ライフサイエンスコーパス: lipid raft

1 ellular membranes in clusters, often called 'lipid rafts'.

2 e dissolution of ordered microdomains (i.e., lipid rafts).

3 s enriched in cholesterol and sphingolipids (lipid rafts).

4 ar structures such as membrane microdomains (lipid rafts).

5 in membrane structure in unique regions like lipid rafts.

6 itol (GPI)-anchored proteins localize in the lipid rafts.

7 its generation can modulate the structure of lipid rafts.

8 cid) had markedly less Tlr4 recruitment into lipid rafts.

9 d proteins on leukocytes that associate with lipid rafts.

10 and platinum colocalized with FAS protein in lipid rafts.

11 nslocation of the GDNF receptor complex into lipid rafts.

12 forces play a major role in the formation of lipid rafts.

13 ead to the formation, stability, and size of lipid rafts.

14 hrough caveolae, membrane invaginations from lipid rafts.

15 integral membrane proteins and formation of lipid rafts.

16 and the redistribution of presenilin 1 from lipid rafts.

17 ial internalization through cholesterol-rich lipid rafts.

18 lated form of the Src family kinase, Fyn, to lipid rafts.

19 ansient domains are observed, reminiscent of lipid rafts.

20 CTF by using gamma-secretase associated with lipid rafts.

21 hrough regulating the cholesterol content of lipid rafts.

22 domain and co-sedimented with caveolin-1 in lipid rafts.

23 DAT internalization arises exclusively from lipid rafts.

24 ontaining high concentrations of sterol-rich lipid rafts.

25 rs and independently of its association into lipid rafts.

26 clustering of the toxin.receptor complex in lipid rafts.

27 lieved to result from their association with lipid rafts.

28 s of naturally occurring nanodomains such as lipid rafts.

29 as well as on their lateral accumulation in lipid rafts.

30 n kinase C activities, and both clathrin and lipid rafts.

31 40 nm that are consistent with the notion of lipid rafts.

32 ding ordered, lipid-driven assemblies termed lipid rafts.

33 hat achieves full activity when recruited to lipid rafts.

34 pressant treatment translocates Galphas from lipid rafts.

35 n filaments and microtubules but also affect lipid rafts.

36 e we show that soluble klotho binds membrane lipid rafts.

37 FC may be incorporated into microdomains or lipid rafts.

38 urally diverse antidepressants accumulate in lipid rafts.

39 ments, the M-PMV MA did not co-localize with lipid rafts.

40 oteins normally restricted to juxtaparanodal lipid-rafts.

41 al synapse, where it regulates filamin A and lipid raft accumulation, as well as T cell activation, i

42 Lipid rafts act as organization centers for biological p

43 Mechanical disruption of the lipid rafts activates PLD2 by mixing the enzyme with its

44 up-regulation of Ack1 and its recruitment to lipid rafts along with TRAIL-R1/2.

45 ing that HtrA and p66 may reside together in lipid rafts also.

46 DAT internalization occurs equivalently from lipid raft and nonraft microdomains, whereas PKC-stimula

47 indomethacin (IND) leads to the formation of lipid rafts and activation of caveolin-1; however, no su

48 Importantly, LMP1 trafficking to lipid rafts and activation of NF-kappaB and PI3K/Akt pat

49 The distribution of membrane lipid rafts and adhesion receptors were analyzed by imag

50 in the motif reduce mGluR1 association with lipid rafts and agonist-induced, mGluR1-dependent activa

51 ficient mutants were found colocalizing with lipid rafts and alive in nonacidic compartments.

52 nds the HIV-1 Gag polyprotein, retains it in lipid rafts and blocks HIV-1 virion production and sprea

53 essential constituents of cell membranes and lipid rafts and can modulate signal transduction events.

54 vitro and in vivo demonstrate that membrane/lipid rafts and caveolin (Cav) organize progrowth recept

55 at infection occurs at the interface between lipid rafts and cellular stress/death pathways via speci

56 CD19 is localized to lipid rafts and constitutively activated by the LMP1/CD4

57 een LMP1 and these proteins was localized to lipid rafts and dependent on LMP1 signaling.

58 two classes of antidepressants accumulate in lipid rafts and effect translocation of Galphas to the n

59 and patient IgG colocalized with markers for lipid rafts and endosomes.

60 miR-33 augments macrophage lipid rafts and enhances proinflammatory cytokine induct

61 ivated Toll-like receptors (TLRs) cluster in lipid rafts and induce pro- and anti-tumor responses.

