ライフサイエンスコーパス: long terminal repeat

1 docking of Tat at the TAR region of the HIV long terminal repeat.

2 s and differential DNA methylation at its 5'-long terminal repeat.

3 ngle-stranded RNA40 (ssRNA40) from the HIV-1 long terminal repeat.

4 of NF-kappaB and its recruitment to the HIV-long terminal repeat.

5 ription through HIF-1 association with HIV-1 long terminal repeat.

6 al cells using the mouse mammary tumor virus long terminal repeat.

7 the HIV Tat protein to transactivate the HIV long terminal repeat.

8 s and histone-modifying enzymes to the HIV-1 long terminal repeat.

9 thers had acquired dual mutations within the long terminal repeat.

10 tions are critical for Tat activation of the long terminal repeat.

11 promoter via trans-activation of the HRES-1 long terminal repeat.

12 prevent downstream transcription from the 3' long terminal repeat.

13 romoter sequences in the U3 region of the 3' long terminal repeat.

14 ta-globin locus within the self-inactivating long-terminal repeat.

15 mating the age of the elements, if they have long terminal repeats.

16 most common hybrid genomes had heterologous long terminal repeats.

17 ion of endogenous retrotransposons that bear long terminal repeats.

18 d three imperfect 21-bp repeats in the viral long terminal repeats.

19 anscripts is promoted by endogenous intronic long terminal repeats.

20 omplex that is associated with repressed HIV long terminal repeats.

21 A mutant is impaired in the synthesis of one-long terminal repeat (1-LTR) and 2-LTR circles.

22 l vector genomes that consisted of linear, 1-long-terminal-repeat (1-LTR), and 2-LTR circular DNAs.

23 verse transcripts, a moderate reduction of 2-long terminal repeat (2-LTR) circles, and a relatively l

24 HIV-1 DNA, including 2-long terminal repeat (2-LTR) circles, and multiply splic

25 Two-long terminal repeat (2-LTR) circles, which are formed i

26 iral DNA to circularization in the form of 2-long terminal repeat (2-LTR) circles.

27 g total HIV DNA, integrated HIV DNA, and two long terminal repeat (2-LTR) circles.

28 as defined by an increase in the level of 2-long terminal repeat (2-LTR) circles.

29 seminal plasma CMV (P = 0.04), detectable 2-long terminal repeat (2-LTR), and lower nadir CD4(+) (P

30 An increase in 2-long-terminal-repeat (2-LTR) circles in the depleted FAC

31 onintegrated viral DNA, particularly the two-long-terminal-repeat (2-LTR) circles.

32 episomes containing two copies of the viral long terminal repeat (2LTR circles) were analyzed in usi

33 ediates showed that integration, but not two-long-terminal-repeat (2LTR) circles or late cDNAs, was r

34 6-bp extension at the viral U5 end of the 3' long terminal repeat (3'-LTR), which is a poor substrate

35 he gene fragment specifically targets the 3' long terminal repeat (3'LTR) in the viral mRNA and block

36 eIF3f and eIF3f specifically targeted the 3' long terminal repeat (3'LTR) region in the viral mRNA.

37 loop structure transcribed from the HIV-1 5' long terminal repeat (5'-LTR).

38 ic drugs and tested these compounds on HIV-1 long terminal repeat-activated transcription.

39 ransactivated gene expression from the HIV-1 long terminal repeat and COT Nef mediated downregulation

40 us under the control of the endogenous HIV-1 long terminal repeat and demonstrated that human cyclin

41 Both P-TEFb recruitment to the HIV long terminal repeat and enhanced HIV processivity were

42 ve and cryptic splice sites in the HIV 5iota long terminal repeat and gag gene as well as in the beta

43 restrictive chromatin structures at the HIV long terminal repeat and limiting P-TEFb levels contribu

44 encoded protein Tax transactivates the viral long terminal repeat and plays a critical role in virus

45 -1(JR-CSF) regulated by the endogenous HIV-1 long terminal repeat and the hu-cycT1 gene under the con

46 ications in the regulatory elements in their long terminal repeats and differed in a helical segment

