ライフサイエンスコーパス: little is known about how

1 However, relatively little is known about how 3D material properties such as

2 N) often fire locked to sensory stimuli, but little is known about how 5-HT affects sensory processin

3 been associated with diabetes risk; however, little is known about how a long-term dietary interventi

4 However, little is known about how ACC activity is influenced by

5 Yet, relatively little is known about how acids are detected by gustator

6 However, little is known about how activity fluctuations of neuro

7 Little is known about how activity of prefrontal neurons

8 RATIONALE: Little is known about how acute kidney injury (AKI) reso

9 avioral and psychiatric vulnerabilities, but little is known about how adolescent VTA neurons encode

10 yperinflation, and alveolar enlargement, but little is known about how age affects small airways.

11 Remarkably little is known about how age affects the sensory signal

12 Despite their likely importance, little is known about how alternate supergene alleles ar

13 the progression of Alzheimer's disease, but little is known about how amyloid-beta deposition relate

14 he treatment of ammonia-rich wastewater, but little is known about how anammox bacteria grow and mult

15 Little is known about how and where effectors are secret

16 olorectal cancer development are well-known, little is known about how and whether epigenetic factors

17 However, remarkably little is known about how animals behaviourally respond

18 Little is known about how animals integrate multiple sen

19 nsation have been extensively characterized, little is known about how animals sense alkaline pH in t

20 Despite the prevalence of this virus, little is known about how antibodies in healthy adults p

21 ith the renal-risk genotype develop CKD, and little is known about how APOL1 variants drive disease.

22 Very little is known about how archaeal cells orchestrate tra

23 Little is known about how archaeal viruses perturb the t

24 However, little is known about how autophagy plays a role in the

25 Yet, little is known about how bodily states impact the expre

26 Little is known about how C. glabrata, an emerging patho

27 t sources of Ca(2+)during cell signaling but little is known about how Ca(2+)enters these stores.

28 However, little is known about how carboxysome biosynthesis and s

29 llular function in all kingdoms of life, yet little is known about how cell shape is controlled.

30 However, little is known about how cell-intrinsic circadian clock

31 Little is known about how cells assemble as systems duri

32 Surprisingly little is known about how cells control these enzymes, g

33 ed glucose causes beta-cell dysfunction, but little is known about how cells handle excess fuels to a

34 However, little is known about how cells sense and respond to mec

35 l-known toxicity of uranium (U) to bacteria, little is known about how cells sense and respond to U.

36 In spite of their essential role, little is known about how centromeres and KTs are assemb

37 cling in undisturbed Cerrado vegetation; yet little is known about how changes in land-use and land-c

38 ctural changes in the prefrontal cortex, but little is known about how changes in prefrontal neuronal

39 However, little is known about how changes in the bacterial commu

40 However, little is known about how cholesterol reduction affects

41 In contrast, little is known about how chromosomes are packed in undi

42 4(+) T cell activation and polarization, but little is known about how CK2 functions in T cells.

43 species' spatial distribution patterns, but little is known about how climate change may affect comm

44 nzyme activity and physiology, comparatively little is known about how climate influences N-fixing tr

45 Little is known about how clinical oncology concepts are

46 However, little is known about how co-occurrence structure change

47 However, little is known about how combinatorial complexity among

48 Little is known about how combining efficacious interven

49 However, little is known about how complex microbial communities

50 As of yet, little is known about how consistent the effects of viru

51 However, little is known about how consolidation processes may tr

52 e to the expression of long-term plasticity, little is known about how constitutive activation of spe

53 Little is known about how contextual processing and neur

54 ve been characterized over the past century, little is known about how coordination influences the st

55 Little is known about how Crc affects the hierarchy of m

56 Little is known about how CRISPR-Cas systems are regulat

57 However, little is known about how crowded DNA modulates facilita

58 However, little is known about how cues from fluid shear stress a

59 Little is known about how DAG kinase activity is regulat

60 However, relatively little is known about how defects in one liquid crystall

61 ridge between the foregut and epidermis, but little is known about how development of these three epi

