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1 rugs [NSAIDs] or aspirin, 17 hormones, and 7 lipid-lowering agents).
2 mg/m2/d plus concomitant levothyroxine and a lipid-lowering agent.
3 l need for more effective and well tolerated lipid-lowering agents.
4 lglutaryl coenzyme A reductase and effective lipid-lowering agents.
5 umed selection biases in the prescription of lipid-lowering agents.
6 clinical investigation of ASBT inhibitors as lipid-lowering agents.
8 in all groups, changes were manageable with lipid-lowering agents and did not warrant discontinuatio
10 justment for the propensity to be prescribed lipid-lowering agents and other potential confounders, p
12 self-reported use of aspirin, beta-blockers, lipid-lowering agents, and their combinations in all CAD
13 torial risk reduction with statins and other lipid-lowering agents, antihypertensive therapies, and a
18 ntion, including aspirin, beta-blockers, and lipid-lowering agents, are underused in these women, esp
20 blocker, and 20% to 25% were not receiving a lipid-lowering agent at 90 days after the index test.
21 her potential confounders, prescription of a lipid-lowering agent at discharge remained associated wi
22 ercury II-Compare the Efficacy and Safety of Lipid Lowering Agents Atorvastatin and Simvastatin With
25 ant differences in other lipids, BMI, use of lipid lowering agents, blood pressure, or smoking status
26 ibility that S1P inhibitors may be effective lipid-lowering agents, but they suggest that nearly comp
30 The recent FIELD study demonstrated that the lipid-lowering agent fenofibrate significantly reduces t
31 et drugs, beta-blockers, ACE inhibitors, and lipid-lowering agents, have proven efficacy in reducing
32 equently, thyromimetic drugs hold promise as lipid-lowering agents if adverse effects can be avoided.
33 alpha ligand that has been widely used as a lipid-lowering agent in the treatment of hypertriglyceri
34 IV infection include lifestyle modification, lipid-lowering agents, insulin sensitizers, and treatmen
35 e implications regarding the clinical use of lipid-lowering agents, like fibric acids, which can acti
36 and older: all individuals who had received lipid-lowering agents (LLAs); all individuals with a cli
39 s Heart Study participants who were using no lipid-lowering agent (n = 2,124) or using only a statin
40 te coronary syndromes who were discharged on lipid-lowering agents (n=3653) with those who were not (
41 reviously: greater use of and more effective lipid-lowering agents, no significant weight gain, no di
44 ted to the lipid pathway, and the effects of lipid-lowering agents on reducing the incidence of OA.
45 in those who used systemic corticosteroids, lipid-lowering agents, or testosterone; those who exerci
46 or antiplatelets/anticoagulants (p<0.05) and lipid-lowering agents (p<0.001) and were maintained at 1
47 rs, and antithrombotic agents in addition to lipid-lowering agents, reduce events, and this may be at
52 4, the British government decided to allow a lipid-lowering agent to be sold as an over-the-counter m
53 emission tomography, changes in aspirin and lipid-lowering agent use was greater after computed tomo
55 ry of medicines, including statins and other lipid-lowering agents, was collected at both assessments
58 G-CoA) reductase, also known as statins, are lipid-lowering agents widely used in the prevention of c
59 Guidelines recommend combining additional lipid-lowering agents with a complementary mode of actio
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