47 The DNA of the element is marked by long terminal repeats and encodes a single large protein

48 and DNMT3a/3b in suppressing retrotransposon long terminal repeats and long interspersed elements, re

49 ersed repetitive elements such as Alu, LINE, long-terminal repeats and simple tandem repeats are freq

50 efines the end of the U5 region in the right long terminal repeat, and the subsequent removal of this

51 s, mutational analysis of the HERV-K (HML-2) long terminal repeat, and treatments with agents that in

52 E. granulatus genome, especially Gypsy-like long terminal repeats, and there has also been an expans

53 detected at satellite, telomeric and active long-terminal repeats, and can spread into proximal uniq

54 wild type enzyme in assembling on the viral long terminal repeat, as each variant required more prot

55 ecific gene family expansions, the number of long-terminal-repeat-based retrotransposons, and mechani

56 rase II (Pol II) binding to the HIV provirus long terminal repeat but did not prevent the induction o

57 or of the human T-cell leukemia virus type 1 long terminal repeat, but multiple lines of evidence sho

58 ntry, post-reverse transcription and pre-two-long-terminal-repeat circle formation, similar to the cy

59 iral load was associated with undetectable 2-long terminal repeat circles (P < .001) and HIV-negative

60 d in a smaller increase in the number of two-long terminal repeat circles than for virus specifically

61 V production (ie, HIV-specific antibodies, 2-long terminal repeat circles) and markers of immune acti

62 virus type 1 (HIV-1) late RT products and 2-long terminal repeat circles.

63 r HIV RNA transcription or more detectable 2-long terminal repeat circles.

64 ion-competent reservoirs, proviral DNA, or 2-long-terminal repeat circles, although APOBEC3G, TRIM5al

65 mutants synthesized normal quantities of two-long-terminal-repeat circles in arrested HeLa cells, ind

66 rectly processed viral ends and abortive two-long-terminal-repeat circles.

67 (P=.013), and frequency of detectable HIV 2-long terminal repeat circular DNA (P=.013) were signific

68 eeds normally in MX2-expressing cells, but 2-long terminal repeat circular forms of HIV-1 DNA are les

69 We demonstrate that the foamy virus long terminal repeats contain an insulator element that

70 he (+)-RNA strand genome of retroviruses and long terminal repeat-containing retrotransposons reflect

71 that many 2C transcripts are initiated from long terminal repeats derived from endogenous retrovirus

72 ds, and recognize an unexpected abundance of long terminal repeat-derived and LINE1-mobilized transpo

73 cultures, hCMV IE-driven, but not the viral long terminal repeat-driven, silent GFP reporter express

74 the effects of different plasmid-based HIV-1 long-terminal-repeat-driven constructs expressing antise

75 We show that a long terminal repeat element inserted into intron 35 exp

76 e we demonstrate that DNA hypomethylation at long terminal repeat elements representing the most rece

77 Self-inactivating vectors devoid of viral long-terminal-repeat enhancers have proven safe; however

78 and HDAC3 contribute to repression of HIV-1 long terminal repeat expression in the HeLa P4/R5 cell l

79 ongation rate included the density of exons, long terminal repeats, GC content of the gene, and DNA m

80 ppaB binding site was engineered into its 3' long terminal repeat, giving rise to SHIV-1157ipd3N4.

81 ng lentiviral vectors with self-inactivating long terminal repeats, have been shown to have improved

82 fied Tat was unable to transactivate the HIV long terminal repeat in U937 human macrophages.

83 heat shock factor binding HREs within their long terminal repeats in seven Brassicaceae species.

84 Insertions of long terminal repeats in the past 5 million years are re

85 (Lamc2(jeb)) due to a murine leukemia virus long terminal repeat insertion in Lamc2 (laminin gamma2

86 human immunodeficiency virus type 1 (HIV-1) long terminal repeat is present on both ends of the inte

87 es show that this retrotransposon with LTRs (Long Terminal Repeats) is widely distributed among the R

88 indicated that sequences at the envelope-3' long terminal repeat junction are required for proper ex

89 A 476-bp fragment that spans the envelope-long terminal repeat junction had activity equivalent to

90 Using the analysis of two long terminal repeat junctions in HIV-infected cells, we

91 on similar to a repeat associated with a non-long terminal repeat-like element and is often found acc

92 ncomitant inhibition of NF-kappaB and HTLV-1 long terminal repeat (LTR) activation.

