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1 nal carcinoma, two chromophobe type, and one lipoma).
2 mour cell types, including fat-cell tumours (lipomas).
3 confidence rating of 4 or 5 as positive for lipoma.
4 well differentiated liposarcomas or atypical lipomas.
5 l segment rearranged in a subset of ordinary lipomas.
6 mosomal segment often rearranged in ordinary lipomas.
7 e and efficacious treatment of large colonic lipomas.
8 ce imaging (MRI) findings of rare pancreatic lipomas.
9 , macroscopic fat (<-30 HU) was present in 9 lipomas.
10 bile duct, stomach, oral cavity tumors, and lipomas.
11 gn ocular tumors, and central nervous system lipomas.
12 low accuracy in the diagnosis of soft-tissue lipomas.
13 ental ages, a cerebellar tumor, and multiple lipomas.
14 on, and have an abnormally high incidence of lipomas.
15 ntial role for HMG I-C in the development of lipomas.
16 nt to perturb adipogenesis and predispose to lipomas.
17 nign tumors of mesenchymal origin, including lipomas.
18 HMGI-C truncation in the generation of human lipomas.
19 ng multiple angiofibromas, collagenomas, and lipomas.
21 tients had myelodysplastic lesions (19 filar lipoma, 14 syringomyelia, 10 intradural lipoma, eight de
22 astomas, 18 (6%) with fibromas, 12 (4%) with lipomas, 28 (9%) with other benign primary cardiac tumor
23 expression was detected in 7 of 11 ordinary lipomas (63.6%) with alterations at 12q14-15 and in one
25 ding frontonasal dysplasia, interhemispheric lipoma, agenesis of the corpus callosum, tibial hemimeli
30 39 well differentiated adipose neoplasms (19 lipomas and 20 ALTs) of known karyotype using polyclonal
31 ith histologically verified fatty tumors (35 lipomas and 25 well-differentiated liposarcomas) were re
32 motor dysfunction, subcutaneous and visceral lipomas and hemangiomas, and intestinal juvenile polypos
35 nal juvenile polyposis, developmental delay, lipomas, and pigmentation spots of the male genitalia.
36 er syndromes characterized by overgrowth and lipomas are part of the PTEN syndrome spectrum, we ascer
40 , progressive skeletal deformities, invasive lipomas, benign and malignant tumors, and deep venous th
41 ertained six individuals with overgrowth and lipomas but who did not meet the diagnostic criteria for
47 s tumors, epithelioid sarcomas, spindle cell lipomas, dermatofibrosarcoma protuberans, and myofibrobl
48 methemoglobin), 3.lipid-containing lesions (lipoma, dermoid cyst, implanted fatty materials, laminar
49 ilar lipoma, 14 syringomyelia, 10 intradural lipoma, eight dermal sinus, five diastematomyelia, five
52 n particular, increased abundance of fat and lipomas, features strikingly similar to those observed i
53 differentiated mesenchymal tumors, including lipomas, fibroadenomas of the breast, salivary gland ade
56 sion of other mesenchymal tumors (leiomyoma, lipoma, gastrointestinal stromal tumor, leiomyosarcoma,
57 feration of mesenchymal cells in leiomyomas, lipomas, hamartomas,and other diseases has been linked t
58 sferase 3) with stroke, and the LHFPL2 gene (lipoma high mobility group protein I-C fusion partner-li
60 f hair cell stereocilia (TMHS, also known as lipoma HMGIC fusion partner-like 5, LHFPL5), both though
61 nding genes, nucleolar protein 4 (NOL4), and lipoma HMGIC fusion partner-like protein 4 (LHFPL4), whi
64 study examined the prevalence of pancreatic lipomas in a sample of patients undergoing abdominal com
65 dministered, are implicated in the growth of lipomas in different anatomic locations including the ep
67 ontaining preferred translocation partner in lipoma), induced by constitutive activation of STAT5, bu
68 ic the mesenchymal submucosal tumors such as lipoma, leiomyoma, and gastrointestinal stromal tumors.
70 69%), including those with either classic or lipoma-like histology, had rearrangements of the 8q12 PL
76 This is the first case of intramedullary lipoma of the spinal cord that may be related to steroid
79 tivity was demonstrated in only two ordinary lipomas: one with 6p21 rearrangement and one with normal
81 a family of proteins that also includes the lipoma preferred partner (LPP) and thyroid receptor-inte
85 Here, we found that CAFs upregulated the lipoma-preferred partner (LPP) gene in microvascular end
86 d receptor-interacting protein 6 (TRIP6) and lipoma-preferred partner (LPP), but not to zyxin itself.
88 rare, karyotypically distinct group of human lipomas, rearrangements of 6p21-23 produce internal dele
89 ed with development of human tumors, such as lipomas, relatively few examples exist of germline mutat
90 igations of primary cardiac tumors (myxomas, lipomas, rhabdomyomas, and fibromas) have provided insig
93 pillary fibroelastomas (SMR 3.17, P=0.0003), lipomas (SMR 5.0, P=0.0003), other benign tumors (SMR 4.
95 k features of BRR: macrocephaly and multiple lipomas, the latter of which occur in a minority of indi
96 ons consisted of six adenomatoid tumors, two lipomas, two epidermoid inclusion cysts, two cases of sa
97 differentiated mesenchymal tumors including lipomas, uterine leiomyomas, and pulmonary chondroid ham
99 he tendon sheath, epidermal inclusion cysts, lipomas, vascular lesions, peripheral-nerve tumours, ski
101 peritoneal hernia repair, the anatomy of the lipomas was studied both at the time of surgery and agai
109 88, average: 65.6 years), with 13 pancreatic lipomas, whose cases constituted the basis for 10 contra
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