ライフサイエンスコーパス: lipid peroxidation

1 methods (ORAC, TRAP, HORAC and inhibition of lipid peroxidation).

2 dopsis (Arabidopsis thaliana) leaves against lipid peroxidation.

3 eduction observed in tissue inflammation and lipid peroxidation.

4 e via reactions that are well precedented in lipid peroxidation.

5 one, endogenous reactive oxygen species, and lipid peroxidation.

6 rolysate was the most effective inhibitor of lipid peroxidation.

7 elevates hydrogen peroxide accumulation and lipid peroxidation.

8 le significance of PUFA ratios in biological lipid peroxidation.

9 s were found in the extent of their membrane lipid peroxidation.

10 n peroxide and malondialdehyde, a product of lipid peroxidation.

11 ty acids that may be caused by activation of lipid peroxidation.

12 emary varieties correlated with tolerance to lipid peroxidation.

13 s of WM integrity and peripheral measures of lipid peroxidation.

14 eatic graft monitoring membrane fluidity and lipid peroxidation.

15 the steatosis score, oil red-O staining, and lipid peroxidation.

16 0% and 2.0%, possibly via modifying membrane lipid peroxidation.

17 rry extract was the most potent inhibitor of lipid peroxidation.

18 the level of unsaturation indicated elevated lipid peroxidation.

19 sly formed mutagenic DNA adduct derived from lipid peroxidation.

20 alization in the hydrophobic region prevents lipid peroxidation.

21 lin also showed strong suppressive effect on lipid peroxidation.

22 uction of reactive oxygen species (ROS), and lipid peroxidation.

23 e also showed a strong suppressive effect on lipid peroxidation.

24 c nitro-radicals (ABTS, DPPH) and to inhibit lipid peroxidation.

25 aused by an increase in oxidative stress and lipid peroxidation.

26 inflammatory gene and protein expression and lipid peroxidation.

27 ited growth advantage with reduced levels of lipid peroxidation.

28 and levels of 4-hydroxynonenal, a marker of lipid peroxidation.

29 iovascular disease, which may be promoted by lipid peroxidation.

30 as the synthetic antioxidant BHT to inhibit lipid peroxidation.

31 protect mouse brains against Fe(2+)-induced lipid peroxidation.

32 used) was the governing factor for effective lipid peroxidation.

33 apoB100 degradation, given its promotion of lipid peroxidation.

34 ciated with a significant decrease in tissue lipid peroxidation.

35 es (ROS), were not sensitive to RNS-mediated lipid peroxidation.

36 tial mediator of the inflammation induced by lipid peroxidation.

37 tion of reactive oxygen species by reversing lipid peroxidation.

38 diate some of the proinflammatory effects of lipid peroxidation.

39 g of TPB extract/kg of TTB) displayed higher lipid peroxidation.

40 eactive oxygen species levels and associated lipid peroxidation.

41 fibrosis by removing fibrogenic products of lipid peroxidation.

42 ve stress and cell membrane damage caused by lipid peroxidation.

43 tion of nitric oxide, protein nitration, and lipid peroxidation.

44 totoxic aldehydes produced by metabolism and lipid peroxidation.

45 ancing vitamin E-mediated protection against lipid peroxidation.

46 70, presence of nitrotyrosine residues, and lipid peroxidation.

47 e iron could initiate carcinogenesis through lipid peroxidation.

48 icating a role for myeloperoxidase-dependent lipid peroxidation.

