ライフサイエンスコーパス: long-term potentiation

1 itatory synaptic currents, modestly enhanced long-term potentiation).

2 reduction of persistent spines, and impaired long-term potentiation.

3 ted inhibition and excitation in hippocampal long-term potentiation.

4 Abeta) oligomers on synapses and hippocampal long-term potentiation.

5 BDNF receptor tyrosine kinase TrkB to elicit long-term potentiation.

6 ned the impact of postnatal loss of Mef2c on long-term potentiation.

7 ndrites and impaired theta burst stimulation long-term potentiation.

8 fter TBI and rescues deficits in hippocampal long-term potentiation.

9 cacy of theta pulse stimulation in reversing long-term potentiation.

10 d synaptic AMPA receptor levels and impaired long-term potentiation.

11 ated genes involved in calcium signaling and long-term potentiation.

12 the ability to subsequently induce synaptic long-term potentiation.

13 r, and reducing induction and maintenance of long-term potentiation.

14 electrophysiologic deficits such as reduced long-term potentiation.

15 receptor (AMPAR) plasticity - namely, anoxic long-term potentiation (a-LTP), and a delayed increase i

16 sonicated mature fibrils inhibit hippocampal long term potentiation, a form of synaptic plasticity im

17 of Crmp2 in the hippocampus leads to reduced long-term potentiation, abnormal NMDA receptor compositi

18 y noradrenaline offers a novel mechanism for long-term potentiation ABSTRACT: Noradrenaline (NA) is a

19 enriched environment but also caused loss of long-term potentiation after theta-burst stimulation.

20 rious hippocampal subfields exhibit impaired long-term potentiation, an electrophysiological correlat

21 dian misalignment causes reduced hippocampal long term potentiation and total CREB expression.

22 eptor (AMPAR) plasticity - an anoxic form of long-term potentiation and a delayed increase in Ca(2+)

23 ion of PDE4A5 in hippocampal neurons impairs long-term potentiation and attenuates the formation of h

24 associated with synaptic function, including long-term potentiation and calcium signaling with higher

25 single gene mutation, demonstrate increased long-term potentiation and decreased long-term depressio

26 tribution, impaired induction of mossy fiber long-term potentiation and deficits in hippocampus-depen

27 Spike timing-dependent long-term potentiation and depression (st-LTP and st-LTD

28 ways responsible for synaptic vesicle cycle, long-term potentiation and depression, and neurotrophin

29 ic strength in the hippocampus, specifically long-term potentiation and depression, depend on new pro

30 ic plasticity genes that are both induced by long-term potentiation and downregulated in the aged bra

31 correlate with IQ and are thought to promote long-term potentiation and enhance memory consolidation.

32 , forniceal DBS restores in vivo hippocampal long-term potentiation and hippocampal neurogenesis.

33 impairs the maintenance of both hippocampal long-term potentiation and hippocampus-dependent spatial

34 ent with synaptic changes including enhanced long-term potentiation and impaired depotentiation ex vi

35 synthase kinase 3beta activity, compromised long-term potentiation and impaired fear-conditioned mem

36 urogranin overexpression in the PFC enhanced long-term potentiation and increased the rates of extinc

37 Consequently, we find that long-term potentiation and long-term depression are inde

38 Dopamine modulated both long-term potentiation and long-term depression in the t

39 spine structural plasticity during chemical long-term potentiation and long-term depression.

40 brain prevents the stabilization of synaptic long-term potentiation and markedly impairs long-term fe

41 that encodes the KIBRA protein critical for long-term potentiation and memory consolidation has prev

42 s, pre- and postsynaptic structural defects, long-term potentiation and miniature postsynaptic curren

43 ed genes were enriched in calcium signaling, long-term potentiation and neuroactive ligand-receptor i

44 increased expression of synaptic markers of long-term potentiation and plasticity, including synapto

45 synaptic currents, prevents the induction of long-term potentiation and prevents spine expansion.

