ライフサイエンスコーパス: loss of vision

1 es may enable treatment to prevent permanent loss of vision.

2 oduct that is mutated in disease, leading to loss of vision.

3 ar deposits, abnormal RPE, and age-dependent loss of vision.

4 Leber's congenital amaurosis, which lead to loss of vision.

5 is a common condition that causes reversible loss of vision.

6 is pigmentosa, which often results in severe loss of vision.

7 neration of retinal pigmented epithelium and loss of vision.

8 ongenital amaurosis (LCA), which all lead to loss of vision.

9 lead to amblyopia, a permanent uncorrectable loss of vision.

10 rogression of retinal disease and subsequent loss of vision.

11 corneal clarity and can result in permanent loss of vision.

12 vascularization that may minimize iatrogenic loss of vision.

13 of eye diseases that result in catastrophic loss of vision.

14 retinal photoreceptor cells and progressive loss of vision.

15 considerable risk of severe and irreversible loss of vision.

16 ted protein methylation leads to progressive loss of vision.

17 loid hemorrhage results in a sudden profound loss of vision.

18 helium, degeneration of the neuroretina, and loss of vision.

19 age related macular degeneration leading to loss of vision.

20 ajor cause of ocular infections and possible loss of vision.

21 ells, causing the deterioration and eventual loss of vision.

22 adaptations to compensate for the unilateral loss of vision.

23 ed cone cell numbers, and led to progressive loss of vision.

24 lude eyelid swelling, ptosis, proptosis, and loss of vision.

25 cally requires aggressive therapy to prevent loss of vision.

26 ial growth factor (VEGF), macular edema, and loss of vision.

27 here are few treatments and none reverse the loss of vision.

28 ner retinal function in ND4 mice, indicating loss of vision.

29 eptor degenerations that lead to progressive loss of vision.

30 min-positive, perisomatic input preceded the loss of vision.

31 itively proved to cause optic neuropathy and loss of vision.

32 f retinal ganglion cells which could lead to loss of vision.

33 s a common cause of profound and intractable loss of vision.

34 ction, which is consistent with irreversible loss of vision.

35 tress, all of which can lead to debilitating loss of vision.

36 n the cornea that leads to tissue damage and loss of vision.

37 ht ameliorate cellular damage and subsequent loss of vision.

38 ion of diabetic retinopathy and the ultimate loss of vision.

39 CEP290) mutations and subjects have profound loss of vision.

40 rve damage, globe enlargement, and permanent loss of vision.

41 acular degeneration that eventually leads to loss of vision.

42 glion cells in the retina, and a progressive loss of vision.

43 glion cell (RGC) pathology and a progressive loss of vision.

44 lly leading to cell death and, consequently, loss of vision.

45 t bystander destruction of normal tissue and loss of vision.

46 These changes cause a loss of vision.

47 h may damage normal RPE function and lead to loss of vision.

48 , pain, double vision, optic neuropathy, and loss of vision.

49 Reasons for loss of vision after BI-KPro implantation most commonly

50 type III (USH3) characterized by progressive loss of vision and hearing is caused by mutations in the

51 Most cases consisted of initial loss of vision and intraocular inflammation without prom

52 A 35-year-old pregnant woman presented with loss of vision and metamorphopsia in her left eye.

53 Loss of vision and ocular motility deficit are the most

54 ficant local side effects that can result in loss of vision and possible amblyogenesis.

55 evidence that these problems actually cause loss of vision and potential therapeutic approaches targ

56 rity in disease is reflected in the complete loss of vision and rapid photoreceptor degeneration in t

57 It is characterized by loss of vision and results from mutations in any one of

58 d SN enhancement coevolved to compensate for loss of vision and to help blind cavefish find food in d

59 growth factor, have been shown to stabilise loss of vision and, in some cases, improve vision in ind

60 rs characterized by slow growth, progressive loss of vision, and limited therapeutic options.

61 ssive constriction of the visual fields, and loss of vision, and pathologically by progressive loss o

62 ammatory markers to increase coincident with loss of vision, and RGC functional loss.

63 Headache and visual disturbances including loss of vision are characteristic of GCA.

64 erience early in life and remains even after loss of vision as long as feedback from the eyes and hea

65 cate that shikonin treatment may prevent the loss of vision associated with DR.

66 e, altered mental functioning, seizures, and loss of vision associated with findings indicating predo

67 ngiogenesis contributes directly to profound loss of vision associated with many diseases of the eye.

68 rea that remains intact for many years after loss of vision attributable to damage to the eyes.

69 hoice for restoring vision or preventing the loss of vision because most blinding diseases originate

70 marked retinopathy consistent with complete loss of vision by 1 month of age.