49 l accumulation), oxidative/nitrative stress (lipid peroxidation, 3-nitrotyrosine formation, and expre

50 ificantly increased ROS content (31-46%) and lipid peroxidation (30-47%), concomitant with decreased

51 KC), neutrophil infiltration (MPO activity), lipid peroxidation (4-HNE), and nitric oxide (iNOS) - we

52 ctor alpha: 64% +/- 24% increase; P < 0.05), lipid peroxidation (4-hydroxynonenal, measured by ELISA:

53 roguanine (8-NO2Gua)) as well as products of lipid peroxidation (8-iso-prostaglandin F2alpha (8-isoPF

54 ion of nontransferrin bound iron, markers of lipid peroxidation-8alpha-isoprostanes, protein oxidatio

55 . 14% of trained mice) and oxidative stress (lipid peroxidation, 9.1 +/- 1.4 vs. 5.2 +/- 0.9 mumol mg

56 Recent advances in our understanding of lipid peroxidation, a degenerative process that is belie

57 o acids, whereas the Flt3 inhibitor prevents lipid peroxidation, a key mechanism of glutamate-mediate

58 Lipid peroxidation, a major consequence of oxidative str

59 Lipid peroxidation, a secondary process that occurs duri

60 r study identified the novel end-products of lipid peroxidation, accumulating in circulation in hyper

61 depolarization, production of free radicals, lipid peroxidation, activation of phospholipase C, IP3 r

62 ibited a significant reducing power and anti-lipid peroxidation activities.

63 -l-cysteine, a potent antioxidant, abolished lipid peroxidation activity and ameliorated EAE in IFN-g

64 Importantly, "free" myelin debris and lipid peroxidation activity at CNS lesions was increased

65 e accumulation of critical concentrations of lipid peroxidation adducts during ALS progression leads

66 itopes (OSE), which are products of enhanced lipid peroxidation and a major target of innate natural

67 In general, AntiOxCINs derivatives prevented lipid peroxidation and acted as inhibitors of the mitoch

68 By measuring markers of lipid peroxidation and antioxidant capacity, we obtained

69 of two tropical fruit juices (FA and FB) on lipid peroxidation and antioxidant enzymes in rats.

70 es typical of neuronal damage with increased lipid peroxidation and cellular oxidant levels but no al

71 ratinocyte adhesion in culture and increases lipid peroxidation and cyclooxygenase-2 (COX-2) levels i

72 re investigated with ferrous salts to induce lipid peroxidation and diethylmaleate (DEM) to reduce GS

73 FA-AKI in mice associates with lipid peroxidation and downregulation of glutathione met

74 ng capability and reduced levels of membrane lipid peroxidation and electrolyte leakage under drought

75 25 mg nCeO2 L(-1), the roots showed enhanced lipid peroxidation and electrolyte leakage, while at 500

76 plants, as indicated by reduced ROS, lowered lipid peroxidation and enhanced photosynthesis.

77 agenic aldehyde, is produced endogenously by lipid peroxidation and exogenously by combustion of orga

78 age through reduction of leaf water loss and lipid peroxidation and increased expression of genes ass

79 and glutathione peroxidase (GPx), vitamin E, lipid peroxidation and liver enzymes in hyperlipidaemia

80 malondialdehyde (MDA) epitopes, products of lipid peroxidation and markers for enhanced oxidative st

81 Our observations suggest that lipid peroxidation and mitochondrial biogenesis are the

82 Our observations suggest that lipid peroxidation and mitochondrial biogenesis are the

83 Also, lipid peroxidation and mitochondrial dysfunction appeare

84 s were seen including elevated mitochondrial lipid peroxidation and mitochondrial membrane defects, a

85 MJ33 increased the levels of lipid peroxidation and mitochondrial O2(* horizontal lin

86 the chemical link between antioxidant load, lipid peroxidation and mitochondrial physiology.

87 Levels of lipid peroxidation and of superoxide anion (O2(* horizon

88 sults identify 5-HT as a potent inhibitor of lipid peroxidation and offer a different perspective on

89 lay reduced selenium levels, which may cause lipid peroxidation and oxidative stress because selenium

90 Lipid peroxidation and polar compounds formation in sunf

91 dismutase [ECSOD] mimetics), porphyrins, and lipid peroxidation and protein carbonylation blockers/in

92 Rice seedlings also exhibited severe lipid peroxidation and protein carbonylation, for oxidat

93 for cytoprotective activity on lymphocytes, lipid peroxidation and protein degradation.