93 actors have been reported to stimulate HIV-1 long terminal repeat (LTR) activity.

94 ly recognizes the terminal sequences of each long terminal repeat (LTR) and cleaves the 3'-end termin

95 : MGEScan-long terminal repeat (LTR) and MGEScan-non-LTR are succe

96 ergent, the two orthologs similarly restrict long terminal repeat (LTR) and non-LTR retrotransposons

97 two NF-kappaB sites in the U3 region of the long terminal repeat (LTR) are critical for Cav-1-mediat

98 in (C/EBP) beta and C/EBP sites in the HIV-1 long terminal repeat (LTR) are crucial for HIV-1 replica

99 mulate RNA polymerase II (RNAP II) on the 5' long terminal repeat (LTR) but not on the 3' LTR.

100 f histone proteins at the HIV type 1 (HIV-1) long terminal repeat (LTR) by histone deactylases (HDACs

101 els of circular Ty1 DNA are present with one-long terminal repeat (LTR) circles and deleted circles c

102 e ART were assayed for total HIV-1 DNA and 2-long terminal repeat (LTR) circles by quantitative polym

103 l-signaling pathways and to characterize the long terminal repeat (LTR) cis-acting elements involved

104 , we found that repressive histone marks and long terminal repeat (LTR) DNA methylation could be dete

105 eve targeted insertion of a gamma-retroviral long terminal repeat (LTR) driving a GFP expression cass

106 ing pattern of RelA recruitment to the HIV-1 long terminal repeat (LTR) during continuous tumor necro

107 erged to the Repbase collection of known ERV/long terminal repeat (LTR) elements to annotate the retr

108 ramer or octamer complex with the viral cDNA long terminal repeat (LTR) ends termed an intasome.

109 myc reporter plasmid indicated that the TBLV long terminal repeat (LTR) enhancer is necessary for T-c

110 r, the transcriptional activity of the viral long terminal repeat (LTR) from Vpr-deficient proviruses

111 cts but 11.7% with intact genomes and normal long terminal repeat (LTR) function.

112 rase cleaves the linear viral DNA within its long terminal repeat (LTR) immediately 3' to the CA dinu

113 status of the elongation complex on the HIV long terminal repeat (LTR) in a repressed state is not k

114 strong transcriptional repressor of the HIV long terminal repeat (LTR) in resting alveolar macrophag

115 on of wild-type WRN transactivates the HIV-1 long terminal repeat (LTR) in the absence of Tat, and WR

116 Here, we show that methylation of the HIV 5' long terminal repeat (LTR) in the latent viral reservoir

117 indings suggest that integration of the XMRV long terminal repeat (LTR) into host DNA could impart an

118 If the terminus of the U3 long terminal repeat (LTR) is aberrant, RSV integrase ca

119 ested, but rather transcription of the HIV-1 long terminal repeat (LTR) is increased in IL-4-producin

120 so identified a transcript that contains the long terminal repeat (LTR) of lambda-olt 2-1 and shows a

121 sequence similarity with the R region of the long terminal repeat (LTR) of the yeast Ty1 retrotranspo

122 rther demonstrate that HspBP1 inhibits HIV-1 long terminal repeat (LTR) promoter activity.

123 HDAC1 are coordinately resident at the HIV-1 long terminal repeat (LTR) promoter and absent from the

124 In addition, we detected Tip110 at the HIV-1 long terminal repeat (LTR) promoter and found that Tip11

125 -1 Tat-mediated transactivation of the viral long terminal repeat (LTR) promoter is essential for HIV

126 lencing of viral transcription driven by the long terminal repeat (LTR) promoter of HIV-1.