62 Little is known about how different antiretrovirals effe

63 However, little is known about how different subsets of anti-infl

64 sures of financial conflicts of interest but little is known about how disclosures of other types of

65 However, little is known about how disorder influences cation tra

66 Little is known about how donor factors, timing of procu

67 Relatively little is known about how dopamine may modulate memories

68 y ecosystem responses to climate change, yet little is known about how drought influences plant-soil

69 ay in shaping dryland landscapes, relatively little is known about how drylands might respond to clim

70 r predicting carbon uptake and storage, very little is known about how eCO2 affects nutrient cycling

71 However, little is known about how electrical coupling strength i

72 However, little is known about how EMILINs may exert their extrac

73 pathogen exposure and epithelial injury, yet little is known about how enteropathogens may circumvent

74 substrate specificity of their transporters, little is known about how evolution of transporter subst

75 Little is known about how expanded insurance coverage af

76 nce on genetically determined circuitry, but little is known about how experience and genetics intera

77 Relatively little is known about how experience changes circuitry i

78 However, little is known about how exposure to childhood trauma i

79 Little is known about how extensively these data have be

80 In addition, little is known about how factors involved in PUFA metab

81 However, despite recent work [2, 4, 17-21], little is known about how flexible mouse innate defensiv

82 However, little is known about how functional connectivity betwee

83 While GA signalling is well understood, little is known about how GA is transported or how GA di

84 However, little is known about how genes are specifically affecte

85 Little is known about how genetic variation and epigenet

86 output side of these reciprocal connections, little is known about how granule cells are excited.

87 At present, relatively little is known about how growth cone collapse occurs pr

88 atial food webs, however to date practically little is known about how habitat configurational fragme

89 netic and signaling communications; however, little is known about how HGG heterogeneity may impact E

90 cal concentrations c-di-GMP is a monomer and little is known about how higher oligomeric complexes as

91 However, little is known about how histone methylation may regula

92 o the paucity of experimental animal models, little is known about how host factors interface with ba

93 en conformational changes of Hsp90; however, little is known about how Hsp90 operates under physiolog

94 Despite its importance, little is known about how human risk-preference can be m

95 o bind and inhibit PP2A are well understood, little is known about how Igo/Ensa are inactivated.

96 w from common lymphoid progenitor cells, but little is known about how ILC2s egress from the bone mar

97 ntrol many important signaling pathways, but little is known about how IMMPs interact with their subs

98 ater emotional regulation and cognition, but little is known about how infant social expressiveness d

99 However, little is known about how influenza virus evolves across

100 But little is known about how information about local object

101 ween object motion and self-motion, although little is known about how information about these distin

102 Despite this, little is known about how insulin regulates FOXO1 and ho

103 her pairwise stabilising processes, and thus little is known about how intransitivity interacts with

104 while it has negative physiological effects, little is known about how it affects human behavior.

105 tion and loss of cell-cell adhesions, though little is known about how it alters cell biophysical pro

106 spite more than a century of research on HR, little is known about how it is so tightly regulated and

107 Nonetheless, surprisingly little is known about how it shapes information processi

108 n depends on the Scc2-Scc4 complex; however, little is known about how it stimulates the cohesion-loa

109 However, little is known about how its structure is influenced by

110 Very little is known about how L-DOPA therapy affects the dyn

111 However, little is known about how life expectancy and mortality

112 However, surprisingly little is known about how LMP1 expression is regulated i

113 Little is known about how local PV interneurons communic

114 Little is known about how long chromosomal variation may

115 In human cells, little is known about how long noncoding RNAs (lncRNAs)

116 Little is known about how long the BCG vaccine protects

117 Yet, very little is known about how low-frequency Vm oscillations

118 meostasis and regulating signaling pathways, little is known about how lysosomal membrane protein lif

119 Little is known about how megakaryocytes may affect meta

120 However, little is known about how metabolic fluxes are coordinat

121 However, little is known about how microbial communities responde

122 st majority of protein-encoding transcripts, little is known about how miRNAs themselves are degraded