46 ormation, ameliorates synapse loss, elevates long-term potentiation and protects memory.

47 restore intrinsic impairment of hippocampal long-term potentiation and spatial learning-memory defec

48 bitor, to adult mBACtgDyrk1a mice normalized long-term potentiation and spine anomalies but not eCB-L

49 nase C isoform, is important for maintaining long-term potentiation and storing memory.

50 nd decreased expression of genes involved in long-term potentiation and synaptic transmission, cancer

51 methyl-D-aspartate (NMDA) receptor-dependent long-term potentiation and transient optogenetic activat

52 ng plasticity in the indirect pathway toward long-term potentiation (and possibly also through more c

53 of T-type currents prevents the induction of long-term potentiation, and also interferes with long-la

54 ctivation, synaptic deficits, suppression of long-term potentiation, and cognitive impairment as comp

55 sulting in higher spine densities, increased long-term potentiation, and enhanced short-term contextu

56 c and axo-spinous synapses, was incapable of long-term potentiation, and less effectively patterned S

57 actor of activated T cells (NFAT) signaling, long-term potentiation, and responsiveness to adrenergic

58 of brain slices with TIMP2 antibody prevents long-term potentiation, arguing for previously unknown r

59 tent nociceptive activity-dependent synaptic long-term potentiation as well as activity-dependent rem

60 KO mice, and they also exhibited attenuated long-term potentiation as well as deficits in spatial na

61 unctionally, only patients' NMDAR-Ab prevent long-term potentiation at glutamatergic synapses, while

62 ynaptic transmission and decreased levels of long-term potentiation at hippocampal Schaffer collatera

63 discover a presynaptically expressed form of long-term potentiation at mossy cell outputs, shedding l

64 We show that optogenetic induction of long-term potentiation at perforant path synapses of den

65 erm depression and blunted depotentiation of long-term potentiation at the Schaffer collateral/cornu

66 rtance of OXTR signaling in the induction of long-term potentiation at the synapses between the entor

67 improvements in synaptic ultrastructure and long-term potentiation, but not a reduction of the Abeta

68 mice displayed a defect in the induction of long-term potentiation, but not long-term depression, at

69 Following the induction of long-term potentiation by high-frequency electrical stim

70 and concomitantly rescues their hippocampal long-term potentiation deficit.

71 chanism controlling synaptic strength during long-term potentiation/depression and homeostatic scalin

72 erm plasticity with heterogeneity, including long-term potentiation/depression and spike-timing-depen

73 biological synapses: unilateral connection, long-term potentiation/depression, a spike-timing-depend

74 nd large eCB transients produce eCB-mediated long-term potentiation (eCB-tLTP).

75 inistered stressed rats showed impairment in long-term potentiation, enhancement in long-term depress

76 ate that Dkk1 triggers synapse loss, impairs long-term potentiation, enhances long-term depression, a

77 acologic blockade of D2Rs led to the loss of long-term potentiation expression, the specific genetic

78 mes: fluorescence analysis of single-synapse long-term potentiation (FASS-LTP).

79 f synaptic activity as inferred by recording long-term potentiation generated at both connections.

80 citatory synaptic transmission, and enhances long-term potentiation in adult wild-type mice.

81 ings of excitatory postsynaptic currents and long-term potentiation in brain slices and assessing the

82 tatory postsynaptic currents and hippocampal long-term potentiation in brain slices.

83 ever, chronic NB-360 treatment did not alter long-term potentiation in CA1 neurons of Sez6(-/-) mice.

84 state that may contribute to an increase in long-term potentiation in CA3 with subsequent stimulatio

85 -dependent enhancement of NMDAR currents and long-term potentiation in D1R-MSN.

