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1 ases in sodium exit from the proximal tubule/loop of Henle.
2 e collecting duct predominantly but also the loop of Henle.
3 rtical collecting ducts, distal tubules, and loop of Henle.
4 by cells of the thick ascending limb of the loop of Henle.
5 op diuretics can curtail their action in the loop of Henle.
6 renal progenitor cells and formation of the loop of Henle.
7 orption in the proximal convoluted tubule or loop of Henle.
8 bsorption in the thick ascending limb of the loop of Henle.
9 in proximal tubules and descending limbs of loops of Henle.
10 expressed by the thick ascending limb of the loops of Henle.
11 essential for the coordinated growth of the loop of Henle, a medullary extension of the nephron that
13 r unchanged or upregulated, and those in the loop of Henle and distal tubule lineages were downregula
15 cal Ang II, to stimulate sodium transport in loop of Henle and the distal nephron, and to induce hype
16 potassium-chloride transporter in the kidney loop of Henle and the KCC2 potassium-chloride transporte
17 inates in the basement membranes of the thin loops of Henle and spreads from there through the inters
19 transport in the thick ascending limb of the loop of Henle; and claudin-4, -7, and -8 as determinants
20 iously reabsorbed in the proximal tubule and loop of Henle; and, second, a stimulus to sodium-cation
21 the convoluted tubules, collecting ducts and loops of Henle as well as within the cytoplasm of tubule
22 ow a distinct distribution pattern along the loop of Henle, but the functional significance of this o
23 ocated in the kidney within the lumen of the loops of Henle, but no intracellular storage sites were
24 concentration was significantly elevated and loop of Henle Cl absorption was reduced in microperfused
25 a4-specific transcripts in proximal tubule, loop of Henle, distal convoluted tubule, and cortical an
26 strate a4 expression in the proximal tubule, loop of Henle, distal tubule, and collecting duct and su
27 e HBD-1 mRNA in the epithelial layers of the loops of Henle, distal tubules, and the collecting ducts
30 the glomerulotubular balance response in the loop of Henle is accompanied by increased Na,K-ATPase ac
32 h is important in sodium reabsorption in the loop of Henle, is maintained or even increased in Foxa1-
34 ctions in proximal stop flow pressure during loop of Henle (LH) perfusion at 40 nl/min with artificia
35 n clinical medicine because they inhibit the loop of Henle Na-K-2Cl cotransporter with much higher af
36 t decrease in THP-, NKCC2- and AQP1-positive loop of Henle nephron segments in mutant DeltaSRM kidney
37 n, and activation of ion transporters in the loop of Henle (NKCC2) and distal nephron (NCC, ENaC, and
38 Cl and water absorption along microperfused loops of Henle of NKCC2A-/- mice were unchanged at norma
40 examined in anaesthetized rats by perfusing loops of Henle of superficial nephrons with solutions co
41 id not change significantly during prolonged loop of Henle perfusion in e-5'NT/CD73(+/+) mice, a comp
42 -flow pressure in response to an increase in loop of Henle perfusion rate from 0 to 30 nl/min was com
44 r in vivo assessment of proximal tubular and loop of Henle sodium handling, to assess sodium exit aft
45 by cells of the thick ascending limb of the loop of HENLE: Subsequent aggregation of these proteins
47 the specific contribution of thick ascending loop of Henle (TALH) -derived HO-1, we generated a trans
48 m and volume delivery to the thick ascending loop of Henle (TALH) and macula densa, providing the err
49 bsorption in the thick ascending limb of the loop of Henle (TALH) in vivo was examined in anaesthetiz
50 of the proximal tubule (S1/S2 segment), the loop of Henle, the intercalated cells of the distal conv
51 previously reabsorbed in proximal tubule and loop of Henle) to the distal nephron in quantities equal
52 ubule and in the thick ascending limb of the loop of Henle, whereas it is transcellular in the distal
53 a and produced kidney cysts primarily in the loops of Henle, whereas inactivation in adult mice did n
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