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1 ncreased liver endothelial cell adhesion and liver metastasis.
2 owing curative intent surgery for colorectal liver metastasis.
3 samples as well as association of AQP5 with liver metastasis.
4 so found to be increased in lung tissue with liver metastasis.
5 upon CCSC detection, all subjects exhibited liver metastasis.
6 topathologic findings, including parenchymal liver metastasis.
7 igh Ki67 proliferation index and presence of liver metastasis.
8 associations with recurrence, survival, and liver metastasis.
9 l ablation (RFA) in patients with colorectal liver metastasis.
10 inical utility of CS-682 in the treatment of liver metastasis.
11 ivity in vivo significantly promoted distant liver metastasis.
12 ly decreased the incidence of lymph node and liver metastasis.
13 G1 (dnG1) construct in a nude mouse model of liver metastasis.
14 provide added value in evaluating carcinoid liver metastasis.
15 y-based drug and sunitinib markedly promotes liver metastasis.
16 herapy alone for patients with breast cancer liver metastasis.
17 mour cell challenge prevented development of liver metastasis.
18 id cells on cancer cells in a mouse model of liver metastasis.
19 a (RCC), colorectal carcinoma, or colorectal liver metastasis.
20 survival in mouse models of breast and lung liver metastasis.
21 large number of patients with CRC also have liver metastasis.
22 exosomal integrin alphavbeta5 was linked to liver metastasis.
23 se expression is a consistent feature of CRC liver metastasis.
24 erapy in patients with resectable colorectal liver metastasis.
25 ociated with CRC in orthotopically generated liver metastasis.
26 by L1 overexpression in driving motility and liver metastasis.
27 eloid populations previously associated with liver metastasis.
28 F-kappaB pathway that has been implicated in liver metastasis.
29 metastasis of melanoma and MC38 colon cancer liver metastasis.
30 lantation procedures and demonstrated robust liver metastasis.
31 tastasis and also plays an important role in liver metastasis.
32 gross metastases in an intrasplenic model of liver metastasis.
33 proved respectability rates in patients with liver metastasis.
34 cant higher risk of advanced tumor stage and liver metastasis.
35 r neutrophils in the early adhesive steps of liver metastasis.
36 osurvival effect of TNF-alpha and to inhibit liver metastasis.
37 mportant role in mediating colorectal cancer liver metastasis.
38 hioate backbone in a mouse model of melanoma liver metastasis.
39 yroidal extension signified a strong risk of liver metastasis (19%, OR 23), whereas no clinical-patho
41 te PET/CT accurately predicts the success of liver metastasis ablation at 1 y and is superior to imme
43 late TGF-beta signature accurately predicted liver metastasis and discriminated HCC cell lines by deg
44 sustaining development of colorectal cancer liver metastasis and identify a potential target for ant
45 stasis to the lung has higher TS levels than liver metastasis and is less responsive to treatment wit
46 A plasmid was an effective treatment against liver metastasis and moderately effective against lung m
49 at Fv-p53 treatment had a profound effect on liver metastasis and represent the first demonstration o
50 ith lung metastasis, 58 patients (8.1%) with liver metastasis, and 34 patients (4.8%) with bone metas
53 malignant pleural effusion, intraparenchymal liver metastasis, and residual tumor size were significa
54 ar survival following surgery for colorectal liver metastasis approaches 50%, over one-half of patien
58 adiofrequency ablation [RFA]) for colorectal liver metastasis between 1982 and 2008 were identified f
59 a critical role in colon cancer invasion and liver metastasis, but the mechanistic features of this p
60 w TNF signaling influences the efficiency of liver metastasis by colon and lung carcinoma in mice tha
62 bition of in vitro proliferation and in vivo liver metastasis by p85alpha or p110alpha siRNA treatmen
65 melanoma cells into mice resulted in reduced liver metastasis compared with CXCR4-positive uveal mela
66 aying the onset and inhibiting the growth of liver metastasis compared with gemcitabine, 5-FU, and co
69 ublished on hepatic resection for colorectal liver metastasis (CRLM) and determine whether a negative
72 , yet anti-lymphangiogenesis does not impact liver metastasis despite abrogating lymph node metastasi
73 verexpression potentiated local invasion and liver metastasis effects, which were suppressed by reexp
74 In a surgical model of pancreatic cancer liver metastasis, eNOS overexpression in the tumor micro
76 sendothelial migration, resulting in reduced liver metastasis formation in vivo Talin1 expression lev
77 er, our findings uncover a new mechanism for liver metastasis formation involving an active contribut