94 ng sugars, sucrose, ethylene, ascorbic acid, lipid peroxidation and reactive oxygen species.

95 d that activation of SAT1 expression induces lipid peroxidation and sensitizes cells to undergo ferro

96 en the fact that D-lactate is a byproduct of lipid peroxidation and that M. tuberculosis lacks the ge

97 fidobacterium longum BB536 on plasma lipids, lipid peroxidation and the faecal excretion of bile acid

98 ducts produced by endogenous processes (e.g. lipid peroxidation) and exposure to bioactivated vinyl m

99 ative stress parameters such as glutathione, lipid peroxidation, and calcium levels along with the gl

100 lent modification and oxidation of proteins, lipid peroxidation, and DNA fragmentation.

101 elevations in pathogenic eicosanoid species, lipid peroxidation, and extracellular receptor kinase 1/

102 eaf visible symptoms (i.e. cell death), less lipid peroxidation, and lower NADPH oxidase activity, in

103 ion and oxidative phosphorylation, increased lipid peroxidation, and neuroinflammmation.

104 ed, with altered antioxidant enzyme content, lipid peroxidation, and oxidative DNA adducts.

105 Cardiomyocyte necrosis and apoptosis, tissue lipid peroxidation, and plasma nitrate and nitrite level

106 s, which can quench singlet oxygen, suppress lipid peroxidation, and prevent oxidative damage.

107 tral region of the cells and were related to lipid peroxidation, and structural changes of nucleic ac

108 ed mitochondrial protein carbonyl formation, lipid peroxidation, and thiol depletion.

109 n sheaths, and that elevated serum levels of lipid peroxidation are reported in BD, these serum measu

110 ia formation of hydroxyl radicals leading to lipid peroxidation as the primary mechanism of bacterial

111 storage roots showed delayed PPD and reduced lipid peroxidation as well as decreased H2O2 accumulatio

112 Lipid peroxidation, as measured by malondialdehyde, incr

113 to disruption of the antioxidant system and lipid peroxidation, as well as alterations in lysosomal

114 Endogenous lipid peroxidation (assay A) and induced lipid peroxidat

115 ous lipid peroxidation (assay A) and induced lipid peroxidation (assay B) were evaluated in liver hom

116 xygen radical absorbance capacity (ORAC) and lipid peroxidation assayed as thiobarbituric acid reacti

117 d proteomics data, enzymatic activities, and lipid peroxidation assays, we identified glutathione per

118 ivity of phenolic compounds in non-enzymatic lipid peroxidation assays.

119 scavenging, beta-carotene-linoleic acid and lipid peroxidation assays; the antibacterial activity wa

120 or </=2 menstrual cycles, with biomarkers of lipid peroxidation being assessed </=8 times per cycle.

121 ges and an increase in concentrations of the lipid peroxidation biomarkers 8-isoprostanes and malondi

122 nd mercury were not associated with elevated lipid peroxidation biomarkers.

123 ation between cadmium, lead, and mercury and lipid peroxidation biomarkers.

124 rably altered antioxidant enzyme content and lipid peroxidation, but better withstood insults.

125 an elevated H(2)O(2) flux, and increases in lipid peroxidation, but no effect on longevity.

126 VL), and kidney dysfunction; Fer-1 inhibited lipid peroxidation, but not mitochondrial reactive oxyge

127 that 5-HT contributes to the termination of lipid peroxidation by direct interaction with active gro

128 These lipids are the substrates for lipid peroxidation by lipoxygenase enzymes.

129 alpha-2, which are implicated in scavenging lipid peroxidation by-products.

130 enzyme with conjugating activity against the lipid peroxidation byproduct 4-hydroxynonenal (4HNE), is

131 XBP1 activation, fueled by lipid peroxidation byproducts, induced a triglyceride bi

132 solute scale of inhibition properties of the lipid peroxidation can be devised.

