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1 subjects with chronic HIV infection, and one long-term nonprogressor.
2 e antiretroviral therapy-treated patients or long term nonprogressors.
3 cted compared with both healthy controls and long-term nonprogressors.
4 ntrols, chronic HIV-1-infected patients, and long-term nonprogressors.
5 ents with acute HIV-1 infection but not from long-term nonprogressors.
6 essors, but much less frequently in aviremic long-term nonprogressors.
7 ly greater than previously observed in adult long-term nonprogressors.
8 odeficiency virus infection as well as HIV-1 long-term nonprogressors.
9 t observed in the minority of HIV-1-infected long-term nonprogressors.
10 ariants arise periodically in HIV-1-infected long-term nonprogressors.
11 human immunodeficiency virus (HIV)-infected long-term nonprogressors.
12 human immunodeficiency virus type 1-infected long-term nonprogressors.
17 e in most patients but have been reported in long-term nonprogressors and in patients treated with hi
19 ls of DC-SIGN compared with cocaine-nonusing long-term nonprogressors and normal progressors, respect
23 es suggested that these patients, as well as long-term nonprogressors, are infected with defective HI
25 irus infection of both progressor (BALB) and long term nonprogressor (C57BL) mouse strains is charact
26 induced CD4 down-modulation, and a subset of long-term nonprogressors carry viruses defective in this
27 rozygotes is increased among HIV-1- infected long-term nonprogressors compared with progressors; howe
29 77 miRNAs in CD4(+) T cells from HIV-1 elite long-term nonprogressors (eLTNPs), naive patients, and m
30 the Vpr mutant identified in HIV-1-infected long-term nonprogressors failed to promote TERT destabil
32 ting CD4(+) T cells in coinfected HLA-DR7(+) long-term nonprogressor HIV subjects with undetectable H
33 right) T cells are elevated in blood of HIV+ long-term nonprogressors in comparison to HIV- donors.
37 iral loads in intestinal mucosal biopsies of long-term nonprogressor (LTNP) patients as compared to c
38 HIV replication in elite controller (EC) and long-term nonprogressor (LTNP) patients has been associa
41 ological clones from two progressors and two long-term nonprogressors (LTNP) in fetal thymic organ cu
42 ble immune-mediated control over HIV, termed long-term nonprogressors (LTNP) or elite controllers, an
43 VZV), and tetanus toxoid in normal controls, long-term nonprogressors (LTNP), and HIV-infected patien
44 (designated Rx <50) and untreated patients (long-term nonprogressors [LTNP]) matched for very low HI
45 fected subjects; the latter group included 8 long-term nonprogressors (LTNPs) and 17 progressors.
46 d in coculture by autologous CD8+ T cells in long-term nonprogressors (LTNPs) and in patients whose v
49 HIV replication in rare individuals, termed long-term nonprogressors (LTNPs) or elite controllers.
50 virus (HIV type 1-infected, untreated white long-term nonprogressors (LTNPs) with a cohort of 605 HI
51 rol of human immunodeficiency virus (HIV) in long-term nonprogressors (LTNPs) with protective human l
52 +/- 74 in 11 healthy donors, 360 +/- 69 in 3 long-term nonprogressors (LTNPs), 186 +/- 52 in 9 newly
53 four patients including typical progressors, long-term nonprogressors (LTNPs), and vertically HIV-inf
54 human immunodeficiency virus (HIV)-infected long-term nonprogressors (LTNPs), it is also present at
55 has not yet been evaluated in HIV-1-infected long-term nonprogressors (LTNPs), who maintain high CD4(
57 ects with a lack of disease progression (ie, long-term nonprogressors [LTNPs]) through 7 years of fol
60 ntiretroviral therapy (n = 21), HIV-infected long-term nonprogressors (n = 10), and HIV-seronegative
61 naive (n = 63), elite controllers (n = 19), long-term nonprogressors (n = 7), and HIV-infected patie
64 gous CD8+ cells from PBMCs from HIV-positive long-term nonprogressors; partial blocking of infection
66 veral cohorts of HIV-1-infected individuals: long-term nonprogressors, progressors to disease, acute
67 disease progression and treatment, including long-term nonprogressors, progressors, and chronically i
69 However, in a cohort of 33 HIV-1-infected long-term nonprogressors, those who were heterozygous fo
70 r AIDS Cohort Study, enriched with rapid and long-term nonprogressors to increase statistical power.
71 Unexpectedly, biologic clones from an adult long-term nonprogressor were noncytopathic in spite of s
73 ins that were secreted when CD8 T cells from long-term nonprogressors with HIV-1 infection were stimu
74 ts were studied including a unique cohort of long-term nonprogressors with low levels of plasma viral
75 IV-specific CD8+ T cells were not limited to long-term nonprogressors with restriction of plasma viru
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