127 of the G-quadruplex structures in the HIV-1 long terminal repeat (LTR) promoter suppresses viral tra

128 iven by the mouse mammary tumor virus (MMTV) long terminal repeat (LTR) promoter were morphologically

129 t in this cell line was specific for the MLV long terminal repeat (LTR) promoter, as normal levels of

130 complex and recruit P-TEFb to the HIV type 1 long terminal repeat (LTR) promoter.

131 uces expression of the HIV-1 promoter in the long terminal repeat (LTR) region in a Tat-independent m

132 olled by the transcriptional activity of its long terminal repeat (LTR) region.

133 NA encoding the viral proteins is flanked by long terminal repeat (LTR) regions from the retrovirus.

134 Es), long interspersed elements (LINEs), and long terminal repeat (LTR) retroelements, which include

135 long interspersed nuclear element (LINE) and long terminal repeat (LTR) retroposons.

136 Thirty-three percent (228/682) of all long terminal repeat (LTR) retrotransposon sequences (LR

137 The long terminal repeat (LTR) retrotransposon Tf1 of Schizo

138 nvestigated the phylogenetic distribution of long terminal repeat (LTR) retrotransposon, long intersp

139 silencing of all 13 intact copies of the Tf2 long terminal repeat (LTR) retrotransposon.

140 esents known repeats including a majority of long terminal repeat (LTR) retrotransposons (13.67%).

141 rough recombination with specific classes of long terminal repeat (LTR) retrotransposons and organize

142 rated from these isoforms appeared to target long terminal repeat (LTR) retrotransposons and other un

143 Long terminal repeat (LTR) retrotransposons are an abund

144 Long terminal repeat (LTR) retrotransposons are closely

145 CENP-B homologues have been shown to silence long terminal repeat (LTR) retrotransposons by recruitin

146 Long terminal repeat (LTR) retrotransposons constitute a

147 a nearly twofold increase in the deletion of long terminal repeat (LTR) retrotransposons via solo LTR

148 endogenous retroviruses (ERVs), also called long terminal repeat (LTR) retrotransposons, begins with

149 Long terminal repeat (LTR) retrotransposons, the most ab

150 genome surveillance role by controlling Tf2 long terminal repeat (LTR) retrotransposons.

151 81% transposable elements, 77% of which are long terminal repeat (LTR) retrotransposons.

152 abundant elements in eukaryotes are the non long terminal repeat (LTR) retrotransposons.

153 ces within the Ty3/gypsy-like superfamily of long terminal repeat (LTR) retrotransposons.

154 ic transcripts that are initiated within ERV long terminal repeat (LTR) sequences and read-through in

155 We detail here the contribution of different long terminal repeat (LTR) sequences for the establishme

156 promoter phenotypically resembles endogenous long terminal repeat (LTR) sequences, pointing to a sele

157 (HIV-1) integrase (IN) with DNA representing long terminal repeat (LTR) termini was previously assemb

158 a 36-bp insulator located in the foamy virus long terminal repeat (LTR) that has high-affinity bindin

159 t Tax activates transcription from the viral long terminal repeat (LTR) through recruitment of cellul

160 genes; (ii) the 5' end extending from the 5' long terminal repeat (LTR) to the beginning of the capsi

161 er junctions in the viral genome from the 5' long terminal repeat (LTR) to the end of pol.

162 s mapped to the NF-kappaB sites in the HIV-1 long terminal repeat (LTR) U3 and could be transferred t

163 Binding to and regulation of the SIV long terminal repeat (LTR) was examined using electropho

164 The transcriptional activity of the XMRV long terminal repeat (LTR) was found to be higher than t

165 oly(A) site and flanking sequences in the 3' long terminal repeat (LTR) was not polyadenylated detect

166 BRG1 associates with Tax at the HTLV-1 long terminal repeat (LTR), and coexpression of BRG1 and

167 Elk-1 activated transcription of the HTLV-1 long terminal repeat (LTR), and mutations within either

168 alyses of SIV polymerase (pol), STLV tax and long terminal repeat (LTR), and SFV pol and LTR sequence

169 following the reduction in H3K27 at the HIV long terminal repeat (LTR), subsequent exposure to the H

170 he intrinsic toggling of HIV's promoter, the long terminal repeat (LTR), to generate bimodal ON-OFF e

171 mobility group protein A1 (HMGA1) and viral long terminal repeat (LTR), which led to higher levels o

172 an intrinsic NF-kappaB activator, increased long terminal repeat (LTR)-dependent XMRV transcription.