133 Increases in lipid peroxidation can cause ferroptosis, a form of cell

134 OA cartilage had higher levels of lipid peroxidation compared to control cartilage, and li

135 ith the control, and decreased the degree of lipid peroxidation compared with the HF diet.

136 dants (myloperoxidase, oxidative stress, and lipid peroxidation) compared with control.

137 The pex11a line showed higher levels of lipid peroxidation content and lower expression of genes

138 t involve ROS generation, carnosol inhibited lipid peroxidation, contrary to carnosic acid.

139 4) mitochondrial function by measuring H2O2, lipid peroxidation, cytochrome c oxidase activity and mi

140 of AhR that protects RPE cells in vitro from lipid peroxidation cytotoxicity mediated by 4-hydroxynon

141 easing levels of reactive oxygen species and lipid peroxidation, depleting and oxidizing glutathione

142 generation by limiting the damage induced by lipid peroxidation derivatives.

143 sis model of assessment (HOMA), and systemic lipid peroxidation determined by plasma F2-isoprostane l

144 rsus UW: 8.5 +/- 4.4 days, P=0.1357), nor in lipid peroxidation during 16-hr cold ischemia (P=0.672),

145 We report here the mechanism of lipid peroxidation during ferroptosis, which involves ph

146 cysteine, histidine, and lysine residues by lipid peroxidation end products such as 4-hydroxy- and 4

147 ls, we report that PPARgamma is activated by lipid peroxidation end products, such as 4-hydroxynonena

148 decreased antioxidant defense, and elevated lipid peroxidation end-products in spite of comparable n

149 eviously unrecognized mechanism for limiting lipid peroxidation, free radical damage, and proinflamma

150 f the olefinic band elicited seizure-induced lipid peroxidation further confirmed by the thiobarbitur

151 Lipid peroxidation generates lipid aldehydes, including

152 Lipid peroxidation have not changed significantly howeve

153 onstrated increased reactive oxygen species, lipid peroxidation, histological evidence of balloon deg

154 IC50=56.51 +/- 3.6 mug/mL) and inhibition of lipid peroxidation (IC50=12.34 +/- 2.3 mug/mL) as compar

155 ty, as well as a good capability to suppress lipid peroxidation in a linoleic acid model system.

156 The ability of these compounds to inhibit lipid peroxidation in a liposome membrane system was exa

157 Lipid peroxidation in articular cartilage from OA patien

158 advances promise to help clarify the role of lipid peroxidation in cell death and human disease.

159 lar glutathione concentrations and decreased lipid peroxidation in cultured hepatocytes.

160 SO is required for lipid peroxidation in DHA-induced apoB100 degradation, b

161 dative-stress genes and elevated products of lipid peroxidation in eyes from abca4(-/-) versus wild-t

162 n of mitochondrial biogenesis, and increased lipid peroxidation in female mouse offspring exposed to

163 Furthermore their effectiveness in retarding lipid peroxidation in fish oil was evaluated by an accel

164 concentration was negatively correlated with lipid peroxidation in foliar tissue under ozone stress a

165 on and prevents anemia, ROS accumulation and lipid peroxidation in Gpx4-deficient cells remain high.

166 conventional red grape juice consumption on lipid peroxidation in healthy individuals.

167 (SA), and correlated with the inhibition of lipid peroxidation in human erythrocytes (LP) and total

168 activity and vitamin E level and on reducing lipid peroxidation in hypercholesterolaemia rabbit, ther

169 mpletion of this protocol, tissue injury and lipid peroxidation in jejunum and ileum were analyzed by

170 These flavonoids were also found to prevent lipid peroxidation in L6 myoblast.

171 egarding the mechanisms of inhibition of the lipid peroxidation in micelles, in view of bibliographic

172 eration, mitochondrial hyperpolarization and lipid peroxidation in neuronal cells, but they do so by

173 beta3pE-42 has an enhanced capacity to cause lipid peroxidation in primary cortical mouse neurons com

