ライフサイエンスコーパス: liver tissue

1 of netrin-1 transcript and protein in murine liver tissue.

2 etastatic liver cancer cells in premalignant liver tissue.

3 after surgical resection of large amounts of liver tissue.

4 ellular carcinoma (HCC) and matched nontumor liver tissue.

5 in the labeling of autophagy organelles from liver tissue.

6 tein, and mild necroinflammation remained in liver tissue.

7 d by the utilisation of decellularised human liver tissue.

8 rformed on formalin-fixed, paraffin-embedded liver tissue.

9 phenotype, and compared with KCs in control liver tissue.

10 ary human hepatocytes, and from HBV-infected liver tissue.

11 l cancer patients compared to their adjacent liver tissue.

12 ration, inflammatory markers in serum and in liver tissue.

13 3), or lower than (grade 4) uptake in normal liver tissue.

14 articipate in the restoration of the damaged liver tissue.

15 mainly because of MeHg in muscle and also in liver tissue.

16 5-fold reduction of heparan sulfate in whole liver tissue.

17 periostin and VCAM-1 was the inflamed sheep liver tissue.

18 lanogaster larval salivary glands, and mouse liver tissue.

19 lear cells were isolated from blood or fresh liver tissue.

20 higher expression in HCC compared to normal liver tissue.

21 ale sex, and HBsAg-positivity in the explant liver tissue.

22 ctural or functional perturbations of normal liver tissue.

23 sulted in hypoferremia and iron retention in liver tissue.

24 umors displayed signal lower than background liver tissue.

25 as a histone H3-specific protease in chicken liver tissue.

26 es of cancer cells, embryonic stem cells and liver tissue.

27 HCC) while being normal in adjacent nontumor liver tissue.

28 ntrast, FHL2 expression was absent in normal liver tissue.

29 l Red O) is demonstrated in a study of human liver tissue.

30 l as in intact explants from HCC and healthy liver tissue.

31 ssenger RNA in human HCC samples and healthy liver tissue.

32 rentiate areas of liver fibrosis from normal liver tissue.

33 actor controlling LPCAT3 expression in mouse liver tissue.

34 em cells might be used to regenerate damaged liver tissue.

35 ratio, heme oxygenase-1 and glyoxalase 1 in liver tissue.

36 levels of XIAP, cIAP1, cIAP2, and cFLIP than liver tissue.

37 o extensively repopulate chronically injured liver tissue.

38 g of proteins in sections of mouse brain and liver tissue.

39 3D, Rab7 and Rab18) were determined in whole liver tissue.

40 ressive fibrosis and nodule formation in the liver tissue.

41 hepatocytes and histopathological changes in liver tissue.

42 ex vivo culture of patient-derived fibrotic liver tissues.

43 ession is very low or undetectable in normal liver tissues.

44 of human HCC tissues compared with non-tumor liver tissues.

45 s and microscopic and histologic analysis of liver tissues.

46 ely been limited due to the paucity of donor liver tissues.

47 ectasia and Rad3 related (ATR) expression in liver tissues.

48 nonalcoholic steatohepatitis than in control liver tissues.

49 1 (CHK1) were higher in HCCs than in normal liver tissues.

50 e AMPK profiles of normal and diabetic human liver tissues.

51 ect the pathological changes of the lung and liver tissues.

52 ovarian tumors, but not nontumor prostate or liver tissues.

53 bolic chambers and by histologic analyses of liver tissues.

54 lymphocytes in vitro and by studies in human liver tissues.

55 ry epithelial cells from healthy or diseased liver tissues.

56 issue microarrays of 105 HCCs and 7 nontumor liver tissues.

57 m samples and matched primary and metastatic liver tissues.

58 samples and their corresponding non-tumorous liver tissues.

59 d in human HCC tissues compared to non-tumor liver tissues.

60 tenin activation in fibrotic/cirrhotic human liver tissues.

61 chronic HCV infection, but were detected in liver tissues.

62 tion of netrin-1 and its receptors in murine liver tissues after I/R injury.