173 We show that long terminal repeat (LTR)-derived transcripts contribut

174 ant ProTalpha protein potently inhibit HIV-1 long terminal repeat (LTR)-driven gene expression in mac

175 We found two instances of long terminal repeat (LTR)-driven provirus transcription

176 D), increases HIV infection by enhancing HIV long terminal repeat (LTR)-driven transcription via the

177 Silencing induced by long terminal repeat (LTR)-encoded cis-acting response e

178 ocytomas transiently transfected with an HIV long terminal repeat (LTR)-luciferase reporter that cont

179 ional elongation complex essential for HIV-1 long terminal repeat (LTR)-mediated and general cellular

180 In the mouse, long terminal repeat (LTR)-retrotransposons, or endogeno

181 ericentromeric sequence, uncovered 45 intact long terminal repeat (LTR)-retrotransposons.

182 the transcription initiation site on the HIV long terminal repeat (LTR).

183 methylation of histones located at the viral long terminal repeat (LTR).

184 (H3K4me2) that colocalizes with a retroviral long terminal repeat (LTR).

185 e element (HRE) localized in the proviral 5' long terminal repeat (LTR).

186 ts, as well as the coactivator CBP, with the long terminal repeat (LTR).

187 P/HEXIM1 complex for activation of the viral long terminal repeat (LTR).

188 n, FoxP3 enhances gene expression from HIV-1 long terminal repeat (LTR).

189 integrase and bases at the end of the viral long terminal repeat (LTR).

190 equired for Tax transactivation of the viral long terminal repeat (LTR).

191 an T-cell lymphotropic virus type 1 (HTLV-1) long terminal repeat (LTR).

192 PBS and repress transcription from the HIV-1 long terminal repeat (LTR).

193 in the CD28-responsive element of the HIV-1 long terminal repeat (LTR).

194 by activating the HIV-1 promoter, termed the long terminal repeat (LTR).

195 -A/61E, in the surface glycoprotein (SU) and long terminal repeat (LTR).

196 rspersed nuclear elements (LINE), but not in long terminal repeats (LTR).

197 BEL/Pao-like long-terminal repeat (LTR) retrotransposons were annotat

198 ions to provide full-length repeats and, for long-terminal repeat (LTR) retrotransposons, calculates

199 n of a variety of retroelements, such as the long-terminal repeat (LTR)-containing MusD and Ty1 eleme

200 d effects on nuclear histone acetylation and long-terminal-repeat (LTR) transcription.

201 nct from their well-characterized effects of long-terminal-repeat (LTR)-driven gene expression.

202 iciency of elongation of the HIV-1 promoter (long terminal repeat [LTR]) transcripts.

203 thylation of, the integrated viral promoter (long terminal repeat [LTR]).

204 We tag hPSCs by GFP, expressed by the long terminal repeat (LTR7) of HERVH endogenous retrovir

205 rs (gammaRV/LV) with self-inactivating (SIN) long terminal repeats (LTRs) and internal moderate cellu

206 CENP-Bs also repress solo long terminal repeats (LTRs) and LTR-associated genes.

207 g endogenous retroviruses and their solitary long terminal repeats (LTRs) compose >40% of the human g

208 irus contains identical 5' and 3' peripheral long terminal repeats (LTRs) containing bidirectional pr

209 from intrachromosomal recombination between long terminal repeats (LTRs) flanking GAP1.

210 Retrotransposons often carry long terminal repeats (LTRs) for retrovirus-like reverse

211 Retrotransposons containing long terminal repeats (LTRs) form a substantial fraction

212 atin insulators, and self-inactivating (SIN) long terminal repeats (LTRs) may have significantly redu

213 omologous recombination between the flanking long terminal repeats (LTRs) of a full-length element, l

214 er Oryza genomes, and based on the dating of long terminal repeats (LTRs) of FRetro3 it was amplified

215 rates of nucleotide substitution between two long terminal repeats (LTRs) of individual orthologous L

216 the strong promoter/enhancer elements in the long terminal repeats (LTRs) of murine leukemia virus (M

217 0 to the HTLV-1 promoter, located within the long terminal repeats (LTRs) of the provirus.