174 We used it to probe the importance of lipid peroxidation in progression of NASH beyond simple

175 MERTK expression and lipid peroxidation in synaptoneurosomes also increased t

176 roscopy, to co-localize amyloid deposits and lipid peroxidation in tissue slides from patients affect

177 NE) is a main endogenous product of cellular lipid peroxidation in tissues and is reported to play pa

178 nd 4-hydroxy-2,6-alkadienals), biomarkers of lipid peroxidation, in exhaled breath condensate of thre

179 tathione-to-glutathione disulfide ratio, and lipid peroxidation indicated that HFD-induced oxidative

180 en species production and hence the level of lipid peroxidation, indicating a role of TAG in protecti

181 We found no significant differences in lipid peroxidation, indicating that oxidative stress may

182 S can be formed after singlet oxygen-induced lipid peroxidation, indicating that RES-stimulated and S

183 tested fractions of sea buckthorn inhibited lipid peroxidation induced by H2O2, however, the non-pol

184 iggers formation of ethylene as a product of lipid peroxidation induced by the respiratory burst.

185 a consequence of sequence-specific repair of lipid peroxidation-induced DNA adduct, 1, N(6)-ethenoade

186 concentration- and time-dependent effect on lipid peroxidation, inducing both pro-oxidant actions at

187 f cruP transcripts under photoinhibitory and lipid peroxidation-inducing conditions, such as high lig

188 ere hepatic iron overload with more advanced lipid peroxidation, inflammation, and portal fibrosis th

189 pe juices produced in Southern Brazil showed lipid peroxidation inhibition abilities in healthy subje

190 arkable antiradical activity and significant lipid peroxidation inhibition activities, with their IC5

191 TCC), free radical scavenging activities and lipid peroxidation inhibition activities.

192 ively correlated with nitrite scavenging and lipid peroxidation inhibition activities.

193 Superoxide radical scavenging, lipid peroxidation inhibition and cupric ion reducing ac

194 polar dicaffeoylquinic acid; whereas higher lipid peroxidation inhibition was attributed to the pres

195 cals scavenging activity, reducing power and lipid peroxidation inhibition) and antitumour potential

196 ity (scavenging activity, reducing power and lipid peroxidation inhibition) and individual phenolic p

197 ical scavenging activity, reducing power and lipid peroxidation inhibition) of dried powder formulati

198 xidant properties (mainly reducing power and lipid peroxidation inhibition), antibacterial activity a

199 crylhydrazyl (DPPH) free radical scavenging, lipid peroxidation inhibition, nitrite scavenging and su

200 Lipid peroxidation is a major consequence of oxidative s

201 Lipid peroxidation is connected to increases in mitochon

202 How BD affects lipid peroxidation is not known.

203 Because lipid peroxidation is ubiquitous and a major component o

204 The reduction of lipid peroxidation levels and activity of hepatic enzyme

205 light and is photoprotective, as it reduces lipid peroxidation levels.

206 A2-VIA is strongly associated with increased lipid peroxidation levels.

207 ted foliar reactive oxygen species (ROS) and lipid peroxidation levels.

208 idemia by sensing a wide range of endogenous lipid peroxidation ligands and activating innate immune

209 A (and/or RD) in BD, and also examined serum lipid peroxidation (lipid hydroperoxides, LPH and 4-hydr

210 Lipid peroxidation (LP) was also determined as an indica

211 onga (CLM and CLW), at 100 mug/mL, inhibited lipid peroxidation (LPO) by 78%, 63%, 81% and 43%, cyclo

212 Lipid peroxidation (LPO) is induced by a variety of abio

213 ons produced endogenously via reactions with lipid peroxidation (LPO) products.