86 Processes that reverse long-term potentiation in field CA1 are not operative in

87 U0409551 enhances NMDAR function and rescues long-term potentiation in hippocampal slices obtained fr

88 ndicates that these connections may underpin long-term potentiation in M1, our findings may lead to n

89 near-complete loss of spike-timing-dependent long-term potentiation in medium spiny neurons (MSNs).

90 Mice exposed to cNIC exhibited long-term potentiation in response to high-frequency sti

91 vels and selectively blocks the induction of long-term potentiation in striatal cholinergic interneur

92 naptic transmission in the dentate gyrus and long-term potentiation in the CA1 region of the hippocam

93 al neurons and enhances excitatory drive and long-term potentiation in the hippocampus of behaving mi

94 fast excitatory transmission and facilitates long-term potentiation in the hippocampus, two effects l

95 At the physiological level, TBI suppressed long-term potentiation in the hippocampus, which was ful

96 ) in the hippocampus, and similarly enhanced long-term potentiation in the hippocampus.

97 affected neuronal plasticity by facilitating long-term potentiation in the hippocampus.

98 ted mGlu5 receptor-dependent facilitation of long-term potentiation in the IL-PFC.

99 probability of LTD without affecting that of long-term potentiation in the insular cortex.

100 ts in object recognition memory and impaired long-term potentiation in the prefrontal cortex.

101 f-administration, similar to cocaine-induced long-term potentiation in the VTA.

102 te-derived L-lactate in long-term memory and long-term potentiation in vivo.

103 calmodulin-binding protein that facilitates long-term potentiation) in the PFC.

104 dition, NMDA receptor-mediated responses and long-term potentiation induced by theta-burst stimulatio

105 e Ras-ERK pathway, is not only essential for long-term potentiation induced with a high frequency sti

106 ity, leads to a complete block of subsequent long-term potentiation induction and a facilitation of l

107 NFH and NFL) markedly depresses hippocampal long-term potentiation induction without detectably alte

108 otine and NRG3 facilitated the conversion of long-term potentiation into long-term depression at cort

109 Long-term potentiation is abolished and long-term depres

110 knocked out (MKP-2(-/-) mice), we show that long-term potentiation is impaired in MKP-2(-/-) mice co

111 bound depolarization, and non-mGluR-mediated long-term potentiation is unaltered.

112 lteration of synaptic strength necessary for long-term potentiation, learning, and memory.

113 Intracortical facilitation and long-term potentiation-like plasticity were impaired in

114 spring) in early postnatal life show reduced long term potentiation (LTP) and impaired hippocampal-de

115 tly reduced and the resulting suppression of long term potentiation (LTP) by Abeta oligomers was prev

116 ic GluN2A-containing NMDARs and induction of long term potentiation (LTP) lead to the translocation o

117 Moreover, long term potentiation (LTP) of CA1 synaptic transmissio

118 d Abeta1-42 oligomers-induced impairments in long term potentiation (LTP).

119 psychiatric disorders-resulted in a lack of long-term potentiation (LTP) (persistent strengthening o

120 mbers (APP, APLP1, APLP2), develop defective long-term potentiation (LTP) and aged mice display spati

121 lic adenosine monophosphate (cAMP) regulates long-term potentiation (LTP) and ameliorates memory in h

122 memantine-induced enhancement of hippocampal long-term potentiation (LTP) and CaMKII activity was tot

123 Synaptic plasticity, such as long-term potentiation (LTP) and depotentiation, was inv

124 Long-term potentiation (LTP) and depression (LTD) at glu

125 Both long-term potentiation (LTP) and depression (LTD) of exc

126 Both long-term potentiation (LTP) and depression (LTD) were s

127 ive GSK3beta caused a significant deficit in long-term potentiation (LTP) and facilitated long-term d

128 e noise, FM tone) recruits distinct forms of long-term potentiation (LTP) and inserts calcium permeab

129 activation is required for the induction of long-term potentiation (LTP) and is generally neuroprote

130 had no effect on increased AMPAR levels with long-term potentiation (LTP) and little effect on decrea

