ライフサイエンスコーパス: log phase

1 urrence of CPTL2 in tobacco and yeast at the log phase.

2 els in early log phase and increased in late log phase.

3 were absent when cells were isolated during log phase.

4 lipids at pH 10.5 versus 3% at pH 7.5 during log phase.

5 the bacteria enter late log phase from early log phase.

6 s significantly reduced when cells grew past log phase.

7 growth phase dependent, peaking at the early log phase.

8 rter clpP-clpX transcript when E. coli is in log phase.

9 ited lower accumulation during acid shock in log phase.

10 east Saccharomyces cerevisiae growing at mid-log phase.

11 accumulated large amounts of sigmaS even in log phase.

12 mt1-451D and precocious induction of FAA4 in log phase.

13 36 levels drop considerably beginning in mid-log phase.

14 to approximately 14 mM) only during the late log phase.

15 air occurred in the NTS control region in WT log phase.

16 se compared to that in bacteria grown to mid-log phase.

17 the inability of cells to grow beyond early log phase.

18 ere grown to stationary phase but not to mid-log phase.

19 Saccharomyces cerevisiae growing through mid-log phase.

20 ementation of aguBA, which is induced during log phase.

21 ag phase for activation of ribosomes for the log phase.

22 uced in vivo compared to in vitro during mid-log phase.

23 arvation, is known to induce rpoS during the log phase 25- to 50-fold.

24 Incubation of early-log-phase A. actinomycetemcomitans cells with conditione

25 When log phase A549 non-small cell lung cancer cells were exa

26 e expression of invasion loci, peaks in late log phase; accordingly, virB expression is enhanced by a

27 the inability of LT2 to display a sustained log-phase acid tolerance response.

28 ial rabbits to M. tuberculosis, 320 to 1,890 log-phase, actively growing inhaled bacilli were require

29 ts required a significant lag phase prior to log-phase aerobic growth, but this lag was not as appare

30 bovis BCG nat results in delayed entry into log phase, altered morphology, altered cell wall lipid c

31 fter treatment with 0.02 microM amsacrine in log phase and 27 x 10(-6) after treatment with 1 microM

32 a 16-kDa cytoplasmic form predominant in the log phase and a 12-kDa membrane-associated form predomin

33 rase strains increased markedly through late log phase and continued to rise in stationary phase in t

34 el of epsilon-protein was highest during the log phase and decreased during the stationary phase.

35 ytic activity is maximal during mid- to late-log phase and decreases in the stationary phase.

36 n degradation was most prominent during late log phase and early stationary phase, due in part to a c

37 on was expressed at very low levels in early log phase and increased in late log phase.

38 es Enterococcus faecalis were grown to early log phase and inoculated on enriched Brucella blood agar

39 firmed that SOD expression is maximal at mid-log phase and is influenced by oxygen, temperature, and

40 acetyltransferase) was first detected in mid-log phase and reached a maximum in stationary phase, sug

41 y depleted from the active rDNA genes during log phase and reassembled during the diauxic shift, larg

42 the expression of sigD is stable throughout log phase and stationary phase but that it declines rapi

43 These physiological changes differ in log phase and stationary phase cells and are controlled

44 h sarA and sarB being most abundant in early log phase and the sarC concentration being highest towar

45 ains of M. tuberculosis were cultured to mid-log phase and used in both adherence and invasion assays

46 umulation gradually declined following early log phase and was maintained at stable levels throughout

47 omyces cerevisiae strain BJ5460 grown to mid-log phase and yielded the largest proteome analysis to d

48 eted by most pathogenic strains, produced in log-phase and controlled by the covR-covS two-component

49 deletion mutant which displayed normal early-log-phase and mid-log-phase growth in bacteriologic medi

50 aprt mutations induced by amsacrine in both log-phase and plateau-phase CHO cells were analyzed.

51 yed in vivo using RecA-GFP foci formation in log-phase and UV-irradiated cells.

52 toxin genes pagA, lef, and cya peaks in late log phase, and steady-state levels of the toxin proteins

53 Maximal activity is produced during early log phase, and the activity is diminished when cells ent

54 the lag phase, remained constant during the log phase, and then rose to starting levels upon entry i

55 During log phase, approximately 50% of the ribosomal DNA (rDNA)

56 clear localization of PKC delta were seen in log phase as compared to confluent C10 cells.

57 e vegetative stage of growth starting at mid-log phase as well as during sporulation.

