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1 onocyte recruitment, foam cell formation and lipoprotein metabolism.
2 activity and also its role in plasma SM and lipoprotein metabolism.
3 erse alterations in glucose, fatty acid, and lipoprotein metabolism.
4 11 genes have been previously implicated in lipoprotein metabolism.
5 ins and in humans plays an important role in lipoprotein metabolism.
6 glyceride, and thereby plays a major role in lipoprotein metabolism.
7 PGC-1alpha/HNF4alpha partnership in hepatic lipoprotein metabolism.
8 netic linkages exist between body weight and lipoprotein metabolism.
9 hospholipid transfer protein are involved in lipoprotein metabolism.
10 influence on body composition and peripheral lipoprotein metabolism.
11 ral SREBP1c and LXR target genes involved in lipoprotein metabolism.
12 g PDZK1 as a novel regulator of high-density lipoprotein metabolism.
13 eptor SR-BI play critical roles in lipid and lipoprotein metabolism.
14 nal fashion, significantly affecting overall lipoprotein metabolism.
15 otein C-III (apoC-III) is a key regulator of lipoprotein metabolism.
16 lial lipase and its relation to high-density lipoprotein metabolism.
17 cterizing the effects of PCSK9 inhibitors on lipoprotein metabolism.
18 ase plays a significant role in high-density lipoprotein metabolism.
19 ic effects of hormone-replacement therapy on lipoprotein metabolism.
20 f phospholipid transfer protein in lipid and lipoprotein metabolism.
21 ondition in mice, independent of its role in lipoprotein metabolism.
22 tial therapeutic alterations of high-density lipoprotein metabolism.
23 c lipase (HL) has a well-established role in lipoprotein metabolism.
24 l cholesterol efflux and plasma high density lipoprotein metabolism.
25 important roles of TR4 in the modulation of lipoprotein metabolism.
26 way whereby LXR and its ligands may modulate lipoprotein metabolism.
27 enic risk that are separate from its role in lipoprotein metabolism.
28 is novel lipase have a significant effect on lipoprotein metabolism.
29 vitro and in vivo, and may thereby influence lipoprotein metabolism.
30 triglyceride and its fatty acids, including lipoprotein metabolism.
31 have a major influence on hepatic lipid and lipoprotein metabolism.
32 n that plays a key role in regulating plasma lipoprotein metabolism.
33 in-1 overexpression in mouse liver on plasma lipoprotein metabolism.
34 mals, we characterized several parameters of lipoprotein metabolism.
35 nd physiological and behavioral variables on lipoprotein metabolism.
36 ings describe a novel pathway for regulating lipoprotein metabolism.
37 largely liver-derived and known to regulate lipoprotein metabolism.
38 n in apo B transgenic mice, without altering lipoprotein metabolism.
39 iated with multiple alterations in lipid and lipoprotein metabolism.
40 en great interest in the effects of MUFAs on lipoprotein metabolism.
41 e mechanisms by which apo E modulates plasma lipoprotein metabolism.
42 ns between body fat, insulin metabolism, and lipoprotein metabolism.
43 ural features of apoB that are important for lipoprotein metabolism.
44 olipid biosynthesis, choline metabolism, and lipoprotein metabolism.
45 observed effects of n-3 fatty acids on human lipoprotein metabolism.
46 hance the value of mice as a model for human lipoprotein metabolism.
47 yme hepatic lipase may play several roles in lipoprotein metabolism.
48 es of these two similar lipolytic enzymes in lipoprotein metabolism.
49 The mouse is a popular animal model for lipoprotein metabolism.
50 at malabsorption or genetic abnormalities in lipoprotein metabolism.
51 te protein is inactive in lipid transfer and lipoprotein metabolism.
52 terol esterase may play an important role in lipoprotein metabolism.
53 apolipoproteins and enzymes participating in lipoprotein metabolism.
54 howing its physiological role in endothelial lipoprotein metabolism.
55 tin-like protein 3 (ANGPTL3), a regulator of lipoprotein metabolism.
56 oprotein E are involved in triglyceride-rich lipoprotein metabolism.
57 erotic disease pathophysiology by regulating lipoprotein metabolism.
58 enhancers of genes linked to fatty acid and lipoprotein metabolism.