62 ynitrite drove TLR4 recruitment into hepatic lipid rafts and inflammation, whereas the in vivo use of

63 m (PrP(Sc)) during prion infection occurs in lipid rafts and is dependent on cholesterol.

64 mAb, lacks the ability to redistribute into lipid rafts and is glycoengineered for augmented antibod

65 lum (ER) membrane protein that is located in lipid rafts and known to be important in ER-associated p

66 location of normal prion protein (PrP(C)) in lipid rafts and lipid cofactors generating infectious pr

67 We observed that SFK and FAK in the lipid rafts and nonrafts are differently regulated by fl

68 Kv1.3 localizes in lipid rafts and participates in the immunological respon

69 ynamics and clustering of SR-B1 contained in lipid rafts and potently inhibits cellular exosome uptak

70 induced localization of TRAF6 and c-SRC into lipid rafts and preventing nuclear accumulation of trans

71 V de novo infection, p130Cas associates with lipid rafts and scaffolds EphrinA2 (EphA2)-associated cr

72 oscopy, we characterized the distribution of lipid rafts and the E-selectin counterreceptor CD44 on t

73 s able to form GM1- and cholesterol-enriched lipid rafts and these raft domains are important for enc

74 re, we confirmed that US9 is associated with lipid-rafts and can target functional enzymes to membran

75 sphingolipid-enriched membrane microdomains (lipid rafts) and are highly expressed in brain tissue.

76 We find that apoA1 rapidly disrupts membrane lipid rafts, and as a consequence, dampens the PI3K/Akt

77 holesterol-rich membrane microdomains called lipid rafts, and can be blocked by non-specific depletio

78 ng ATP, protein kinase A or the formation of lipid rafts, and does not require ion flux through the c

79 evels, increases membrane fluidity, disrupts lipid rafts, and redistributes CD44, which is the primar

80 by preventing phagolysosomal maturation in a lipid raft- and O antigen-dependent manner.

81 Lipid rafts are cholesterol- and sphingolipid-enriched m

82 Here we show that lipid rafts are dynamic compartments that inactivate the

83 Lipid rafts are hypothesized to facilitate protein inter

84 upport the notion that sialogangliosides and lipid rafts are membrane receptors for sKlotho and that

85 Lipid rafts are specialized dynamic microdomains of the

86 Lipid rafts are submicron proteolipid domains thought to

87 Lipid rafts are widely believed to be an essential organ

88 esistant membrane nanodomains, also known as lipid rafts, are the primary response element in EF sens

89 Lipid heterogeneities, such as lipid rafts, are widely considered to be important for t

90 , providing a new perspective on the role of lipid rafts as concentrators of membrane proteins.

91 ect parallels the movement of Galphas out of lipid rafts as determined by cold detergent membrane ext

92 ngomyelin (SM) content and aggregation of ER lipid rafts, as visualized using Erlin1-GFP.

93 &HNF4alpha on CPT2, the lipid droplet and ER-lipid-raft associated PLIN3 and Erlin1.

94 oichiometries of intact Nanodiscs containing lipid-raft associated sphingomyelin.

95 These Ia.2(+) class II conformers are lipid raft-associated and able to drive both tyrosine ki

96 SLE patients displayed an altered profile of lipid raft-associated glycosphingolipids (GSLs) compared

97 -Src downregulated the expression of Pag1, a lipid raft-associated inhibitor of Src, which was restor

98 In summary, this study identifies AnxA2 as a lipid raft-associated trafficking factor for NKCC2 and p

99 ed with Dex treatment alone, suggesting that lipid raft association of the GR has a role in enhancing

100 red among various enveloped viruses, such as lipid raft association, membrane curvature, or ESCRT dep

101 e lack of BACE1 S-palmitoylation and reduced lipid raft association.

