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1 cyte marker gene expression and subcutaneous lipoatrophy.
2 ients but can worsen glucose homeostasis and lipoatrophy.
3 exercise-induced hypothalamic amenorrhea and lipoatrophy.
4 ssue basis of the increased REE shown in HIV lipoatrophy.
5 uscle is hypermetabolic in patients with HIV lipoatrophy.
6 resting hypermetabolism of patients with HIV lipoatrophy.
7 sed relationships between these variants and lipoatrophy.
8 ve been implicated in the development of HIV lipoatrophy.
9 ohypertrophy, and fat wasting, also known as lipoatrophy.
10 ients with relative leptin deficiency due to lipoatrophy.
11 low levels of adiponectin and leptin due to lipoatrophy.
12 ood model for humans with severe generalized lipoatrophy.
13 le in causing the metabolic complications of lipoatrophy.
14 5%; P = .02) and were less likely to develop lipoatrophy after adjustment for age, sex, race, and ART
15 rt stature, intrauterine growth restriction, lipoatrophy and a facial gestalt involving a triangular
16 ockout mice exhibit a prominent phenotype of lipoatrophy and are resistant to high-fat diet-induced o
18 and dermal fibrosis as well as subcutaneous lipoatrophy and counteracted the up-regulation of collag
23 maraviroc counterregulated ritonavir-induced lipoatrophy and interlelukin-6 gene expression in epidid
25 of cells caused extreme growth retardation, lipoatrophy, and hypoglycemia, a clinical constellation
26 cells and the observable levels of fibrosis, lipoatrophy, and lymphocytic infiltration were reduced i
27 atitis, peripheral neuropathy, lipodystrophy/lipoatrophy, and pancreatitis, whereas the most common l
28 tion in patients with lipoatrophy, including lipoatrophy associated with HIV or highly active antiret
29 s poly-L-lactic acid were first approved for lipoatrophy associated with human immunodeficiency virus
30 IRS-1 and IRS-3 developed early-onset severe lipoatrophy associated with marked hyperglycemia, hyperi
32 resulting in similar growth retardation and lipoatrophy, caused diabetes without beta-cell hyperplas
36 erized by a prematurely aged appearance with lipoatrophy, epidermal and dermal atrophy along with hyp
39 Here we have identified evidence of partial lipoatrophy in Reep1 null mice in addition to prominent
41 roves metabolic dysfunction in patients with lipoatrophy, including lipoatrophy associated with HIV o
42 strated positive effects of rosiglitazone on lipoatrophy; insulin sensitivity; and metabolic indices,
44 immunodeficiency virus (HIV)-related facial lipoatrophy is a devastating adverse effect of antiretro
46 nal obesity and stavudine-induced peripheral lipoatrophy, might contribute to the higher prevalence o
47 ed by a progressive cellular and peristromal lipoatrophy of epithelial cell cytology and metabolic pa
48 We find that Lpin1 null mutations lead to lipoatrophy of the epineurium, and to the dysregulation
50 although less severe, is reminiscent of the lipoatrophy phenotype observed in the most common form o
52 tients with HIV-associated FLA, a Carruthers Lipoatrophy Severity Scale (CLSS) grade of 2 or greater
53 asured REE remained significantly greater in lipoatrophy subjects after routine adjustment for lean b
57 lammation, dermal fibrosis, and subcutaneous lipoatrophy via PPAR-gamma in a mouse model of scleroder
58 need to be different in those patients with lipoatrophy when compared with patients with central fat
60 d an age-dependent loss of adipose tissue or lipoatrophy, with all observed adipose depots dramatical
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