59 ecreased in number and exhibit dysfunctional lipoprotein metabolism.
60 acellular matrix organization, and lipid and lipoprotein metabolism.
61 nappreciated complexity of triglyceride-rich lipoprotein metabolism.
62 alterations e.g. in energy, amino acid, and lipoprotein metabolism.
63 cts of hepatic insulin action on glucose and lipoprotein metabolism.
64 s enriched in pathways involved in lipid and lipoprotein metabolism.
65 ne phospholipase activity and is involved in lipoprotein metabolism.
66 thogenesis of NAFLD and in regulating plasma lipoprotein metabolism.
67 drome, long-term physical activity, diet and lipoprotein metabolism.
68 which was attributed to the dysregulation of lipoprotein metabolism.
69 g the apolipoprotein E protein that mediates lipoprotein metabolism.
70 ch as host defense, tissue repair, and lipid/lipoprotein metabolism.
71 no animal model entirely recapitulates human lipoprotein metabolism.
72 ting translatable model of human hepatic and lipoprotein metabolism.
73 portance of microRNAs (miRNAs) in regulating lipoprotein metabolism.
74 ice, a well-established model for human-like lipoprotein metabolism.
75 nesis, independent of its regulatory role in lipoprotein metabolism.
76 hin the locus, as sortilin regulates hepatic lipoprotein metabolism.
77 ors, heart disease risk reduction, and human lipoprotein metabolism.
78 ms for FXR-dependent regulation of lipid and lipoprotein metabolism.
79 expression of several key genes involved in lipoprotein metabolism.
80 ervous system and classically exert roles in lipoprotein metabolism.
81 Tregs inhibit atherosclerosis by modulating lipoprotein metabolism.
82 pression of key intestinal genes involved in lipoprotein metabolism.
83 with close similarity to humans in terms of lipoprotein metabolism.
84 l signaling, regulating different aspects of lipoprotein metabolism.
85 the fetal liver, was metabolically active in lipoprotein metabolism.
86 transfer protein (PLTP) activity influences lipoprotein metabolism.
87 understanding of the molecular physiology of lipoprotein metabolism.
88 ch had never been previously associated with lipoprotein metabolism.
89 fatty acid beta oxidation, and high-density lipoprotein metabolism.
90 d has other potentially favorable actions on lipoprotein metabolism.
91 which has not previously been implicated in lipoprotein metabolism.
92 scores of loci not previously implicated in lipoprotein metabolism.
93 tially overlapping or complementary roles in lipoprotein metabolism.
94 undant role of HL and EL in apolipoprotein B lipoprotein metabolism.
95 tion and this has consequences for lipid and lipoprotein metabolism.
96 many involved in cholesterol, bile acid, and lipoprotein metabolism.
97 lipids, mediating altered functionalities in lipoprotein metabolism - affecting the risk of coronary
98 (TM6SF2) gene has been implicated in plasma lipoprotein metabolism, alcoholic and non-alcoholic fatt
99 e formation, signal transduction, and plasma lipoprotein metabolism, all of which may well have an im
101 rect in vivo effects of the apoE isoforms on lipoprotein metabolism and atherosclerosis are not yet f
102 f Apoc3 antisense oligonucleotides (ASOs) on lipoprotein metabolism and atherosclerosis in a mouse mo
103 ortant to learn how this receptor influences lipoprotein metabolism and atherosclerosis in people.
104 r CHD and may help better define the role of lipoprotein metabolism and atherosclerosis in the pathog
105 study was to investigate the role of SCD1 in lipoprotein metabolism and atherosclerosis progression.
107 ng function of hepatic lipase (HL) to plasma lipoprotein metabolism and atherosclerosis, we compared
109 ew the role of brown and beige adipocytes in lipoprotein metabolism and atherosclerosis, with evidenc
111 receptor family that play important roles in lipoprotein metabolism and atherosclerotic disease.
112 es a variety of genes that may contribute to lipoprotein metabolism and atherosclerotic lesion format
113 ed to be mediated indirectly by an effect on lipoprotein metabolism and by a direct effect on the ves
114 APOA1/C3/A4/A5 are key components modulating lipoprotein metabolism and cardiovascular disease risk.
115 Originally recognized for their role in lipoprotein metabolism and cardiovascular disease, apoli
117 rotein that plays an important role in lipid/lipoprotein metabolism and cardiovascular diseases.