58 tical determinant of looping, peaks in early log phase at the proximal poly(A) site, but as growth ph

59 l to the stationary phase ATR but not to the log phase ATR.

60 Results from experiments with intact late log phase bacteria and cell walls indicated homogeneous

61 etermined by injecting eyes with 1000 CFU of log phase bacteria plus either 200 ng active purified pr

62 Log-phase bacteria grown in Stainer-Scholte (SS) broth p

63 Compared to the inoculum (mid-log-phase bacteria), H. ducreyi harvested from pustules

64 in was secreted at the highest levels during log-phase bacterial growth.

65 proof-of-principle study, actively growing (log phase) breast cancer (MCF7, MDA-MB-231, and hTERT-HM

66 h curve on PET exhibits the distinct lag and log phases, but the generation time is more than twice l

67 d docking in the internal thylakoid lumen of log phase C. reinhardtii.

68 ated by toxins from stationary phase but not log phase CA-MRSA, and alpha-hemolysin was singularly id

69 nt at about 24,000 copies of monomer per mid-log phase cell, binds double-stranded DNA with a site si

70 quences were identified, six associated with log phase cells and four with stationary phase cells.

71 ese sites reduced both the basal activity in log phase cells and the cell cycle regulated activity of

72 usceptibility to the induction of apoptosis (log phase cells arrested in G1 for 48 hours by isoleucin

73 antial leaky expression could be effected in log phase cells by adding cyclic AMP and acetate at pH6.

74 foci twofold to threefold in a population of log phase cells grown in minimal medium.

75 that render stationary cells as sensitive as log phase cells identifies only mutations that block res

76 It is proposed that log phase cells produce DSBs that do not induce the SOS

77 In extracts from log phase cells, E2F complexes contained predominantly E

78 In log phase cells, foci were not localized to any particul

79 In late log phase cells, the most upregulated mRNA encoded the n

80 parasites, scbetaAraT activity is maximal in log phase cells.

81 opy of 4',6-diamidino-2-phenylindole-stained log phase cells.

82 but was observed in As(III)-unexposed, late-log-phase cells and (ii) treating As(III)-unexposed, ear

83 in comparisons of Tf1 protein extracted from log-phase cells and that extracted from stationary-phase

84 aoxAB was absent in As(III)-unexposed early-log-phase cells but was observed in As(III)-unexposed, l

85 The log-phase cells contained equal molar amounts of Gag and

86 lular glycogen content was observed in early log-phase cells grown on trehalose, which was followed b

87 has been shown that, while many (15 to 25%) log-phase cells have RecA filaments, few (about 1%) are

88 posed in a typical assay for acid tolerance (log-phase cells in minimal glucose medium) was shown to

89 t RecA's ability to induce SOS expression in log-phase cells is repressed because of the potentially

90 ith HeLa cells demonstrated that the tips of log-phase cells of GUH-2 adhered to and penetrated the s

91 ockout mice) were infected intranasally with log-phase cells of N. asteroides GUH-2.

92 to pulmonary infection of the murine lung by log-phase cells of Nocardia asteroides.

93 icroscopy demonstrated that only the tips of log-phase cells penetrated pulmonary epithelial cells fo

94 )H isotope ratio of intracellular water from log-phase cells showed an appreciably larger contributio

95 to a change in iron level, iron-starved late-log-phase cells were diluted in fresh low- and high-iron

96 s and (ii) treating As(III)-unexposed, early-log-phase cells with ethyl acetate extracts of As(III)-u

97 ted X-shaped DNA molecules at the origins in log-phase cells, but these were not directly associated

98 tions dominated the spectrum of mutations in log-phase cells, but were much less prevalent in plateau

99 comprises 0.36 to 0.55% of the dry weight of log-phase cells, depending on culture medium, and this a

100 In log-phase cells, dinI mutants have fewer foci than wild

101 the protein in cells, thin sections of late-log-phase cells, sporulating cells, and free spores were

102 By introducing Psi93 into log-phase cells, we further show that Psi93 has a role i

103 were more mutagenic in plateau-phase than in log-phase cells.

104 cyclin/Cdc28 complexes and not Cln/Cdc28 in log-phase cells.

105 s-)resistance to a 60-min PmB challenge than log-phase cells.

106 nt mechanisms that repress SOS expression in log-phase cells.