131 wn to support such bidirectional changes are long-term potentiation (LTP) and long-term depression (L

132 lutamate receptors (NMDAR, AMPAR), including long-term potentiation (LTP) and long-term depression (L

133 l synaptic plasticity has largely focused on long-term potentiation (LTP) and long-term depression (L

134 t studies compare the absolute magnitudes of long-term potentiation (LTP) and long-term depression (L

135 Long-term potentiation (LTP) and long-term depression (L

136 tum and assessed neuroplasticity by inducing long-term potentiation (LTP) and long-term depression (L

137 term changes in synaptic strength, including long-term potentiation (LTP) and long-term depression (L

138 ral plasticity might involve mechanisms like long-term potentiation (LTP) and long-term depression (L

139 Competing mechanisms of long-term potentiation (LTP) and long-term depression (L

140 At glutamatergic synapses, both long-term potentiation (LTP) and long-term depression (L

141 essary and sufficient for the maintenance of long-term potentiation (LTP) and long-term memory (LTM).

142 but not monomers, produces an impairment of long-term potentiation (LTP) and memory, independent of

143 nt concentrations of Abeta are necessary for long-term potentiation (LTP) and memory.

144 ant synaptic protein CaMKII is necessary for long-term potentiation (LTP) and memory.

145 de in the healthy brain, enhance hippocampal long-term potentiation (LTP) and memory.

146 are associated with altered corticostriatal long-term potentiation (LTP) and specific reduction of d

147 SIGNIFICANCE STATEMENT: Various types of long-term potentiation (LTP) are correlated with distinc

148 odic memory, and synaptic changes induced by long-term potentiation (LTP) are thought to underlie mem

149 equired for hippocampal-dependent memory and long-term potentiation (LTP) at CA1 Schaffer collateral

150 Deficits in long-term potentiation (LTP) at central excitatory synap

151 vented Abeta oligomers-induced inhibition of long-term potentiation (LTP) at concentrations that did

152 ization of postsynaptic substructures during long-term potentiation (LTP) at individual dendritic spi

153 al injury relaxes the timing rules governing long-term potentiation (LTP) at mouse primary afferent s

154 p.) impaired short-term plasticity (STP) and long-term potentiation (LTP) at perforant path-DG synaps

155 synaptic and postsynaptic activity can evoke long-term potentiation (LTP) at sensory synapses onto ad

156 deed, genetic deletion of Calstabin2 reduced long-term potentiation (LTP) at the hippocampal CA3-CA1

157 Several forms of long-term potentiation (LTP) at the Schaffer collateral

158 Long-term potentiation (LTP) at the Schaffer collateral-

159 Remarkably, long-term potentiation (LTP) can override the masking ef

160 , so we asked whether basal transmission and long-term potentiation (LTP) differed in slices of adult

161 creased inducibility of associative synaptic long-term potentiation (LTP) due to saturation after sle

162 t calpain-1 is required for the induction of long-term potentiation (LTP) elicited by theta-burst sti

163 The synapse specificity of long-term potentiation (LTP) ensures that no interferenc

164 onstrate that TBI inhibits the expression of long-term potentiation (LTP) evoked by high-frequency st

165 Long-term potentiation (LTP) had a higher threshold in L

166 scovery by Bliss and Lomo, the phenomenon of long-term potentiation (LTP) has been extensively studie

167 Since the discovery of long-term potentiation (LTP) in 1973, thousands of paper

168 h-frequency stimulation, which evoked robust long-term potentiation (LTP) in brain slices from LV con

169 compound 5b completely inhibits induction of long-term potentiation (LTP) in CA3-CA1 but not in MF-CA

170 nsistent with previous studies, we show that long-term potentiation (LTP) in cortico-striatal circuit

171 nRNP K regulates dendritic spine density and long-term potentiation (LTP) in cultured hippocampal neu