118 ) with lipid surfaces plays crucial roles in lipoprotein metabolism and cholesterol homeostasis.
120 oviding a potentially important link between lipoprotein metabolism and distal PPAR alpha transcripti
121 ression has potentially important effects on lipoprotein metabolism and foam-cell lesion development.
122 n-protein interactions, may be important for lipoprotein metabolism and for protein-membrane binding.
123 s into the mechanisms governing high-density lipoprotein metabolism and function and open new avenues
124 ation signals should expand our knowledge of lipoprotein metabolism and generate targets for pharmaco
125 rease our understanding of the regulation of lipoprotein metabolism and hepatic lipoprotein export an
128 d HDL receptor activities of SR-BI in normal lipoprotein metabolism and in SR-BI's ability to protect
130 ys a rate-limiting role in triglyceride-rich lipoprotein metabolism and is expressed in most tissues.
131 Lipoprotein lipase (LPL) is a key enzyme for lipoprotein metabolism and is responsible for hydrolysis
134 ol intake from any type of beverage improves lipoprotein metabolism and lowers cardiovascular mortali
135 ascent HDL particles plays a central role in lipoprotein metabolism and macrophage cholesterol homeos
136 Apolipoprotein E (apoE) plays a key role in lipoprotein metabolism and may have other important biol
137 r SPT activity could result in regulation of lipoprotein metabolism and might have an impact on the d
138 of CVD risk factor traits (ie, indicators of lipoprotein metabolism and oxidative stress) were measur
141 th the liver being the main organ modulating lipoprotein metabolism and plasma lipid levels) uptake s
142 CA1 modulates HDL as well as apoB-containing lipoprotein metabolism and reduces atherosclerosis in vi
143 (CETP) plays a central role in high density lipoprotein metabolism and reverse cholesterol transport
144 linical and genetic-based factors related to lipoprotein metabolism and risk for AMD in the All of Us
146 evidence for a novel regulatory pathway for lipoprotein metabolism and suggest that modulation of th
147 esis of proteins involved in cholesterol and lipoprotein metabolism and the coagulation cascade.
148 hanisms by which hepatic lipase may modulate lipoprotein metabolism and the development of atheroscle
149 ies, there is a strong focus on high-density lipoprotein metabolism and the reverse cholesterol trans
150 isms by which these apolipoproteins regulate lipoprotein metabolism and thereby influence vascular bi
151 Therefore, we aimed to further explore their lipoprotein metabolism and to characterize key hepatic s
152 evelop a broader biological understanding of lipoprotein metabolism and to identify new therapeutic o
153 the etiology of ARM include cholesterol and lipoprotein metabolism and transport, extracellular matr
155 causal effects of lipoprotein(a) on overall lipoprotein metabolism and we assess potential pleiotrop
156 eins that may be linked to altered immunity, lipoprotein metabolism, and accelerated vasculopathy in
159 energy delivery to tissues and in modulating lipoprotein metabolism, and could impact on atherogenesi
160 ting from hyperinsulinemia, abnormalities of lipoprotein metabolism, and endothelial dysfunction, wil
161 eryl ester, plays a key role in high-density lipoprotein metabolism, and has been believed to be crit
162 postprandially, on liver injury, glucose and lipoprotein metabolism, and markers of early atheroscler
163 e as a key regulator in apoptosis, lipid and lipoprotein metabolism, and other cell regulatory pathwa
164 burden of atherosclerosis, triglyceride-rich lipoprotein metabolism, and platelet activation), or to
166 ical studies describing the role of SORT1 in lipoprotein metabolism, and recent work that has begun t
167 athophysiological link between inflammation, lipoprotein metabolism, and the development of atheroscl
168 ereas four metabolic covariates representing lipoprotein metabolism (apoAII, apoAI, triglycerides and
171 alterations in insulin signaling with plasma lipoprotein metabolism are incompletely understood.
