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1 ls adjacent to the central vein in the liver lobule.
2 cant decrease in the right inferior parietal lobule.
3 uscular fatty structures typical of the alar lobule.
4 sion of non-melanoma skin cancer on the alar lobule.
5 tion (rTMS) applied to the inferior parietal lobule.
6 s and intraparietal sulcus/inferior parietal lobule.
7 rrelations observed in the inferior parietal lobule.
8 with the identity of the injected cerebellar lobule.
9  frontal and parietal lobes, and paracentral lobule.
10 eral prefrontal cortex and inferior parietal lobule.
11 rea and bilaterally in the inferior parietal lobule.
12 or temporal gyri, and left superior parietal lobule.
13 th activation in the right inferior parietal lobule.
14 us, postcentral gyrus, and inferior parietal lobule.
15  by per cent damage to the inferior parietal lobule.
16 tal cortex and the rostral superior parietal lobule.
17 2 and C1 zones in crus II and the paramedian lobule.
18 nalogue represents a cross-section through a lobule.
19 ich facilitates migration within the hepatic lobule.
20 obular region and finally replace the entire lobule.
21 ting at a size much smaller than a choroidal lobule.
22 rior hippocampus and right inferior parietal lobule.
23  from activity in the left inferior parietal lobule.
24 gyrus, the putamen and the superior parietal lobules.
25 re widespread throughout multiple cerebellar lobules.
26  occipital lobe and of the superior parietal lobules.
27           Fibrous septae separated adipocyte lobules.
28 ent forms of sensory input across cerebellar lobules.
29  of hexagonal prisms represented the hepatic lobules.
30 basement membrane, primarily in the anterior lobules.
31 ally used for functional restoration of alar lobules.
32 erior lobules and vestibular input in caudal lobules.
33  asymmetrical inferior and superior parietal lobules.
34  disorganized and had excess sebaceous gland lobules.
35 u using multiphoton microscopy in pancreatic lobules.
36 xpressed in the epithelium of both ducts and lobules.
37 f which are also required for patterning the lobules.
38  had only minor effects on stellate cells in lobules.
39 al decrease in the dimensions of the hepatic lobules.
40 signal generation in normal mouse pancreatic lobules.
41   Confocal z-stack images were obtained from Lobule 1 and analyzed with Imaris software to quantify t
42                                              Lobules 10 and 9 in the caudal posterior vermis [also kn
43 ted by Cav3-Kv4 interactions proves to allow lobule 2 granule cells to respond more effectively to ta
44 nse of granule cells to mossy fiber input in lobules 2 and 9 of the rat cerebellum.
45 ely to tactile stimulus-like burst input and lobule 9 cells to slow shifts in input frequency charact
46 pressed at a substantially higher density in lobule 9 cells, acting to increase A-type current availa
47 ta-burst stimulation of mossy fiber input in lobule 9 granule cells lowered the current threshold to
48 peripheral vestibular sensors but similar to lobules 9 and 10 of the cerebellar vermis (nodulus and u
49 or surgical reconstruction of the nasal alar lobule after two-layer excision of non-melanoma skin can
50 representation in the left inferior parietal lobule and a significant decrease in the right inferior
51                              The paracentral lobule and cuneus had the highest resting metabolic stat
52 creased diffusivity in the superior parietal lobule and increased diffusivity in the hippocampus.
53 hat was localized to the right intraparietal lobule and left Brodmann area 9 (BA9).
54 e Cohort, by determining number of acini per lobule and lobular area.
55 ignal in the left anterior inferior parietal lobule and posterior inferior temporal gyrus and sulcus
56 activity patterns in right superior parietal lobule and premotor cortex, and also left frontopolar co
57 m to start at the periphery of the pulmonary lobule and progressively extend toward the core of this
58 actional anisotropy in the superior parietal lobule and reduced mean diffusivity in the thalamus in t
59 the right precuneus, right inferior parietal lobule and right middle occipital gyrus.