214 yde (MDA), which is a significant product of lipid peroxidation (LPO), total oxidant status (TOS), to

215 he left maxilla was used for the analysis of lipid peroxidation (malondialdehyde [MDA]) and antioxida

216 Resistance to lipid peroxidation maps genetically to transmembrane and

217 and 450 +/- 360%, respectively, and urinary lipid peroxidation marker malondialdehyde was decreased

218 serum levels of malondialdehyde (MDA), as a lipid peroxidation marker, and 8-hydroxydeoxyguanosine (

219 ctions of several SPs, we found increases in lipid peroxidation markers in Trsp-deficient epithelial

220 Brain death (BD)-related lipid peroxidation, measured as serum malondialdehyde (M

221 therapeutic uses for ferrostatins, and that lipid peroxidation mediates diverse disease phenotypes.

222 loss of normal PLA2G6 gene activity leads to lipid peroxidation, mitochondrial dysfunction and subseq

223 o determine the effects of SOD2 depletion on lipid peroxidation, mtDNA damage, and mitochondrial resp

224 oated-NPs exposed snails did not undergo any lipid peroxidation nor change in the antioxidant content

225 Confocal microscopy and lipid peroxidation observations show that Cu and citric

226 vidence suggests that this process, known as lipid peroxidation, occurs in vivo under a variety of co

227 Finally, we show that nonenzymatic lipid peroxidation of arachidonic and other polyunsatura

228 he inhibitory effect of HLP on oxidation and lipid peroxidation of LDL was defined in vitro.

229 None of the compounds modified lipid peroxidation or glutathione concentrations (GSH) i

230 s free radicals by mitochondria thus causing lipid peroxidation, oxidative and acidic stress, which c

231 < 0.0001), blood pressure (P < 0.0001), and lipid peroxidation (P = 0.001) were also observed for th

232 idative stress (peroxiredoxin-3/5) and for a lipid peroxidation product (hydroxynonenal).

233 In GDM cells, the lipid peroxidation product 4-hydroxynonenal (HNE) failed

234 CaARP-expressing plants showed lower lipid peroxidation product content in presence or absenc

235 .026) and lower plasma concentrations of the lipid peroxidation product F2-isoprostanes (18.5 pg/mL,

236 ng is activated by 4-hydroxynonenal (HNE), a lipid peroxidation product generated naturally during ox

237 of genotoxic N-nitroso compounds (NOCs) and lipid peroxidation products (LPOs) in the gut.

238 hemically diverse group of products, such as lipid peroxidation products (LPP).

239 These data suggest that lipid peroxidation products play a role in progression o

240 Tissue content of lipid peroxidation products was increased in G6PDX mice

241 hat preventing the damage to biomolecules by lipid peroxidation products, a novel concept in vision r

242 the diet and traditional medicines and from lipid peroxidation products, in human prostate and renal

243 These could be lipid peroxidation products, including isolevuglandins (

244 best discriminators of SLE included elevated lipid peroxidation products, MDA, gamma-glutamyl peptide

245 Lipid peroxidation products, such as 4-hydroxy-trans-2-n

246 formed from 2,3-epoxyaldehydes of endogenous lipid peroxidation products, were present in all subject

247 e available for the generation of neurotoxic lipid peroxidation products.

248 ating antibodies against protein adducted by lipid peroxidation products.

249 minant nonphenolic structures is mediated by lipid peroxidation products.

250 ation of oxidative stress signals related to lipid peroxidation, protein carbonylation, and nitration

251 anti-oxidant enzymes were elevated, as were lipid peroxidation, protein nitrosylation, and ROS.

252 onsequences of nitrooxidative stress include lipid peroxidation, protein oxidation and DNA damage.

253 -type HCV replicase is uniquely regulated by lipid peroxidation, providing a mechanism for attenuatin

254 The left kidneys were used to determine lipid peroxidation, quantification of reactive oxygen sp

255 The SLE metabolome exhibited profound lipid peroxidation, reflective of oxidative damage.

256 Lipid peroxidation, regulated in part through sphingosin

257 displayed an increased membrane leakage and lipid peroxidation relative to Cu-GGH and OV-3 alone.