79 ng research of the mechanisms and tropism of liver metastasis from breast cancer will hopefully resul
83 from the primary tumor to the development of liver metastasis greater than 2 years was a significant
84 ealed that CD44 knockdown increased lung and liver metastasis >10-fold, while metastasis was decrease
86 following surgical management of colorectal liver metastasis have largely focused on overall surviva
87 r 2; HR, 1.46), loss of appetite (HR, 1.62), liver metastasis (HR, 1.32), >/= four metastatic sites (
88 acted from normal colon, primary cancer, and liver metastasis in a patient with metastatic colon canc
90 ly impaired the growth of primary tumors and liver metastasis in an orthotopic mouse model of PDAC (m
91 ic cancer cells suppressed tumorigenesis and liver metastasis in an orthotopic nude mouse model, sugg
92 stimulated tumor cell invasion, and induced liver metastasis in an orthotopic xenograft mouse model
97 es positively correlate with tumor grade and liver metastasis in human pancreatic neuroendocrine tumo
101 2, reduced polyp numbers in Apc(Min/+) mice, liver metastasis in NSG mice with orthotopic tumors, and
104 cing lung metastasis but not affecting B16F1 liver metastasis, in fact, liver metastasis was enhanced
106 ls from a monogenomic primary tumour and its liver metastasis indicated that a single clonal expansio
108 al for a patient with a solitary, resectable liver metastasis is better than that for a patient with
112 ificantly correlated with the development of liver metastasis, its function in pancreatic cancer meta
113 he primary tumor (KMS 72; P = 0.0001) to the liver metastasis (KMS 25; tumor compared with metastasis
114 000 proteins, we found unexpectedly that all liver metastasis lesions displayed a reproducible, zonal
115 merase I in lung metastasis as compared with liver metastasis, lung tumors had a 5-fold increase in E
124 promoter (RANTES-NIS-MSC) in a colon cancer liver metastasis mouse model established by intrasplenic
125 nt SL, 95 had disseminated disease [(surface liver metastasis (n = 29) and peritoneal deposits (n = 6
126 e liver metastasis (n = 5), deep parenchymal liver metastasis (n = 4), peritoneal deposits (n = 1), n
127 l 75 had unresectable disease due to surface liver metastasis (n = 5), deep parenchymal liver metasta
128 cer: n = 1 or metastatic disease [colorectal liver metastasis]: n = 14, ovarian cancer: n = 1, gastri
129 sion profiles in primary mouse colon tumour, liver metastasis of colon cancer and naive colon tissues
130 mphangiogenic gene expression in primary and liver metastasis of colorectal cancer (CRC) and identify
132 x 10(9) PU; once a week, twice) directly to liver metastasis of five individuals with colon cancer a
134 ts showed that TrkBT1 overexpression induces liver metastasis of pancreatic cancer and uncovered a un
135 CD24 increases retroperitoneal invasion and liver metastasis of pancreatic cancer cells in an orthot
138 interactive effects of CYP2B6 genotypes and liver metastasis on the prognosis of metastatic breast c
139 MP-1 levels were correlated with synchronous liver metastasis or distant metastasis-associated diseas
140 ver resection in patients with breast cancer liver metastasis proved to be cost-effective when compar
141 ver resection in patients with breast cancer liver metastasis proved to be cost-effective when compar
143 onmetastatic CRC cells prevented lung and/or liver metastasis, relative to control cells that did met
146 RFP cancer cells were coinjected in the PV, liver metastasis resulted that contained GFP spleen cell
147 polymerase chain reaction on a cohort of 64 liver metastasis samples from patients with complete fol
148 in colon cancer 1 (MACC1) levels measured in liver metastasis specimens to predict further recurrence
149 vanced stages of colorectal cancer (CRC) and liver metastasis, suggesting that it may play a role in
150 iagnosis, Hispanics were less likely to have liver metastasis than whites (30% v 44%, respectively; P
151 ate of patients with unresectable colorectal-liver metastasis to complete resection with hepatic-arte
154 vival data following treatment of colorectal liver metastasis using RFA with or without hepatic resec
155 th a splenic injection model of experimental liver metastasis using syngeneic MC38 colon cancer cells
158 t affecting B16F1 liver metastasis, in fact, liver metastasis was enhanced in the absence of platelet
159 and mechanisms involved in colorectal cancer liver metastasis, which could be potential targets for d
160 leads to their escape from treatment and to liver metastasis, which in turn makes pancreatic ductal
161 also significantly inhibited lymph node and liver metastasis, which led to a significant increase in
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