as constructed for the single bilayer binary lipid system.

281 directly capture lipid antigens within CD1d-lipid-TCR complexes, while excluding CD1d bound to nonan

282 is a key regulator of intracellular neutral lipids that has been recently identified as a tumor supp

283 Using a ternary mixture of lipids, the modulated-intensity profile of elastic back-

284 ter diameter, can be produced by varying the lipid-to-polymer ratio.

285 Acyl position in structural and storage lipids together with acyl-CoA analysis further help to d

286 iants are likely to mediate their effects on lipid traits by regulating gene expression.

287 In the Mediterranean area, lipid transfer proteins (LTPs) are important causes of p

288 ansport between the two bilayers mediated by lipid transfer proteins.

289 The details in subsequent lipid-transfer process, however, have not yet been compl

290 n the role of ER-PM contacts in nonvesicular lipid transport between the two bilayers mediated by lip

291 Cytoplasmic lipid droplets (LDs) of neutral lipids (triacylglycerols [TAGs], sterylesters, etc.) are

292 ncreases the expression of genes involved in lipid turnover (ACADM) and insulin signalling (IRS2) in

293 e conclude that morning fasting up-regulates lipid turnover genes in SCAT of lean individuals.

294 ced CD36 translocation and further increased lipid uptake.

295 Sugar metabolism and lipid utilization are linked to the spatiotemporal seque

296 Total extracted lipid values on roe weight were 5.8-6.3%.

297 leoyl-sn-glycero-3-phosphocholine (DC18:1PC) lipid vesicles using a fluorescence assay for gA channel

298 DHA was confined to these lipids, while plastidial lipids of prokaryotic type were

299 nchor binds selected plasma membrane anionic lipids with defined head groups and lipid side chains.

300 /-2.9wt% and even more concentrated in polar lipids, with 1.4+/-0.2vs 8.5+/-1.1wt%.

301 , advanced extraction techniques showed good lipid yields and furthermore, the produced echium oil ha