ライフサイエンスコーパス: liver repair

1 n-1 influences later mechanisms that lead to liver repair.

2 in BM-MSCs all led to increased efficacy for liver repair.

3 isingly exacerbated liver injury and delayed liver repair.

4 mmatory and anti-fibrotic actions to enhance liver repair.

5 complex signaling network that orchestrates liver repair.

6 are targets of injury, but also orchestrate liver repair.

7 ors but impaired monocyte recruitment during liver repair.

8 id-specific Foxm1 deletion caused a delay in liver repair.

9 hepatic MMPs are responsible for successful liver repair.

10 rarely and contribute to a lesser extent to liver repair.

11 tor (G-CSF) may be effective in facilitating liver repair.

12 cell reaction, and the contribution of BM to liver repair.

13 ic liver injury in humans, is a regulator of liver repair.

14 rokinase-type plasminogen activator (uPA) in liver repair.

15 ince been determined to be very important in liver repair.

16 uting to the resolution of inflammation, and liver repair.

17 ndogenous stem/progenitors to participate in liver repair.

18 are injury models that favor stem cell-based liver repair.

19 ype 2 macrophage activation was required for liver repair after bacterial infection, but resulted in

20 Complement has been implicated in liver repair after toxic injury.

21 The role of progenitor cells in liver repair and fibrosis has been extensively described

22 cells (HPCs), regulating the balance between liver repair and fibrosis.

23 s hematopoietic stem cells (HSCs) can aid in liver repair and improve survival in an animal model of

24 Foxm1 is critical for liver repair and is required for the recruitment of mono

25 that MSCs are a valuable source of cells for liver repair and regeneration and that, by the alteratio

26 The process of liver repair and regeneration following hepatic injury i

27 es play important roles in the mechanisms of liver repair and regeneration through their effects on h

28 data suggest that TNF-alpha participates in liver repair and regeneration, in part, by directly indu

29 progenitor cells (HPCs) play a major role in liver repair and regeneration.

30 f monocytes to Foxm1-deficient mice restored liver repair and rescued liver function.

31 We suggest that AhR may serve to adjust liver repair and to block tumorigenesis by modulating st

32 e cells (HpSC), well known to participate in liver repairing and fibrosis, mediate potent immunomodul

33 activation markers Ym1 and Fizz1, increased liver repair, and reduced hepatotoxicity.

34 nase activity and collagen resorption during liver repair, and speculate that PMN-derived MMP8 or PMN

35 an-1 HS halt disease progression and promote liver repair by enhancing hepatocyte survival in AILI.

36 the fate of key cell types involved in adult liver repair by modulating epithelial-to-mesenchymal-lik

37 onclusion, Foxf1 +/- mice exhibited abnormal liver repair, diminished activation of hepatic stellate

38 To evaluate this possibility, we examined liver repair during different types of liver injury afte

39 e report a potential role of beta-catenin in liver repair, especially in enhancing the resolution of

40 extracellular matrix, we examined Foxf1 +/- liver repair following carbon tetrachloride injury, a kn

41 Molecular mechanisms regulating liver repair following cholestatic injury remain largely

42 ess in multiple organ systems and influences liver repair following hepatotoxic damage or regeneratio

43 Here, we investigated whether the defective liver repair in mice lacking Plg is due to impaired acti

44 rrected the functional deficits and improved liver repair in Plg-deficient mice.

45 lso correlated significantly with markers of liver repair, including numbers of hepatic progenitors a

46 Liver repair involves phenotypic changes in hepatic stel

47 cirrhosis, a disabling outcome of defective liver repair, involves deregulated accumulation of myofi

48 Antibody treatment slightly delayed liver repair processes, as evidenced by extending the pe

49 potentiating hepatotoxicity and/or aiding in liver repair processes.

50 with stem-like properties that contribute to liver repair/regeneration under different pathophysiolog

51 s; however, the mechanisms and potential for liver repair remain unclear.

52 ly modulate regeneration and fibrosis during liver repair remains to be defined.

53 While hepatic inflammation is critical for liver repair, the transcriptional mechanisms required fo

54 Because this has potential implications for liver repair, this review will summarize current knowled

55 ontribute to liver injury, it may also block liver repair through inhibition of hepatocyte proliferat