ライフサイエンスコーパス: loratadine

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1 es) obtained from rats orally dosed with 14C-loratadine.

2 Finally, loratadine (10 microM) failed to inhibit HERG K+ channel

3 ed on several metabolites and derivatives of loratadine (a long-acting and nonsedating tricyclic anti

4 Loratadine, a clinically used histamine H1 receptor anta

5 A series of loratadine analogues were synthesized to gain insight in

6 nd simvastatin (relative risk or RR = 1.69); loratadine and alprazolam (RR = 1.86); loratadine and du

7 .69); loratadine and alprazolam (RR = 1.86); loratadine and duloxetine (RR = 1.94); loratadine and ro

8 of LC/MS/MS methods for the quantitation of loratadine and its metabolite, descarboethoxyloratadine,

9 In the present study, two commercial drugs, Loratadine and Miconazole, were identified as potential

10 .86); loratadine and duloxetine (RR = 1.94); loratadine and ropinirole (RR = 3.21); and promethazine

11 ntified with their p-values less than 1E-06: loratadine and simvastatin (relative risk or RR = 1.69);

12 itro drug metabolism and inhibition potency, loratadine and simvastatin and tegaserod and promethazin

13 were treated orally with the H1R antagonist, loratadine, and/or the H4R antagonist, JNJ7777120.

14 patch-clamp electrophysiology, we found that loratadine blocked Kv1.5 current measured from inside-ou

15 Loratadine did not alter the kinetics of Kv1.5 current a

16 alicylate, caffeine, l-leucine, l-histidine, loratadine, ibuprofen, acetaminophen, acetylsalicylic ac

17 Treatment with montelukast and loratadine inhibited the release of cysteinyl leukotrien

18 It is concluded that loratadine is an effective blocker of Kv1.5 that interac

19 the effects of the nonsedating antihistamine loratadine on a rapidly activating delayed-rectifier K+

20 Treatment with loratadine or JNJ7777120 individually partially suppress

21 ene antagonist montelukast and antihistamine loratadine or two matched placebos were administered for

22 gram amounts of the pharmaceutical compounds loratadine, oxycodone, and atenolol deposited on glass,

23 eceptor antagonists such as diphenhydramine, loratadine, ranitidine, and cimetidine, but has modest a

24 This interaction suggests a potential for loratadine to alter cardiac excitability in vivo.

25 This effect of loratadine was achieved by a reduced number of openings

26 the single-channel level, the main effect of loratadine was to reduce the mean probability of opening

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