63 sed to study lipid droplets (LDs) ex vivo in liver tissue and also in vitro in a single endothelial c

64 We analyzed liver tissue and blood samples obtained before initiatio

65 essed by p16(INK4a) in situ hybridization in liver tissue and by senescence-associated beta-galactosi

66 prevented ischemia-induced cell swelling in liver tissue and dramatically improved LVR outcomes in s

67 iet, we observed higher MC concentrations in liver tissue and fecal matter.

68 onsideration the limits recognized for adult liver tissue and for other forms of stem cells.

69 DAC6 protein was increased sixfold in cystic liver tissue and in cultured cholangiocytes isolated fro

70 at GCs stabilize HIF protein in intact human liver tissue and isolated hepatocytes.

71 HCC without inducing toxicity to tumor-free liver tissue and might therefore represent a promising n

72 tic fibrogenesis flux rates both directly in liver tissue and noninvasively in blood.

73 ortant for physiologically-relevant in vitro liver tissue and organ models, it is most often complete

74 novel insights into genome function in human liver tissue and provides a valuable resource for assess

75 study reinforces our previous findings using liver tissue and reveals new genes and likely tissue-spe

76 We studied HLA-G expression in liver tissue and serum of adult liver transplant recipie

77 state cancer samples, as well as 10 nontumor liver tissues and 20 nontumor prostate tissues, collecte

78 B cells were isolated from mouse and human liver tissues and analyzed by flow cytometry and enzyme-

79 ate clustering between the healthy/cancerous liver tissues and between the bacterial species.

80 and subsequent distribution in the targeted liver tissues and hepatic tumors confirmed with MRI and

81 miR-433 levels in nuclear receptor Shp(-/-) liver tissues and liver tumors compared with wild-type m

82 ly, miR-139-5p was upregulated in metastatic liver tissues and negatively correlated with genes assoc

83 This population was absent in nondiseased liver tissues and peripheral blood.

84 ne the nature of hepatic FGF21 activation in liver tissues and tissue sections from several mouse liv

85 Imaging was performed in normal liver tissues and tumors before and after IRE.

86 R procedure is demonstrated on conventional (liver tissue) and very delicate (fish eggs) samples, for

87 Volumes of interest were drawn in the liver tissue, and hepatic metabolic clearance of (18)F-F

88 s increased in disease compared with control liver tissue, and in both nondiseased and diseased liver

89 in a mouse model downregulated CIDEB in the liver tissue, and knockout of the CIDEB gene in a hepato

90 Serum, liver tissues, and primary hepatocytes were collected fr

91 erate metastatic stem cells in preneoplastic liver tissue are not well understood.

92 of intact glycopeptides isolated from mouse liver tissue are presented to illustrate characteristic

93 expression with fibrotic septa in cirrhotic liver tissues as well as with desmoplastic regions of hu

94 The gold content of tumor and liver tissue at 1 hour or 24 hours after injection was q

95 igates the effects of culturing in S. aurata liver tissue at the molecular level using Fourier Transf

96 ker, von Willebrand factor, in human and rat liver tissue, at advanced stages of fibrosis caused by e

97 effector function, was enhanced in infected liver tissue, B6 mice deficient in these granular protei

98 cell-to-cell DNA methylation variability in liver tissue, based on comparing the methylation status

99 Based on these results, liver tissue bioenergetics is increased 3 hr after ConA

100 dies were performed in human control and PSC liver tissue biopsies, serum, and bile.

101 Anti-CD3 did not differentially reach BM and liver tissues but was more effective in reducing graft a

102 For experiments on liver tissue, calibration by spiked tissue homogenates a

103 To determine whether lipid profiling of liver tissue can identify metabolic signatures associate

104 with PSC were CD28(-), compared with control liver tissue (CD4(+): 30.3% vs 2.5%, P < .0001; and CD8(

105 with increased transcriptional status of the liver tissue cells were observed in the cultured fish ti

106 f genome-wide messenger RNA expression using liver tissues collected from mice fed a standard chow di

107 physiologic and metabolic aspects of intact liver tissue compared with two-dimensional culture syste

108 trahepatic T cells from healthy and diseased liver tissues compared with T cells from blood.

