ライフサイエンスコーパス: liver parenchyma

1 slet allografts can survive long term in the liver parenchyma.

2 tions of cells in perivascular areas and the liver parenchyma.

3 cement was compared with that of surrounding liver parenchyma.

4 cell proliferation and rapid restoration of liver parenchyma.

5 tion of tolerance to islet allografts in the liver parenchyma.

6 the largest area of the tumor and background liver parenchyma.

7 mean fluorescence intensity in the adjacent liver parenchyma.

8 (BS)-mediated toxic injury of the "upstream" liver parenchyma.

9 tify tissue attenuation properties of ICG in liver parenchyma.

10 bile ducts developed in the Prox1-deficient liver parenchyma.

11 HCC tissues compared with the nonneoplastic liver parenchyma.

12 ficient (D*), and perfusion fraction (PF) in liver parenchyma.

13 unique tissue architecture of the developing liver parenchyma.

14 carcinoma cases arise within a noncirrhotic liver parenchyma.

15 te interactions between cancer cells and the liver parenchyma.

16 al liver metastases and nontumorous-adjacent liver parenchyma.

17 igated the physiological role of HOIP in the liver parenchyma.

18 synchronous liver metastases and/or adjacent liver parenchyma.

19 stinct signal voids dispersed throughout the liver parenchyma.

20 n the donor hepatocyte mass engrafted in the liver parenchyma.

21 hrough the recruitment of lymphocytes to the liver parenchyma.

22 the surgical resection clips and the normal liver parenchyma.

23 arameters between primary HCC and background liver parenchyma.

24 rotein in either the lymphoid compartment or liver parenchyma.

25 tion and tend to deposit iron throughout the liver parenchyma.

26 new cells are added and incorporated to the liver parenchyma.

27 ence of numerous cysts spread throughout the liver parenchyma.

28 with ascites), and lesion isointensity with liver parenchyma.

29 epatocyte hyperplasia and removal of injured liver parenchyma.

30 area of transgene expression throughout the liver parenchyma.

31 and integration of transplanted cells in the liver parenchyma.

32 s, and the rare occurrence of mitoses in the liver parenchyma.

33 could repopulate a mean of 32% of recipient liver parenchyma.

34 foci eventually become confluent and replace liver parenchyma.

35 splenically injected hepatocytes engraft in liver parenchyma; 2) a mean of 6.6% of splenically injec

36 In the presence of the primary tumor, the liver parenchyma adjacent to the synchronous liver metas

37 tion selectively and quantitatively into the liver parenchyma after inducing apoptosis.

38 large liver vessels (at 24 hours) and in the liver parenchyma (after day 3).

39 on (PVL), and in situ splitting (ISS) of the liver parenchyma along the falciform ligament were perfo

40 liver tumors while minimizing the impact on liver parenchyma and avoiding systemic effects.

41 tic adenocarcinoma with those in surrounding liver parenchyma and healthy subjects.

42 hich resulted in the atrophy of the affected liver parenchyma and induction of hyperplasia in the tra

43 ted into hepatocytes when engrafted into the liver parenchyma and into bile epithelial cells when eng

44 Transplanted cells integrated in the liver parenchyma and liver architecture were preserved n

45 Average CNR between liver parenchyma and segmental veins were measured in ea

46 mutant mice presented with severely necrotic liver parenchyma and significantly larger hypoxic areas,

47 rats, transplanted cells integrated into the liver parenchyma and survived in vascular spaces.

48 troglitazone for 3.5 months, massive loss of liver parenchyma and symptoms of liver failure developed

49 differences in SH U 508A uptake between the liver parenchyma and the lesion for metastases and chola

50 requency of five morphologic findings of the liver parenchyma and two intrahepatic biliary findings i

51 gnificantly enhanced the number of DC in the liver parenchyma and within the liver metastases, as wel

52 d more transplanted cells to be deposited in liver parenchyma, and decreased hepatic ischemia and end

53 t of multiple cysts scattered throughout the liver parenchyma, and include different entities, classi

54 factant proteins in lung alveoli, albumin in liver parenchyma, and lipase in the stomach lining.

55 mage models, the extent of the injury to the liver parenchyma, and potential species-specific differe

56 rtal vein or tail vein, or directly into the liver parenchyma, and the forms of rAAV vector DNA extra

57 were hypoenhancing compared with surrounding liver parenchyma, and they reached their nadir signal in

58 giogenic factors was encountered in adjacent liver parenchyma as compared to the metastases.

59 rge region of interest, inclusive of as much liver parenchyma as possible in the right lobe while avo

60 period of replication and expansion into the liver parenchyma, as well as penetration into the centra

61 ce of finding the metal stent outside of the liver parenchyma at the time of transplantation is repor

62 ment (the ratio of signal intensities of the liver parenchyma before and 20 minutes after intravenous

63 atio of signal intensity measurements of the liver parenchyma before and 20 minutes after intravenous

64 nosis, and one biopsy specimen showed benign liver parenchyma; both ablated lesions showed no residua

65 evealed VWF-specific staining throughout the liver parenchyma but not in endothelial cells.

