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1 ed to high-dose inhaled corticosteroids plus long-acting beta2 agonists.
2 c despite taking inhaled corticosteroids and long-acting beta(2)-agonists.
3 g high doses of inhaled corticosteroids plus long-acting beta(2)-agonists.
4 79 [CI, 0.67 to 0.94]) but not compared with long-acting beta2-agonists alone (relative risk, 0.82 [C
6 offer insights into the mechanisms by which long-acting beta(2)-agonists and glucocorticoids might r
8 d on inhaled corticosteroids with or without long-acting beta2-agonists and in patients with COPD wit
9 dysplasia and exposure to the combination of long-acting beta2-agonists and inhaled corticosteroids (
10 for renal dysplasia associated with combined long-acting beta2-agonists and inhaled corticosteroids.
11 aler therapy with an inhaled corticosteroid, long-acting beta2-agonist, and long-acting muscarinic an
13 n inhalers that contain a corticosteroid and long-acting beta(2)-agonist as reliever therapy in addit
15 -severe asthma on inhaled corticosteroid and long-acting beta(2)-agonist but not maintenance oral cor
18 nts with COPD from an inhaled corticosteroid/long-acting beta2-agonist combination treatment to tripl
19 responsiveness to an inhaled corticosteroid/long-acting beta2-agonist combination versus a long-acti
21 corticosteroids (ICS) or low-dosage ICS plus long-acting beta2 agonist fixed-combination therapy at s
22 o 7 (maximum 14) days inhaled corticosteroid/long-acting beta2-agonist fluticasone furoate/vilanterol
24 hort-acting beta2-agonist albuterol, and the long-acting beta2-agonists formoterol and olodaterol.
25 to high-dosage inhaled corticosteroids plus long-acting beta(2)-agonists (ICS plus LABA) and a histo
26 S) and inhaled corticosteroids combined with long-acting beta2-agonist (ICS/LABA) are standard treatm
27 e on high-dosage inhaled corticosteroids and long-acting beta2-agonists (ICS plus LABA) in the previo
30 ycopyrrolate), a fixed-dose combination of a long-acting beta2-agonist (indacaterol) and a long-actin
31 l/glycopyrronium 110/50 mug with twice-daily long-acting beta2-agonist/inhaled corticosteroid salmete
32 -to-high-dose inhaled corticosteroids plus a long-acting beta2 agonist, irrespective of baseline eosi
33 erapy, including inhaled corticosteroids and long-acting beta2-agonists, is effective in patients for
34 costeroid (ICS) fluticasone furoate (FF) and long-acting beta(2) -agonist (LABA) vilanterol (VI) on e
36 ids (ICSs) are insufficient include adding a long-acting beta(2)-agonist (LABA) or increasing the ICS
37 erapy with dual inhaled corticosteroid (ICS)/long-acting beta2-agonist (LABA) therapy in patients wit
41 umulating for the efficacy of adding inhaled long-acting beta(2)-agonists (LABAs) to corticosteroids
44 OPD and treated with inhaled corticosteroid, long-acting beta2 agonist, long-acting muscarinic antago
45 cerbations) study, which compared once-daily long-acting beta2-agonist/long-acting muscarinic antagon
46 ng-acting beta2-agonist combination versus a long-acting beta2-agonist/long-acting muscarinic antagon
48 ugh there is still controversy as to whether long-acting beta2-agonists may increase the risk of asth
49 = 7), long-acting anticholinergics (n = 10), long-acting beta2-agonists (n = 22), corticosteroids (n
50 tment (high-dose inhaled corticosteroids and long-acting beta2-agonists or medium- to high-dose inhal
51 reas the reduction in airway eosinophilia by long-acting beta(2)-agonists probably operates through a
52 nary disease (COPD) and, in combination with long-acting beta2 agonists, reduce exacerbations and imp
54 -to-high-dose inhaled corticosteroids plus a long-acting beta2 agonist require additional treatment o
55 rolled, crossover study of the effect of the long-acting beta(2)-agonist salmeterol on airway inflamm
56 tested the hypothesis that inhaled combined long-acting beta2-agonist (salmeterol) and corticosteroi
57 fluticasone propionate (F), with an inhaled long-acting beta(2)-agonist, salmeterol (S), to treat th
58 d that higher doses of inhaled steroids with long-acting beta2 agonists should be used for total cont
59 in addition to inhaled corticosteroids plus long-acting beta2-agonist therapy could improve the live
61 r without a leukotriene receptor antagonist; long-acting beta2-agonist therapy was not permitted duri
62 vious 12 months, with adjustment for ICS and long-acting beta(2)-agonists use (odds ratio, 2.7; 95% c
64 -to-high-dose inhaled corticosteroids plus a long-acting beta2 agonist were eligible for participatio
65 roids combined with oral corticosteroids and long-acting beta2-agonists) were extracted from 65 Dutch
67 s year, and receiving inhaled corticosteroid/long-acting beta2-agonist with or without LAMA daily for
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