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1 mory such as working, episodic, spatial, and long term memory.
2 milar context until the link crystallizes in long term memory.
3 estored spine density prevents the rescue of long-term memory.
4 engram cells restores both spine density and long-term memory.
5 resulting in chronic deficits in working and long-term memory.
6 g and short-term memory while also impairing long-term memory.
7 between physical activity during the day and long-term memory.
8 f, generally ineffective training to produce long-term memory.
9 neprilysins are involved in middle-term and long-term memory.
10 tic regulation of gene expression underlying long-term memory.
11 esponsive genes, including those involved in long-term memory.
12 and unstable memory transforms into a stable long-term memory.
13 yonic axis specification to the formation of long-term memory.
14 rophysiological signal hypothesized to index long-term memory.
15 T cell response, promoting the formation of long-term memory.
16 ) it is involved in spontaneous retrieval of long-term memory.
17 he first probe trial, and exhibited impaired long-term memory.
18 e same time enabling self-renewal needed for long-term memory.
19 established LTP and disrupt several forms of long-term memory.
20 ct the B-cell compartment and maintenance of long-term memory.
21 schema congruence has a strong influence on long-term memory.
22 an immediate-early gene that is required for long-term memory.
23 ong-lasting forms of synaptic plasticity and long-term memory.
24 n synthesis necessary for the maintenance of long-term memory.
25 NA plays a critical role in consolidation of long-term memory.
26 d versus massed training in the formation of long-term memory.
27 2 significantly boosted contextual and trace long-term memory.
28 ich are the cellular analogs of two forms of long-term memory.
29 stabilized for consolidation of associative long-term memory.
30 oach to help elucidate the synaptic basis of long-term memory.
31 for both persistent synaptic plasticity and long-term memory.
32 function and improve synaptic plasticity and long-term memory.
33 KII and GluA1 levels and impairs associative long-term memory.
34 oted asynchronous GABA release, and impaired long-term memory.
35 ventral tegmental area predicted associative long-term memory.
36 c plasticity, a cellular mechanism mediating long-term memory.
37 erent kinds of information in the service of long-term memory.
38 ects of CaMKIINtide on both GluA1 levels and long-term memory.
39 specifically improve olfactory learning and long-term memory.
40 to be necessary for both LTP maintenance and long-term memory.
41 impaired the ability of CD8+ T cells to form long-term memory.
42 necessary for the conversion from short- to long-term memory.
43 functional consequence of which is impaired long-term memory.
44 protein synthesis at activated synapses, and long-term memory.
45 nding and intron removal selectively impairs long-term memory.
46 plasticity contributes to the maintenance of long-term memory.
47 ck into working memory during retrieval from long-term memory.
48 consolidating the learned association into a long-term memory.
49 that forms functional amyloids necessary for long-term memory.
50 ole in binding temporally adjacent events in long-term memory.
51 res synapse number, synaptic plasticity, and long-term memory.
52 differed markedly on subsequent measures of long-term memory.
53 wn to be a key component in the formation of long-term memory.
54 JJJ2 enhances the animal's capacity to form long-term memory.
55 vation mediated IEG expression implicated in long-term memory.
56 er, this is at the expense of forming stable long-term memory.
57 N-terminal targeting domain, does not affect long-term memory.
58 genously boost memory consolidation and thus long-term memory.
59 own to be important for Orb2 aggregation and long-term memory.
60 to be crucial for converting experience into long-term memory.
61 KC isoform proposed to maintain late-LTP and long-term memory.
62 involved in the formation and maintenance of long-term memory.
63 crucial for expression and likely storage of long-term memory.
64 imental to the maintenance of information in long-term memory.
65 identified as relating to the maintenance of long-term memory.
66 or, consistent with impaired ability to form long-term memories.
67 ng the synaptic changes required for storing long-term memories.
68 ibutes both to the encoding and retrieval of long-term memories.
69 architecture that supports it and shapes our long-term memories.
70 dependent changes in neural circuits mediate long-term memories.
71 vity via inhibitors is sufficient to enhance long-term memories.
72 ith a decrease in the influence of others on long-term memories.
73 communication aid transformation into stable long-term memories.
74 hallenge the idea that stable synapses store long-term memories.
75 ns during the retrieval of short-term versus long-term memories.
76 uates the formation of hippocampus-dependent long-term memories.
77 % of the responding clones were recruited to long-term memory 3 months postvaccination, (iii) the mos
78 r LTP generation, which may be implicated in long-term memory acquisition and in its deterioration in
79 gle-strand DNA damage, was also required for long-term memory acquisition in a variety of learning pa
81 areas and mark those synapses for forming a long-term memory after the prion form is established.