109 Human liver tissues contained a significant proportion of IgA-

110 2013 through May 2017, along with tumor-free liver tissues (control tissues) and peripheral blood sam

111 d and accurate production of human acellular liver tissue cubes (ALTCs) using normal liver tissue uns

112 ned with a spatial-temporal model simulating liver tissue damage and regeneration after CCl4 intoxica

113 ysoPC (18:0) might have a role in signalling liver tissue damage due to warm ischemia before transpla

114 During ruxolitinib treatment, liver tissue damage receded concomitantly with a decreas

115 ised to respond to the release of IL-33 upon liver tissue damage through expression of type 2 cytokin

116 mation and neutrophil recruitment results in liver tissue damage whereas type II NKT cells protect fr

117 Analysis of chronically inflamed liver tissue demonstrated accumulation of SCARF-1 at sit

118 Confocal experiments of liver tissues demonstrated that adiponectin delivery als

119 ncy, ChIP-seq against endogenous TR in mouse liver tissue demonstrates considerable hormone-induced T

120 (at the messenger RNA and protein levels) in liver tissue, despite preserved induction of SHP.

121 eNOS dysfunction observed in platelets and liver tissue didn't match with FMD.

122 tribute to optimal lymphocyte positioning in liver tissue during chronic hepatitis.

123 position is essential for the advancement of liver tissue engineering.

124 n = 16; P < .05), compared with nonfibrotic liver tissue (F0, n = 12).

125 changes of the chromatin landscape in intact liver tissue following glucocorticoid injection.

126 interest around tumors and adjacent healthy liver tissue for up to 3 lesions per patient on (99m)Tc-

127 thway components in tumor and non-neoplastic liver tissue from 7 pediatric patients with moderately d

128 tein levels were evaluated in Huh7 cells, in liver tissue from a rat model of NAFLD, and in liver bio

129 antitative IMS experiments were performed on liver tissue from an animal dosed in vivo.

130 antial levels of hepatocyte TSLP in fibrotic liver tissue from chronic HCV patients.

131 ssion in both HCV-infected Huh7 cells and in liver tissue from chronic hepatitis C patients.

132 ency of cII target gene was 3.46 x 10(-5) in liver tissue from control fish, which increased by 1.4-f

133 Notably, liver tissue from Ercc1(/-) mice fed ad libitum showed p

134 Compared with normal liver tissue from healthy individuals, the amount of IL-

135 ing locations of four liver-essential TFs in liver tissue from human, macaque, mouse, rat, and dog.

136 HCV-infected Huh7 and Huh7.5.1 cells and in liver tissue from in HCV-infected patients, suggesting t

137 In fibrotic liver tissue from mice and patients, hepatic S1P levels

138 n systems, and in vivo ribosome profiling of liver tissue from mice carrying genomic deletions of 3'

139 ire metabolic profiles of muscle, heart, and liver tissue from normally slaughtered and dead on arriv

140 f JNK in drug-induced liver injury (DILI) in liver tissue from patients and in mice with genetic dele

141 KCs in liver tissue from patients with chronic HBV were analyze

142 MCPIP1 expression was higher in liver tissue from patients with chronic HCV infection co

143 ese monocyte subsets in peripheral blood and liver tissue from patients with chronic inflammatory and

144 Liver tissue from patients with PSC contained high level

145 ata from healthy volunteers (n = 11), normal liver tissue from public repositories and patients witho

146 tric data; plasma samples; and SAT, VAT, and liver tissues from 113 obese patients undergoing bariatr

147 pared gene expression patterns with those of liver tissues from 25 patients with nonalcoholic steatoh

148 atitis B surface antigen and core antigen in liver tissues from 26 patients with chronic HBV infectio

149 ed virological factors and AKT expression in liver tissues from 56 HCC patients with better prognoses

150 We collected and analyzed liver tissues from C57BL/6 (control), LysM-EGFP, B6 ACTb

151 n comparing gene expression profiles between liver tissues from different mouse models of NAFLD and p

152 Liver tissues from FXR-knockout mice had reduced express

153 vitro (Huh7-S10-3 liver cells) and in vivo (liver tissues from HEV-infected pigs); however, ISG15 is

154 patients with hepatocellular carcinoma, and liver tissues from live donors served as control.

