ライフサイエンスコーパス: long-term synaptic depression

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1 ry synaptic plasticity, and have accentuated long-term synaptic depression.

2 sential for certain types of both short- and long-term synaptic depression.

3 , which is an important mechanism underlying long-term synaptic depression.

4 tein O-GlcNAcylation induces a novel form of long-term synaptic depression at hippocampal CA3-CA1 syn

5 resveratrol blocked endocannabinoid-mediated long-term synaptic depression in VTA dopamine neurons.

6 apses onto inhibitory interneurons undergo a long-term synaptic depression (interneuron LTD; iLTD).

7 ) (FMRFa) can induce transcription-dependent long-term synaptic depression (LTD) in Aplysia sensorimo

8 xamine long-term synaptic potentiation (LTP)/long-term synaptic depression (LTD) in brain slices of r

9 artate (NMDA) to hippocampal slices produces long-term synaptic depression (LTD) in CA1 that is (1) s

10 onic acid-type glutamate receptors caused by long-term synaptic depression (LTD) in cerebellar Purkin

11 Here, we demonstrate that long-term synaptic depression (LTD) in hippocampal neuro

12 noid (eCB) signaling mediates short-term and long-term synaptic depression (LTD) in many brain areas.

13 Cerebellar long-term synaptic depression (LTD) is a model system of

14 Cerebellar long-term synaptic depression (LTD) is a model system of

15 methyl-D-aspartate (NMDA) receptor-dependent long-term synaptic depression (LTD) is associated with a

16 to characterize the dependence of cerebellar long-term synaptic depression (LTD) on postsynaptic Ca(2

17 campal long-term potentiation, their role in long-term synaptic depression (LTD) remains unclear.

18 estigated the site of expression of striatal long-term synaptic depression (LTD) using analysis of Sr

19 ) stimulates dendritic protein synthesis and long-term synaptic depression (LTD), but it remains uncl

20 M1 mAChRs induce long-term synaptic depression (LTD), but little is known

21 s, which exhibit exaggerated mGlu1/5-induced long-term synaptic depression (LTD).

22 aptic and postsynaptic spikes, we can induce long-term synaptic depression (LTD).

23 ation is necessary and sufficient to trigger long-term synaptic depression (LTD).

24 o G(i/o) protein, can mediate short-term and long-term synaptic depression (LTD).

25 , long-term synaptic facilitation (LTF), and long-term synaptic depression (LTD).

26 ptors (CB1Rs) are believed to participate in long-term synaptic depression (LTD).

27 on, ArcGFP+ neurons preferentially displayed long-term synaptic depression (mGluR-LTD) and robust inc

28 tor (mGluR)-stimulated protein synthesis and long-term synaptic depression (mGluR-LTD) are altered in

29 lutamate receptors (mGluRs) induce a form of long-term synaptic depression (mGluR-LTD) in area CA1 of

30 binoid receptors (CB1Rs), mediate short- and long-term synaptic depression of neurotransmitter releas

31 etion did not rescue altered mGluR-dependent long-term synaptic depression or translational control o

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