71 Most diseases that cause catastrophic loss of vision do so as a result of abnormal angiogenesi

72 Since most diseases that cause catastrophic loss of vision do so as a result of abnormal ocular angi

73 nces, there is still significant unnecessary loss of vision due to cataract among older African Ameri

74 sa is a sight-threatening disease leading to loss of vision due to corneal inflammation.

75 s attention because it can lead to permanent loss of vision due to foveal involvement by inflammation

76 lly impaired individuals who exhibit partial loss of vision due to inherited retinal dystrophies (IRD

77 cultures of rat retina and, in vivo, prevent loss of vision due to light-induced degeneration of phot

78 ve blinding disease characterized by gradual loss of vision due to optic neuropathy and retinal gangl

79 It may lead to loss of vision due to retinal detachment and chronic inf

80 fications that may play an important role in loss of vision during aging and cataract formation.

81 opacities in the cornea result in bilateral loss of vision, eventually necessitating corneal transpl

82 al manifestations of which include the early loss of vision followed by deterioration of brain functi

83 eutic approaches to prevent the irreversible loss of vision from this disease.

84 esultant hyperopic shift, opacification, and loss of vision has recently become a concern.

85 ation and early treatment can prevent severe loss of vision in affected patients.

86 n developed countries and often causes rapid loss of vision in age-related macular degeneration.

87 was referred to our center with progressive loss of vision in both eyes 10 days after unintentional

88 Loss of vision in glaucoma is due to apoptotic retinal g

89 Loss of vision in glaucoma occurs because retinal gangli

90 Loss of vision in glaucoma results from the selective de

91 -old woman presented with a sudden, painless loss of vision in her left eye.

92 an lady presented with sudden and persisting loss of vision in her right eye, ocular pain and vomitin

93 -year-old man presented with sudden painless loss of vision in his left eye.

94 generative disorders results in irreversible loss of vision in humans.

95 and young adults, often resulting in severe loss of vision in later life.

96 work demonstrates a metabolic-stress-induced loss of vision in mammals, which has not been described

97 Linkage between low blood glucose and loss of vision in mice may highlight the importance for

98 nsgene in flies led to an activity-dependent loss of vision in older animals and we hypothesized that

99 al disease which causes progressive profound loss of vision in patients during their 4th decade.

100 ew treatment for the leading cause of severe loss of vision in patients with age-related macular dege

101 tural history of AMD demonstrates relentless loss of vision in persons who developed advanced AMD.

102 ration (AMD) is one of the leading causes of loss of vision in the industrialized world.

103 e treatment, the patient still complained of loss of vision in the left eye with a central scotoma.

104 with choroidal neovascularization causing a loss of vision in the majority of patients.

105 utcomes of visual impairment attributable to loss of vision in the non-amblyopic eye.

106 l amblyopia who were visually impaired after loss of vision in their non-amblyopic eye but had no oth

107 Age-dependent loss of vision in these mutant mice is associated with p

108 Here, we report that loss of vision, in the form of dark exposure (DE) for 1

109 anges in these behaviors will result in less loss of vision is needed because of the expected increas

110 lly, our only method of slowing glaucomatous loss of vision is to reduce intraocular pressure (IOP),

111 ge-related macular degeneration (AMD), where loss of vision is typically acute and treatment leads to

112 evere neuromotor retardation and progressive loss of vision, leading to blindness by the second decad

113 here is Charles Bonnet syndrome (CBS), where loss of vision leads to complex, vivid visual hallucinat

114 Understanding the processes that lead to loss of vision may lead to preventive strategies.

115 roughout life, serious complications such as loss of vision, mother-to-fetus transmission, and fatal

116 Irreversible loss of vision occurred in one participant who underwent

117 and Cfh(-/-) mice, RPE damage accompanied by loss of vision occurred only in old Cfh(+/-) mice.

118 No clinically visible damage to the eye or loss of vision occurred.

119 Consequently, the loss of vision occurs before the deterioration of brain

120 One patient met a primary endpoint with a loss of vision of 10 letters or more from baseline in on

121 For instance, loss of vision or audition leads to the brain areas norm

122 s, MEK inhibitors did not cause irreversible loss of vision or serious eye damage.

123 l hypertension (IIH) can present with severe loss of vision or with ongoing vision loss despite maxim

124 stress have been found to correlate with the loss of vision, particularly in cones, the type of photo

125 Our results suggest that loss of vision produces distinct circuit changes in the

126 cutive patients with diabetes without recent loss of vision referred for retinal control were assesse

127 The 10-year probability of loss of vision related to glaucoma was 1.0% after EK, 2.

128 as associated with vision deterioration, but loss of vision remained exceptional.

129 Cortical blindness refers to the loss of vision that occurs after destruction of the prim

130 ively in the primary visual cortex, in which loss of vision to one eye permanently degrades cortical

131 id not affect photoreceptor function, and no loss of vision was seen with kinase-dead transgenics.

132 CRB1 mutations lead to early-onset severe loss of vision with thickened, disorganized, nonseeing r