60 fied decreased MTR in left inferior parietal lobule and right superior parietal lobule in suicide att
61 x differences in the right inferior parietal lobule and superior marginal gyrus, and displayed revers
62 nstruction of the 3D architecture of a liver lobule and the development of an experimental model of t
63  cortex and both the right superior parietal lobule and the left lateral occipital cortex) included t
64 portantly, activity in the inferior parietal lobule and the left middle frontal gyrus varied with a f
65 irror neurons (MNs) in the inferior parietal lobule and ventral premotor cortex (PMv) can code the in
66 nal connectivity with left inferior parietal lobule and ventral premotor cortex, indicating that each
67 cated in fibrotic septa between the exocrine lobules and adjacent to the ductal system of the pancrea
68           TFF3 is expressed in normal breast lobules and ducts, at higher levels in areas of fibrocys
69 s for adipose tissue, fibrous stroma, breast lobules and ducts, cysts and microcysts, as well as in s
70 xamined were identified throughout the liver lobules and in portal tracts, although portal tracts wer
71 emporal gyri, inferior and superior parietal lobules and precuneus, all of which were unilaterally ac
72 ithin the cerebellum embodied by a series of lobules and Purkinje cell (PC)-striped gene expression.
73 ile receptor afferents prevalent in anterior lobules and vestibular input in caudal lobules.
74 perimental models: ex vivo (mouse pancreatic lobules) and in vitro (human cells).
75 (Broca's area and the left inferior parietal lobule), and with stronger negative relationships betwee
76 ri, middle occipital lobe, inferior parietal lobule, and also cingulate, paracentral, and precentral
77 lateral prefrontal cortex, inferior parietal lobule, and cerebellum.
78 onal connectivity with the inferior parietal lobule, and children with ASD showed atypical functional
79  superior occipital gyrus, superior parietal lobule, and dorsal premotor area) was relevant for monit
80 orbital prefrontal cortex, superior parietal lobule, and hippocampus; right claustrum/putamen, latera
81 eft middle temporal gyrus, inferior parietal lobule, and inferior frontal gyrus as videos were rated
82 yri, right precuneus, left inferior parietal lobule, and left postcentral gyrus.
83 transverse temporal gyrus, superior parietal lobule, and paracentral, lateral orbitofrontal, and late
84  frontal cortex, posterior inferior parietal lobule, and parahippocampus.
85 tion sensitive area MT/V5, superior parietal lobule, and primary visual cortex, while showing decreas
86 group (left temporal pole, inferior parietal lobule, and superior temporal gyrus) corresponded to reg
87  of the prefrontal cortex, inferior parietal lobule, and temporoparietal junction, as well as the ins
88  the cerebellum, area Spt, inferior parietal lobule, and the anterior cingulate cortex.
89 aparietal sulcus, anterior superior parietal lobule, and the ventral object-specific lateral occipita
90 ., middle occipital gyrus, inferior parietal lobule, and ventral premotor area) was specifically invo
91  fields, both superior and inferior parietal lobules, and regions within the prefrontal cortex.
92 cortex, right superior and inferior parietal lobules, and right superior frontal, middle temporal, mi
93 ght frontal pole, the right lateral parietal lobules, and the left posterior cingulate cortex.
94 de network, precuneus, and inferior parietal lobule; and, within the dorsal attention network, intrap
95 er metabolic values in the inferior parietal lobule, anterior cingulate, inferior temporal lobule, th
96 n the sagittal plane, directed away from the lobule apex.
97 motor cortex face area and inferior parietal lobule are both implicated in the cortical mirror-neuron
98 eflect the status of a woman's normal breast lobules are associated with breast cancer risk.
99 structed by enlarged hepatic lobules; no new lobules are formed during this process.
100                                       Breast lobules are the anatomic sites of origin of breast cance
101 er than the presence of a full complement of lobules, are critical for generating PC protein stripes
102               In macaques, superior parietal lobule area 5 has been described as occupying an extensi
103 al cortices (precuneus and inferior parietal lobule), as expected.
104 onded to activation in the inferior parietal lobule, as well as to activation around the inferior fro
105 recise targeting of mossy fibers to distinct lobules, as well as their subsequent resolution into dis
106 ble selection approach, was used to identify lobules associated with motor function, language, execut
107 orsolateral PFC, and right inferior parietal lobule at rest in the treatment group compared with the
108 premotor cortex (BA 6) and inferior parietal lobule (BA 40) for the same contrast.
109 or area, premotor area and superior parietal lobule, based on the anatomic location of the hand-motor
110 ast map included bilateral superior parietal lobule, bilateral dorsolateral prefrontal cortex (DLPFC)
111 genitors destined to form the posterior-most lobule causes this unique phenotype.
