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1 , CYLD ameliorates hepatocellular damage and liver fibrogenesis.
2 nalyze whether microbiota may interfere with liver fibrogenesis.
3 give rise to HSCs and myofibroblasts during liver fibrogenesis.
4 tion, and survival of HSCs and protects from liver fibrogenesis.
5 dentify potential key regulatory proteins of liver fibrogenesis.
6 es the synthesis of extracellular matrix and liver fibrogenesis.
7 th IL-10R2 and IL-22R1, thereby ameliorating liver fibrogenesis.
8 pigenetic regulatory mechanisms impacting on liver fibrogenesis.
9 HSC) activation is an essential event during liver fibrogenesis.
10 A levels, suggesting a direct role of HIV in liver fibrogenesis.
11 GF-beta1) production play important roles in liver fibrogenesis.
12 al insight into intrahepatic immunity during liver fibrogenesis.
13 erates hepatic myofibroblasts, which promote liver fibrogenesis.
14 rgo myofibroblastic trans-differentiation in liver fibrogenesis.
15 tellate cell activation is a main feature of liver fibrogenesis.
16 gy for inducing HSC apoptosis and inhibiting liver fibrogenesis.
17 ANGO1 in procollagen I secretion in HSCs and liver fibrogenesis.
18 (alphaSMA) and collagen I is a key event in liver fibrogenesis.
19 s to myofibroblastic cells to participate in liver fibrogenesis.
20 (HSC), the primary cellular event underlying liver fibrogenesis.
21 tivation regulated by a specific cytokine in liver fibrogenesis.
22 ation called "myofibroblastic activation" in liver fibrogenesis.
23 Ang II on HSCs and plays a critical role in liver fibrogenesis.
24 patocyte injury is mechanistically linked to liver fibrogenesis.
25 ndependent determinant of chemically induced liver fibrogenesis.
26 tic stellate cells (HSC), a pivotal event in liver fibrogenesis.
27 dative stress, which play important roles in liver fibrogenesis.
28 transcription 3 (STAT3) had been involved in liver fibrogenesis.
31 e EGF receptor (EGFR) inhibitor erlotinib on liver fibrogenesis and hepatocellular transformation in
32 fector T-cell responses, which may encourage liver fibrogenesis and progression to end-stage liver di
33 r mechanisms underlying activation of HSC in liver fibrogenesis and the potential therapeutic value o
34 g-term TCS exposure, especially on enhancing liver fibrogenesis and tumorigenesis, and the relevance
36 in the processing of type I collagen during liver fibrogenesis, and that TGF-beta1 and TNF-alpha reg
37 mice were protected against CCl(4)-mediated liver fibrogenesis, as evidenced by reduced collagen typ
41 senchymal transition (EndMT) and spontaneous liver fibrogenesis in EC-specific constitutive hemi-defi
45 upon liver injuries and functions to inhibit liver fibrogenesis induced by either carbon tetrachlorid
46 negative endotoxin producers, may accelerate liver fibrogenesis, introducing dysbiosis as a cofactor
49 n of pathologic angiogenesis, attenuation of liver fibrogenesis partly mediated through inhibition of
53 the potential function of E-type cyclins for liver fibrogenesis, we repetitively treated constitutive
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