66 Presence of core protein in the liver parenchyma but not in lymphoid cells protects from

67 /immunohistochemistry combination within the liver parenchyma but not within hepatocytes.

68 epatic cryoablation of 30% to 35% or more of liver parenchyma, but this complication has not been rep

69 Histologically, the liver parenchyma contained randomly distributed, occasio

70 howed that the hepatocytes that comprise the liver parenchyma do not activate RNase L when infected w

71 innate and adaptive immune components of the liver parenchyma during acute viral hepatitis.

72 changes in perfusion to HCC and surrounding liver parenchyma during MR-IR-monitored TACE.

73 Any disruption of the liver parenchyma during transplantation should be secure

74 dard model to score precisely bile ducts and liver parenchyma features.

75 n of Tc-99m-RBC in hemangiomas and in normal liver parenchyma (HEM/liv), and to verify, whether the q

76 lthough being classically located inside the liver parenchyma, hemangiomas may occasionally develop o

77 1.029, P=0.012) and only partial drainage of liver parenchyma (HR 4.158, P=0.040).

78 SH U 508A uptake between the lesion and the liver parenchyma (ie, subjective conspicuity score [SCS]

79 R-2 gene expression was abundant in adjacent liver parenchyma in all 3 groups.

80 Liver parenchyma in both patient groups enhanced signifi

81 ent of necroinflammation and fibrosis in the liver parenchyma in chronic HCV infection.

82 ration and more prominent iron deposition in liver parenchyma in vivo, retained iron export function

83 g-4/VCAM-1 interactions to transmigrate into liver parenchyma in vivo.

84 zation; to reveal incomplete coverage of the liver parenchyma involved by the tumor, which may be rel

85 rvations show that sporozoite entry into the liver parenchyma involves a complex cascade of events, f

86 The accumulation of the fat in the liver parenchyma is accompanied by downregulation of gen

87 e course, whereas T cell infiltration of the liver parenchyma is associated with progressive liver in

88 ve bile ductular hyperplasia in which normal liver parenchyma is largely replaced with well-different

89 ontaining small (<5 cm) cysts with preserved liver parenchyma is safe and appropriate.

90 that the distribution of microspheres in the liver parenchyma is uniform.

91 assessed lesions (HEM) and in the uninvolved liver parenchyma (liv).

92 Transplanted cells integrated in the liver parenchyma more rapidly in CP-treated animals, and

93 IgM in mouse blood, because IgM deposited in liver parenchyma most likely initiated complement activa

94 interfered with neither cell integrations in liver parenchyma nor secretory function of transplanted

95 9 uveal melanoma cells was injected into the liver parenchyma of 105 CB17 SCID mice through a 1-cm ab

96 r injecting 2.5 million H4IIE cells into the liver parenchyma of ACI rats, they typically form a 1-cm

97 nd they constitute approximately 0.5-2.5% of liver parenchyma of all donor ages.

98 m hESCs and hiPSCs could be engrafted in the liver parenchyma of immune-deficient transgenic mice car

99 were the only ErbB proteins detected in the liver parenchyma on embryonic day 19.

100 vasculature and in the kidney but not in the liver (parenchyma or vasculature).

101 neurotrophin receptor (p75NTR): HSCs in the liver parenchyma; perivascular mesenchymal cells express

102 in consensus) for the imaging pattern of the liver parenchyma, presence and grade of intrahepatic bil

103 ly, significantly different from surrounding liver parenchyma relaxation times of 840 msec +/- 113 an

104 tegration of transplanted hepatocytes in the liver parenchyma required cell membrane regenesis, with

105 crosphere accumulation and regions of normal liver parenchyma that demonstrated no apparent microsphe

106 After integration in liver parenchyma, the position of transplanted cells was

107 etic resonance (MR) imaging with surrounding liver parenchyma to determine the relationship between t

108 planted cell survival and integration in the liver parenchyma was determined by histochemical analysi

109 Objective noise in the liver parenchyma was measured, and the tube current was

110 ry or histology in patients whose background liver parenchyma was normal on SPIO-enhanced MRI.

111 The necrotic liver parenchyma was seen as high and low intensity on T

112 inusoids, albeit integration of cells in the liver parenchyma was slower in gadolinium-treated rats a

113 of the complex set of cues arising from the liver parenchyma, we cocultured adult rat liver SECs, id

114 which transplanted cells integrate into the liver parenchyma, we used dipeptidyl peptidase IV-defici

115 rtosis, skewness) MRI parameters] in HCC and liver parenchyma were compared using Wilcoxon signed-ran

116 d distribution of hepatocytes throughout the liver parenchyma, whereas an intraperitoneal injection r

117 arge, well-defined granulomas throughout the liver parenchyma, whereas CCR2-deficient mice had much f

118 es, and to accelerate cell integrations into liver parenchyma will advance applications of hepatocyte

119 l hepatectomy in mice combine destruction of liver parenchyma with hepatocyte mitoinhibition.