ライフサイエンスコーパス: liver parenchymal cell

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1 m of Ab-mediated destruction of transplanted liver parenchymal cells.

2 ides whose concentration is regulated by the liver parenchymal cells.

3 l for PPARalpha-regulated gene expression in liver parenchymal cells.

4 n to occur via a regulatory protein found in liver parenchymal cells.

5 Liver parenchymal cell allografts initiate both CD4-depe

6 nant hexokinase in pancreatic beta-cells and liver parenchymal cells and functions as a critical comp

7 present not only in immune cells but also in liver parenchymal cells and the complexity of the cell p

8 These findings suggest that liver parenchymal cells are the dominant source of Type

9 FGFR type 4 (FGFR4) in liver parenchymal cells binds only FGF-1, whereas FGFR1

10 ates in the regulation of innate immunity in liver parenchymal cells both in vitro and in vivo and to

11 nd that p62/SQSTM1, a protein upregulated in liver parenchymal cells but downregulated in HCC-associa

12 e hypothesis that acute damage of allogeneic liver parenchymal cells by the CD4-dependent pathway is

13 cKK-E12 was highly selective toward liver parenchymal cell in vivo, with orders of magnitude

14 miR-194, which is specifically expressed in liver parenchymal cells, in preventing liver cancer cell

15 Mice lacking NEMO in liver parenchymal cells (LPC) spontaneously develop stea

16 While ablating either RIPK1 or RelA in liver parenchymal cells (LPCs) did not cause spontaneous

17 etic inhibition of catalytic IKK activity in liver parenchymal cells (LPCs; IKKalpha/beta(LPC-KO) ) w

18 cells that form a barrier between blood and liver parenchymal cells, NS2(H126R) activates RNase L, w

19 , we report that targeted deletion of PBP in liver parenchymal cells (PBP(Liv-/-)) results in the abr

20 te that MyD88 in immune cells rather than in liver parenchymal cells plays an important role in infla

21 To do so, we created mice harboring liver parenchymal cell-specific deletion of HOIP (Hoip(D

22 lls located in the subendothelial space, and liver parenchymal cells, take on the roles of antigen-pr

23 HOIP deficiency in liver parenchymal cells triggered tumorigenesis at 18 mo

24 re, we show that targeted deletion of PBP in liver parenchymal cells, using the Cre-loxP system, resu

25 aloglycoprotein-receptor (ASGP-R) located on liver parenchymal cells was originally identified and ch

26 ole of linear ubiquitination specifically in liver parenchymal cells, we investigated the physiologic

27 acid (0.5% w/w) for 2 wk, killed, and their liver parenchymal cells were isolated.

28 g water for 1 mo, were sacrificed, and their liver parenchymal cells were isolated.

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