258 plants showed no increase in ROS content or lipid peroxidation relative to well-watered controls, de

259 arbituric acid (TBA) number, an indicator of lipid peroxidation responsible for off-flavour generatio

260 dicate that photoexcited SWCNTs can catalyze lipid peroxidation similarly to lipoxygenases.

261 in the male germ line, with the products of lipid peroxidation stimulating free radical generation b

262 -beta1 concentrations to oxidant burden (ie, lipid peroxidation), T(H)2-mediated eosinophilic inflamm

263 4HNE) and acrolein, generated as a result of lipid peroxidation, target the mitochondria of human spe

264 0) were predominantly associated with higher lipid peroxidation (TBARS) [exp(beta) = 1.09-1.78, p < 0

265 ntioxidant activity (FRAP, ABTS), as well as lipid peroxidation (TBARS) were determined at the end of

266 total antioxidant capacity, DPPH assays and lipid peroxidation test).

267 is an electrophilic aldehyde produced during lipid peroxidation that forms covalent adducts on protei

268 se (epsilon-base) DNA lesions induced by the lipid peroxidation that is stimulated by reactive oxygen

269 ntramuscular glycogen, and oxidative stress (lipid peroxidation) that occurred following approximatel

270 g activity, reducing power and inhibition of lipid peroxidation, the antitumour potential was tested

271 g activity, reducing power and inhibition of lipid peroxidation; the antitumour potential was tested

272 PSEE exhibited a protection of lipid peroxidation threefold higher than a positive cont

273 titudes, which paralleled with reductions in lipid peroxidation, thus suggesting plants from the high

274 be used in TTB as a protective agent against lipid peroxidation to extend its shelf-life up to two mo

275 Lipid peroxidation took place after 3 weeks of storage i

276 dehyde levels in both endogenous and induced lipid peroxidation up to 35% and 70%, respectively.

277 Increased lipid peroxidation via oxidative stress was also detecte

278 Reducing LD accumulation in glia and lipid peroxidation via targeted lipase overexpression an

279 Hepatic lipid peroxidation was also elevated in the nose-only gr

280 tioxidant status (SOD, CAT, GPX and GSH) and lipid peroxidation was also studied.

281 fatty acids illustrated that, in the light, lipid peroxidation was predominantly due to the producti

282 after sham laparotomy, but this increase in lipid peroxidation was prevented by preconditioning with

283 stress as measured by glutathione levels and lipid peroxidation was significantly reduced in rapamyci

284 Graft lipid peroxidation was significantly reduced in the pres

285 oxidation compared to control cartilage, and lipid peroxidation was similarly elevated in SOD2-deplet

286 nce on the antioxidant role of Vitamin E, as lipid peroxidation was suppressed in HeLa cells both und

287 After reperfusion, radical mediated lipid peroxidation was twofold higher in the MPanox grou

288 Urinary malondialdehyde (MDA), a marker of lipid peroxidation, was measured in 24 hour urine collec

289 However, MDA adduct formation, a marker of lipid peroxidation, was not affected by any of the four

290 ecies (ROS) and their downstream products of lipid peroxidation, we investigated the effect of nerve

291 To determine possible underlying causes of lipid peroxidation, we investigated the renal redox bala

292 ipid accumulation, apoptosis, and changes in lipid peroxidation were attenuated.

293 Enhanced electrolyte leakage and lipid peroxidation were found in the shoots of seedlings

294 onylation, amount of glutathione stores, and lipid peroxidation were similar irrespective of the insu

295 d polyunsaturated fatty acids, which inhibit lipid peroxidation, were able to partially rescue the lo

296 ow-density lipoprotein, serum amyloid A, and lipid peroxidation, were significantly altered by polyba

297 ate, ascorbic acid degradation, and membrane lipid peroxidation, which enhanced total phenolics conte

298 All compounds inhibited lipid peroxidation with an inhibitory concentration 50%

299 s an early and integral component of in vivo lipid peroxidation with important clinical implications

300 adical anion (and indirectly, a causative of lipid peroxidation) within the mitochondria matrix.