1 ered significantly by degree of beta-blocker lipid solubility.

2 pendent on general physical features such as lipid solubility and molar refractivity rather than on c

3 n of future neuroactive steroids because the lipid solubility and related accessibility properties of

4 ug resistance was negatively correlated with lipid solubility at physiological pH (r2 = 0.90, p < 0.0

5        The findings indicate that increasing lipid solubility decreases the spinal cord bioavailabili

6 s of the MOR-1 gene) and/or pharmacokinetic (lipid solubility) factors.

7 a-blockers by generation (early vs late) and lipid solubility (high vs low to moderate).

8                                 Although the lipid solubility of AZT, as determined by the 1-octanol/

9  modify host-parasite immunobiology, and the lipid solubility of plant oils might offer alternative,

10 sistance was also negatively correlated with lipid solubility (r2 = 0.88, p < 0.0001).

11 is difference appears to result from a lower lipid solubility rather than saturable [(14)C]ALA transp