155 Using liver tissues from liver cirrhosis patients, we observed

156 Whole-transcriptome sequence analyses of liver tissues from mice and patients with Alagille syndr

157 We collected liver tissues from mice that express a constitutively ac

158 performed quantitative proteomic analyses of liver tissues from mice to evaluate these functions and

159 Liver tissues from mice with liver-specific hemizygous d

160 yhead like transcription factor 2 (GRHL2) in liver tissues from mice.

161 AIL combined with BZB exerted no toxicity in liver tissues from nonalcoholic fatty liver disease pati

162 rs of chronic HCV patients that is absent in liver tissues from nonviral hepatitis or healthy subject

163 mRNA, a target of MIR122, were increased in liver tissues from patients and mice with ALD, compared

164 a systematic transcriptome-wide analysis of liver tissues from patients at different stages of NAFLD

165 nt cirrhotic liver samples, and 10 non-tumor liver tissues from patients undergoing resection for hep

166 Levels of ALR were lower in liver tissues from patients with advanced alcoholic live

167 Levels of GRHL2 also were increased in liver tissues from patients with ALD, compared with cont

168 Liver tissues from patients with and without advanced li

169 Liver tissues from patients with BA had 4.6-fold higher

170 IL17 is up-regulated in liver tissues from patients with BA, compared with contr

171 Liver tissues from patients with chronic HCV infection h

172 In addition, we collected liver tissues from patients with chronic HCV infection o

173 We obtained nontumor liver tissues from patients with colorectal carcinoma un

174 In liver tissues from patients with PBC and dnTGF-betaRII m

175 evels of tumor necrosis factor (TNF)alpha in liver tissues from patients with PSC and the effects of

176 mean percentage of CD4(+)CD28(-) T cells in liver tissues from patients with PSC was significantly h

177 eased levels of CPEB1 and CPEB4 in cirrhotic liver tissues from patients, compared with control tissu

178 Levels of NRF2 were measured in liver tissues from rats with cholestasis (induced by adm

179 neutrophil extracellular traps in kidney and liver tissues from untreated rats, consistent with the t

180 rm long-term expanding organoids that retain liver tissue function and genetic stability.

181 therapeutically lower uptake than in healthy liver tissue, however, showed high uptake within radioem

182 histopathologic examination of gingival and liver tissues; immunohistochemistry to cells positive fo

183 Analysis of tumors and surrounding normal liver tissue in DEN-treated HNF4alpha knockout mice show

184 ffective protocols aimed at recreating human liver tissue in vitro.

185 enome-wide scale using ChIP-seq in heart and liver tissues in a panel of rat recombinant inbred and t

186 Pathologic changes in kidney and liver tissues in a rat model of major burn and endotoxin

187 LPS challenge can activate CREBH in mouse liver tissues in a toll-like receptor (TLR)/MyD88-depend

188 Rbeta expression is increased in adipose and liver tissues in obese high-fat fed mice.

189 quiescent Cd39 null hepatocytes in vitro and liver tissues in vivo.

190 estinal system, especially for intestine and liver tissues in which aberrant and deregulated repair r

191 healthy individuals but highly expressed in liver tissue, indicating an ectopic origin from the live

192 lfate (a genotoxicity marker) accumulated in liver tissue, indicating that Mrp3 is involved in the si

193 ied by liver growth, occur in the absence of liver tissue injury and hepatic progenitor cells can be

194 the ultrasound field to fractionate porcine liver tissue into subcellular debris without inducing fu

195 c sinusoidal channels and positioning within liver tissue is a critical event in the development and

196 iciency in mouse embryo fibroblasts or mouse liver tissue is associated with replicative stress and m

197 Liver tissue is characterized by groups of hepatocytes w

198 onfirmed that its expression in HCV-infected liver tissue is inversely related to HCV infection.

199 Serum and liver tissue lipids were also quantified.

200 learance of (18)F-FDG (mL of blood/100 mL of liver tissue/min) was estimated.

201 Recently, a liver tissue model conducive to hepatotoxicity testing w

202 Different Rb genetic profiles were found in liver tissue near each tumor phenotype, giving insight i

203 sected tumor tissue and corresponding normal liver tissue of 156 patients with Klatskin tumors (n = 7

204 he motor cortex, hippocampus, cerebellum and liver tissue of 5 year old transgenic sheep and matched

205 l K- and Ru L2,3-edges) in tumor, kidney and liver tissue of a SW480 bearing mouse.