112 eased to 10% (p < 0.001), at which point the lobule ceased to expand further and was counterbalanced
113  PEPCK1 protein was located around the liver lobule central vein and was low in CTR fetuses but rose
114  representation within the inferior parietal lobule changes, with a decrease of the ipsilateral hemif
115 pically of repeating units of various sizes (lobules) consisting of CD34-postive, GLUT-1-negative end
116 ounterbalanced by the number of contributing lobules containing microspheres that increases as r(2).
117 that activity in the human inferior parietal lobule correlates with the divergence of such outcome di
118 eral prefrontal cortex and superior parietal lobule, corresponded to the decision variables resulting
119 tanding of the mechanisms by which ducts and lobules develop is derived from model organisms and thre
120           Defects involve fusion of adjacent lobules, disrupted layering of neurons and glia, and fra
121 bility gave for the less irradiated tissue a lobule dose distribution centered around 103 Gy (full wi
122  can replace all hepatocytes along the liver lobule during homeostatic renewal.
123              GM volume decrease in posterior lobules (especially vermis VI) was associated with reduc
124               Atrophy of specific cerebellar lobules explains different aspects of motor and cognitiv
125 d by switch cues in medial superior parietal lobule for both domains of control, revealing a single d
126 ft (r = -0.839, P = 0.037) superior parietal lobules for the 3Let task.
127 tomic force microscopy analysis across liver lobules from normal and fibrotic livers.
128  the default mode network, superior parietal lobule, fusiform gyrus and anterior insula.
129 thickness in the pars orbitalis, paracentral lobule, fusiform gyrus and inferior temporal gyrus was l
130 from 103 down to 50 Gy, and about 17% of the lobules got a dose lower than 40 Gy to their different s
131                        The inferior parietal lobule, held to be a homologue of the monkey parietal mi
132 ggest vulnerability of the superior parietal lobule, hippocampus, and thalamus to glycemic extremes d
133 ion has been linked to the superior parietal lobule; however, the neural substrates associated with s
134  ipsilateral cerebellar cortex in cerebellar lobule HVI and in lobule I.
135 ociated with the anterior lobe and posterior lobule HVI.
136 ellar cortex in cerebellar lobule HVI and in lobule I.
137 S, FTD and PSP (Crus I/II), and MSA and PSP (lobules I-IV), respectively.
138  expressed in a striking array of stripes in lobules I-V (anterior zone; AZ) and VIII-anterior IX (po
139  in the anterior portions of the cerebellum (lobules II, V, and VI) and strong in lobules IX and X.
140 mulation over right medial superior parietal lobule impaired target discrimination after a shift of a
141  to establish the functional significance of lobules implicated in cognitive and motor functions in n
142 gular gyrus, precuneus and superior parietal lobule in carriers compared to non-carriers, with trend-
143  parietal lobule and right superior parietal lobule in suicide attempters relative to both non-attemp
144 iddle frontal gyrus to the superior parietal lobule in the right hemisphere in healthy controls, at-r
145 ry temporal region and the inferior parietal lobule in the right hemisphere.
146 g of clinical dysfunctions to the cerebellar lobules in disease populations is necessary to establish
147 mparison) and between PwMS and PwCIS for all lobules in subregions VI and left crus I (p<0.05).
148 erations to venom-treated excised pancreatic lobules in vitro.
149 ructural alterations in individual posterior lobules, in which cognitive functioning seems prepondera
150 y somatosensory cortex and superior parietal lobule influences brain networks associated with tactual
151 erior intraparietal sulcus/inferior parietal lobule interfered with perceptual conflict processing.
152 paradigm, we show that the inferior parietal lobule (IPL) (corresponding to the supramarginal gyrus)
153 ateralized network, where the infra-parietal lobule (IPL) activation was lateralized to the left, whi
154 emporal network; bilateral inferior parietal lobule (IPL) activity was larger in HC versus SZ and HC
155 ls of HSPs in hippocampus, inferior parietal lobule (IPL) and cerebellum of subjects with aMCI.
156 lently bound by HNE in EAD inferior parietal lobule (IPL) compared to age-related control brain.