172 ors (betaARs) enhances both the induction of long-term potentiation (LTP) in hippocampal CA1 pyramida

173 ABSTRACT: NMDA receptor independent long-term potentiation (LTP) in hippocampal stratum orie

174 reduction of fear and enhanced induction of long-term potentiation (LTP) in PW pups, in contrast to

175 imulation of corticostriatal fibres produces long-term potentiation (LTP) in striatal projection neur

176 Here we characterized two forms of long-term potentiation (LTP) in the anterior cingulate c

177 hannels completely suppressed development of long-term potentiation (LTP) in the CA1-subiculum, but n

178 soform, completely suppressed development of long-term potentiation (LTP) in the CA1-subiculum, but n

179 a low concentration of TTX) are required for long-term potentiation (LTP) in the distal apical dendri

180 g-dependent long-term depression switches to long-term potentiation (LTP) in the former, timing-depen

181 Electrophysiology experiments showed that long-term potentiation (LTP) in the hippocampus, which i

182 Although long-term potentiation (LTP) in the lateral amygdala (LA

183 Long-term potentiation (LTP) in the rat hippocampus is t

184 me displayed a robust twofold enhancement in long-term potentiation (LTP) induction accompanied by a

185 ut did not fully abolish, the probability of long-term potentiation (LTP) induction by either single

186 Long-term potentiation (LTP) is a form of synaptic plast

187 Long-term potentiation (LTP) is a persistent increase in

188 Long-term potentiation (LTP) is a rapid and persistent i

189 Long-term potentiation (LTP) is an activity-dependent an

190 The molecular mechanism underlying long-term potentiation (LTP) is critical for understandi

191 learning rules at retinogeniculate synapses, long-term potentiation (LTP) is intact but long-term dep

192 rticostriatal long-term depression (LTD) and long-term potentiation (LTP) is proposed to be critical

193 Long-term potentiation (LTP) is widely perceived as a me

194 ocampus, a form of NMDA receptor-independent long-term potentiation (LTP) occurs at excitatory synaps

195 itutively repress the expression of synaptic long-term potentiation (LTP) of C-fiber-evoked potential

196 imulation (LFS) to the sciatic nerve induced long-term potentiation (LTP) of C-fiber-evoked potential

197 y in rat hippocampal slices, we explored how long-term potentiation (LTP) of different excitatory syn

198 s of drug addiction indicate that persistent long-term potentiation (LTP) of excitatory synaptic tran

199 Long-term potentiation (LTP) of excitatory synaptic tran

200 We found that long-term potentiation (LTP) of mossy fiber input invoke

201 nsive analysis of APA following induction of long-term potentiation (LTP) of mouse hippocampal CA3-CA

202 Long-term potentiation (LTP) of NMDA receptor (NMDAR)-me

203 ess in rats causes persistent enhancement of long-term potentiation (LTP) of NMDA receptor-mediated g

204 Physiologically, CRS reduced long-term potentiation (LTP) of Schaffer collateral/comm

205 Mossy fiber input is known to exhibit a long-term potentiation (LTP) of synaptic efficacy throug

206 Long-term potentiation (LTP) of synaptic strength betwee

207 d odor exposure, induces glomerulus-specific long-term potentiation (LTP) of synaptic strength select

208 re, we report that CCNY negatively regulates long-term potentiation (LTP) of synaptic strength throug

209 and cognition are thought to require normal long-term potentiation (LTP) of synaptic strength, which

210 Surprisingly, we find that long-term potentiation (LTP) of synaptic transmission at

211 occurs locally at individual synapses during long-term potentiation (LTP) or long-term depression (LT

212 of the neuronal synapse, phenomena known as long-term potentiation (LTP) or long-term depression (LT

213 Cortical plasticity mechanisms of long-term potentiation (LTP) or of long-term depression

214 array and characterized by downregulation of long-term potentiation (LTP) related transcripts and upr

215 atum has been characterized, their effect on long-term potentiation (LTP) remains unknown.