123 Little is known about how mitochondrial lipids reach inn

124 However, little is known about how mitotic RNA processing contrib

125 s has not yet been shown experimentally, and little is known about how multicellular signal processin

126 For poxviruses, little is known about how multiple adaptive mutations em

127 identifying and manipulating single engrams, little is known about how multiple engrams interact to i

128 by nucleoid-associated proteins (NAPs), but little is known about how NAP activity is coupled to cel

129 Little is known about how NGF elicits faster axon outgro

130 e implicated in nicotine's actions, but very little is known about how nicotinic acetylcholine recept

131 onal modifications, such as phosphorylation, little is known about how non-degradative ubiquitination

132 r from sources other than precipitation, yet little is known about how non-rainfall water inputs infl

133 Little is known about how olfactory inputs are processed

134 However, little is known about how or whether Abeta influences sy

135 In contrast, little is known about how other major global change driv

136 tional impact of reward-predictive cues, but little is known about how other neuromodulatory systems

137 e of metacognition for many aspects of life, little is known about how our metacognitive system opera

138 However, little is known about how PA-X induces host shutoff.

139 fections on the developing fetus, relatively little is known about how pathogens associated with cong

140 However, little is known about how patient outcomes have evolved.

141 al role in shaping the developing brain, yet little is known about how Pb perturbs NSC functions and

142 d in a "bottom-up" sensory-driven framework, little is known about how perceptual size information is

143 However, little is known about how perturbation of allosteric aut

144 However, little is known about how phage polyvalence (i.e., broad

145 However, little is known about how phenotypic switches arise and

146 However, relatively little is known about how phylogenetic patterns in envir

147 Background: Little is known about how physician time is allocated in

148 ns responsive to pitch have been identified, little is known about how pitch information is extracted

149 e key role of vegetation in carbon dynamics, little is known about how plants influence the source of

150 by which chilling is sensed, but relatively little is known about how plants sense freezing.

151 Cs) has been extensively studied, relatively little is known about how post-transcriptional modulatio

152 However, little is known about how postprandial sleep is regulate

153 However, little is known about how predicted temperature increase

154 Little is known about how pro-obesity diets regulate tis

155 Little is known about how projection neurons with distin

156 licing is regulated by phosphorylation, very little is known about how protein kinases phosphorylate

157 However, little is known about how Reelin controls the cytoskelet

158 e termini of their anteroposterior axis, but little is known about how regeneration signaling restore

159 In comparison, little is known about how regulation at the protein leve

160 problematic contaminant at nuclear sites and little is known about how repeated microbiologically med

161 ated with many inflammatory diseases, though little is known about how resident microbes may contribu

162 ers to EGFR inhibitors have been identified, little is known about how resistant clones evolve during

163 Little is known about how risk calculators inform clinic

164 to qualify as biomarkers for clinical care, little is known about how routine imaging acquisition te

165 auge the professional impact of a scientist, little is known about how scientific impact emerges and

166 ctivity in the mammalian olfactory bulb, yet little is known about how sensory input activates granul

167 However, little is known about how sleep neuron function is deter

168 However, relatively little is known about how slow hemodynamic oscillations

169 Despite the importance of social influences, little is known about how social interactions modulate v

170 ect large numbers of people and communities, little is known about how social structures affect menta

171 degradation is a serious global problem, but little is known about how soil microbial communities res

172 ntial effectors are poorly defined, and very little is known about how spacious vacuoles are formed a

173 However, little is known about how specific cell types within ind

174 espite the size and growth of this spending, little is known about how spending on each condition var

175 Little is known about how spontaneous attentional deploy

176 Little is known about how stony corals build their calca

177 However, little is known about how stopover habitat, essential fo

178 lities are often present in these disorders, little is known about how stress affects the sensory cor

179 However, very little is known about how stress response, neural proces

180 one of the major symptoms of narcolepsy, but little is known about how strong, positive emotions trig