102 Tim-3 was found within CD8(+) T cell lipid rafts at the immunological synapse.

103 ntractile actomyosin ring and coalescence of lipid rafts between reticulocyte and pyrenocyte, steps w

104 Inhibition of FAK in the lipid rafts blocked SFK response to fluid flow, while in

105 activation of PDGFR specifically located in lipid rafts but not outside rafts, implying the role of

106 X7 receptors associate with cholesterol-rich lipid rafts, but it is unclear how this affects the prop

107 Ralpha colocalized with GM1 ganglioside-rich lipid rafts, but MHC I clusters retracted to smaller sub

108 phenotype was accompanied by a reduction of lipid rafts, but was independent of the peroxisomal or t

109 Therefore, the recruitment of Ret into lipid rafts by GFRalpha1 is required for the physiologic

110 Lipid rafts, chemically distinct membrane lipid microdom

111 al function in uninfected cells to fine-tune lipid raft cholesterol that regulates innate immunity to

112 R proinflammatory responses due to augmented lipid raft cholesterol.

113 cell surface glycosaminoglycans and induces lipid raft clustering, increasing the incorporation of C

114 naling and internalization by disrupting BCR-lipid raft coclustering and by inducing the endocytosis

115 to be tightly regulated by cholesterol-rich "lipid rafts." Collectively, these data show that RIDalph

116 rinuclear tubular structures, an increase of lipid raft components, and increased lipid raft partitio

117 is defective Ret signaling owing to improper lipid raft composition or function.

118 in that is an important player in remodeling lipid raft composition.

119 sion of CD1d accompanied by an alteration in lipid raft content on the plasma membrane of thymocytes

120 ANGPTL4-deficient CMPs have higher lipid raft content, are more proliferative and less apop

121 ses in cellular and membrane cholesterol and lipid raft contents as determined by Raman spectroscopy

122 oscale assemblies in cell membranes known as lipid rafts, coself-assembly of 1-decanol into cetyltrim

123 t focal adhesion kinase pathway and toward a lipid raft-dependent caveolin-Fyn-Shc pathway.

124 and fusion, and through macropinocytosis and lipid raft-dependent endocytosis.

125 HMVEC-d), its naturalin vivotarget cells, by lipid raft-dependent macropinocytosis.

126 cells could mediate HCV RNA replication in a lipid raft-dependent manner, as the depletion of cholest

127 ts on fibronectin in a protease-independent, lipid raft-dependent manner.

128 Annexins mediate lipid raft-dependent trafficking of transmembrane protei

129 on and clustering within the plasma membrane lipid rafts, dimerization and autophosphorylation.

130 In addition, the EPA-induced lipid raft disorganization, caveolin-1 inactivation, and

131 s, and provide cytoprotection, consequent to lipid raft disorganization.

132 was cell-contact dependent and unaffected by lipid raft disruption of donor TEC.

133 Pretreatment with lipid raft disruptor (Methyl-beta-cyclodextrin, MbetaCD)

134 position throughout brain development, their lipid raft distribution and biological relevance is poor

135 NHE3 complex formation and changes the NHE3 lipid raft distribution, which cause changes in specific

136 dentified proteins and LMP1 was localized to lipid raft domains and was dependent on LMP1-induced sig

137 Cholesterol-conjugated antibodies bind to lipid raft domains on the membrane, and because of this

138 LMP1 is localized to lipid raft domains to induce signaling.

139 fe cycle stages possess chemically different lipid rafts due to different sterol utilization.

140 The trafficking behavior of the lipid raft-dwelling US9 protein from Herpes Simplex Viru

141 Lipid raft-enriched fractions were isolated and purified

142 ion of its Gsalpha target which resides in a lipid raft environment.

143 Association of NKCC2 with lipid rafts facilitates its AVP-induced apical transloca

144 Subsequently, clathrin-, caveolin-, lipid raft-, flotillin-, cholesterol-, and dynamin-indep

145 ent of TCR-associated signaling molecules to lipid rafts, followed by abrogation of protein tyrosine

146 The forces that drive lipid raft formation are poorly understood.

147 of switch-activated protein 70 (SWAP-70) in lipid raft formation of dendritic cells.

148 been largely overlooked as major players in lipid raft formation.

149 like control cells Neph1 was retained in the lipid raft fractions in the transduced cells following t

150 ly, CRFR1 and gamma-secretase co-localize in lipid raft fractions, with increased gamma-secretase acc

151 aracterized the lipid and protein content of lipid rafts from control E. huxleyi cells and those infe

152 pletion of cholesterol, a major component of lipid rafts, from autophagosomes abolished HCV RNA repli

153 To specifically interfere with lipid raft function in vivo, we focused on the Sterol Ca

154 GM3, a lipid raft ganglioside synthesized by GM3 synthase (GM3S

155 ce statement: Membrane microdomains known as lipid rafts have been proposed to be unique subdomains i

156 Based on in vitro studies, lipid rafts have been reported to be necessary for the f

157 dered membrane domains, often referred to as lipid rafts, have been highly debated by cell biologists

158 Since cholesterol is a critical component of lipid rafts, here we tested the hypothesis of whether th

159 The lipid raft hypothesis presents insights into how the cel

160 The lipid raft hypothesis proposes lateral domains driven by

161 This was associated with increased lipid rafts in APOepsilon3/APOepsilon4 monocytes.

162 istently, GFRalpha1 correctly partitioned to lipid rafts in brain tissue.