216 Long-lasting forms of long-term potentiation (LTP) represent one of the major

217 DA (N-methyl-d-aspartate) receptor-dependent long-term potentiation (LTP) shapes neural circuits and

218 ly more mushroom spines and higher-magnitude long-term potentiation (LTP) than SR synapses.

219 We show changes in DG long-term potentiation (LTP) that parallel behavioral re

220 esynaptic, NMDA-receptor-independent form of long-term potentiation (LTP) that requires postsynaptic

221 receptors (AMPARs), and promotes hippocampal long-term potentiation (LTP) through AMPAR trafficking.

222 tatic plasticity and NMDA receptor-dependent long-term potentiation (LTP), a form of Hebbian plastici

223 ual fear memory and increase the duration of long-term potentiation (LTP), a form of hippocampal syna

224 n is an essential step for the expression of long-term potentiation (LTP), a long-lasting, activity-d

225 yield diverse outcomes: In vivo induction of long-term potentiation (LTP), a model of learning, is re

226 lasting changes in synaptic strength such as long-term potentiation (LTP), a putative cellular mechan

227 Long-term potentiation (LTP), a well characterized form

228 ngth and the induction of CP-AMPAR-dependent long-term potentiation (LTP), an anti-Hebbian form of LT

229 dendritic integration of excitatory inputs, long-term potentiation (LTP), and spatial memory formati

230 put specificity is a fundamental property of long-term potentiation (LTP), but it is not known if lea

231 models demonstrated exaggerated hippocampal long-term potentiation (LTP), consistent with deficits i

232 emory and its electrophysiological surrogate long-term potentiation (LTP), effects that may be mediat

233 and memory that are accompanied by enhanced long-term potentiation (LTP), impaired long-term depress

234 We show that long-term potentiation (LTP), in the CA1 region of hippo

235 plasticity, such as glycine-induced chemical long-term potentiation (LTP), known to evoke synaptic pl

236 Biological synaptic behaviors such as long-term potentiation (LTP), long-term depression (LTD)

237 -aspartate (NMDA) receptor (NMDAR)-dependent long-term potentiation (LTP), remains unclear.

238 One is the discovery of long-term potentiation (LTP), the phenomenon that brief,

239 ts, wash-in of letrozole virtually abolished long-term potentiation (LTP), whereas it did not prevent

240 ments is most likely mediated by fast-acting long-term potentiation (LTP), which relies on the precis

241 ecessary and sufficient for the induction of long-term potentiation (LTP).

242 thesis required to sustain the late phase of long-term potentiation (LTP).

243 memory impairments, we examined hippocampal long-term potentiation (LTP).

244 ally reduces basal synaptic transmission and long-term potentiation (LTP).

245 aptic plasticity loss evident by a decreased long-term potentiation (LTP).

246 ethyl-d-aspartate receptor (NMDAR)-dependent long-term potentiation (LTP).

247 riments examining the effects on hippocampal long-term potentiation (LTP).

248 icantly enriched in proteins associated with long-term potentiation (LTP).

249 lular processes underlying learning, such as long-term potentiation (LTP).

250 aptic transmission and are unable to sustain long-term potentiation (LTP).

251 ted memory deficits and impaired hippocampal long-term potentiation (LTP).

252 , and blocked D1 dopamine receptor dependent long-term potentiation (LTP).

253 , and alleviates Abeta-mediated reduction of long-term potentiation (LTP).

254 eviously shown to have increased hippocampal long-term potentiation (LTP).

255 hiatric disorders, and frequently depends on long-term potentiation (LTP).

256 le of cholesterol in synaptic changes during long-term potentiation (LTP).

257 ction of both long-term depression (LTD) and long-term potentiation (LTP).

258 ngth by facilitating an anti-Hebbian form of long-term potentiation (LTP).

259 nd ablates Reelin stimulation of hippocampal long-term potentiation (LTP).

