ライフサイエンスコーパス: lordosis

1 ation to reinstate feminine sexual behavior (lordosis).

2 eptors, which is expected in turn to promote lordosis.

3 of the female rodent reproductive behavior, lordosis.

4 atosensory, not just hormonal, influences on lordosis.

5 oceptors also facilitates estrogen-dependent lordosis.

6 al hormone treatments sufficient to activate lordosis.

7 avity of wedged lumbar vertebrae, known as a lordosis.

8 and eventually develop bowed legs and lumbar lordosis.

9 cell group crucial for estrogen induction of lordosis.

10 quantified as the copulatory stance known as lordosis.

11 OR and induces the sexual receptive behavior lordosis.

12 hr amide reversed LPNY-induced inhibition of lordosis.

13 for a significant proportion of variance in lordosis.

14 cated in alpha(1)-adrenergic facilitation of lordosis.

15 lieving estrogen inhibition and facilitating lordosis.

16 inea pigs displayed progesterone-facilitated lordosis (85.7% vs. 5.8%, respectively, p<0.05), there w

17 gulation of the female reproductive behavior lordosis, a behavior dependent upon the sequential activ

18 Genetic influences on lordosis, a mammalian social behavior, are amenable for

19 al beta isoform of the ER gene showed normal lordosis and courtship behaviors, extending in some case

20 a,5alpha-THP; 100 or 200ng/side)-facilitated lordosis and its enhancement by D1 (SKF38393; 100ng/side

21 stent with the behavioral incompatibility of lordosis and ultrasound production and suggest that the

22 ion and time course of the lesion effects on lordosis and ultrasound production suggest that the VMN

23 inhibited estrogen plus progesterone-induced lordosis, and the MOR-selective antagonist D-Phe-Cys-Tyr

24 into the medial preoptic nucleus attenuated lordosis, and their effects were blocked with the MOR an

25 ce the quality of the reproductive behavior, lordosis, and to reduce the EMG of lumbar back muscles i

26 (genital sniffing of females by male mice), lordosis (arched-back mating posture in female rats), co

27 wed a transient, but significant, decline in lordosis behavior 5 and 10 min after restraint.

28 aminobutyric acid (GABA) modulate female rat lordosis behavior and appear to interact in their contro

29 Therefore, NO regulation of lordosis behavior and gonadotropin release in female rat

30 in the VMN are involved in the modulation of lordosis behavior and lead to the suggestion that 5-HT2C

31 As expected, 8-OH-DPAT reduced lordosis behavior and muscimol attenuated this inhibitio

32 tion of 5-HT(2A/2C) receptors can facilitate lordosis behavior and that the VMN is one site at which

33 th EB showed significantly greater levels of lordosis behavior compared with OVX females treated with

34 ving 200 ng 8-OH-DPAT exhibited a decline in lordosis behavior following infusion.

35 E- and P-binding neurons important for lordosis behavior have been located within the ventromed

36 nucleus of the hypothalamus (VMN), inhibited lordosis behavior in all hormone-treated conditions.

37 alpha(1)-adrenoceptor antagonist prazosin on lordosis behavior in E(2)- and P-treated female rats.

38 itatory effects of alpha(1)-adrenoceptors on lordosis behavior in female rats, and previous exposure

39 sary for the expression of hormone-dependent lordosis behavior in female rats.

40 e protein kinase G inhibitor KT5823 inhibits lordosis behavior in hormone-treated female rats.

41 l (E(2)) and progesterone (P) facilitate rat lordosis behavior in part by regulating the expression o

42 st action at 5-HT(2A/2C) receptors increased lordosis behavior in rats with low sexual receptivity.

43 s of T3 showed significantly lower levels of lordosis behavior in response to estradiol benzoate (EB)

44 ) receptor antagonist, ketanserin, inhibited lordosis behavior in sexually receptive rats.

45 tor subtypes contribute to the modulation of lordosis behavior in the female rat.