157  motor cortex (M1) and the inferior parietal lobule (IPL) have been identified with supporting the le
158  success pattern, a larger inferior parietal lobule (IPL) region tracked the validity of the memory c
159 arginal gyrus (SMG) of the inferior parietal lobule (IPL) where we observed a double dissociation bet
160 ior temporal gyrus (pSTG), inferior parietal lobule (IPL), and ventral central sulcus (vCS) that were
161 tronger signal in the left inferior parietal lobule (IPL), bilateral precuneus (PCN), bilateral premo
162 posterior cingulate gyrus, inferior parietal lobule (IPL), postcentral gyrus) areas.
163  was possible in the right inferior parietal lobule (IPL).
164 ench, activity in the left inferior parietal lobule (IPL).
165 und for the left and right inferior parietal lobules (IPL), the left superior parietal lobule (SPL) a
166  a specific sector of left inferior parietal lobule is devoted to tool use in humans, but not in monk
167 s to determine whether the superior parietal lobule is indeed necessary for working memory.
168 signal processing across multiple cerebellar lobules is controlled differentially by postsynaptic ion
169 s) and SCA (contraction of total cerebellar, lobule IV, and Crus I volumes with additional X- or Y-ch
170 ults demonstrate that simple spike firing in lobules IV-VI is significantly correlated with position,
171 pyridine-3-ol hydrochloride, into cerebellar lobules IV-VI, in vivo, significantly reduced their alco
172 acingulate gyri (ACG/ApCG), left cerebellum (lobules IV/V and VIII), bilateral superior frontal gyrus
173 ule, the dentate nucleus, and the cerebellar lobules IV/V, VI, and VIII bilaterally corresponding to
174 bellum (lobules II, V, and VI) and strong in lobules IX and X.
175  strongest in the gyrus region of cerebellar lobules IX and X.
176 x/retrosplenial (PCC/Rsp), inferior parietal lobule, lateral temporal cortex, and hippocampus regions
177 igured periportal-to-CV gradients to exhibit lobule-location dependent behaviors.
178 k node (the left posterior inferior parietal lobule, lpIPL) induced two topographically distinct chan
179 permatids are released into the seminiferous lobule lumen (SLL), where they develop into spermatozoa
180 erior intraparietal sulcus/inferior parietal lobule may resolve perceptual conflicts selectively when
181 ady visualization of features such as ducts, lobules, microcysts, blood vessels, and arterioles and t
182 s, while the 3D matrix with a modified liver lobule microstructure allows toxins to be trapped effici
183 tream, specifically in the inferior parietal lobule, middle frontal gyrus, and dorsal parts of the in
184  a common region of medial superior parietal lobule (mSPL) as a domain-independent source of cognitiv
185 mic activity in the medial superior parietal lobule (mSPL), previously implicated in voluntary (as op
186  between locations [medial superior parietal lobule (mSPL)].
187 eactive metabolite) formation within hepatic lobules (NAPQI zonation) are necessary and sufficient pr
188 ted liver is constructed by enlarged hepatic lobules; no new lobules are formed during this process.
189 membrane of central hepatocytes in the liver lobule of control mice.
190  presence of rare Thy1(+) cells in the liver lobule of normal animals, occasionally forming small col
191 transfection of CFs in the caudal cerebellar lobules of rats.
192               Histological features included lobules of small vessels within the dermis, resembling a
193 ging picture in which increasingly posterior lobules of the anterior cerebellar cortex are associated
194          Gray matter atrophy of the superior lobules of the cerebellum (IV, V, VI), and lobules VIII
195 ways, and small vessels within the secondary lobules of the lung.
196 nfiltrative growth that replaced the hepatic lobule or histologic nodular growth in the portal triad
197 gregation of mossy fibers across 10 distinct lobules over the rostrocaudal axis, with tactile recepto
198 er risk with increasing numbers of acini per lobule (P = .0004).
199  a decrease in the number of hepatocytes per lobule (p = 0.029).
200 ts suggest a significant impact of posterior lobules pathology in corticocerebellar loop disruption r
201 d greater activation in the cuneus, parietal lobule, precentral gyrus, and superior temporal gyrus.
202 activation of Notch signaling in the hepatic lobule promotes ectopic biliary differentiation and tubu
203                                In pancreatic lobules, PSC responded to agonists consistent with the p
204  of regions, including the superior parietal lobule (r = -0.66; p < 0.01).
205                     Hepatocyte area (HA) and lobule radius (LR) were also measured.
206 ctions between the central inferior parietal lobule region and the anterior prefrontal cortex.