218 ing enabled the accurate prediction of 20.5% long terminal repeats (LTRs) that doubled the previous e

219 d enrichment in transposable elements (TEs): long terminal repeats (LTRs) were randomly located acros

220 clustering of telomeres and retrotransposon long terminal repeats (LTRs)) were observed throughout t

221 genous retroviral elements (ERVs) containing long terminal repeats (LTRs), are silenced through trime

222 ) contains identical DNA sequences, known as long terminal repeats (LTRs), at its 5' and 3' ends.

223 YC; this is flanked by two loxP sites in its long terminal repeats (LTRs).

224 young transposable elements (TEs), including Long-Terminal-Repeats (LTRs) and SINE-VNTR-Alus (SVAs),

225 hat Tax-mediated activation of luciferase in long terminal repeat-luciferase (LTR-LUC) mice serves as

226 red with transgenic NF-kappaB reporters (HIV-long terminal repeat/luciferase [HLL]), we found exagger

227 trol of the mouse mammary tumor virus (MMTV) long-terminal repeat (MMT mice).

228 DNA repair response from expression of a non-long terminal repeat (non-LTR) retrotransposon in mammal

229 The non-long terminal repeat (non-LTR) retrotransposon R2 is ins

230 Non-long terminal repeat (non-LTR) retrotransposons are a cl

231 Non-long terminal repeat (non-LTR) retrotransposons are high

232 Many non-long terminal repeat (non-LTR) retrotransposons lack int

233 The Drosophila non-long terminal repeat (non-LTR) retrotransposons TART and

234 R2 elements are non-long terminal repeat (non-LTR) retrotransposons with a s

235 ill have the hallmarks that characterize non-long terminal repeat (non-LTR) retrotransposons; they ha

236 he retrotransposition activity of the L1 non-long-terminal-repeat (non-LTR) retrotransposon in both H

237 -1 (LINE-1 or L1) elements are abundant, non-long-terminal-repeat (non-LTR) retrotransposons that com

238 irus promoter is not diminished, whereas the long terminal repeat of a retrovirus, like the ICP0 prom

239 ird 21-bp transcription element found in the long terminal repeats of HTLV-1 and HTLV-2 but instead c

240 The long terminal repeats of lymphomagenic P-MLVs are differ

241 as we found TINATs to be encoded in solitary long terminal repeats of the ERV9/LTR12 family, which ar

242 he inhibition of Tat-dependent activation of long terminal repeat promoter leading to reduced GFP exp

243 the control of the mouse mammary tumor virus long terminal repeat promoter, develop multifocal hyperp

244 RP1 protein enhances Tat-mediated HIV-1 LTR (long terminal repeat) promoter activity.

245 c protein (WAP) or mouse mammary tumor virus-long terminal repeat promoters, develop mammary tumors.

246 the binding of C/EBPbeta and p65 to the SIV long terminal repeat region in colonic lamina propria ce

247 ased Tat-mediated transactivation of the HIV long terminal repeat region, and this functionality was

248 F/EYFP mice, which are transgenic for both a long terminal repeat-regulated full-length infectious HI

249 trate that this model can be useful to study long terminal repeat regulation, as previously character

250 families of long interspersed element 1 and long terminal repeat retroelements, which are disparatel

251 Large long terminal repeat retrotransposon clusters occupy sig

252 domolecules, including coverage of the major Long Terminal Repeat retrotransposon families.

253 host-silencing pathways, particularly copia long terminal repeat retrotransposon in Drosophila melan

254 Previous studies of Tf1, a long terminal repeat retrotransposon in Schizosaccharomy

255 r gene mutation is due to the insertion of a long terminal repeat retrotransposon in the Or allele.

