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1 eplaced instead by a novel truncated form of lipomannan.
2 nown intermediate and of the known precursor lipomannan.
3 ol mannosides and linear and mature branched lipomannan and lipoarabinomannan are prominent phospholi
4 cothiol, but phosphatidylinositol mannoside, lipomannan and lipoarabinomannan levels were not altered
5 sence of the phosphatidylinositol-containing lipomannan and lipoarabinomannan, replaced instead by a
6 hway of phosphatidyl-myo-inositol mannoside, lipomannan, and lipoarabinomannan, which are key glycoli
7 way of phosphatidyl-myo-inositol mannosides, lipomannan, and lipoarabinomannan, which are key glycoli
9 phosphatidyl-myo-inositol mannosides (PIMs), lipomannan, and mannose-capped lipoarabinomannan (ManLAM
10 smegmatis produced increased levels of LAM, lipomannan, and phosphatidylinositol mannosides (PIMs) c
11 e and thus indirectly for lipoarabinomannan, lipomannan, and the higher-order phosphatidyl-myo-inosit
12 rans, attached to either a galactofuran or a lipomannan, are the primary constituents of mycobacteria
13 e lipoglycans, such as lipoarabinomannan and lipomannan, but a mechanism for lipoglycans to traffic f
14 etion of NCgl1505 resulted in the absence of lipomannan (Cg-LM-A), lipoarabinomannan (Cg-LAM) and a m
15 sulted in the formation of a novel truncated lipomannan (Cg-t-LM) and a complete ablation of LM/LAM b
18 only 5 to 20 Manp residues as compared with lipomannan from the wild type strain consisting of 21-34
19 unrecognized feature of the biosynthesis of lipomannan/lipoarabinomannan allows a significant revisi
21 e essential, mycobacterial genes involved in lipomannan (LM) and lipoarabinomannan (LAM) biosynthesis
24 n altered growth and inability to synthesize lipomannan (LM) but accumulation of a previously unchara
25 (PI)-containing lipoarabinomannan (LAM) and lipomannan (LM) of Mycobacterium spp. follows a conserve
26 phosphatidylinositol mannoside-6 (PIM6) and lipomannan (LM) were identified as factors responsible f
27 actions and spheroplasts showed that LAM and lipomannan (LM) were primarily found in a cell wall-enri
29 .tb-exposed cell envelope molecules, such as lipomannan (LM), contain subtle structural variations th
33 ates that the biosynthetic precursor of LAM, lipomannan (LM), which is also present in the cell wall,
35 ted Corynebacterineae synthesize a family of lipomannans (LM) and lipoarabinomannans (LAM) that are a
36 using LAMs and their biosynthetic precursor lipomannans (LMs) isolated from different mycobacterial
37 was mannosyl-lipoarabinomannan, followed by lipomannan, phosphatidylinositol mannoside, arabinosyl-l
38 ght/mass spectrometry of the mutated form of lipomannan shows a family of phosphatidylinositol-anchor
39 erase, required for the proper elongation of lipomannan to its normal state and subsequent synthesis
40 In contrast with the MR, the PIM(2)f and lipomannan were recognized by DC-SIGN comparable to high
41 ws a family of phosphatidylinositol-anchored lipomannans with from only 5 to 20 Manp residues as comp
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