207 ey system in the precuneus/superior parietal lobule region with reduced functional connectivity, whic
208             We have quantified the extent of lobule regression on a benign breast biopsy in 85 patien
209 cantly reduced MTR in left inferior parietal lobule relative to controls, as well as an MTR reduction
210  middle temporal gyrus and inferior parietal lobule, respectively.
211 tilage grafts allow safe and functional alar lobule restoration.
212                    Comparison of DISC across lobules revealed that it was weak in the anterior portio
213 mentary motor area (SMA), bilateral parietal lobule, right hippocampus, bilateral middle frontal gyru
214 nterior hippocampus, right inferior parietal lobule, right posterior cingulate cortex, and right vent
215 es, temporoparietal junction and paracentral lobule, right superior temporal and parietal lobes, temp
216 mice is that necrosis begins adjacent to the lobule's central vein (CV) and progresses outward.
217 al evidence supporting the superior parietal lobule's purported role in working memory has been lacki
218 lyses with hippocampal and inferior parietal lobule seed regions and the rest of the brain also revea
219 olded along the anterior-posterior axis into lobules separated by fissures, allowing the large number
220 active during lick-related movement across a lobule-specific region of the cerebellum showing high te
221               Superior and inferior parietal lobule (SPL and IPL) possessed both types of structure.
222  frontal eye fields (FEF), superior parietal lobule (SPL) and intraparietal sulcus (IPS).
223 al lobules (IPL), the left superior parietal lobule (SPL) and the right precuneus-SPL, which were all
224 etal sulcus (IPS) and left superior parietal lobule (SPL) differing in time and sign for recognized o
225 d the critical role of the superior parietal lobule (SPL) in shifting spatial attention, a finding no
226  frontal eye fields (FEF), superior parietal lobule (SPL), and right supramarginal gyri (SMG).
227 1-IPS5) and an area in the superior parietal lobule (SPL1)] to examine their spatial attention signal
228            Atypical RCrusI-inferior parietal lobule structural connectivity was also evident in the P
229 e of BSEP from zone 1 to zone 3 of the liver lobule, suggesting that the mutation identified here may
230 ortical activity in medial superior parietal lobule, suggesting that this may be the source of a doma
231 fied a region in the right superior parietal lobule that responded to both types of visuomotor load a
232 cerebellum consists of an intricate array of lobules that arises during the process of foliation.
233  axis that are organized with respect to the lobules that divide the Cb along the anterior-posterior
234  is region specific: it is most prominent in lobules that regulate eye movement and process vestibula
235                     The proportion of normal lobules that were involuted was categorized by an expert
236 obule, anterior cingulate, inferior temporal lobule, the dentate nucleus, and the cerebellar lobules
237 ferior parietal sulcus and superior parietal lobule, the frontal eye-movement field, and the inferior
238 ntral and postcentral gyrus, the paracentral lobule, the superior temporal gyrus, the middle cingulat
239                                 Within liver lobules, this shuttle may allow flexible transfer of bil
240 dermal and mesodermal origins in a hexagonal lobule unit.
241 ast tissue samples of morphologically normal lobules up to 12 years before the development of breast
242 nd that the right cerebellar vermis and left lobule V of cerebellar anterior lobe were additionally a
243  cerebellar peduncles, cerebellar vermis and lobules V and VI, and corpus callosum.
244 rror trials was observed in the hand area of lobules V and VI.
245 tate, the ipsilateral motor representations (lobules V/VI), and most interestingly the contralateral
246                We injected rabies virus into lobules VB-VIIIB of the vermis and used retrograde trans
247                                     Instead, lobules VB-VIIIB represent a site where the cortical mot
248 PwMS and HS for the right (p<0.001) and left lobule VI (p<0.01), left crus I, right VIIb and entire c
249 audal dorsal premotor cortex, and cerebellar lobule VI (t >/= 4.18, whole-brain familywise error corr
250 that the anterior cerebellum, extending into lobule VI, and parts of lobule VIII were smaller than no
251 rior cerebellum, specifically in hemispheric lobule VI, Crus I, and VIIb.
252 were most pronounced in, but not limited to, lobules VI and interposed nuclei.