46 onsistent with reports that GABA facilitates lordosis behavior in this hypothalamic structure.

47 e pulses could be reversed and still achieve lordosis behavior induction.

48 dence that the effects of 5-HT on female rat lordosis behavior involve the integrated activity of at

49 ) receptor-mediated inhibition of female rat lordosis behavior is also discussed.

50 onadotropin-releasing hormone release and of lordosis behavior of female rats.

51 tive to hormone-primed, ovariectomized rats, lordosis behavior of proestrous females was less affecte

52 th several reports that muscimol facilitates lordosis behavior of suboptimally hormonally primed fema

53 tion of estradiol, an estrogen necessary for lordosis behavior to occur, heightens this effect.

54 Lordosis behavior was examined following bilateral infus

55 tor interaction in the control of female rat lordosis behavior was examined.

56 le for VMN 5-HT3 receptors in the control of lordosis behavior was examined.

57 Estradiol-induced lordosis behavior was observed in response to a stud mal

58 Lordosis behavior was observed prior to VMN infusion, du

59 both routes of administration, inhibition of lordosis behavior was seen following treatment with the

60 In contrast, DZ facilitates lordosis behavior when infused into the midbrain central

61 8-OH-DPAT inhibited lordosis behavior within 15 min of the infusion and ever

62 olymerization with cytochalasin D attenuated lordosis behavior, indicating the importance of estradio

63 reduced the ability of 8-OH-DPAT to inhibit lordosis behavior, rats were preprimed with 10 microg EB

64 t was given progesterone and then tested for lordosis behavior, the induction of which requires the g

65 lamic ventromedial nucleus (VMN) facilitated lordosis behavior-inducing genomic actions of estrogen.

66 e hypothalamus, these changes are needed for lordosis behavior.

67 he effects of 5-HT2 receptor-active drugs on lordosis behavior.

68 A receptor antagonist, effectively inhibited lordosis behavior.

69 leus of the hypothalamus (VMN) to facilitate lordosis behavior.

70 roduce two groups of rats differing in their lordosis behavior.

71 HT(2A/2C) receptor agonists could facilitate lordosis behavior.

72 ors mediating inhibition and facilitation of lordosis behavior.

73 effective in preventing DOI from increasing lordosis behavior.

74 rts a dual role in the control of female rat lordosis behavior.

75 le for VMN 5-HT3 receptors in the control of lordosis behavior.

76 tron producing the most consistent change in lordosis behavior.

77 cause, alone, 1500 ng mCPBG slightly reduced lordosis behavior.

78 tion to the role of 5HT in the regulation of lordosis behavior.

79 oth sexes were gonadectomized and tested for lordosis behavior.

80 blood levels of progesterone, and eliminated lordosis behavior.

81 wild type females, CX43+/- mice had reduced lordosis behavior.

82 These cells are essential for lordosis behavior.

83 tors in the mediobasal hypothalamus inhibits lordosis behavior.

84 Muscimol alone also reduced lordosis behavior.

85 pylamino)tetralin (8-OH-DPAT), on female rat lordosis behavior.

86 altering the 5-HT(1A) receptor modulation of lordosis behavior.

87 knocked out show that ERalpha is crucial for lordosis behavior.

88 y female sex behavior in laboratory animals, lordosis behavior.

89 the ventromedial hypothalamus (VMN) depress lordosis but increase ultrasonic vocalization in female

90 ine D1 receptor stimulation, facilitation of lordosis by D1 receptor stimulation in estradiol-primed

91 the VMN blocked the subsequent induction of lordosis by D1-like agonists.

92 itation of one component of mating behavior, lordosis, by vaginal-cervical stimulation does not requi

93 upports the proposed hierarchical network of lordosis control.

94 Sexual proceptivity and receptivity (lordosis) during the postpartum estrus were virtually el

95 determine if either drug would attenuate the lordosis-facilitating effects of DOI.