172 most, if not all of the effects of PDZK1 on lipoprotein metabolism are likely because of the effects
174 with FL formation through the regulation of lipoprotein metabolism as hsa-miR-122-5p levels associat
175 hich is to stabilize surface pressure during lipoprotein metabolism as lipids move in and out of the
176 uman genetics to validate candidate genes in lipoprotein metabolism as well as in the functional vali
177 C3(rs138326449) loss of function mutation in lipoprotein metabolism, as well as the effects of LPL(rs
179 w the effects of apoE4 domain interaction in lipoprotein metabolism, atherosclerosis, and neurodegene
180 proliferation, motility and differentiation, lipoprotein metabolism, blood coagulation, inflammation,
181 of distinct biological effects on lipid and lipoprotein metabolism, blood pressure, platelet functio
182 only from underlying species differences in lipoprotein metabolism but also from differences in expe
183 lts indicate that apoCIII not only modulates lipoprotein metabolism but also may directly contribute
184 use appropriate animal models when studying lipoprotein metabolism but also to feed the animals comp
185 relevant to genes with known roles in plasma lipoprotein metabolism but has, thus far, failed to iden
186 gh-density lipoprotein (HDL) is critical for lipoprotein metabolism, but despite its importance, the
187 important role in the liver in high-density lipoprotein metabolism, but it is also thought to partic
188 eted and multifunctional player in lipid and lipoprotein metabolism, but much additional work will be
190 n animal tissues and plays multiple roles in lipoprotein metabolism, but the function of peripheral P
192 ( approximately 150 ng/ml), apoA-V modulates lipoprotein metabolism by binding to glycosylphosphatidy
193 or ApoF in the control of plasma and hepatic lipoprotein metabolism by favoring VLDL-TG secretion and
194 hepatic heparan sulfate in triglyceride-rich lipoprotein metabolism by inactivating the biosynthetic
197 ism, separable from the role of HL in plasma lipoprotein metabolism, by which HL modulates atherogeni
200 tions indicate that a primary disturbance in lipoprotein metabolism can result in several traits asso
201 f the energy metabolism and the lipid/sterol/lipoprotein metabolism categories revealed that CAG leng
202 lesterolemia (FH) is an inherited disease of lipoprotein metabolism caused by a defect in the LDL rec
204 te genes APOA1/C3/A4/A5 cluster (involved in lipoprotein metabolism), COL22A1, CDO1, CTNAA2, and CYP4
205 To further evaluate the role of SR-BI in lipoprotein metabolism, compound apolipoprotein E knock-
206 -BI) in hepatocytes in vivo, and thus normal lipoprotein metabolism, depend on its four PDZ domain (P
209 and its potential role in the modulation of lipoprotein metabolism during inflammatory conditions, i
210 F1 gene allelic variants on serum indices of lipoprotein metabolism, early markers of asymptomatic at
212 ular and mitochondrial fatty acid transport, lipoprotein metabolism, fatty acid oxidation, branched-c
213 A role for hepatocyte heparan sulfate in lipoprotein metabolism has now been genetically establis
215 ent role in cholesterol transport and plasma lipoprotein metabolism, has recently emerged as a major
218 insulin, HDL subfractions, and indicators of lipoprotein metabolism (HDL-cholesterol fractional ester
219 y crucial role in various processes, such as lipoprotein metabolism, hemostasis, fetal development, e
220 ps support an important role for sortilin in lipoprotein metabolism; however, the directionality of t
221 atherosclerosis-prone model with a humanized lipoprotein metabolism, IC7Fc markedly lowered plasma tr
222 we examine the complex interactions between lipoprotein metabolism, immunosuppressive drug therapy,
224 expression of several key genes involved in lipoprotein metabolism in a subgroup of participants (n
227 further found that the impairment in TG-rich lipoprotein metabolism in gammaEC/BM-KO mice was associa
229 the role of insulin resistance and abnormal lipoprotein metabolism in NASH, determining the pathogen
233 ascular disease prevention but its impact on lipoprotein metabolism in subjects with dyslipidemia ass
236 ripe for new insights into how LPL-mediated lipoprotein metabolism in the brain impacts CNS processe
237 role in reproductive processes, in lipid and lipoprotein metabolism in the central nervous system, an
239 thesis that EL plays a physiological role in lipoprotein metabolism in vivo, we have used gene target
241 therogenic and alters hepatic and macrophage lipoprotein metabolism, in part, by enhancing uptake of
242 esults in diverse abnormalities of lipid and lipoprotein metabolism, in particular hypertriglyceridae
243 and DHA have differences in their effects on lipoprotein metabolism, in which EPA, with a more potent
244 cates that statins exert multiple effects on lipoprotein metabolism, including chylomicrons and HDLs.