253                             Right cerebellar lobules VI and VII (including Crus I/II) are engaged dur
254 ct of grey matter (GM) volume alterations in lobules VI to VIIIb on IPS in persons with clinically is
255 s, with cerebellar lesion volume, cerebellar Lobules VI, Crus I and VIIIa atrophy being independent p
256 arily from reduced gray matter in crus I and lobules VI, VIIB, and VIIIA.
257 rotein 25 (HSP25) is expressed in stripes in lobules VI-VII (central zone, CZ) and posterior IX-X (no
258 identified P-cells in the oculomotor vermis, lobules VIc and VII.
259 found that a region in posterior cerebellum (lobule VII crus 1) was engaged specifically when a tempo
260 theta burst stimulation (iTBS) to the vermis lobule VII or right lateral cerebellar Crus I/II, subreg
261                                The vermis of lobule VII receives a prominent input from the retrosple
262 erebral default mode network, whereas vermal lobule VII stimulation influences the cerebral dorsal at
263 ngly the contralateral posterior cerebellum (lobule VII) impede the benefits of motor execution on pe
264 on in ventral cerebellum, in the vicinity of lobules VII/VIII.
265 ctional connections between right cerebellar lobule VIIb and the posterior parietal cortex.
266 e findings suggest that the right cerebellar lobule VIIb interacts with the posterior parietal cortex
267 ated neural signal from the right cerebellar lobule VIIb, specifically during the late encoding phase
268 me in the occipital lobe and left cerebellar lobule VIIb, which is functionally connected with prefro
269  attention tasks robustly recruit cerebellar lobules VIIb and VIIIa, in addition to canonical cortica
270 tween task-responsive portions of cerebellar lobules VIIb/VIIIa and cortex.
271 nnectivity patterns revealed that cerebellar lobules VIIb/VIIIa group with cortical nodes of the dors
272 trates that a cerebellar subdivision (mainly lobules VIIb/VIIIa), which exhibits strong intrinsic fun
273 ork should be expanded to include cerebellar lobules VIIb/VIIIa.
274 rsal attention network to include cerebellar lobules VIIb/VIIIa.
275 minates as climbing fibres in lateral vermal lobule VIII (pyramis).
276                               The C1 zone of lobule VIII receives a more prominent projection from th
277 llum, extending into lobule VI, and parts of lobule VIII were smaller than normal in children with au
278 r lobules of the cerebellum (IV, V, VI), and lobules VIII also correlated with worse posturometric va
279  of total cerebellum, flocculus, and Crus II-lobule VIIIB volumes in males) and SCA (contraction of t
280 romosomes; X-specific contraction of Crus II-lobule VIIIB).
281                                   Cerebellar lobule volumes were derived from a graph-cut based segme
282 nd the right IFG and right inferior parietal lobule was also significantly correlated with age of acq
283  the postcentral gyrus and superior parietal lobule was sensitive to dot periodicity.
284 ere present when different zones of the same lobule were injected.
285 odel of the testis was designed in which the lobules were approximated by a cross-section of seminife
286 atter displayed numerous aggregates, whereas lobules were mildly affected.
287                                   Cerebellum lobules were segmented using SUIT V.3.0 to estimate thei
288 e found bilaterally in the inferior parietal lobule when prisms, but not plain glasses, were used.
289 perfunctional islets are confined within few lobules, whereas hypofunctional islets are present throu
290 d to the adjacent acinar cells in pancreatic lobules; whereas taurolithocholic acid 3-sulfate primari
291 the presence of a partially formed posterior lobule which echoes the posterior vermis DW 'tail sign'
292  movement direction in the superior parietal lobule, which may underlie a transformation from the loc
293 of gray matter in the left posterior insular lobule, which we hypothesize may be related to the depen
294                It contains a large number of lobules, which in turn are composed of convoluted semini
295 tely around pericentral areas of the hepatic lobule, while there was no transgene expression in perip
296 or temporal gyrus, and the inferior parietal lobule, while those of patients with atypical language l
297 ted numerous melanocytic nuclei in the tumor lobules, while MART-1 and HMB-45 revealed the dendritic
298 stimulus-driven responses of interneurons in lobules X (nodulus) and IXc,d (ventral uvula) of the cau
299 stimulus-driven responses of interneurons in lobules X (nodulus) and IXc,d (ventral uvula) of the mac
300 of the cerebellar nodulus and ventral uvula (lobules X and IXc,d of the vermis) for vestibular proces

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