96 sion of a membrane P receptor, 25-Dx, during lordosis facilitation.

97 (10 or 20 mg/kg) significantly reduced both lordosis frequency and quality and reduced (but not sign

98 SA or P&BSA showed significant elevations in lordosis in 5 min.

99 Present studies show that KT5823 attenuates lordosis in a dose-dependent manner when infused bilater

100 of the hypothalamus (VMH), OFQ/N facilitates lordosis in female rats through estrogen and progesteron

101 ance the display of progesterone-facilitated lordosis in juvenile females, increase levels of hypotha

102 enhanced display of progesterone-facilitated lordosis in prepubertal guinea pigs following MPOA lesio

103 sm by which acute E exposure facilitates the lordosis-inducing genomic actions of estrogens.

104 tions in the hypothalamus can potentiate its lordosis-inducing genomic actions on behavior and may be

105 aspects of mating, including facilitation of lordosis, induction of sexual receptivity, abbreviation

106 le both estrogen and progesterone reduce the lordosis-inhibiting effect of 8-OH-DPAT, the mechanisms

107 subchronically treated with fluoxetine, the lordosis-inhibiting effect of an acute injection with fl

108 DOI attenuated the lordosis-inhibiting effect of ketanserin, but ketanserin

109 g quipazine or TFMPP were protected from the lordosis-inhibiting effects of 8-OH-DPAT, alone.

110 gesterone, prepriming with EB attenuated the lordosis-inhibiting effects of systemic treatment with 8

111 The lordosis-inhibiting effects of the 5-HT1A receptor agoni

112 /M)>/=0.5 were used to examine the potential lordosis-inhibiting effects of the 5-HT2A receptor antag

113 The neural circuit for lordosis involves a supraspinal loop, which is controlle

114 mechanism through which estrogen facilitates lordosis is by remodeling synaptic connectivity within t

115 e the EMG of lumbar back muscles involved in lordosis is exerted through a reticulospinal pathway wit

116 Duration and intensity of lordosis is mediated by actions of the progesterone (P)

117 ong evidence that dopaminergic modulation of lordosis is mediated by the novel D5 dopamine receptor.

118 subtype mediating E(2) and P facilitation of lordosis is the alpha(1)-adrenoceptor.

119 luded anterolisthesis, osteophytic spurring, lordosis, kyphosis, and nerve root impingement.

120 about the local VMH microcircuitry governing lordosis nor how estrogen alters synaptic connectivity w

121 enous P (200 microg) significantly increased lordosis of all mice within 10 min of P, but vehicle inf

122 uced plasma 3alpha,5alpha-THP and attenuated lordosis of all mice.

123 RU38486, a PR antagonist, attenuated lordosis of C57 and C57x129, but not PRKO, mice; epostan

124 alpha-ol-20-one (3alpha,5alpha-THP) enhanced lordosis of ovariectomized, sexually experienced C57BL/6

125 oxysteroid oxidoreduced product, facilitates lordosis of rodents in part via agonist-like actions at

126 We have found that progestogen-facilitated lordosis of rodents is enhanced by activation of dopamin

127 vived and developed progressive dwarfism and lordosis of the cervical spine.

128 metabolites' effects on sexual receptivity (lordosis) of mice was examined.

129 , compared with that seen in mice with lower lordosis on initial mating.

130 For sexually naive mice, greater lordosis on initial sexual experience corresponded to gr

131 for the display of progesterone-facilitated lordosis or perfused, and their hypothalamic tissue proc

132 ount and duration and reduced the latency of lordosis, over that seen with vehicle infusion, in PRKO

133 ts sensorimotor integration of the analogous lordosis posture displayed by sexually receptive female

134 gment of the motor pathway that produces the lordosis posture, the hallmark of female rat sexual beha

135 A typical sex difference in lordosis quotient (LQ) was observed and neonatal RU-486

136 VMHVL resulted in a significant reduction in lordosis quotient compared to control (reverse sense) OD

137 midbrain central gray infusions of DZ on the lordosis quotient.