206 OS, we propose that PFOS-induced mutation in liver tissue of lambda transgenic medaka may be mediated

207 cations on all four bases and using DNA from liver tissue of mice exposed to the tobacco-specific nit

208 wed significantly elevated expression in the liver tissue of NASH patients.

209 As compared to nonrecovered patients, liver tissue of spontaneous survivors revealed not only

210 ereas miR-122 was elevated in both serum and liver tissue of those patients.

211 at proinflammatory cytokines were induced in liver tissues of C57/BL6 mice infected with SFTSV, which

212 rs of ACSL1, we examined ACSL1 expression in liver tissues of hamsters fed a normal diet, a high fat

213 number of IL17a-positive gammadelta T cells liver tissues of mice with BA was associated with increa

214 bryonic fibroblasts as well as in muscle and liver tissues of mutant mice.

215 f CD4(+) and CD8(+) T cells that infiltrated liver tissues of patients with PSC were CD28(-), compare

216 decrease in oxysterol formation in brain and liver tissues of the newborn Dhcr7-knockout pups.

217 ellular carcinomas (HCC), but not in healthy liver tissue or other organs.

218 as the presence of the HCV genome in either liver tissue or peripheral blood monocytes, despite cons

219 ts; 17 were excluded because of insufficient liver tissue or use of hepatotoxic medications, so 148 r

220 ssues, compared with T cells from tumor-free liver tissues or blood.

221 ary sequence, were negligible in human fetal liver tissues or in the differentiating hESCs, and stabl

222 SUV ratio (SUVmax tumor to SUVmax nontumoral liver tissue, or T/NT ratio) of 2.5 or greater were cons

223 urrent single-cell model, whether of primary liver tissue origin, from liver cell lines, or derived f

224 alue in healthy subjects (1.41 mmol/min/L of liver tissue (P < 0.0001).

225 ficantly higher than in corresponding normal liver tissue (P < 0.001).

226 D4 T cells colocated within freshly isolated liver tissue (p < 0.001).

227 ess abundant within HCC compared to adjacent liver tissue (p-value =0.08) and normal control tissue (

228 er levels of IL17 messenger RNA than control liver tissues (P = .02).

229 Curiously, despite having more marked liver tissue pathology, 9142-primed mice also had spleni

230 mice with knockdown of XBP1, we observed of liver tissue persistent endoplasmic reticulum stress, de

231 but relevant complication that appears when liver tissue primed by cirrhosis or prior and subsequent

232 3 present on organelles isolated from murine liver tissue prior to CE-LIF analysis.

233 ctly from the surface of ultrathin slices of liver tissue prior to detection by high-resolution mass

234 Elevation of CXCL1 in premetastatic liver tissue recruited CXCR2-positive myeloid-derived su

235 n of PPARdelta in cultured hepatic cells and liver tissue reduced LPCAT3 mRNA levels, and exogenous o

236 th hepatitis C virus-associated cirrhosis or liver tissues removed during transplantation; healthy hu

237 in reaction analyses of peripheral blood and liver tissue revealed an acute hepatitis E virus infecti

238 Analysis of liver tissue revealed steatosis in regions with human, b

239 and UGT2B17) genes were quantified in human liver tissue samples (n = 125) using real-time PCR.

240 e analysis of immunostaining were applied to liver tissue samples from infants with BA.

241 sphingolipids, and ceramides among 154 human liver tissue samples.

242 using ChIP-seq data for 20 DAPs in two human liver tissue samples.

243 the balance from restoration to progressive liver tissue scarring are not well understood.

244 urthermore, evaluation of Vdr(-/-) BDL mouse liver tissue sections indicated altered E-cadherin stain

245 Histopathological analysis of liver tissue showed reduction of liver apoptosis and enh

246 Chromatin immunoprecipitation analyses of liver tissue showed that at 6 hours after PH, liver XBP1

247 Finally, analysis of clinical datasets from liver tissues showed a negative correlation between expr

248 this signal is counteracted in noncirrhotic liver tissue (showing various levels of inflammation) by

249 PTX3 was evaluated in cultured human cells, liver tissue slices, and mice with acute-on-chronic live

250 We obtained peripheral blood and liver tissue specimens from 20 patients with BA, collect

251 pectrum of clinical disorders and changes in liver tissue that can be detected by pathology analysis.