82 s) are important for neuronal functions like long-term memory and are well-characterized in mammals b
83 rocess during the formation and retrieval of long-term memory and define general memory-implicated ge
84 priately timed physical exercise can improve long-term memory and highlight the potential of exercise
85 tical role of astrocyte-derived L-lactate in long-term memory and long-term potentiation in vivo.
86 igosaccharide analogs of ST-5 CPS RU induced long-term memory and protective immune responses in rabb
87 mation is buffered when being retrieved from long-term memory and reconcile current theories of memor
88 odic memories are gradually assimilated into long-term memory and this process is strongly influenced
89 lesions may selectively affect orthographic long-term memory and working memory processes, relativel
91 this strategy versus exploring new ones from long-term memory, and (ii) another from polar to lateral
95 sodic memories, it is not known whether such long-term memories are necessary for particular features
99 bout the word's letters and their order from long-term memory as well as the maintenance and processi
100 increase the precision of hippocampal-based long-term memory associations by assessing the salience
101 f antagonists, and coexistence of short- and long-term memory at loci with weak and strong constituti
103 ependent of alpha'/beta' and gamma MBNs, and long-term memory becomes completely dependent on alpha/b
106 ptic remodeling associated with formation of long-term memories, but the mechanisms that regulate thi
107 binding protein Orb2 has been implicated in long-term memory, but how conformational conversion of O
108 to associate different stimuli is vital for long-term memory, but how neural ensembles encode associ
111 e further studied their capacity to maintain long-term memory by measuring cellular and serologic res
114 ll generation or in postmitotic cells, while long-term memory can survive multiple rounds of cell div
115 rapidly forgets, or is not yet able to form long-term memories, can exert such a long-lasting and im
119 Canonical models suggest that mechanisms of long-term memory consist of a synapse-specific, protein
120 before fear learning significantly impaired long term memory consolidation, whereas short-term memor
124 s and short-term memory, but exhibited broad long-term memory deficits as revealed by several behavio
125 working memory processes, with orthographic long-term memory deficits centred in either the left pos
127 erexpression in aged animals ameliorated the long-term memory deficits observed in control animals.
128 place cell representations could explain the long-term memory deficits observed in previous behaviora
129 ce showed reduced prepulse inhibition (PPI), long-term memory deficits, and spontaneous locomotor hyp
132 o internal representations, whether semantic long-term memory (e.g., letters, digits, words), sensory
135 ss the real-time contribution of the mPFC to long-term memory encoding, we performed temporally preci
138 re of well-organized relevant information in long-term memory, enhanced attitude extremity and access
139 g-term memory formation and are required for long-term memory enhancement by a class of broad-acting
141 n the unfolding therapeutic process generate long-term memories for reconsolidated emotional material
142 In addition, we found selectively enhanced long-term memory for contextual but not cued fear memory
143 PHC), and hippocampus differentially support long-term memory for item information, spatial context,
150 tate from astrocytes, which is necessary for long-term memory formation and for underlying molecular
151 lular and molecular mechanisms that regulate long-term memory formation and storage may provide alter
152 n Arc (also known as Arg3.1) is required for long-term memory formation and synaptic plasticity.
154 ously uncharacterized in the brain, supports long-term memory formation in the amygdala and suggests
155 le their sucrose intake at an early stage of long-term memory formation initiated the investigation o
157 as an essential and well established role in long-term memory formation throughout a diverse set of o
158 encoding-induced hippocampal activation and long-term memory formation, and have important implicati
159 ement-binding protein, a crucial mediator in long-term memory formation, correlated anatomically and
160 To evaluate what determines the efficacy of long-term memory formation, we developed an extensive se
171 ociation system segregation is predictive of long-term memory function, independent of an individual'
174 Although persistence is a key feature of long-term memory, how this occurs, despite the rapid tur
175 amine-depleted rats, hence amnesic, restored long-term memory; however, the time frame of memory resc
181 d these defects are associated with impaired long-term memory in both the exposed fathers and their o
184 Decreasing the levels of caspase-2 restored long-term memory in mice that had existing deficits.
185 n of astrocytic Gs-coupled signaling reduced long-term memory in mice without affecting learning.
190 stimulation enhanced the short-, but not the long-term memory in the novel-object recognition task.