256 Ty1, a long terminal repeat retrotransposon of Saccharomyces, i

257 on of the GP(Y/F) domain in the IN of Tf1, a long terminal repeat retrotransposon of Schizosaccharomy

258 obase gene duplication event mediated by the long terminal repeat retrotransposon Rider.

259 This reduced capacity for long terminal repeat retrotransposon silencing and remov

260 The long terminal repeat retrotransposon Tf1 of Schizosaccha

261 Like its retroviral relatives, the long terminal repeat retrotransposon Ty1 in the yeast Sa

262 The Saccharomyces cerevisiae long terminal repeat retrotransposon Ty3 assembles its G

263 The Saccharomyces cerevisiae long terminal repeat retrotransposon Ty3 integrates with

264 In the Saccharomyces cerevisiae genome, the long terminal repeat retrotransposon Ty3 is found at RNA

265 ement is not a DNA transposon but instead is long terminal repeat retrotransposon-like with human end

266 Tf1, a long-terminal repeat retrotransposon in Schizosaccharomy

267 We initially investigated 510 long terminal repeat-retrotransposon (LTR-RT) families c

268 Analyzing the long terminal repeat-retrotransposon (LTR-RT) type of TE

269 Long terminal repeat retrotransposons (LTR-RTs) are prev

270 of transposable elements (TEs), particularly long terminal repeat retrotransposons (LTR-RTs), in reco

271 d primarily by the periodic amplification of long terminal repeat retrotransposons (LTR-RTs).

272 mologous end joining, mediates clustering of long terminal repeat retrotransposons at centromeres in

273 tion and evolutionary and genomic studies of long terminal repeat retrotransposons in other genomes.

274 Integrases (INs) of retroviruses and long terminal repeat retrotransposons possess a C-termin

275 iniature (TRIMs) are a unique group of small long terminal repeat retrotransposons that are difficult

276 ensis genome due to a rapid amplification of long terminal repeat retrotransposons that occurred 38

277 s in T. halophila were found to contain five Long Terminal Repeat retrotransposons, MuDR DNA transpos

278 These repeat clusters are almost exclusively long terminal repeat retrotransposons, of which the pale

279 Unlike other long terminal repeat retrotransposons, TRIMs are enriche

280 ement activity predominately driven by Gypsy long terminal repeat retrotransposons, which extended th

281 ely derived from internal deletions of large long terminal repeat retrotransposons.

282 ersity of the replication strategies used by long terminal repeat retrotransposons.

283 Finally, analysis of KERV long terminal repeat sequences using massively parallel

284 o the consensus sequence in both protein and long terminal repeat sequences.

285 endogenous retroviruses [ERVs] and 488 solo long terminal repeats [sLTRs]) within the C57BL/6J mouse

286 l replication-competent avian leukosis virus long terminal repeat, splice acceptor (RCAS)/TVA system

287 of uridine-rich ssRNA derived from the HIV-1 long terminal repeat (ssRNA40) on activation of NK cells

288 ete proviruses and proviruses devoid of a 5' long terminal repeat, suggesting that the expression of

289 t least five nucleosomes, is found at the 5' long terminal repeat, the location and modification stat

290 ween the 3' end of the coding region and the long terminal repeat, this retrotransposon family contai

291 Tax transactivates the viral long-terminal repeat through a series of protein-protein

292 r contains additional NF-kappaB sites in the long terminal repeats to enhance viral replicative capac

293 otide (nt) pri-miRNA, encoded within the BFV long terminal repeat U3 region, that is subsequently cle

294 of transcription factors to the HIV provirus long terminal repeat using chromatin immunoprecipitation

295 of functional annotations (e.g. centromeres, long terminal repeats) using 3D genome reconstructions f

296 transcription; M. minutoides cells produce 2-long-terminal-repeat viral DNA circles and linear viral

297 sponsive MMTV-LTR (mouse mammary tumor virus-long terminal repeat), we show that BRG1 and BRM are rec

298 rtions of the viral envelope gene and the 3' long terminal repeat were tested in the presence and abs

299 ial cells and the replication-competent ASLV long terminal repeat with a splice acceptor/tv-a glioma

300 Given the shift toward self-inactivating long terminal repeats with weaker promoters to control t