181 ite the clinical challenge this shift poses, little is known about how such dysfunctional immune stat

182 However, little is known about how survival signalling is regulat

183 oke rehabilitation to cognitive enhancement, little is known about how tDCS affects the human brain,

184 motility and generating traction forces, yet little is known about how tension borne by an individual

185 have relatively low trabecular density, but little is known about how that density varies throughout

186 However, to date little is known about how the abundance of these species

187 However, little is known about how the activity of these proteins

188 NusG/Spt5 and RNA polymerase in prokaryotes, little is known about how the binding of eukaryotic Spt4

189 ies have focused on the cyst epithelium, and little is known about how the blood and lymphatic microv

190 Little is known about how the brain differentiates betwe

191 Little is known about how the brain supports representat

192 However, little is known about how the CD2 interacts with ssDNA.

193 However, little is known about how the cell wall is regulated in

194 However, little is known about how the choice is made between ger

195 However, little is known about how the crosstalk between Mvarphi

196 ficant proliferative stages after birth, but little is known about how the developmental changes impa

197 endent on the choice of antibodies; however, little is known about how the different properties of th

198 Unfortunately, little is known about how the dopamine (DA) system funct

199 s are fairly well understood, but relatively little is known about how the extracellular matrix (ECM)

200 However, little is known about how the formation and maintenance

201 ong habitats and local species interactions, little is known about how the identity and origin of dis

202 been reported to play a role in asthma, but little is known about how the insulin-like growth factor

203 nfected by human cytomegalovirus (HCMV), but little is known about how the memory-like NK cell pool i

204 Little is known about how the microbial composition of a

205 Very little is known about how the most common local interneu

206 Despite its fundamental importance, little is known about how the NMDAR transitions between

207 Little is known about how the normal atria of the heart

208 However, little is known about how the parasite performs most mol

209 ed a suite of environmental issues, however, little is known about how the partial pressure of CO2 (p

210 Little is known about how the persistence of picky eatin

211 Yet little is known about how the phase behaviour of a prote

212 idely used as a popular folk drink; however, little is known about how the post-harvest operations af

213 Surprisingly, little is known about how the progressive assembly of ub

214 Little is known about how the proportions of dependency

215 However, little is known about how the protein interactome change

216 cification and patterning, but comparatively little is known about how the regeneration blastema asse

217 mGluR5-antagonists prevent fear and anxiety, little is known about how the same receptor in the amygd

218 is critical to prevent genomic instability, little is known about how the steady state levels of DNA

219 rious up- and downstream signaling pathways, little is known about how the surrounding microenvironme

220 have been shown to shape plasticity of ILCs, little is known about how the tissue microenvironment in

221 However, little is known about how the transition from inflammato

222 Little is known about how the trophic life forms, which

223 s have been shown to arise from the somites, little is known about how the two segments are specified

224 mplicated in many chronic diseases, but very little is known about how the unfolded protein response

225 ons critically regulate gene expression, yet little is known about how their coordinated activities c

226 Little is known about how their spatial distribution is

227 However, little is known about how these assemblies modulate reac

228 echanisms underlying emotional associations, little is known about how these associations guide behav

229 While considered obligate aerobes, little is known about how these bacteria respond to peri

230 However, little is known about how these cathelicidins affect TLR

231 However, little is known about how these extracellular factors in

232 athway have been identified and studied, but little is known about how these factors function collect

233 be involved in Acanthamoeba infections, but little is known about how these facultative parasites in

234 eir evolutionary and ecological success, but little is known about how these features developed as ty

235 t underpin learning about reward and danger, little is known about how these interact to solve basic

236 Overall, little is known about how these interactions are affecte

237 However, little is known about how these lamina-associated domain

238 role in polarizing differentiated cells, but little is known about how these microtubules are organiz

239 However, little is known about how these neurons behave when choi

240 However, little is known about how these NPs affect fungal cell f

241 Ca(2+) and cGMP control microneme secretion, little is known about how these pathways are naturally a

242 However, little is known about how these populations compare in t

243 issue regeneration follow tissue damage, but little is known about how these processes are coordinate

244 Little is known about how these progenitor cells expand

245 ns in nitrate have been identified; however, little is known about how these regulators interact to o

246 ic relays on their way to a destination, but little is known about how these relays form and the func

247 So far, little is known about how these resistance modules inter

248 combined nutritional deficiencies, yet very little is known about how these signalling pathways are

249 yme during lung branching morphogenesis, but little is known about how these signals are integrated.