193 econd, PKMzeta is essential for late-LTP and long-term memory in wild-type mice, and PKMzeta-null mic
194 e in hippocampus blocks late-LTP and spatial long-term memory in wild-type mice, but not in PKMzeta-n
195 genetic removal of these receptors enhanced long-term memory in young and aging mice and increased t
196 Sleep is important for the formation of long-term memory, in humans as well as in other animals,
197 of several types of working, short-term and long-term memories, including fear conditioning, object
198 s2(-/-) mice displayed impaired formation of long-term memory, increased risk taking and stimulus see
201 been assumed that information retrieved from long-term memory is represented in working memory, we la
205 vivo, and identify the microRNA miR-21 as a long-term memory keeper of the fibrogenic program in MSC
207 studies suggested that NK cells may display long-term, memory-like responses to murine cytomegalovir
208 changes using aversive olfactory associative long-term memory (LTAM) and identified three major gene
209 ds have existing representations in subjects long-term memories (LTM) and that face stimuli used in p
211 lly involved in middle-term memory (MTM) and long-term memory (LTM) and that their expression is requ
212 uced performance in Morris Water Maze (MWM), long-term memory (LTM) contextual fear testing, and rota
213 le that activates mTORC2 (A-443654) reverses long-term memory (LTM) deficits in both aged mice and fl
214 the fact that PDE11A is required for social long-term memory (LTM) formation during adolescence and
218 brain are required for middle-term (MTM) and long-term memory (LTM) in the dorsal paired medial (DPM)
219 l synaptic plasticity.SIGNIFICANCE STATEMENT Long-term memory (LTM) induced by repeated trials spaced
222 f spaced training trials in the formation of long-term memory (LTM) is widely appreciated, surprising
224 l cortex in the temporoparietal junction and long-term memory (LTM) retrieval processes are localized
226 transform a subthreshold learning event into long-term memory (LTM), and hSyn-HM4D completely impaire
230 ngs provide initial evidence to suggest that long-term memory may be enhanced by more carefully atten
231 in the storage of target representations in long-term memory may underlie rapid changes in the effic
232 evidence has indicated that, in addition to long-term memory, MTL subregions may similarly contribut
235 ced memory reactivation during sleep renders long-term memories of negative experiences more negative
237 mission of food preference (STFP), mice form long-term memory of food odors presented by a social par
238 unclear whether this inheritance can direct long-term memory of individual gene expression states (c
241 within hours after Ag exposure, resulting in long-term memory or abortive effector responses, correla
246 e encoding protein KIBRA, is associated with long-term memory performance, we hypothesized that commo
249 s PKMzeta is essential for wild-type LTP and long-term memory, persistent PKCiota/lambda activation c
251 mory CD8 T cells, including acquisition of a long-term memory phenotype (i.e., CD27(hi), CD62L(hi), K
255 reflex, we compared acquisition learning and long-term memory recall of uninfected (control) honey be
256 eep transforms episodic representations into long-term memories, redistributes them toward extrahippo
258 ntenance results from the interactions among long-term memory representations and basic processes, in
259 ive control task if control processes engage long-term memory representations that are supported by t
267 We propose a model whereby persistence of long-term memory results from the assembly of CPEB3 into
270 e basic brain processes: visual recognition, long-term memory, short-term memory, action selection, a
273 trong neurobiological evidence suggests that long-term memory storage involves formation of new synap
277 P degradation also prevents the formation of long-term memory, suggesting rapid SCOP breakdown is nec
278 e to NMDA receptor activation is crucial for long term memory, synaptic tagging, and epigenetic signa
279 ed to reflect the use of a separate episodic long-term memory system, rather than working memory.
281 paired performance on an associative spatial long-term memory task, whereas right CA3 silencing had n
282 pite the fact that these mice are amnesic in long-term memory tests when natural recall cues are used
283 id single-colored stimuli and were tested in long-term-memory tests 1 or 3 days later, they only init
286 ynaptic substrate for a socially conditioned long-term memory that operates at the level of the initi
287 tems in prokaryotes and animals give rise to long-term memory through modification of specific genomi
288 promote the storage of new information into long-term memory through the activation of the SN/VTA-Hi
289 apid working memory during ongoing tasks and long-term memory to guide future action, respectively).
292 primates is especially complex and requires long-term memory, value comparison, strategic planning,
294 found that the retrieval of information from long-term memory was limited to just a few simple object
295 h-frequency stimulation, both the short- and long-term memories were robustly improved in the novel-o
296 However, a striking asymmetry emerged in long-term memory, wherein only left CA3 silencing impair
299 the capability to integrate information from long-term memory with information from novel spike input
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