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1 MMIC activity in hepatic stellate cells and liver sinusoidal endothelial cells.
2 ived factor-1 receptors, CXCR7 and CXCR4, in liver sinusoidal endothelial cells.
3 fic uptake of 125I-HA at 37 degrees C by rat liver sinusoidal endothelial cells.
4 ion is largely restricted to lymph nodes and liver sinusoidal endothelial cells.
5 clude lymphatic endothelial cells (LECs) and liver sinusoidal endothelial cells.
6 - 6.4%), hepatic B cells (81.5 +/- 9.3%) and liver sinusoidal endothelial cells (64.6 +/- 13.7%) inte
9 After acute injury, CXCR7 upregulation in liver sinusoidal endothelial cells acts with CXCR4 to in
10 like receptor 3 (TLR3)-activated Kupffer and liver sinusoidal endothelial cells and further controlle
11 g dispersion atomization, to direct genes to liver sinusoidal endothelial cells and hepatocytes, resp
13 lizidine alkaloid, with reported toxicity in liver sinusoidal endothelial cells and Kupffer cells.
14 y BM-derived APCs including Kupffer cells or liver sinusoidal endothelial cells, and apparently can o
15 sident liver cell types such as progenitors, liver sinusoidal endothelial cells, and hepatic stellate
16 ble contributions of liver progenitor cells, liver sinusoidal endothelial cells, and hepatic stellate
17 n liver, CD302 was expressed by hepatocytes, liver sinusoidal endothelial cells, and Kupffer cells.
19 rget hepatic stellate cells, myofibroblasts, liver sinusoidal endothelial cells, and macrophages to i
21 n Willebrand factor, which is synthesized by liver sinusoidal endothelial cells but not hepatocytes,
22 ptor-dependent manner and cross-presented by liver sinusoidal endothelial cells, but not dendritic ce
23 n Fab binding was evaluated on primary human liver sinusoidal endothelial cells by flow cytometry and
24 vel location of C4d staining in proximity to liver sinusoidal endothelial cell capillarization and st
25 t ligation, constitutive FGFR1 signalling in liver sinusoidal endothelial cells counterbalanced CXCR7
26 gh activation of KCs, inhibited APAP-induced liver sinusoidal endothelial cell damage and improved he
27 vascular niche predominantly represented by liver sinusoidal endothelial cells deploys paracrine tro
29 atment significantly attenuated APAP-induced liver sinusoidal endothelial cell dysfunction and amelio
30 mechanisms underlying death of cultured rat liver sinusoidal endothelial cells exposed to chemical h
31 aluronic acid uptake are reliable markers of liver sinusoidal endothelial cell function and that norm
32 nduced ICAM-1 and VCAM-1 expression in human liver sinusoidal endothelial cells (HLSECs) and the adhe
33 how pro-regenerative angiocrine signals from liver sinusoidal endothelial cells is subverted to promo
34 ther major FcgammaRIIB-expressing cell type, liver sinusoidal endothelial cells, is required for both
36 The endocytic hyaluronan (HA) receptor of liver sinusoidal endothelial cells (LECs) is responsible
38 ndothelial cell targeting, we isolated mouse liver sinusoidal endothelial cells (LSEC) and examined c
39 r-tagged LPS in liver became associated with liver sinusoidal endothelial cells (LSEC) and only appro
40 receptor (FcgammaRIIb, RIIb) is expressed on liver sinusoidal endothelial cells (LSEC) and that the l
43 show that the coculture of hepatocytes with liver sinusoidal endothelial cells (LSEC) significantly
45 .e., the liver-resident macrophages) and the liver sinusoidal endothelial cells (LSEC) which line the
46 proving z-axis resolution, we identified the liver sinusoidal endothelial cells (LSEC), marked by Fcg
49 cells lining the hepatic sinusoids, such as liver sinusoidal endothelial cells (LSECs) and hepatic s
50 fVIII mRNA in purified populations of murine liver sinusoidal endothelial cells (LSECs) and hepatocyt
52 creases in hepatocyte growth factor (HGF) in liver sinusoidal endothelial cells (LSECs) are thought t
55 sure the association of human platelets with liver sinusoidal endothelial cells (LSECs) by immunohist
61 ific difference, the roles of Kupffer cells, liver sinusoidal endothelial cells (LSECs), hepatocytes,
62 characterized by loss of differentiation of liver sinusoidal endothelial cells (LSECs), precedes the
67 in myeloid cell recruitment more than either liver sinusoidal endothelial cells or Kupffer cells.
68 ivo on porcine aortic, femoral arterial, and liver sinusoidal endothelial cells (PAEC/PFAEC/PLSEC).
69 IMS: After liver injury, bone marrow-derived liver sinusoidal endothelial cell progenitor cells (BM S
70 Thus, Sleeping Beauty transposon targeted to liver sinusoidal endothelial cells provided long-term ex
71 ion by stellate cells and CD34 expression by liver sinusoidal endothelial cells remained stable, cons
73 resulted in marked pathologic remodeling in liver sinusoidal endothelial cells (SECs), including SEC
75 rrying the neuronal NOS gene (nNOS) targeted liver sinusoidal endothelial cells, stellate cells, and
76 show that divergent angiocrine signals from liver sinusoidal endothelial cells stimulate regeneratio
77 R7 expression shifted angiocrine response of liver sinusoidal endothelial cells, stimulating prolifer
78 ied several microenvironmental regulators of liver sinusoidal endothelial cells that prolong their ph
80 othelium could be replaced with transplanted liver sinusoidal endothelial cells, we developed an anim
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