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1 cyte marker gene expression and subcutaneous lipoatrophy.
2 ients but can worsen glucose homeostasis and lipoatrophy.
3 exercise-induced hypothalamic amenorrhea and lipoatrophy.
4 ssue basis of the increased REE shown in HIV lipoatrophy.
5 uscle is hypermetabolic in patients with HIV lipoatrophy.
6 resting hypermetabolism of patients with HIV lipoatrophy.
7 sed relationships between these variants and lipoatrophy.
8 ve been implicated in the development of HIV lipoatrophy.
9 ohypertrophy, and fat wasting, also known as lipoatrophy.
10 ients with relative leptin deficiency due to lipoatrophy.
11  low levels of adiponectin and leptin due to lipoatrophy.
12 ood model for humans with severe generalized lipoatrophy.
13 le in causing the metabolic complications of lipoatrophy.
14 5%; P = .02) and were less likely to develop lipoatrophy after adjustment for age, sex, race, and ART
15 rt stature, intrauterine growth restriction, lipoatrophy and a facial gestalt involving a triangular
16 ockout mice exhibit a prominent phenotype of lipoatrophy and are resistant to high-fat diet-induced o
17      Pparg(Delta/Delta) mice exhibited total lipoatrophy and complete absence of sebaceous glands.
18  and dermal fibrosis as well as subcutaneous lipoatrophy and counteracted the up-regulation of collag
19                                     Variable lipoatrophy and cutis laxa are the basis for a progeroid
20 ence of both insulin and adiponectin, severe lipoatrophy and hyperlipidemia lead to lethality.
21            Ncb5or deficiency also results in lipoatrophy and increased hepatocyte sensitivity to cyto
22 nectin levels, in HIV-infected patients with lipoatrophy and insulin resistance.
23 maraviroc counterregulated ritonavir-induced lipoatrophy and interlelukin-6 gene expression in epidid
24  in children with atrophic disorders such as lipoatrophy and morphea.
25  of cells caused extreme growth retardation, lipoatrophy, and hypoglycemia, a clinical constellation
26 cells and the observable levels of fibrosis, lipoatrophy, and lymphocytic infiltration were reduced i
27 atitis, peripheral neuropathy, lipodystrophy/lipoatrophy, and pancreatitis, whereas the most common l
28 tion in patients with lipoatrophy, including lipoatrophy associated with HIV or highly active antiret
29 s poly-L-lactic acid were first approved for lipoatrophy associated with human immunodeficiency virus
30 IRS-1 and IRS-3 developed early-onset severe lipoatrophy associated with marked hyperglycemia, hyperi
31           Increased REE in subjects with HIV lipoatrophy cannot be explained by differences in organ-
32  resulting in similar growth retardation and lipoatrophy, caused diabetes without beta-cell hyperplas
33            Severe adipose tissue deficiency (lipoatrophy) causes insulin-resistant diabetes, elevated
34                                 Subcutaneous lipoatrophy characteristically accompanies dermal fibros
35 aplogroup J gave relative protection against lipoatrophy during ART in A5005s.
36 erized by a prematurely aged appearance with lipoatrophy, epidermal and dermal atrophy along with hyp
37                                       Facial lipoatrophy (FLA) is associated with human immunodeficie
38 l filler in patients with HIV-related facial lipoatrophy (FLA).
39  Here we have identified evidence of partial lipoatrophy in Reep1 null mice in addition to prominent
40 ant role in the pathogenesis of fibrosis and lipoatrophy in SSc.
41 roves metabolic dysfunction in patients with lipoatrophy, including lipoatrophy associated with HIV o
42 strated positive effects of rosiglitazone on lipoatrophy; insulin sensitivity; and metabolic indices,
43      Antiretroviral therapy (ART)-associated lipoatrophy involves mitochondrial dysfunction.
44  immunodeficiency virus (HIV)-related facial lipoatrophy is a devastating adverse effect of antiretro
45            Focal adipose deficiency, such as lipoatrophy, lumpectomy or facial trauma, is a formidabl
46 nal obesity and stavudine-induced peripheral lipoatrophy, might contribute to the higher prevalence o
47 ed by a progressive cellular and peristromal lipoatrophy of epithelial cell cytology and metabolic pa
48    We find that Lpin1 null mutations lead to lipoatrophy of the epineurium, and to the dysregulation
49                                              Lipoatrophy persisted in KO animals in which the confoun
50  although less severe, is reminiscent of the lipoatrophy phenotype observed in the most common form o
51 t function, as suggested by the diabetes and lipoatrophy phenotypes in Ncb5or null mice.
52 tients with HIV-associated FLA, a Carruthers Lipoatrophy Severity Scale (CLSS) grade of 2 or greater
53 asured REE remained significantly greater in lipoatrophy subjects after routine adjustment for lean b
54                                      The HIV lipoatrophy syndrome is characterized by loss of subcuta
55 y estimate a higher prevalence of peripheral lipoatrophy than does MRI in HIV+ subjects.
56 E was significantly greater in subjects with lipoatrophy than in control subjects.
57 lammation, dermal fibrosis, and subcutaneous lipoatrophy via PPAR-gamma in a mouse model of scleroder
58  need to be different in those patients with lipoatrophy when compared with patients with central fat
59     aP2-cre-mediated GRP78 deletion leads to lipoatrophy with approximately 90% reduction in gonadal
60 d an age-dependent loss of adipose tissue or lipoatrophy, with all observed adipose depots dramatical

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