245 s major monogenic and polygenic disorders of lipoprotein metabolism, including familial hypercholeste
246 a theoretical framework for cholesterol and lipoprotein metabolism; information on the role of chole
248 tionship between lipid transfer proteins and lipoprotein metabolism is expected to be an important fr
254 pe 9 (PCSK9), a key regulator of low-density lipoprotein metabolism, is induced by leptin and resisti
255 c) particles are impaired in some aspects of lipoprotein metabolism, it is of upmost interest to bioc
256 ietin-like 3 (ANGPTL3) is a key regulator of lipoprotein metabolism, known for its potent inhibition
257 t these two proteins play essential roles in lipoprotein metabolism: liver-derived Angptl3 inhibits l
259 ts of ANGPTL3 deficiency on both glucose and lipoprotein metabolism make it an attractive therapeutic
262 ds during liver dysfunction, or disorders of lipoprotein metabolism, may have important implications
263 r other mouse lines featuring alterations in lipoprotein metabolism, may provide new mouse models for
264 A limitation is that, owing to the nature of lipoprotein metabolism, measures related to the composit
265 e understanding of how these miRNAs modulate lipoprotein metabolism promises to reveal new therapeuti
266 isease related abnormalities of postprandial lipoprotein metabolism related to clearance mechanisms f
267 of lipoprotein receptor activity influences lipoprotein metabolism, related physiology and pathophys
268 of action of PCSK9 monoclonal antibodies on lipoprotein metabolism remains to be fully evaluated.
269 etabolic pathways of hepatic cholesterol and lipoprotein metabolism, resulting in lowering of plasma
271 e HIF-2alpha regulates genes associated with lipoprotein metabolism, ribosome biogenesis and E2F and
273 In addition to their prominent effects on lipoprotein metabolism, statins can regulate the small G
274 on loci identify genes involved in lipid and lipoprotein metabolism, substrate transport and inflamma
276 ndent regulation of hepatic SR-BI and, thus, lipoprotein metabolism supports the proposal that this a
277 sider the evidence that PCSK9 has effects on lipoprotein metabolism that are in addition to its role
278 o identify defects of apolipoprotein (apo) B lipoprotein metabolism that characterize hypertriglyceri
279 pattern B is a common inherited disorder of lipoprotein metabolism that has been shown to have a sig
280 ice, a well-established model for human-like lipoprotein metabolism that unlike hyperlipidemic Apoe(-
281 ally more important than group IIa enzyme in lipoprotein metabolism, that the sPLA(2) activities are
283 standard cell culture model of human hepatic lipoprotein metabolism, there is a limited availability
284 ANGPTL3, which were previously implicated in lipoprotein metabolism through classical wet bench appro
285 e demonstrate that LXRs also regulate plasma lipoprotein metabolism through control of the phospholip
286 indicate that WWOX disruption alters HDL and lipoprotein metabolism through several mechanisms and ma
289 est that FXR may play a role in high density lipoprotein metabolism via the regulation of PLTP gene e
290 ue within this family, Angptl3 and 4 inhibit lipoprotein metabolism via their ability to inhibit the
291 ddress the impact of liver-expressed PLTP on lipoprotein metabolism, we created a mouse model that ex
292 signaling pathways that could rapidly affect lipoprotein metabolism, we examined whether acute exposu
293 To evaluate the role of SR-BI in plasma lipoprotein metabolism, we have generated transgenic mic
294 To evaluate the in vivo role of PLTP in lipoprotein metabolism, we used homologous recombination
296 d fat on various aspects of carbohydrate and lipoprotein metabolism were evaluated in 10 healthy, pos
297 ntrations and sizes and proteins involved in lipoprotein metabolism were measured by calibrated diffe
298 ver histology and derangement in glucose and lipoprotein metabolism, which contribute to the presenta
299 chanisms by which different statins regulate lipoprotein metabolism will lead to improved strategies
300 In summary, apo A-II plays a complex role in lipoprotein metabolism, with some antiatherogenic proper