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1 nd expand upon the clinical phenotype of the logopenic variant.
2 found in nfvPPA, but not in the semantic or logopenic variants.
3 n the distinction of the agrammatic from the logopenic variant and left considerable latitude to clin
4 non-fluent/agrammatic variant, and four with logopenic variant), as well as 28 age-matched healthy co
5 be classified into non-fluent, semantic and logopenic variants based on motor speech, linguistic and
8 s with primary progressive aphasia including logopenic variant (n = 14, age = 61 +/- 9 years), non-fl
11 The aim is to explore the evolution of the logopenic variant of primary progressive aphasia as a di
12 terior cortical atrophy, 4 subjects with the logopenic variant of primary progressive aphasia, 6 age-
13 including posterior cortical atrophy and the logopenic variant of primary progressive aphasia, differ
14 role of short-term memory in a discussion of logopenic variant PPA, and components of language associ
16 nset, <65 y; memory and executive deficits), logopenic variant primary progressive aphasia (language
18 e (age at onset <65 years), 12 patients with logopenic variant primary progressive aphasia and 13 pat
21 interest, the left language network for the logopenic variant primary progressive aphasia region of
22 disease and posterior cortical atrophy than logopenic variant primary progressive aphasia) and highe
23 than in early-onset Alzheimer's disease and logopenic variant primary progressive aphasia), with a t
24 ypical variants (posterior cortical atrophy, logopenic variant primary progressive aphasia, and corti
29 and occipital cortices in patients with the logopenic variant, within the left inferior frontal cort
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