163 y much resembles the assembly process of the lipid rafts in cell membranes and triggers orders of mag

164 cAMP (Galphas) is increasingly localized to lipid rafts in depressed subjects and that chronic antid

165 nism of maintaining the integrity of BCRs in lipid rafts in DLBCLs with low or high NF-kappaB.

166 a GFRalpha1, the existence and importance of lipid rafts in GDNF-Ret signaling under physiologic cond

167 1 cascade, which requires Fyn-Src kinase and lipid rafts in human taste bud cells (TBCs).

168 ross-talk that involves co-localization with lipid rafts in LbetaT2 cells.

169 is possibility and also points to a role for lipid rafts in milk product secretion.

170 ke receptor (TLR)-4 recruitment into hepatic lipid rafts in nonalcoholic steatohepatitis (NASH) are u

171 nce of specialized signalling domains called lipid rafts in schistosomes and propose that correct sig

172 P) partitioned in GM1 ganglioside-containing lipid rafts in the plasma membrane of live cells.

173 ycoprotein hemagglutinin is localized within lipid rafts in virus-infected cells, whereas M2 is assoc

174 esides in detergent-resistant outer membrane lipid rafts in which conversion to the pathogenic misfol

175 phorylceramides (GIPCs), major components of lipid rafts, in the membrane requirement for B.

176 sociated with uncompacted myelin domains and lipid rafts, including flotillin-1, cholesterol, and GM1

177 ced NHE3 complex size, reduced expression in lipid rafts, increased BB mobile fraction, and reduced b

178 d signal pathway for migration away from the lipid raft-independent focal adhesion kinase pathway and

179 man bronchial epithelial cells in vitro in a lipid raft-independent manner, is subsequently trafficke

180 ellular factors (e.g. actin cytoskeleton and lipid rafts) influence the assembly of ligand-receptor c

181 B1 oligomers in the membrane disrupts planar lipid raft integrity and causes apoptosis via activating

182 activity and that this modulation depends on lipid raft integrity.

183 in actin-dependent cellular functions and/or lipid raft integrity.

184 B cell receptors (BCRs), a process requiring lipid rafts, interferes with PM repair.

185 d HDL-mediated cholesterol depletion reduces lipid rafts, interferes with VEGFR2 (also known as KDR)

186 t Tax recruitment of autophagic molecules to lipid rafts is a dominant strategy to deregulate autopha

187 mation of higher order receptor complexes in lipid rafts is an equally important aspect.

188 s we show that clustering of gangliosides in lipid rafts is important.

189 dy suggest that the association of LMP1 with lipid rafts is mediated at least in part through interac

190 s suggests that the association of LMP1 with lipid rafts is mediated through interactions with actin-

191 cavenger receptor type B-1 (SR-B1), found in lipid rafts, is a receptor for cholesterol-rich high-den

192 ft membranes, with a significant increase in lipid rafts isolated from ASA knockout mice.

193 this quantitative analysis to detergent-free lipid rafts isolated from wild-type mice and arylsulfata

194 , and C24:0) in central nervous system (CNS) lipid rafts isolated without using detergent.

195 into cholesterol-rich membrane microdomains (lipid rafts), its compartmentalization has not been demo

196 , which reduced IFNG signaling by disrupting lipid rafts, leading to reduced phosphorylation (activat

197 th a clustering of CD44 and CD24 in membrane lipid rafts, leading to the activation of Src Family Kin

198 The MAM region of the ER is a lipid raft-like domain closely apposed to mitochondria i

199 This is complicated by HA and NA residing in lipid raft-like domains, whereas M2, although an integra

200 In particular, mitochondrial lipid raft-like microdomains appear to function as platf

201 Mitochondrial lipid raft-like microdomains, experimentally also termed

202 it mitochondrial antiviral signaling through lipid raft-like microdomains.