107 likely to detach than clustered ones in the log phase; clustered bacteria in micro-colonies have str

108 orneas were intrastromally injected with 100 log phase colony-forming units (CFU) of S. aureus 8325-4

109 t oriC may stimulate initiation during early log phase compared with the lesser activity of the alpha

110 lls treated with rapamycin and cells in post-log phase confirm this conclusion and demonstrate that t

111 brief period of reduced CRP binding in early log phase corresponds to a transient burst of P1 transcr

112 ase of growth rates in batch cultures during log phase, culminating with the onset of stationary phas

113 acetate extracts of As(III)-unexposed, late-log-phase culture supernatants also resulted in aoxAB in

114 he main lethal factor present in type C late-log-phase culture supernatants.

115 s detectable only in supernatant fluids from log phase cultures.

116 st exclusively within the cytoplasm in fresh log-phase cultures assessed by cryo- immunoelectron micr

117 ayed for altered localization of HrpZ in mid-log-phase cultures by immunoblotting sodium dodecyl sulf

118 nd to be undetectable in supernatants of mid-log-phase cultures containing >1% glucose but abundant i

119 Accumulation of this protein was observed in log-phase cultures following a shift to oxygen-limiting

120 We have used log-phase cultures of body cavity-based lymphoma 1 cells

121 Streptococci harvested from log-phase cultures readily bound FH, but binding was gre

122 r frequency analyses of both synchronous and log-phase cultures showed that origin utilization was cl

123 In log-phase cultures, ComK is trapped in a complex compose

124 In early-log-phase cultures, LipL36 is one of the most abundant L

125 direct addition of linear DNA constructs to log-phase cultures.

126 esent in conditioned media derived from late-log-phase cultures.

127 addition of antibodies to Dkk-1 in the early log phase decreased proliferation.

128 ne fractions from cells harvested in the mid-log phase demonstrated very little nucleoside triphospha

129 of the intracellular water hydrogen atoms in log-phase E. coli cells are isotopically distinct from t

130 d intensities with intensities estimated for log-phase E. coli on the basis of the knowledge of its k

131 age intensities has been made for spectra of log-phase E. coli.

132 During rapid growth in mid-log phase, E. coli generates 15% more negative supercoil

133 wth, up to 70% of the intracellular water in log-phase Escherichia coli cells was actually generated

134 of cells exposed to Cefazolin at the end of log phase exhibited a different behavior.

135 ceptions are the gene cad, which has reduced log phase expression and serum induction in c-myc null c

136 ttCxattI recombination products were seen in log phase for both strains; however, in stationary phase

137 In the past two years we have entered the log phase for unraveling the molecular clockworks.

138 When cells were grown in LB medium to log phase, four transcription initiation sites could be

139 gulated 14-fold when the bacteria enter late log phase from early log phase.

140 49 alveolar epithelial cells were exposed to log-phase GBS or stabilized hemolysin extracts of GBS cu

141 ha-toxin or perfringolysin O) levels in late-log-phase genotype D supernatants and lethality.

142 early S- and log-phase HeLa cells, and from log-phase GM06990, a karyotypically normal lymphoblastoi

143 eletion mutant (Deltaacr2) was unimpaired in log phase growth and persisted in IFN-gamma-activated hu

144 A supported editing under conditions of late log phase growth and stationary phase.

145 ehave similar to wild-type astrocytes during log phase growth but demonstrate increased saturation de

146 in the wild type strain under aerobic, late log phase growth conditions.

147 ed to express AGS1/RASD1 (Ad.AGS1) inhibited log phase growth in vitro and increased the percentage o

148 om at the 4-position of tryptanthrin stopped log phase growth of E. coli cultures at concentrations a

149 in contrast to other reports, did not affect log phase growth rates.

150 cripts revealed maximum transcription during log phase growth, an observation confirmed by promoter f

151 rylation sites did not form eisosomes during log phase growth, indicating that phosphorylation is cri

152 ificantly elevated in the rye mutants during log phase growth.

153 Microscopic observation of early-log-phase growth (2 to 3 h) in a shaking broth was the b

154 fter UV treatment differed from those during log-phase growth and may reflect the different DNA subst

155 obtain a census of proteins expressed during log-phase growth and measurements of their absolute leve

156 In vitro, hpyIM expression was higher during log-phase growth and was required for normal bacterial m

157 strated a small but reproducible decrease in log-phase growth but achieved the same plateau density.