138 Receptivity (lordosis quotients and ratings) and proceptivity (dartin

139 etween intromissions and ejaculation, higher lordosis quotients and ratings, more pacing of their sex

140 us oxytocin infused controls, as measured by lordosis quotients and receptivity scores, at 40, and 90

141 howed reduced intromission-like behavior and lordosis quotients compared with vehicle and scrambled c

142 Lordosis quotients were reduced about 50% in nonandrogen

143 The simplest hypothesis was that lordosis quotients would be significantly reduced as a r

144 al role of hypothalamic opioid expression in lordosis reflex 16-mer oligodeoxynucleotide (ODN) direct

145 ng the neuraxis previously implicated in the lordosis reflex are indeed serially connected.

146 The lordosis reflex is a hormone-dependent behavior displaye

147 mount (L/M) ratio and in the quality of the lordosis reflex with 500 ng tropisetron producing the mo

148 receptor agonist altered the quality of the lordosis reflex.

149 tal development of this limbic-hypothalamic, lordosis regulating circuit, and to determine the age at

150 expression in the adult limbic-hypothalamic lordosis regulating circuit, which are not functional be

151 d may be subject to estrogenic influences in lordosis-relevant neurons in the ventrolateral subdivisi

152 It is suggested that muscimol may enhance lordosis responding in suboptimally hormonally primed ra

153 However, both drugs limited the duration of lordosis responding initiated by DOI.

154 ,907 in reducing the DOI-induced increase in lordosis responding.

155 -HT1A receptor antagonist did not facilitate lordosis responding.

156 In Experiment 2, SP diminished the lordosis response as it did in Experiment 1.

157 exposure to SP for ten weeks attenuated the lordosis response following sequential treatment with es

158 The expression of the lordosis response is modulated by OT action in the ventr

159 1) alpha(1)-adrenoceptors in the HYP enhance lordosis responses by activating the nitric oxide (NO)-c

160 h past results, unilateral lesions disrupted lordosis responses to contralateral flank stimulation.

161 ale guinea pigs rarely display adult-typical lordosis responses to ovarian steroid hormones until 40-

162 The lordosis stabilizes the upper body over the lower limbs

163 he arcuate nucleus or preoptic area, induced lordosis, suggesting the functional presence of D5 dopam

164 wild type mice suggest that P may facilitate lordosis through actions at substrates other than intrac

165 on-traditional actions in the VTA to enhance lordosis through D1 and/or GABA(A) include activity of P

166 Females with a lordosis to mount (L/M) ratio < 0.5 were used to test th

167 ere was a dose-dependent decline in both the lordosis to mount (L/M) ratio and in the quality of the

168 Tropisetron reduced the lordosis to mount (L/M) ratio in every animal investigat

169 Rats with a lordosis to mount ratio (L/M)>/=0.5 were used to examine

170 ha-THP-, SKF38393-, and muscimol-facilitated lordosis was attenuated by infusions of the PLC inhibito

171 Progestin facilitation of lordosis was correlated with a reduction of estrogen-ind

172 lses (15 min to 2 h duration, and 5 h apart) lordosis was induced.

173 ular progestin receptors (PRs) to facilitate lordosis was investigated in PR knockout (PRKO) mice, PR

174 Lordosis was observed in response to solicitous males in

175 les raised on a diet high in phytoestrogens, lordosis was reduced in comparison with females of both

176 ) activation, the steroid-dependent behavior lordosis was used in estrogen-primed ovariectomized Spra

177 sthetics, which would block E's induction of lordosis when administered at the time of estrogen appli

178 ed to bovine serum albumin (P:BSA) increased lordosis when applied bilaterally to both the VMH and VT

179 rdosis with i.v. P infusion and increases in lordosis when P's effects are relegated to the membrane

180 The rapid facilitation of lordosis with i.v. P infusion and increases in lordosis