260 ouse model of Alzheimer's disease (AD), late long-term potentiation (LTP; L-LTP) and its associative

261 epression (LTD) of synaptic transmission but long-term-potentiation (LTP) of synaptic signals in HIL

262 Synaptic plasticity (e.g., long-term potentiation [LTP]) is considered the cellular

263 erosynaptic and is expressed as an increase (long-term potentiation, LTPGABA) or a decrease (long-ter

264 is not the case for hippocampal presynaptic long-term potentiation (LTPpre), which is expressed as a

265 ation of the Group II mGluR, mGluR3, induces long-term potentiation of electrical synapses.

266 Long-term potentiation of excitatory synapses on pyramid

267 hippocampal stratum oriens exhibit a form of long-term potentiation of excitatory transmission that i

268 ression in rat hippocampal neurons precludes long-term potentiation of glutamatergic synapses specifi

269 showed reduced initiation and maintenance of long-term potentiation of glutamatergic synapses.

270 eatment, lOFC neurons displayed a persistent long-term potentiation of glutamatergic synaptic transmi

271 dent stimuli of the lateral amygdala induces long-term potentiation of lateral-basal amygdala excitat

272 the absence of impaired sensory processes or long-term potentiation of the Schaffer collateral pathwa

273 of the neurons, basal synaptic transmission, long-term potentiation or expression of LTD.

274 of Mef2c did not impact learning and memory, long-term potentiation, or social and repetitive behavio

275 es revealed processes including NMDA-related long-term potentiation, PKA, immune response signaling,

276 ffects combined with PE-induced anti-Hebbian long-term potentiation reported in a previous study coul

277 epression (required for Dc-ODP), and CREB in long-term potentiation (required for Pc-ODP).SIGNIFICANC

278 avage, ameliorates synapse loss and augments long-term potentiation, resulting in protection of memor

279 Testing spatial memory and hippocampal long-term potentiation revealed an impairment in APP/PS1

280 in single dendritic spines during structural long-term potentiation (sLTP) in hippocampal CA1 pyramid

281 escribe a three-molecule model of structural long-term potentiation (sLTP) of murine dendritic spines

282 of neuronal plasticity, including structural long-term potentiation (sLTP), which is a correlate of a

283 in hippocampal basal synaptic transmission, long-term potentiation, spatial learning, and memory in

284 undergone theta-burst stimulation to produce long-term potentiation (TBS-LTP) and compared them to co

285 imilar to the memory deficits, theta-induced long-term potentiation (theta-LTP) in the nucleus accumb

286 n naive male mice, to spike-timing-dependent long-term potentiation (tLTP) in DID mice, an effect tha

287 ltiple cellular pathways may contribute to a long term potentiation-type response.

288 ber synapses in area CA3 and found increased long-term potentiation upon depletion of IFT20 or disrup

289 Hippocampal long-term potentiation was decreased in both chronic BAC

290 tly different between the two MA groups, but long-term potentiation was greater in ampakine-treated r

291 a was applied on hippocampal slices ex vivo, long-term potentiation was lowered.

292 More importantly, hippocampal CA1 long-term potentiation was markedly impaired in parabiot

293 d with a potent BACE1 inhibitor, hippocampal long-term potentiation was reduced.

294 platform stress paradigm, and extinction and long-term potentiation were examined.

295 urrents in pyramidal neurons and hippocampal long-term potentiation were reduced in animals treated w

296 ence and magnitude of spike timing-dependent long-term potentiation were significantly higher at musc

297 n postsynaptic densities and NMDAR-dependent long-term potentiation, which is critical for learning a

298 fusion lead to CaMKII activation and calcium long-term potentiation, which support cardiomyocyte cont

299 water maze, and a significant disruption of long-term potentiation without alteration of long-term d

300 e tolerance, OIH and pronociceptive synaptic long-term potentiation without altering antinociception.