158 f HB-EGF-stimulated growth by heparin and of log-phase growth by CRM 197, a specific inhibitor of HB-

159 microbial killing of M. tuberculosis during log-phase growth ceases because of the depletion of this

160 ags of 2 to 5 days prior to the induction of log-phase growth compared to wild-type strains.

161 ugh transcription of tcpPH is reduced at mid-log-phase growth in an El Tor strain, transcription is t

162 ich displayed normal early-log-phase and mid-log-phase growth in bacteriologic medium but survived le

163 e find that CLN3 mRNA levels are high during log-phase growth in glucose medium, low in postdiauxic c

164 Acid induction during log-phase growth in minimal medium appears to occur thro

165 while transcription of tcpPH differs at mid-log-phase growth in ToxR-inducing conditions between the

166 l strains of V. cholerae is activated at mid-log-phase growth in ToxR-inducing conditions, while tran

167 g studies were performed with ethambutol and log-phase growth Mycobacterium tuberculosis to identify

168 n urine, the oxyRS mutant exhibited the same log-phase growth rate (broth) and plateau density (broth

169 HFS-TB studies included log-phase growth studies under ambient air, semidormant

170 our genes decreased as cells progressed from log-phase growth to stationary phase and increased after

171 e evaluated for toxin production during late-log-phase growth via quantitative Western blotting and b

172 ef-lacZ and cya-lacZ expression during early-log-phase growth was increased only two- to threefold in

173 s vector) CD20+ human Raji lymphoma cells in log-phase growth were incubated with or without rituxima

174 that LuxO represses hapR expression early in log-phase growth, and constitutive expression of hapR bl

175 During log-phase growth, B. pertussis releases the muramyl pept

176 ificant amounts from some pneumococci during log-phase growth, because autolysis was not believed to

177 wed regulated signaling that was high during log-phase growth, low during contact-inhibited different

178 til it was induced during the second half of log-phase growth, with expression becoming maximal durin

179 te that pbpC is transcribed primarily during log-phase growth, with lower amounts expressed during sp

180 significantly increased during mid- to late-log-phase growth.

181 r ADA3/NGG1 deletion mutants are examined in log-phase growth.

182 each gene occurring during the last half of log-phase growth.

183 rains was rapid and occurred near the end of log-phase growth.

184 ynthesized 1 to 2 weeks following the end of log-phase growth.

185 er decline in viability following the end of log-phase growth.

186 oxin (CPA) and perfringolysin O (PFO) during log-phase growth.

187 alf-lives of all transcripts produced during log-phase growth.

188 may be required for the entry of cells into log-phase growth.

189 ion of pulmonary epithelial cell surfaces by log-phase GUH-2 and inhibited spread to the brain.

190 orbed IMS preferentially labeled the tips of log-phase GUH-2 cells.

191 ere is only moderate concordance between the log-phase HeLa bubble map and published maps of small na

192 ive human origin libraries from early S- and log-phase HeLa cells, and from log-phase GM06990, a kary

193 detectable in the membrane fraction at early log phase in comparison with the levels present at late

194 The rel(Bbu) mutant grew well during log phase in complete BSK-H but reached lower cell conce

195 Strain W3110 was grown to early log phase in complex broth buffered at pH 4.9, 6.0, 8.0,

196 rain and a gcvB deletion strain grown to mid-log phase in Luria-Bertani broth.

197 hypothetical proteins) was activated in late log phase in OG1RF versus the fsrB deletion mutant, expr

198 rities to the responses induced by exit from log phase, including decreased virulence factor transcri

199 men (cecum) was compared with that of a late-log-phase LB broth culture using a whole-genome microarr

200 Log phase lcb4-deleted yeast cells make no LCB phosphate

201 (+/-)) littermates when inoculated i.v. with log-phase Listeria monocytogenes.

202 About 5% of the log-phase mutants and 16% of the plateau-phase mutants w

203 ing changes in mitotic activity at the early log phase of cell growth.

204 We found that cultured CASMCs during log phase of growth and smooth muscle cell layer of the

205 rate induces yhcS transcription in the early log phase of growth under anaerobic conditions and that

206 as a monocistronic message, in the early mid-log phase of growth, and this coincides with the appeara

207 s also demonstrate that during the early mid-log phase of growth, LuxS regulates the transcript level

208 In log phase of growth, when compared with wild type (WT) o

209 e found in the outside medium during the mid-log phase of growth, when the optical density at 650 nm

210 ferential and high affinity for hMSCs in the log phase of proliferation.

