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1 letion of lmo2185 in the srtB region reduced listerial [(59) Fe]-Hn transport, and purified Lmo2185 b
3 s lytic activity potentiates presentation of listerial Ags by class I MHC and inhibits presentation v
5 he delayed-type hypersensitivity response to listerial antigen while not significantly altering cellu
8 stitute a significant proportion of the anti-listerial CD8+ T cell repertoire, the kinetics and magni
10 to a dominant MHC class II (H-2k)-restricted listerial determinant, when coinjected i.p. with murine
11 a pigs that led to the identification of 201 listerial genes important for infection of the placenta
12 ast cell culture systems were permissive for listerial growth and cell-to-cell spread and revealed th
13 ce gene expression significantly impairs the listerial growth rate (mu) and maximum growth (A) in ric
14 concentration, and the srtA-independence of listerial Hn/Hb uptake distinguished it from comparable
16 quired to recruit neutrophils to the site of listerial infection in the decidua basalis, and infectio
18 cules (CD40, CD80, and CD86) were induced by listerial infection only when the bacteria invaded the c
23 of CD8+ T lymphocytes in host resistance to listerial infections of the liver may be due to their ca
24 xplore the role of NK cells in resistance to listerial infections of the liver, lymphokine-activated
26 se severity (measured by lethality) and (ii) listerial infectivity (measured by liver and spleen colo
28 ate of unfavorable outcome among adults with listerial meningitis has increased over a 14-year period
29 RB6-8C5-treated mice appeared to result from listerial multiplication in the spleen and liver rather
30 al administration of a profoundly attenuated listerial mutant can safely elicit solid protective immu
31 Gbp10--conferred cell-autonomous immunity to listerial or mycobacterial infection within macrophages
32 previously unrecognized as playing a role in listerial pathogenesis were identified: knockdowns affec
35 n, indicating its potential significance for listerial persistence in the environment and for food sa
36 cV mutation precludes the binding of certain listerial phages and produces a profound attenuation cha
38 Hepatocytes constitute the principal site of listerial replication in the livers of mice infected i.v
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