250 the brain responds to identical stimuli, but little is known about how these states, and transitions

251 ociations with dangerous taste stimuli, very little is known about how these stored emotional associa

252 etoxify ROS and prevent cellular damage, but little is known about how these systems manage to functi

253 However, little is known about how these two cellular events are

254 he extent and nature of the mosquito virome, little is known about how these viruses persist, spread,

255 mones, and neurotransmitters, but relatively little is known about how they are formed.

256 critical roles in recovery from injury, but little is known about how they are regulated within the

257 arately linked to the prefrontal cortex, but little is known about how they combine to support learni

258 frequently dysregulated in human cancer, yet little is known about how they control tumorigenesis.

259 ance migrants accomplish natal homing [1-8], little is known about how they do so.

260 derlie a plethora of cellular processes, but little is known about how they fold.

261 known about these separate learning systems, little is known about how they interact to shape and gui

262 ncers and H3K4me3 for broad domains, however little is known about how they interact with each other

263 ute one of the two major classes of CTS, but little is known about how they interact with NKA.

264 erine ecosystems is increasingly recognized, little is known about how they may be influenced by glob

265 s are present in >125 bacterial species, yet little is known about how this biofilm exopolysaccharide

266 ls receive input from their environment, but little is known about how this collective cell migration

267 Little is known about how this conversion alters soil ca

268 significant role in Acanthamoeba infections, little is known about how this facultative parasite infl

269 To date, very little is known about how this information is encoded by

270 ain regions thought to mediate such changes, little is known about how this information is integrated

271 However, little is known about how this interaction controls phen

272 ffects of object motion (or vice versa), but little is known about how this is accomplished neurally.

273 However, little is known about how this is happening in practice

274 ion influences what is later remembered, but little is known about how this occurs in the brain.

275 rders that stem from traumatic memories, yet little is known about how this process changes the origi

276 on hepatocytes, leading to their death, but little is known about how this process contributes to ma

277 profile of nociceptors in normal conditions, little is known about how this profile changes in pathol

278 in the United States continues to rise, yet little is known about how this spending varies by condit

279 Little is known about how this trajectory compares with

280 a fundamental aspect of human cognition, yet little is known about how this type of information is en

281 ores made by neighboring organisms, although little is known about how this ubiquitous behaviour is r

282 However, little is known about how those niches are formed.

283 However, remarkably little is known about how tissues coordinate their movem

284 However, little is known about how TLS develop in cancer, how the

285 op, because ECs are difficult to culture and little is known about how to direct them to stably integ

286 Little is known about how tonsils contribute to the loca

287 Despite this, remarkably little is known about how TP53 point mutations are acqui

288 However, very little is known about how Tpl2 modulates the development

289 d to be muted among experienced traders, but little is known about how trading experience reduces the

290 In particular, relatively little is known about how transcriptional processes are

291 However little is known about how trigeminal neuralgia (TN), a c

292 Yet little is known about how value signals for food and oth

293 ition of ex vivo slices of cancerous tumors, little is known about how variations in the cellular pro

294 Yet, very little is known about how various geometrical parameters

295 However, little is known about how viruses resist IFITM inhibitio

296 Yet, very little is known about how VP neuron responses contribute

297 Little is known about how well postoperative pain is man

298 al progeroid disorder Cockayne syndrome, but little is known about how XPG loss results in this devas

299 fied by different fatty acids; however, very little is known about how zDHHC enzymes contribute to ac

300 However, little is known about how zoonotic bacteria maintain the