203 a complement-independent manner and required lipid raft localization for CSC maintenance and cisplati

204 hippocampal expression of Cav-1 and membrane/lipid raft localization of postsynaptic density protein

205 Mechanistically, SQS contributed to a lipid-raft-localized enrichment of tumor necrosis factor

206 nd RhoA GTPase signal molecules early during lipid raft (LR)-dependent productive macropinocytic entr

207 glioside is a major constituent of mammalian lipid rafts (LRs) and known to react with cholera toxin

208 lfate proteoglycans and cholesterol-enriched lipid rafts (LRs).

209 opus pronephric kidney were positive for the lipid raft markers ganglioside GM1 and Caveolin-1.

210 or cell electrosensing and provide a role in lipid raft mechanotransduction.

211 Both clathrin- and caveolae/lipid raft-mediated endocytosis pathways are involved in

212 least two clathrin-independent, cholesterol/lipid raft-mediated pathways that do not require ubiquit

213 Furthermore, macropinocytosis and lipid raft-mediated were shown here as mechanisms of MkM

214 cysteine residue enables Fas to localize to lipid raft microdomains and induce apoptosis in cell lin

215 e recruitment of key signaling components to lipid raft microdomains by LMP1 was analyzed.

216 Lipid raft microdomains typically serve as platforms for

217 Many channels localize in lipid raft microdomains, which are enriched in cholester

218 complex containing Beclin1 and Bif-1 to the lipid raft microdomains.

219 n with no cytosolic domain that localizes to lipid raft microdomains.

220 ber of the MAGUK family, recruits Kv1.3 into lipid-raft microdomains and protects the channel against

221 in vitro demonstrate that neuronal membrane/lipid rafts (MLRs) establish cell polarity by clustering

222 Membrane lipid rafts (MLRs) within the plasma membrane of most ce

223 nd GnRHR cross-talk within Flot-1-containing lipid rafts modulates cell proliferation via PKC activat

224 These findings illuminate differences in the lipid rafts of an organism employing life cycle-specific

225 geting glycoprotein ligands for selectins to lipid rafts of leukocytes.

226 ters with interleukin-2 and -15 receptors in lipid rafts of T cells.

227 holesterol metabolism is pathogens targeting lipid rafts of the host plasma membrane.

228 conclude that VHH JM4, when targeted to the lipid rafts of the plasma membrane, efficiently neutrali

229 attachment signal, VHHs are targeted to the lipid rafts of the plasma membranes.

230 attachment signal, VHHs are targeted to the lipid rafts of the plasma membranes.

231 VV binding strongly colocalized with lipid rafts on the surfaces of all VV binding-susceptibl

232 known how these glycoproteins associate with lipid rafts or whether this association is required for

233 embranes by inducing several signals through lipid raft organization after membrane incorporation, wh

234 ion or genetic inactivation of ACAT decrease lipid raft palAPP levels by up to 76%, likely resulting

235 ease of lipid raft components, and increased lipid raft partitioning of APP.

236 LMP1 signaling requires oligomerization, lipid raft partitioning, and binding to cellular adaptor

237 ) I-A(k) class II conformers and controlling lipid raft partitioning.

238 xtracellular prosaposin and was disrupted by lipid raft perturbation using methyl-beta-cyclodextrin o

239 ipotoxicity, which leads to disruption of ER lipid rafts, perturbation of protein trafficking, and in

240 rable attention, especially in connection to lipid raft phenomena in cells.

241 temperature: additional host factors such as lipid rafts place CTA1 in the folded conformation requir

242 KII signaling and sequestration of CD44 into lipid rafts, preventing differentiation.

243 Current models of lipid rafts propose that lipid domains exist as nanoscal

244 ll population of the GR co-localize with the lipid raft protein flotillin-1 (Flot-1) at the plasma me

245 omic analysis confirmed flotillin as a major lipid raft protein, along with a variety of proteins aff

246 nce of specialized membrane domains, such as lipid rafts, protein-lipid complexes, receptor complexes

247 e latter ones often called lipid domains or "lipid rafts." Recent findings highlight the dynamic natu

248 isms by which membrane microdomains, such as lipid rafts, reduce these effects, and thus, enhance ass

249 brane cholesterol content or perturbation of lipid rafts regulates agonist-dependent activation of ER

250 The receptor for GDNF comprises the lipid raft-resident, glycerophosphatidylinositol-anchore

251 with one another because of competition for lipid rafts, revealing how frequent membrane injury and

252 P90, and HER2 within specific actin-rich and lipid raft-rich membrane signaling domains.

253 Here, we show that the lipid raft scaffolding protein caveolin-1 interacts with

254 membrane, contradicting previous reports of lipid raft segregation.