211 res displayed a lag phase of about 5 days, a log phase of rapid growth of about 5 days, and then a st

212 lower than that observed at mid-log and end-log phases of growth.

213 was most abundant in cells grown to the late log phase on acetate but barely detectable in cells grow

214 e grown at 37 degrees C and harvested at mid-log phase or early stationary phase to serve as the targ

215 iscrimination was possible with cells in mid-log phase or in lag phase.

216 studied, using bacterial cells in either the log phase or stationary phase of aerobic growth, and usi

217 sulting in synergistic lethality to cultured log-phase or quiescent malignant cells.

218 e organisms bound only 10% as much C3 as mid-log-phase organisms; this was only in part due to capsul

219 Conditioned medium from a late-log-phase P. gingivalis culture induced the luciferase o

220 total CP (serotypes 1 and 5 CP combined) in log phase (P = 0.0012) but significantly more total CP i

221 ely affects the transition from lag phase to log phase, perhaps through increasing oxidative stress,

222 scription from this promoter starts in early log phase, prior to the transcription of speB.

223 Transcript abundance in vitro (mid-log phase) ranged from about 0.004 (feoB and hpaA) to 20

224 taAraTs away from the Golgi apparatus during log phase, regulation of activated Ara precursors may co

225 or 97% and 87% of the ORFs in stationary and log phases, respectively.

226 re scarified and topically inoculated with a log phase S. aureus parent strain (8325-4), an alpha-tox

227 Log-phase S. aureus, expressing minimal capsule, was inc

228 Interestingly, (unadapted) log-phase S. typhimurium rpoS and stiA mutants were very

229 8 and 12%, respectively, and pretreatment of log-phase S. typhimurium with 15% bile dramatically incr

230 emolytic toxin was purified from extracts of log-phase spirochetes, and the N-terminal amino acid seq

231 One hundred colony-forming units of log phase Staphylococcus aureus was injected intrastroma

232 These results suggest that log-phase susceptibility to complement is not due to var

233 A squash technique was developed for log phase Tetrahymena pyriformis which permitted the res

234 t it no longer expressed 27 proteins in late log phase that were present in its isogenic parent.

235 When grown to the mid-log phase, the double mutant was significantly more resi

236 In log phase, the level of the longer transcript is higher

237 E. coli cells grown in minimal medium to mid-log phase, the ratio of free to DNA-bound Lrp was about

238 During the late log phase, the RS-2 cells decreased in number and regene

239 When grown for several days in log phase, the tlc1Delta cells initially display wild-ty

240 vity, resulting in increased sigmaS level in log-phase, the transcription from promoter Px1 during lo

241 ssion in M. tuberculosis cultures from early log phase through late stationary phase indicates that e

242 olymerases change during the transition from log phase to long-term stationary phase.

243 ity is required throughout the transition of log phase to stationary phase (diauxic shift) for effect

244 61 array elements during the transition from log phase to stationary phase, including declining trans

245 It was found that the majority of log-phase transcripts (90%) have a short half-life (<5 m

246 When late-log-phase vegetative culture supernatants were analyzed

247 al toxins in supernatants prepared from late-log-phase vegetative cultures of a large collection of g

248 Similar to natural type C infection, log-phase vegetative cultures of wild-type CN3685 caused

249 growth conditions (anaerobic vs. aerobic and log-phase vs. stationary-phase bacterial cells) had no n

250 , the transcription from promoter Px1 during log phase was increased.

251 , a quorum signaling molecule active in late log phase, was synthesized by Shigella species and enter

252 When cells in the log phase were compared, Lrp levels were 3- to 4-fold hi

253 cal analyses showed that growth rates during log phase were not controlled primarily by the availabil

254 to APEC's microaerobic growth at the lag and log phases when cultured in duck serum and that ArcA pla

255 r multiply septated sclerotic bodies in post-log phase, when the G14V-altered protein was diminished.

256 pronounced effects on transcription in early log phase, where gene expression was repressed in the mu

257 anslation were expressed at higher levels in log phase, whereas many genes known to be involved in th

258 on electron microscopy of freeze-substituted log-phase wild-type cells, fibril material was observed

259 script encoding LysRS2 was detectable during log phase with either substrate.

260 re activation of each gene in cells at early log phase, with activation values ranging from 6- to 26-

261 When irradiated log phase WT cells were in rich medium, two TS domains w

262 Experiments show that while log phase xthA cells have twofold more double-stranded b