252 The liver tissue that surrounded the tumors showed no histol

253 In ex vivo bovine liver tissue, the thermal coagulation columns were found

254 We evaluated HCV RNA sequences in serum and liver tissue to distinguish relapse from reinfection.

255 an hepatocellular carcinoma (HCC) and benign liver tissue to identify miRNA target sites transcriptom

256 quencing and ribosome profiling in heart and liver tissues to investigate strain-specific translation

257 Rejection resulted in distinct blood and liver tissue transcriptional changes in patients who wer

258 Compared with surrounding nontumor liver tissue, tumors from all 3 strains had increased ac

259 ular liver tissue cubes (ALTCs) using normal liver tissue unsuitable for transplantation.

260 for investigation of hepatotoxicity in human liver tissue upon applying drug concentrations relevant

261 evelopment of EMT-positive mCSCs in HCC-free liver tissue upon chronic inflammation.

262 We assessed cytokine profiles of liver tissues using a multiplexed array.

263 ples including 27 HCC and 20 adjacent normal liver tissues using the Illumina HumanMethylation450 Bea

264 Liver tissue was analyzed for Sirius Red staining and hy

265 Liver tissue was obtained 2-4 hours before each IFN inje

266 emoved during transplantation; healthy human liver tissue was obtained from a commercial source (cont

267 Liver tissue was obtained from healthy liver donors (n =

268 Lipid analysis in fatty liver tissue was performed using a dietary mouse model o

269 Human liver tissue was perfused over a 30 hour period with hou

270 Attenuation in ablated liver tissues was reduced compared with that in control

271 Based on studies of mice and human liver tissues, we found the liver to be a site of IgA pr

272 Serum, hepatocytes and liver tissue were analyzed.

273 mRNA levels in flash-frozen HCC and adjacent liver tissue were determined by quantitative RT-PCR.

274 Liver tissues were analyzed by histochemistry, hydroxypr

275 Liver tissues were analyzed by histology and by immunobl

276 Liver tissues were analyzed by histology using validated

277 re examined for features of asthma; lung and liver tissues were analyzed for transcription of DEP-reg

278 Liver tissues were collected 14 months after delivery; g

279 Liver tissues were collected and analyzed by bioluminesc

280 Liver tissues were collected and analyzed by histologic

281 Pancreata and liver tissues were collected and analyzed by histology,

282 Liver tissues were collected and analyzed by immunohisto

283 of liver failure (alanine aminotransferase); liver tissues were collected and analyzed by quantitativ

284 Liver tissues were collected and analyzed by quantitativ

285 Mice were weighed, and serum and liver tissues were collected and analyzed for histology,

286 Liver tissues were collected and bile acid pool sizes an

287 Lymph node, lung, and liver tissues were collected and evaluated by flow cytom

288 Liver tissues were collected at different timepoints dur

289 Liver tissues were collected before surgery and at 6 and

290 Liver tissues were collected from ALR-L-KO mice and ALR(

291 Liver tissues were collected from mice and analyzed hist

292 Serum and liver tissues were collected; hepatocytes and liver mono

293 Cirrhotic and noncirrhotic liver tissues were obtained from patients with hepatocel

294 cytotoxicity for HCC, but not for tumor-free liver tissue, which could even be verified in liver expl

295 the PET study was 0.019 L of blood/min/L of liver tissue, which was not significantly different from

296 n hepatic V(max)(PET) was 0.57 mmol/min/L of liver tissue, which was significantly lower than the val

297 lating the spatial distribution of lipids in liver tissue with disease progression using advanced mas

298 15 could recognize S. japonicum eggs laid in liver tissues with a detection ratio of 80.5%.

299 tex or adjacent to the renal medulla; and in liver tissue, with and without metallic or plastic plate

300 esions per patient and also adjacent healthy liver tissue without evidence of tumor.