255 Instead, lipid rafts shifted disordered CTA1 to a folded conforma

256 estingly, inhibition of myosin-9, actin, and lipid-rafts, shown to be involved in PNX1-hemichannel fu

257 ar cholesterol and disrupted plasma membrane lipid rafts, similar to positive control methyl-beta-cyc

258 Lipid rafts, specialized membrane microdomains in the pl

259 ling a dynamic spatiotemporal control of the lipid raft structure with light.

260 This GlyT2.PMCA2,3.NCX1 complex is found in lipid raft subdomains where GlyT2 has been previously fo

261 ol-glycolipid-rich membrane regions known as lipid rafts, suggesting that HtrA and p66 may reside tog

262 h the colocalization of platinum with FAS in lipid rafts support an extrinsic apoptotic signaling mec

263 Moreover, expression of lipid raft-targeted Bif-1 or Beclin1 was sufficient to i

264 cytoskeleton necessary for the clustering of lipid rafts, TCR, and costimulatory receptors toward the

265 The O antigen targets B. parapertussis to lipid rafts that are retained in the membrane of phagoso

266 This very much resembles the role of the lipid rafts that sharply increases the reaction rate of

267 l in which GFRalpha1 is no longer located in lipid rafts, that the developmental functions of GDNF in

268 omyelin, cholesterol, and select proteins in lipid rafts-the dynamic functional subdomains of the pla

269 otility by sequestering Src to caveolin-rich lipid rafts, thereby disengaging Src from FAK-associated

270 sion by EZCs reduces movement of BMPR1b into lipid rafts, thereby limiting the known deleterious effe

271 HGAL phosphorylation and redistribution from lipid raft to bulk membrane and cytoplasm, followed by d

272 ed that HCV could induce the localization of lipid rafts to autophagosomes to mediate its RNA replica

273 Our results identify ganglioside-enriched lipid rafts to be receptors that mediate soluble klotho

274 s enveloped viruses utilize cholesterol-rich lipid rafts to bud from the host cell membrane, and it i

275 llactose-containing gangliosides enriched in lipid rafts to inhibit raft-dependent PI3K signaling.

276 used as a tool to study the contribution of lipid rafts to neurodegenerative disease conditions wher

277 gered relocalization of gamma-secretase from lipid rafts to nonlipid rafts where it cleaved Notch.

278 echanistically, uPAR sequestered TACE within lipid rafts to prevent Notch1 activation, thereby promot

279 and decreased recruitment of both LFA-1 and lipid rafts to the immunological synapse, which correlat

280 exosomal LMP1 release that is distinct from lipid raft trafficking.

281 tegrin-acylated Fyn signaling complexes into lipid rafts upon uPAR ligation through protein-protein i

282 biosensor was further targeted in or outside lipid rafts via different lipid modification signals.

283 in vitro is augmented by translocation into lipid rafts via GFRalpha1, the existence and importance

284 To explain why klotho preferentially targets lipid rafts we show that clustering of gangliosides in l

285 to specifically modify Galphas localized to lipid rafts, we sought to determine whether structurally

286 V binding-susceptible PHL subsets, even when lipid rafts were relocated to cell uropods upon cell pol

287 Expression and recruitment of TLR4 into the lipid rafts were significantly greater in rodent and hum

288 nexin A2, which are proteins associated with lipid rafts, were also identified.

289 ed rather than oxidized DJ-1 translocated to lipid rafts, where it associated with Lyn, an interactio

290 n recruitment to invadopodia is dependent on lipid rafts, whereas ezrin/moesin proteins mediate podop

291 Caveolae, specific lipid rafts which concentrate caveolins, harbor signalin

292 e involved in the formation of nanodomains ("lipid rafts"), which serve as important signaling platfo

293 dence suggests that membrane domains, termed lipid rafts, which are enriched in sphingomyeline and ch

294 on creates specific lipid domains, including lipid rafts, which determine the distribution of many me

295 e activation is key in TLR4 recruitment into lipid rafts, which in turn up-regulates NF-kappaB transl

296 could specifically induce the association of lipid rafts with autophagosomes for its RNA replication.

297 The association of lipid rafts with autophagosomes was specific to HCV, as

298 The association of lipid rafts with HCV-induced autophagosomes was confirme

299 cantly increased partitioning of BMPR1b into lipid rafts with increased SMAD1/5/8 and p38 signaling.

300 tion by preventing translocation of AKT into lipid rafts without altering the activation of receptor-